Search results for "Viral Evolution"

showing 10 items of 72 documents

Enhanced adaptation of vesicular stomatitis virus in cells infected with vaccinia virus.

2008

Infections involving different viruses (multiple infections) are common in nature and can take place between different strains of the same virus or between different virus species, including DNA and RNA viruses. The influence of multiple infections on viral evolution has been previously studied using different populations of the same virus. Here, we took a step forward by studying the evolution of an RNA virus (vesicular stomatitis virus, VSV) in the presence of a resident DNA virus (vaccinia virus, VV). Cell cultures were infected with a constant amount of VV, and VSV was added at four different post-VV-inoculation times and four different population sizes. The results showed that the pres…

Microbiology (medical)virusesPopulationAdaptation BiologicalVaccinia virusBiologyMicrobiologyVirusMicrobiologyCell Linechemistry.chemical_compoundCricetinaeGeneticsAnimalseducationMolecular BiologyEcology Evolution Behavior and SystematicsVirus classificationeducation.field_of_studyRNA virusDNA virusVesiculovirusbiology.organism_classificationVirologyBiological EvolutionInfectious DiseaseschemistryVesicular stomatitis virusViral evolutionVacciniaInfection, genetics and evolution : journal of molecular epidemiology and evolutionary genetics in infectious diseases
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Insights into virus evolution and membrane biogenesis from the structure of the marine lipid-containing bacteriophage PM2.

2008

Recent, primarily structural observations indicate that related viruses, harboring no sequence similarity, infect hosts of different domains of life. One such clade of viruses, defined by common capsid architecture and coat protein fold, is the so-called PRD1-adenovirus lineage. Here we report the structure of the marine lipid-containing bacteriophage PM2 determined by crystallographic analyses of the entire approximately 45 MDa virion and of the outer coat proteins P1 and P2, revealing PM2 to be a primeval member of the PRD1-adenovirus lineage with an icosahedral shell and canonical double beta barrel major coat protein. The view of the lipid bilayer, richly decorated with membrane protein…

Models MolecularViral proteinProtein ConformationvirusesMolecular Sequence DataBiologymedicine.disease_causeCrystallography X-Ray03 medical and health sciencesProtein structuremedicineLipid bilayerMolecular Biology030304 developmental biology0303 health sciences030306 microbiologyCorticoviridaeVirionCell BiologyVirologyBiological EvolutionLipidsCell biologyBeta barrelMembrane proteinCapsidViral evolutionMembrane biogenesisVirusesCalciumCapsid ProteinsMolecular cell
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Experimental Evolution in Viruses

2011

Experiments in which evolution takes place in real time can help us establish cause–effect relationships that are difficult to infer from the analysis of natural populations. The simplicity, rapid evolution and biomedical relevance of viruses make them a particularly interesting model system for experimental evolution. Bacterial, animal and plant viruses can be passaged under a variety of conditions, either in simple cell culture systems or in vivo to test population biology hypotheses, study the genetic basis of evolution, or predict evolutionary change in nature. Experimental evolution is a conceptually simple and flexible tool which allows us to address issues ranging from the molecular …

Mutation rateExperimental evolutionGenetic driftved/biologyViral evolutionved/biology.organism_classification_rank.speciesMutation (genetic algorithm)ZoologyPopulation geneticsComputational biologyBiologyAdaptationModel organismeLS
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Variability in the mutation rates of RNA viruses

2014

ABSTRACT:  It is well established that RNA viruses show extremely high mutation rates, but less attention has been paid to the fact that their mutation rates also vary strongly, from 10-6 to 10-4 substitutions per nucleotide per cell infection. The causes explaining this variability are still poorly understood, but candidate factors are the viral genome size and polarity, host-specific gene expression patterns, or the intracellular environment. Differences between animal and plant viruses, or between arthropod-borne and directly transmitted viruses have also been postulated. Finally, RNA viruses may be able to regulate the rate at which new mutations spread in the population by modifying f…

Mutation rate[SDE.MCG]Environmental Sciences/Global ChangesPopulationBiology03 medical and health sciences[SDV.EE.ECO]Life Sciences [q-bio]/Ecology environment/Ecosystems[SDV.MHEP.MI]Life Sciences [q-bio]/Human health and pathology/Infectious diseasesVirologyPlant virusGene expressioneducationGenome sizeComputingMilieux_MISCELLANEOUS030304 developmental biologyGenetics[SDV.EE.SANT]Life Sciences [q-bio]/Ecology environment/Health0303 health scienceseducation.field_of_study[SDV.MHEP.ME]Life Sciences [q-bio]/Human health and pathology/Emerging diseases030302 biochemistry & molecular biologyRNAVirology[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology3. Good healthViral replicationViral evolution[SDV.MP.VIR]Life Sciences [q-bio]/Microbiology and Parasitology/Virology[SDE.BE]Environmental Sciences/Biodiversity and Ecology
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The external domains of the HIV-1 envelope are a mutational cold spot

2015

In RNA viruses, mutations occur fast and have large fitness effects. While this affords remarkable adaptability, it can also endanger viral survival due to the accumulation of deleterious mutations. How RNA viruses reconcile these two opposed facets of mutation is still unknown. Here we show that, in human immunodeficiency virus (HIV-1), spontaneous mutations are not randomly located along the viral genome. We find that the viral mutation rate experiences a threefold reduction in the region encoding the most external domains of the viral envelope, which are strongly targeted by neutralizing antibodies. This contrasts with the hypermutation mechanisms deployed by other, more slowly mutating …

Mutation ratevirusesGeneral Physics and AstronomyHIV InfectionsBiologymedicine.disease_causeArticleGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesCytidine deaminationMutation RateViral Envelope ProteinsViral envelopeViral entrymedicineViral structural proteinHumans030304 developmental biologyGenetics0303 health sciencesMutationMultidisciplinary030302 biochemistry & molecular biologyRNAGeneral ChemistryVirologyProtein Structure Tertiary3. Good healthViral evolutionHIV-1Nature Communications
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Variation in RNA virus mutation rates across host cells.

2014

It is well established that RNA viruses exhibit higher rates of spontaneous mutation than DNA viruses and microorganisms. However, their mutation rates vary amply, from 10−6 to 10−4 substitutions per nucleotide per round of copying (s/n/r) and the causes of this variability remain poorly understood. In addition to differences in intrinsic fidelity or error correction capability, viral mutation rates may be dependent on host factors. Here, we assessed the effect of the cellular environment on the rate of spontaneous mutation of the vesicular stomatitis virus (VSV), which has a broad host range and cell tropism. Luria-Delbrück fluctuation tests and sequencing showed that VSV mutated similarly…

Mutation ratevirusesVirus Replicationmedicine.disease_causeMice[SDV.MHEP.MI]Life Sciences [q-bio]/Human health and pathology/Infectious diseasesCricetinaeBaby hamster kidney celllcsh:QH301-705.50303 health sciencesMutation[SDV.MHEP.ME]Life Sciences [q-bio]/Human health and pathology/Emerging diseases030302 biochemistry & molecular biology3. Good healthViral evolution[SDV.MP.VIR]Life Sciences [q-bio]/Microbiology and Parasitology/VirologyResearch Articlelcsh:Immunologic diseases. Allergy[SDE.MCG]Environmental Sciences/Global ChangesImmunologyBiologyMicrobiologyEvolution Molecular03 medical and health sciences[SDV.EE.ECO]Life Sciences [q-bio]/Ecology environment/EcosystemsCell Line TumorVirologyGeneticsmedicineAnimalsBiologyMolecular BiologyTropism030304 developmental biology[SDV.EE.SANT]Life Sciences [q-bio]/Ecology environment/HealthEvolutionary BiologyPoint mutationRNA virusVesiculovirusbiology.organism_classificationVirologyMolecular biology[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/BacteriologyViral replicationlcsh:Biology (General)MutationMicrobial EvolutionParasitology[SDE.BE]Environmental Sciences/Biodiversity and Ecologylcsh:RC581-607Population GeneticsPLoS Pathogens
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Emergence and Phylodynamics of Citrus tristeza virus in Sicily, Italy

2013

Citrus tristeza virus (CTV) outbreaks were detected in Sicily island, Italy for the first time in 2002. To gain insight into the evolutionary forces driving the emergence and phylogeography of these CTV populations, we determined and analyzed the nucleotide sequences of the p20 gene from 108 CTV isolates collected from 2002 to 2009. Bayesian phylogenetic analysis revealed that mild and severe CTV isolates belonging to five different clades (lineages) were introduced in Sicily in 2002. Phylogeographic analysis showed that four lineages co-circulated in the main citrus growing area located in Eastern Sicily. However, only one lineage (composed of mild isolates) spread to distant areas of Sici…

Nonsynonymous substitutionCitrusGenetic-variationLineage (evolution)Population Dynamicslcsh:MedicinePopulation geneticsPlant Sciencelcsh:SciencePhylogenetic analysesPhylogenyGeneticsMultidisciplinarybiologyPhylogenetic treeGeographyCitrus tristeza virusAgriculturePhylogeneticsItalyRNA ViralEvolutionary dynamicsCross-protectionSequence AnalysisResearch ArticleClosterovirusDNA ComplementaryMolecular Sequence DataPlant PathogensCropsMicrobiologyViral EvolutionFruitsGenetic driftSpecies SpecificityVirologyMosaic-virusGenetic variationCTV Phylodynamics SicilyEvolutionary SystematicsPopulation-structureHost passageBiologyPlant DiseasesEvolutionary BiologyMaximum-likelihoodlcsh:RSettore AGR/12 - Patologia VegetaleComputational BiologyGenetic VariationBayes TheoremSequence Analysis DNAPlant Pathologybiology.organism_classificationAgronomyViral phylodynamicsDNA polymorphismEvolutionary biologyMolecular evolutionlcsh:Q
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NATURAL SELECTION AND THE ORGAN-SPECIFIC DIFFERENTIATION OF HIV-1 V3 HYPERVARIABLE REGION

2004

The existence of organ-specific HIV-1 populations within infected hosts has been studied for many years; nonetheless results reported by different authors are somewhat discrepant. To tackle this problem, we used a population genetics approach to analyze previously published data from the V3 hypervariable region of the envelope env gene. Our results are compatible with a population subdivision by organs in 95% of individuals analyzed at autopsy. In addition, populations infecting the nervous system and testicles clearly appear as differentiated subsets of the so-called macrophage-tropic variants. Liver and kidney may harbor differentiated populations as well. Although it is widely accepted t…

Nonsynonymous substitutionPopulationPopulation geneticsHIV Envelope Protein gp120BiologyEvolution MolecularGeneticsCluster AnalysisHumansSelection GeneticeducationEcology Evolution Behavior and SystematicsGeneticsAnalysis of VarianceLikelihood Functionseducation.field_of_studyNatural selectionBase SequenceModels GeneticMechanism (biology)HIVPeptide FragmentsHypervariable regionGenetics PopulationOrgan SpecificityViral evolutionAdaptationDatabases Nucleic AcidGeneral Agricultural and Biological SciencesSequence AlignmentEvolution
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From foes to friends: Viral infections expand the limits of host phenotypic plasticity

2020

Phenotypic plasticity enables organisms to survive in the face of unpredictable environmental stress. Intimately related to the notion of phenotypic plasticity is the concept of the reaction norm that places phenotypic plasticity in the context of a genotype-specific response to environmental gradients. Whether reaction norms themselves evolve and which factors might affect their shape has been the object of intense debates among evolutionary biologists along the years. Since their discovery, viruses have been considered as pathogens. However, new viromic techniques and a shift in conceptual paradigms are showing that viruses are mostly non-pathogenic ubiquitous entities. Recent studies hav…

Phenotypic plasticityHuman evolutionary geneticsBiologyEnvironmental stressAdaptation PhysiologicalBiological EvolutionEvolutionary biologyVirus DiseasesViral evolutionVirusesAnimalsHumansNorm (social)Evolutionary dynamicsPhysiological HomeostasisVirus Physiological Phenomena
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CRISPR-Cas12a-Based Detection of SARS-CoV-2 Harboring the E484K Mutation

2021

The novel respiratory virus SARS-CoV-2 is rapidly evolving across the world with the potential of increasing its transmission and the induced disease. Here, we applied the CRISPR-Cas12a system to detect, without the need of sequencing, SARS-CoV-2 genomes harboring the E484K mutation, first identified in the Beta variant and catalogued as an escape mutation. The E484K mutation creates a canonical protospacer adjacent motif for Cas12a recognition in the resulting DNA amplicon, which was exploited to obtain a differential readout. We analyzed a series of fecal samples from hospitalized patients in Valencia (Spain), finding one infection with SARS-CoV-2 harboring the E484K mutation, which was t…

PolymersBiomedical EngineeringBiosensing TechniquesBiologyBiochemistry Genetics and Molecular Biology (miscellaneous)Genomechemistry.chemical_compoundCOVID-19 TestingPeptide LibraryTechnical NoteCRISPRCRISPR diagnosticsHumansGeneticsvirus evolutionSARS-CoV-2Epidemiological surveillanceepidemiological surveillanceCOVID-19General MedicineDNAAmpliconSurface Plasmon ResonanceVirus evolutionProtospacer adjacent motifHEK293 CellschemistryGenetic TechniquesSpainViral evolutionImmunoglobulin GMutation (genetic algorithm)DNA ViralMutationRespiratory virusCRISPR-Cas SystemsDNAACS Synthetic Biology
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