Search results for "Viral Replication"

showing 10 items of 157 documents

Ub surprised: viral ovarian tumor domain proteases remove ubiquitin and ISG15 conjugates.

2007

Ubiquitin (Ub) and interferon stimulated gene product 15 (ISG15) reversibly conjugate to proteins via a conserved LRLRGG C-terminal motif, mediating important innate antiviral responses. The ovarian tumor (OTU) domain represents a superfamily of predicted proteases found in eukaryotic, bacterial and viral proteins, some of which have Ub-deconjugating activity. We show that the OTU domain-containing proteases of nairoviruses and arteriviruses hydrolyze Ub and ISG15 from cellular target proteins. This broad activity contrasts with the target specificity of known mammalian OTU domain-containing proteins. The biological significance of this activity of viral OTU domain-containing proteases was …

Cancer ResearchProteasesCellPeptideBiologyMicrobiologyArticleOvarian tumorViral ProteinsUbiquitinImmunology and Microbiology(all)VirologymedicineHumansMolecular BiologyUbiquitinschemistry.chemical_classificationVirologyISG15Immunity InnateCell biologyNeoplasm ProteinsProtein Structure Tertiarymedicine.anatomical_structureViral replicationchemistrybiology.proteinCytokinesParasitologyConjugatePeptide HydrolasesSignal TransductionCell hostmicrobe
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Oncolytic parvovirus H1 induces release of heat-shock protein HSP72 in susceptible human tumor cells but may not affect primary immune cells.

2003

Certain autonomous parvoviruses preferentially replicate in and kill in vitro-transformed cells and may reduce the incidence of spontaneous and implanted tumors in animals. Hence, these viruses and their derivatives are currently under evaluation as antitumor vectors. However, the mechanisms underlying their tumor-suppressing properties are not yet understood. We asked whether the lytic parvovirus H1 may enhance the immunogenicity of infected tumor cells. Out of human melanoma and gastrointestinal tumor cells, we selected the cell line SK29-Mel-1 being very susceptible to H1-induced apoptotic killing. Here, no upregulation of HLA class I and costimulatory molecules could be observed followi…

Cancer ResearchTime FactorsCell SurvivalGenetic VectorsApoptosisHSP72 Heat-Shock ProteinsVirusParvovirusImmune systemCell Line TumorHumansHSP70 Heat-Shock ProteinsTransgenesMolecular BiologyMelanomaCells CulturedHeat-Shock ProteinsbiologyParvovirusImmunogenicityHSC70 Heat-Shock Proteinsbiology.organism_classificationVirologyOncolytic virusUp-RegulationCell killingViral replicationCell cultureCancer researchMolecular MedicineCarrier ProteinsCancer gene therapy
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Novel cell culture systems for the hepatitis C virus.

2001

Infections with the hepatitis C virus (HCV) are a major cause of acute and chronic liver disease. The high prevalence of the virus, the insidious course of the disease and the poor prognosis for long-term persistent infection make this pathogen a serious medical and socioeconomical problem. The identification of the viral genome approximately 10 years ago rapidly led to the delineation of the genomic organization and the structural and biochemical characterization of several virus proteins. However, studies of the viral life cycle as well as the development of antiviral drugs have been difficult because of the lack of a robust and reliable cell culture system. Numerous attempts have been un…

Carcinoma HepatocellularVirus CultivationvirusesHepacivirusHepatitis C virusGenome ViralHepacivirusmedicine.disease_causeTransfectionVirus ReplicationVirusFlaviviridaeViral life cycleVirologymedicineTumor Cells CulturedAnimalsHumansCells CulturedPharmacologybiologyLiver Neoplasmsbiology.organism_classificationVirologyViral replicationCell cultureDrug DesignImmunologyRepliconViral diseaseAntiviral research
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Replication of subgenomic hepatitis C virus RNAs in a hepatoma cell line.

1999

An estimated 170 million persons worldwide are infected with hepatitis C virus (HCV), a major cause of chronic liver disease. Despite increasing knowledge of genome structure and individual viral proteins, studies on virus replication and pathogenesis have been hampered by the lack of reliable and efficient cell culture systems. A full-length consensus genome was cloned from viral RNA isolated from an infected human liver and used to construct subgenomic selectable replicons. Upon transfection into a human hepatoma cell line, these RNAs were found to replicate to high levels, permitting metabolic radiolabeling of viral RNA and proteins. This work defines the structure of HCV replicons funct…

Carcinoma HepatocellularVirus CultivationvirusesHepatitis C virusDrug ResistanceGenome ViralHepacivirusBiologyViral Nonstructural Proteinsmedicine.disease_causeTransfectionVirus ReplicationViruschemistry.chemical_compoundmedicineTumor Cells CulturedHumansCloning MolecularNS5ANS5BSubgenomic mRNAGeneticsNS3MultidisciplinaryLiver NeoplasmsVirologyHepatitis CNS2-3 proteaseViral replicationchemistryRNA ViralRepliconGentamicinsScience (New York, N.Y.)
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Mind-body medicine: stress and its impact on overall health and longevity.

2005

During evolution, DNA viruses have captured a broad array of cellular genes involved in immune recognition and growth control that are nonessential for viral replication. The encoded virokines and viroceptors may act as mimetics or antagonists of their cellular homologues, altering signal transduction and cell communication towards survival of virus-infected cells. Human herpesvirus type 8 (HHV8) is the most recently identified human oncogenic herpesvirus. It is associated with Kaposi's sarcoma and lymphoproliferative diseases, such as pleural effusion lymphomas and multicentric Castleman's disease. HHV8 has captured a unique number of cellular regulatory genes, which redirect gene expressi…

Cell signalingTumor suppressor genemedicine.medical_treatmentLongevityBiologyVirokineGeneral Biochemistry Genetics and Molecular BiologyMind-Body Relations MetaphysicalParacrine signallingHistory and Philosophy of ScienceStress PhysiologicalNeoplasmsmedicineHumansDiseaseAutocrine signallingGeneral Neurosciencevirus diseasesBrainPsychoneuroimmunologyCytokineViral replicationHealthImmunologyCancer researchSignal transductionAnnals of the New York Academy of Sciences
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Role of lipid rafts in virus infection

2009

Rafts are domains of the plasma membrane, enriched in cholesterol and sphingolipids; they form a platform for signaling proteins and receptors. The lipid rafts are utilized in the replication cycle of numerous viruses. Internalization receptors of many viruses localize to rafts or are recruited there after virus binding. Arrays of signal transduction proteins found in rafts contribute to efficient trafficking and productive infection. Some viruses are dependent on raft domains for the biogenesis of their membranous replication structures. Finally, rafts are often important in virus assembly and budding. Subsequently, raft components in the viral envelope may be vital for the entry to a new…

Cell signalingvirusesmedia_common.quotation_subjectBiologySphingolipidVirologyVirusCell biologyViral envelopeViral replicationVirologylipids (amino acids peptides and proteins)Signal transductionInternalizationLipid raftmedia_commonFuture Virology
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T helper cell- and CD40-dependent germline IgM prevents chronic virus-induced demyelinating disease

2012

Generation of antiviral IgM is usually considered as a marker of a short-lived initial antibody response that is replaced by hypermutated and more-efficient IgG. However, once viruses have established a particular niche for their persistence (e.g., within the CNS), the immune system has to specifically mobilize a broad range of antimicrobial effectors to contain the pathogen in the long term. Infection of the CNS with the mouse hepatitis virus (MHV) provides a unique model situation in which the extent of inflammatory CNS disease is determined by the balance between antiviral immune control, viral replication, and immune-mediated damage. We show here that whereas antibody- or B cell-defici…

Central Nervous SystemEnzyme-Linked Immunospot AssayFluorescent Antibody TechniqueVirusMice03 medical and health sciences0302 clinical medicineImmune systemCytidine DeaminaseActivation-induced (cytidine) deaminaseDemyelinating diseasemedicineAnimalsCD40 Antigens030304 developmental biologyMice KnockoutAnalysis of VarianceB-LymphocytesMurine hepatitis virus0303 health sciencesMultidisciplinaryCD40biologyT-Lymphocytes Helper-InducerT helper cellBiological SciencesFlow Cytometrymedicine.diseaseVirology3. Good healthmedicine.anatomical_structureImmunoglobulin MViral replicationImmunologybiology.proteinAntibodyDemyelinating Diseases030215 immunologyProceedings of the National Academy of Sciences
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Roflumilast inhibits respiratory syncytial virus infection in human differentiated bronchial epithelial cells.

2013

Respiratory syncytial virus (RSV) causes acute exacerbations in COPD and asthma. RSV infects bronchial epithelial cells (HBE) that trigger RSV associated lung pathology. This study explores whether the phosphodiesterase 4 (PDE4) inhibitor Roflumilast N-oxide (RNO), alters RSV infection of well-differentiated HBE (WD-HBE) in vitro. WD-HBE were RSV infected in the presence or absence of RNO (0.1-100 nM). Viral infection (staining of F and G proteins, nucleoprotein RNA level), mRNA of ICAM-1, ciliated cell markers (digital high speed videomicroscopy, β-tubulin immunofluorescence, Foxj1 and Dnai2 mRNA), Goblet cells (PAS), mRNA of MUC5AC and CLCA1, mRNA and protein level of IL-13, IL-6, IL-8, T…

CyclopropanesScienceAminopyridinesBronchiCell CountRespiratory Syncytial Virus InfectionsBiologyMucin 5ACImmunofluorescenceVirus ReplicationVirusAntioxidantsChloride ChannelsTubulinGene expressionmedicineHumansCiliaRNA MessengerRespiratory systemRoflumilastMessenger RNAMetaplasiaMultidisciplinarymedicine.diagnostic_testQRvirus diseasesCell DifferentiationEpithelial CellsForkhead Transcription FactorsAxonemal Dyneinsrespiratory systemViral LoadVirologyMolecular biologyRespiratory Syncytial VirusesOxidative StressViral replicationBenzamidesMedicineCytokinesTumor necrosis factor alphaGoblet CellsReactive Oxygen SpeciesBiomarkersmedicine.drugResearch ArticlePloS one
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ICTV Virus Taxonomy Profile: Cystoviridae

2017

The family Cystoviridae includes enveloped viruses with a tri-segmented dsRNA genome and a double-layered protein capsid. The innermost protein shell is a polymerase complex responsible for genome packaging, replication and transcription. Cystoviruses infect Gram-negative bacteria, primarily plant-pathogenic Pseudomonas syringae strains. This is a summary of the International Committee on Taxonomy of Viruses (ICTV) Report on the taxonomy of the Cystoviridae, which is available at http://www.ictv.global/report/cystoviridae.

Cystoviridae0301 basic medicinebacteriophagesGenes Viralviruksetviruses030106 microbiologyGenome ViralVirus ReplicationGenomebakteriofagitICTVtaxonomy03 medical and health sciencesViral envelopeVirologyGram-Negative BacteriaPseudomonas syringaevirusesPseudomonas phage phi6PolymeraseVirus classificationbiologyta1183Bacteriophage phi 6VirologyICTV Virus Taxonomy Profiles3. Good health030104 developmental biologyCapsidViral replicationbiology.proteinPhageRNA ViralCapsid ProteinsJournal of General Virology
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Intracellular route of canine parvovirus entry.

1998

ABSTRACT The present study was designed to investigate the endocytic pathway involved in canine parvovirus (CPV) infection. Reduced temperature (18°C) or the microtubule-depolymerizing drug nocodazole was found to inhibit productive infection of canine A72 cells by CPV and caused CPV to be retained in cytoplasmic vesicles as indicated by immunofluorescence microscopy. Consistent with previously published results, these data indicate that CPV enters a host cell via an endocytic route and further suggest that microtubule-dependent delivery of CPV to late endosomes is required for productive infection. Cytoplasmic microinjection of CPV particles was used to circumvent the endocytosis and membr…

CytoplasmMicroinjectionsParvovirus CanineEndosomeanimal diseasesvirusesImmunologyEndocytic cycleBiologyVirus ReplicationEndocytosisMicrotubulesMicrobiologyCell LineDogsVirologyAnimalsMicroinjectionParvovirusNocodazoleTemperatureCanine parvovirusLipid bilayer fusionbiology.organism_classificationVirologyEndocytosisVirus-Cell InteractionsMicroscopy FluorescenceViral replicationInsect Science
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