Search results for "WNT"

showing 10 items of 166 documents

<em>Crassula ovata</em>, una nueva especie alóctona para China continental

2015

Se reporta por primera vez la presencia de Crassula ovata, o planta de jade, en China continental. Se han descubierto dos pequeñas poblaciones en el centro de la ciudad de Chengdu (provincia de Sichuan, oeste de China).

Mainland ChinaGeographybiologyDowntownEcologyCrassula ovataSmall population sizePlant ScienceAlienChinabiology.organism_classificationJADE (particle detector)Collectanea Botanica
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Drug-induced chromatin accessibility changes associate with sensitivity to liver tumor promotion

2019

This work explores quantitative chromatin accessibility, transcriptional and cis-acting gene regulatory variations underlying mouse strain–specific differences in drug-induced liver tumor promotion sensitivity.

Male0301 basic medicine63Health Toxicology and MutagenesisGene regulatory networkPlant ScienceSMADBiologyBiochemistry Genetics and Molecular Biology (miscellaneous)Epigenesis GeneticMice03 medical and health sciences0302 clinical medicinemedicineAnimalsHumansGene Regulatory NetworksEpigeneticsWnt Signaling PathwayTranscription factorResearch ArticlesEcologyGene Expression ProfilingLiver NeoplasmsWnt signaling pathwayComputational Biology11Chromatin Assembly and Disassemblymedicine.diseaseChromatin3. Good healthChromatin030104 developmental biologyPhenobarbital030220 oncology & carcinogenesisCancer researchTumor promotionLiver cancerResearch ArticleLife Science Alliance
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Wnt3a Neutralization Enhances T-cell Responses through Indirect Mechanisms and Restrains Tumor Growth

2018

Abstract The Wnt/β-catenin pathway regulates T-cell functions, including the repression of effector functions to the advantage of memory development via Tcf1. In a companion study, we demonstrate that, in human cancers, Wnt3a/β-catenin signaling maintains tumor-infiltrating T cells in a partially exhausted status. Here, we have investigated the effects of Wnt3a neutralization in vivo in a mouse tumor model. Abundant Wnt3a was released, mostly by stromal cells, in the tumor microenvironment. We tested whether Wnt3a neutralization in vivo could rescue the effector capacity of tumor-infiltrating T cells, by administering an antibody to Wnt3a to tumor-bearing mice. This therapy restrained tumor…

Male0301 basic medicineCancer Researchanimal structuresStromal cellT cellmedicine.medical_treatmentImmunologyAdenocarcinomaCD8-Positive T-LymphocytesDendritic CellSettore MED/0403 medical and health sciencesLymphocytes Tumor-Infiltrating0302 clinical medicineImmunology; Cancer Research; Wnt; Beta-catenin.Cell Line TumorWnt3A ProteinmedicineAnimalsHumansWnt Signaling PathwayColonic NeoplasmTumor microenvironmentAnimalChemistryEffectorStromal CellWnt signaling pathwayCD8-Positive T-LymphocyteDendritic CellsImmunotherapyDendritic cellCell biologyMice Inbred C57BLbody regions030104 developmental biologymedicine.anatomical_structureLymphocyte Transfusion030220 oncology & carcinogenesisColonic Neoplasmsembryonic structuresImmunotherapyStromal CellsCD8HumanCancer Immunology Research
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WNT Signaling Perturbations Underlie the Genetic Heterogeneity of Robinow Syndrome

2018

International audience; Locus heterogeneity characterizes a variety of skeletal dysplasias often due to interacting or overlapping signaling pathways. Robinow syndrome is a skeletal disorder historically refractory to molecular diagnosis, potentially stemming from substantial genetic heterogeneity. All current known pathogenic variants reside in genes within the noncanonical Wnt signaling pathway including ROR2, WNT5A, and more recently, DVL1 and DVL3. However, ∼70% of autosomal-dominant Robinow syndrome cases remain molecularly unsolved. To investigate this missing heritability, we recruited 21 families with at least one family member clinically diagnosed with Robinow or Robinow-like pheno…

Male0301 basic medicineCandidate geneFrizzledGROWTH-PLATEDEP DOMAINlnfectious Diseases and Global Health Radboud Institute for Molecular Life Sciences [Radboudumc 4]PROTEINskeletal dysplasiaCraniofacial Abnormalities0302 clinical medicineLocus heterogeneityChromosome SegregationChild[ SDV.GEN.GH ] Life Sciences [q-bio]/Genetics/Human geneticsWnt Signaling PathwayGenetics (clinical)Genes DominantGeneticsWnt signaling pathwayMiddle AgedRobinow syndromeMENDELIAN-INHERITANCEPhenotypeChild PreschoolFemaleNEURAL-TUBE DEFECTSVERTEBRATE GASTRULATIONhuman embryonic developmentRare cancers Radboud Institute for Health Sciences [Radboudumc 9]AdultAdolescentCELL POLARITYLimb Deformities CongenitalMutation MissenseDwarfismBiologyArticledual molecular diagnosisDiagnosis DifferentialGenetic Heterogeneity03 medical and health sciencesFrizzledAll institutes and research themes of the Radboud University Medical CenterSkeletal disorderGeneticsmedicineHumansGenetic Association StudiesNeurodevelopmental disorders Donders Center for Medical Neuroscience [Radboudumc 7]Base SequenceGenetic heterogeneityMUTATIONSROR2medicine.diseaseDROSOPHILA TISSUE POLARITY030104 developmental biology[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsUrogenital AbnormalitiesAUTOSOMAL-DOMINANT030217 neurology & neurosurgery
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The canonical but not the noncanonical wnt pathway inhibits the development of allergic airway disease

2018

Abstract Asthma is a syndrome with multifactorial causes, resulting in a variety of different phenotypes. Current treatment options are not curative and are sometimes ineffective in certain disease phenotypes. Therefore, novel therapeutic approaches are required. Recent findings have shown that activation of the canonical Wnt signaling pathway suppresses the development of allergic airway disease. In contrast, the effect of the noncanonical Wnt signaling pathway activation on allergic airway disease is not well described. The aim of this study was to validate the therapeutic effectiveness of Wnt-1–driven canonical Wnt signaling compared with Wnt-5a–driven noncanonical signaling in murine mo…

Male0301 basic medicineOvalbuminT-LymphocytesT cellImmunologyMedizinWnt1 ProteinLymphocyte ActivationWnt-5a ProteinImmunomodulationMice03 medical and health sciences0302 clinical medicineImmune systemMetaplasiaRespiratory HypersensitivitymedicineAnimalsHumansImmunology and AllergyWnt Signaling PathwayCells CulturedMice Inbred BALB CGoblet cellbusiness.industryWnt signaling pathwayDendritic CellsAllergensrespiratory systemPhenotypeAsthmaIn vitroMice Inbred C57BLDisease Models Animal030104 developmental biologymedicine.anatomical_structureCancer researchFemalemedicine.symptombusinessFunction (biology)030215 immunology
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Activated Thyroid Hormone Promotes Differentiation and Chemotherapeutic Sensitization of Colorectal Cancer Stem Cells by Regulating Wnt and BMP4 Sign…

2016

Abstract Thyroid hormone is a pleiotropic factor that controls many cellular processes in multiple cell types such as cancer stem cells (CSC). Thyroid hormone concentrations in the blood are stable, but the action of the deiodinases (D2–D3) provides cell-specific regulation of thyroid hormone activity. Deregulation of deiodinase function and thyroid hormone status has been implicated in tumorigenesis. Therefore, we investigated the role of thyroid hormone metabolism and signaling in colorectal CSCs (CR-CSC), where deiodinases control cell division and chemosensitivity. We found that increased intracellular thyroid hormone concentration through D3 depletion induced cell differentiation and s…

Male0301 basic medicineThyroid Hormonesendocrine systemCancer Researchmedicine.medical_specialtyendocrine system diseasesCellular differentiationDeiodinaseBone Morphogenetic Protein 4Colorectal NeoplasmMice03 medical and health sciencesCancer stem cellCell Line TumorInternal medicinemedicineAnimalsHumansThyroid HormoneWnt Signaling PathwayHormone activityThyroid hormone receptorbiologyAnimalThyroidWnt signaling pathwayCell DifferentiationMiddle Aged030104 developmental biologyEndocrinologymedicine.anatomical_structureOncologyNeoplastic Stem CellsCancer researchbiology.proteinNeoplastic Stem CellColorectal NeoplasmsHumanSignal TransductionHormoneCancer Research
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WNT2b activates epithelial-mesenchymal transition through FZD4: relevance in penetrating Crohns disease.

2020

Abstract Background and Aims Epithelial-mesenchymal transition [EMT] has been related to fibrosis and fistula formation, common complications associated with Crohn´s disease [CD]. The WNT signalling pathway mediates EMT, and specific WNT/FZD interactions have been related to the activation of this process in several diseases. We aim to analyse the relevance of EMT and WNT ligands and receptors in the penetrating behaviour of CD. Methods Intestinal surgical resections were obtained from control and CD patients with a stenotic or penetrating behaviour. Fibrosis was determined by the histological analysis of collagen deposition and EMT by confocal microscopy. The expression of WNT ligands, inh…

Male0301 basic medicineWNT pathwayVimentin0302 clinical medicineCrohn DiseaseFibrosisMedicineIntestinal MucosaReceptorWnt Signaling PathwayAged 80 and overbiologyGastroenterologyWnt signaling pathwayGeneral MedicineMiddle AgedCrohn's disease10219 Clinic for Gastroenterology and Hepatology030220 oncology & carcinogenesisembryonic structuresFemaleHT29 CellsAdultEpithelial-Mesenchymal TransitionAdolescentColonBlotting Western610 Medicine & healthReal-Time Polymerase Chain ReactionYoung Adult03 medical and health sciencesHT29 CellsHumansImmunoprecipitation2715 GastroenterologyEpithelial–mesenchymal transitionCrohn´s disease WNT pathway fibrosisAgedGlycoproteinsCadherinbusiness.industryfibrosismedicine.diseaseFibrosisFrizzled ReceptorsIn vitroWnt Proteins030104 developmental biologyCancer researchbiology.proteinbusiness
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Mutations inWNT10Aare frequently involved in oligodontia associated with minor signs of ectodermal dysplasia

2012

Ectodermal dysplasias (ED) are a clinically and genetically heterogeneous group of hereditary disorders that have in common abnormal development of ectodermal derivatives. Hypohidrotic ectodermal dysplasia (HED) is characterized by abnormal development of eccrine sweat glands, hair, and teeth. The X-linked form of the disease, caused by mutations in the EDA gene, represents the majority of patients with the hypohidrotic form. Autosomal dominant and autosomal recessive forms are occasionally seen, and result from mutations in at least three genes (WNT10A, EDAR, or more rarely EDARADD). We have screened for mutations in EDAR (commonly involved in the hypohidrotic form) and WNT10A (involved in…

MaleEctodermal dysplasiaGenotypeMolecular Sequence Datamedicine.disease_causeCompound heterozygosityEctodermal DysplasiaGeneticsmedicineHumansAmino Acid SequenceHypohidrotic ectodermal dysplasiaGenetic Association StudiesGenetics (clinical)AnodontiaGeneticsMutationEDARADDEdar ReceptorGenetic heterogeneitybusiness.industrymedicine.diseaseWnt ProteinsHypodontiaPhenotypeMutationFemaleEctodysplasin AbusinessSequence AlignmentAmerican Journal of Medical Genetics Part A
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M2 Macrophages Activate WNT Signaling Pathway in Epithelial Cells: Relevance in Ulcerative Colitis

2013

Macrophages, which exhibit great plasticity, are important components of the inflamed tissue and constitute an essential element of regenerative responses. Epithelial Wnt signalling is involved in mechanisms of proliferation and differentiation and expression of Wnt ligands by macrophages has been reported. We aim to determine whether the macrophage phenotype determines the expression of Wnt ligands, the influence of the macrophage phenotype in epithelial activation of Wnt signalling and the relevance of this pathway in ulcerative colitis. Human monocyte-derived macrophages and U937-derived macrophages were polarized towards M1 or M2 phenotypes and the expression of Wnt1 and Wnt3a was analy…

MaleFarmacologiaBeta-cateninMedicinaCellular differentiationlcsh:MedicineWnt1 ProteinProto-Oncogene Proteins c-mycAntigens CDWnt3A ProteinHumanslcsh:ScienceWnt Signaling Pathwaybeta CateninMultidisciplinarybiologyU937 cellMacrophageslcsh:RWnt signaling pathwayLGR5Cell DifferentiationLRP5U937 CellsWnt3A ProteinEnterocytesAparell digestiu Malaltiesbiology.proteinCancer researchColitis UlcerativeFemalelcsh:QEnterocyte differentiationCaco-2 CellsResearch ArticlePLoS ONE
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Quiescence Modulates Stem Cell Maintenance and Regenerative Capacity in the Aging Brain.

2018

The function of somatic stem cells declines with age. Understanding the molecular underpinnings of this decline is key to counteract age-related disease. Here, we report a dramatic drop in the neural stem cells (NSCs) number in the aging murine brain. We find that this smaller stem cell reservoir is protected from full depletion by an increase in quiescence that makes old NSCs more resistant to regenerate the injured brain. Once activated, however, young and old NSCs show similar proliferation and differentiation capacity. Single-cell transcriptomics of NSCs indicate that aging changes NSCs minimally. In the aging brain, niche-derived inflammatory signals and the Wnt antagonist sFRP5 induce…

MaleNeurogenesisSubventricular zoneInflammationBiologyGeneral Biochemistry Genetics and Molecular BiologyTranscriptome03 medical and health sciencesMice0302 clinical medicineNeural Stem CellsmedicineAging brainsFRP5stem cell agingAnimalsHomeostasisquiescenceStem Cell Nichereproductive and urinary physiologyCellular Senescence030304 developmental biologyneural stem cellsCell Proliferation0303 health sciencesWnt signaling pathwayAge Factorssubventricular zoneBrainmodelingCell DifferentiationinterferonWnt signalingNeural stem cellCell biologynervous system diseasesNerve RegenerationMice Inbred C57BLmedicine.anatomical_structurenervous systeminflammationsimulationsmedicine.symptomStem cellbiological phenomena cell phenomena and immunitysingle-cell transcriptomics030217 neurology & neurosurgeryCell DivisionAdult stem cellCell
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