Search results for "White People"

showing 10 items of 206 documents

Genome-wide and gene-centric analyses of circulating myeloperoxidase levels in the charge and care consortia

2013

Increased systemic levels of myeloperoxidase (MPO) are associated with the risk of coronary artery disease (CAD). To identify the genetic factors that are associated with circulating MPO levels, we carried out a genome-wide association study (GWAS) and a gene-centric analysis in subjects of European ancestry and African Americans (AAs). A locus on chromosome 1q31.1 containing the complement factor H (CFH) gene was strongly associated with serum MPO levels in 9305 subjects of European ancestry (lead SNP rs800292; P = 4.89 × 10(-41)) and in 1690 AA subjects (rs505102; P = 1.05 × 10(-8)). Gene-centric analyses in 8335 subjects of European ancestry additionally identified two rare M…

AdultMaleGenotypeLocus (genetics)Single-nucleotide polymorphismGenome-wide association studyCoronary Artery Disease030204 cardiovascular system & hematologyBiologyPolymorphism Single NucleotideGene Expression Regulation EnzymologicWhite PeopleYoung Adult03 medical and health sciences0302 clinical medicineGenotypeGeneticsHumansSNPMolecular BiologyGenetic Association StudiesGenetics (clinical)AgedPeroxidase030304 developmental biology0303 health sciencesAssociation Studies ArticlesCase-control studyGenetic VariationGeneral MedicineMiddle Aged3. Good healthBlack or African AmericanCase-Control StudiesComplement Factor HFactor HMyeloperoxidaseImmunologybiology.proteinFemaleGenome-Wide Association StudyHuman Molecular Genetics
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Specific association of IL17A genetic variants with panuveitis.

2015

Background/aims A pathogenic role of Th17 cells in uveitis has become clear in recent years. Therefore, in the present study, we aimed to evaluate the possible influence of the IL 17A locus on susceptibility to non-anterior uveitis and its main clinical subgroups. Methods Five IL17A polymorphisms (rs4711998, rs8193036, rs3819024, rs2275913 and rs7747909), selected by tagging, were genotyped using TagMan assays in 353 Spanish patients with non-anterior uveitis and 1851 ethnically matched controls. Results The case/control analysis yielded a consistent association between two of the analysed genetic variants, rs8193036 and rs2275913, and the presence of panuveitis under a dominant model (p(FD…

AdultMaleGenotyping TechniquesImmunologyLocus (genetics)DiseaseReal-Time Polymerase Chain ReactionPolymorphism Single NucleotideWhite PeopleCellular and Molecular NeuroscienceGene FrequencyGenetic modelPanuveitismedicineGeneticsHumansGenetic Predisposition to DiseaseInflammationbusiness.industryPanuveitisInterleukin-17Middle Agedmedicine.diseaseSensory SystemsOphthalmologymedicine.anatomical_structureImmunologyIntermediate uveitisTh17 CellsFemaleIL17AChoroidbusinessUveitisThe British journal of ophthalmology
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Perilipin polymorphism interacts with saturated fat and carbohydrates to modulate insulin resistance

2010

Macronutrient intakes and genetic variants have been shown to interact to alter the risk of insulin resistance, but replication of gene-nutrient interactions across independent populations may be difficult to achieve. Saturated fat and carbohydrate were previously shown to interact with genotype for insulin resistance for a variant of perilipin (PLIN), the major adipocyte-associated protein and a regulator of adipocyte metabolism. We investigated the same interaction for PLIN 11482G>A (rs894160) on insulin resistance in US men(n=462) and women(n=508) (mean ±SD, 49±16 years). In multivariate linear regression models, we found a significant (PA for HOMA-IR (homeostasis model assessment of ins…

AdultMaleHeterozygotePerilipin-1medicine.medical_specialtyAdolescentMinnesotaEndocrinology Diabetes and MetabolismSaturated fatmedicine.medical_treatmentMedicine (miscellaneous)BiologyPolymorphism Single NucleotideWhite PeopleArticleYoung Adultchemistry.chemical_compoundNutrigenomicsInsulin resistanceUtahAdipocyteInternal medicineDietary CarbohydratesmedicineHumansInsulinGenetic Association StudiesAgedAged 80 and overSex CharacteristicsNutrition and DieteticsInsulinMetabolismMiddle AgedCarbohydratePhosphoproteinsmedicine.diseaseDietary FatsEndocrinologychemistryPerilipinFemaleInsulin ResistanceCarrier ProteinsCardiology and Cardiovascular MedicineHomeostasisNutrition, Metabolism and Cardiovascular Diseases
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Evaluation of clinical diagnosis criteria of familial ligand defective apoB 100 and lipoprotein phenotype comparison between LDL receptor gene mutati…

2007

Familial hypercholesterolemia (FH) and familial defective apoB 100 (FDB) are characterized by increased plasma low-density lipoprotein cholesterol (LDLc) levels and risk of coronary heart disease (CHD). FDB is clinically indistinguishable from FH. The aims of this study were to evaluate clinical diagnosis criteria for FDB and to compare the lipoprotein phenotype between carriers of LDL receptor (LDLR) gene mutations that affect the ligand-binding domain and subjects with the R3500Q mutation in apoB gene. We studied 213 subjects (113 probands) with FH and 19 heterozygous FDB subjects. Genetic diagnosis was determined by following a protocol based on Southern blot and polymerase chain reactio…

AdultMaleHeterozygotemedicine.medical_specialtyGenotypeApolipoprotein BPopulationMutation MissenseCoronary DiseaseFamilial hypercholesterolemiaGene mutationBiologyWhite PeopleHyperlipoproteinemia Type IIchemistry.chemical_compoundPhysiology (medical)Internal medicinemedicineHumansMissense mutationeducationPolymorphism Single-Stranded Conformationaleducation.field_of_studyBinding SitesCholesterolGenetic Carrier ScreeningBiochemistry (medical)Public Health Environmental and Occupational HealthCholesterol LDLGeneral MedicineMiddle Agedmedicine.diseaseFounder EffectProtein Structure TertiaryEuropePhenotypeEndocrinologyReceptors LDLchemistryApolipoprotein B-100LDL receptorbiology.proteinFemalelipids (amino acids peptides and proteins)LipoproteinTranslational Research
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Functional polymorphism in the neuropeptide Y gene promoter (rs16147) is associated with serum leptin levels and waist-hip ratio in women

2013

<b><i>Objective:</i></b> The neuropeptide-Y (NP-Y) gene is a strong candidate gene in the pathophysiology of obesity-linked behavior, and several single-nucleotide polymorphisms of NP-Y have already been linked to body weight and appetite. However, the results from current studies remain inconclusive. The aim of the present study was to test whether a certain functional genetic variant (SNP rs16147) in the NP-Y promoter gene is associated with serum leptin levels and body fat distribution. <b><i>Method:</i></b> We genotyped and measured the serum leptin levels of the NP-Y rs16147 polymorphism in 1,097 Caucasian subjects in the context of a pop…

AdultMaleLeptinCandidate genemedicine.medical_specialtyGenotypeblood [Leptin]Medicine (miscellaneous)610 Medicine & healthmacromolecular substancesPolymorphism Single NucleotideWhite PeopleStatistics NonparametricWaist–hip ratiogenetics [Obesity]Sex FactorsMedizinische FakultätInternal medicineGenotypemedicineHumansNeuropeptide YObesityddc:610Genephysiology [Neuropeptide Y]Nutrition and Dieteticsbusiness.industryWaist-Hip RatioLeptinCase-control study2701 Medicine (miscellaneous)Middle AgedNeuropeptide Y receptormedicine.diseaseObesitygenetics [European Continental Ancestry Group]EndocrinologyCase-Control Studies10054 Clinic for Psychiatry Psychotherapy and Psychosomatics2916 Nutrition and DieteticsFemalegenetics [Neuropeptide Y]businessphysiology [Polymorphism Single Nucleotide]
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Replication of previous genome-wide association studies of psychiatric diseases in a large schizophrenia case-control sample from Spain.

2014

Genome wide association studies (GWAS) has allowed the discovery of some interesting risk variants for schizophrenia (SCZ). However, this high-throughput approach presents some limitations, being the most important the necessity of highly restrictive statistical corrections as well as the loss of statistical power inherent to the use of a Single Nucleotide Polymorphism (SNP) analysis approach. These problems can be partially solved through the use of a polygenic approach. We performed a genotyping study in SCZ using 86 previously associated SNPs identified by GWAS of SCZ, bipolar disorder (BPD) and autistic spectrum disorder (ASD) patients. The sample consisted of 3063 independent cases wit…

AdultMaleMultifactorial InheritanceAdolescentBipolar disorderSingle-nucleotide polymorphismGenome-wide association studyBiologyPolymorphism Single NucleotideODZ4White PeopleYoung AdultPolygenic scoremedicineGWASSNPHumansGenetic Predisposition to DiseaseBipolar disorderAlleleGenotypingBiological PsychiatryAgedGeneticsAged 80 and overMembrane GlycoproteinsModels GeneticCase-control studyMiddle Agedmedicine.diseasePsychiatry and Mental healthROC CurveSchizophreniaSpainArea Under CurveCase-Control StudiesReplication studySchizophreniaFemaleGenome-Wide Association StudySchizophrenia research
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Angiotensin type 2 receptor is important in the normal development of the ureter

1999

In humans, the actions of angiotensin II are transduced through the AT1 and AT2 receptors which have recently been implicated in renal organogenesis. Polymorphisms in the human angiotensin II receptor genes have been linked to cardiovascular and nephrological disorders. In this study we evaluated 35 patients with either primary obstructive megaureter or posterior urethral valves. Each was genotyped for the A1166 AT1 polymorphism and the recently described A-1332G AT2 transition. The incidence of these genetic variants was also evaluated in normal controls without any ultrasonographic urological abnormalities. Similar to our previous findings in congenital urological abnormalities, the AT1 r…

AdultMaleNephrologymedicine.medical_specialtyAngiotensin receptorAdolescentGenotypeMegaureterUrologyReceptor Angiotensin Type 2Receptor Angiotensin Type 1White PeopleGene FrequencyUrethraInternal medicineUrethral DiseasesmedicineHumansChildReceptorReceptors AngiotensinAngiotensin II receptor type 1business.industryCase-control studyInfantmedicine.diseaseAngiotensin IIEndocrinologyNephrologyChild PreschoolPediatrics Perinatology and Child HealthGene polymorphismUreterbusinesshormones hormone substitutes and hormone antagonistsUreteral ObstructionPediatric Nephrology
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Association of attentional network function with exon 5 variations of the CHRNA4 gene

2007

Mutational analyses in xenopus oocyte and mice models indicate that the positive effect of nicotine on attention may be modulated by genetic variations within exon 5 of the alpha4 subunit of the nicotinergic acetylcholine receptor gene CHRNA4. The potential relevance of exon 5 is further emphasized by two recent family-based association studies of nicotine dependence because subgroups of nicotine-dependent subjects are thought to 'self-medicate' attentional deficits with nicotine. We investigated a synonymous single nucleotide polymorphisms (SNP): rs1044396, which has recently been associated with nicotine-dependence, plus two adjacent synonymous SNPs rs1044394 and rs1044393 in exon 5 of n …

AdultMaleNicotineXenopusPosterior parietal cortexSingle-nucleotide polymorphismReceptors NicotinicBiologyModels BiologicalWhite PeopleMiceExonParietal LobeGeneticsmedicineAnimalsHumansAttentionMolecular BiologyOddball paradigmGenetics (clinical)Genetic associationGeneticsSupplementary motor areamedicine.diagnostic_testParietal lobeExonsTobacco Use DisorderGeneral MedicineMagnetic Resonance ImagingRadiographymedicine.anatomical_structureAttention Deficit Disorder with HyperactivityFemaleFunctional magnetic resonance imagingHuman Molecular Genetics
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Nine Loci for Ocular Axial Length Identified through Genome-wide Association Studies, Including Shared Loci with Refractive Error

2013

Refractive errors are common eye disorders of public health importance worldwide. Ocular axial length (AL) is the major determinant of refraction and thus of myopia and hyperopia. We conducted a meta-analysis of genome-wide association studies for AL, combining 12,531 Europeans and 8,216 Asians. We identified eight genome-wide significant loci for AL (RSPO1, C3orf26, LAMA2, GJD2, ZNRF3, CD55, MIP, and ALPPL2) and confirmed one previously reported AL locus (ZC3H11B). Of the nine loci, five (LAMA2, GJD2, CD55, ALPPL2, and ZC3H11B) were associated with refraction in 18 independent cohorts (n = 23,591). Differential gene expression was observed for these loci in minus-lens-induced myopia mouse …

AdultMaleRefractive errorAdolescentGene ExpressionLocus (genetics)Genome-wide association studyBiologyOcular Axial LengthPolymorphism Single NucleotideWhite PeopleArticle03 medical and health sciences0302 clinical medicineAsian PeopleSDG 3 - Good Health and Well-beingmedicineGeneticsHumansGWASGenetic Predisposition to DiseaseGenetics(clinical)RSPO1Eye ProteinsGeneGenetics (clinical)030304 developmental biologyGenetic associationAgedGenetics0303 health sciencesta1184HeritabilityMiddle Agedta3121medicine.diseaseRefractive ErrorsAxial Length EyeGenetic Loci030221 ophthalmology & optometryEye disorderFemaleGenome-Wide Association StudySignal Transduction
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Point mutations associated with Leber hereditary optic neuropathy in a Latvian population

2013

Purpose To study mutations associated with Leber hereditary optic neuropathy (LHON) in patients suspected of having this mitochondrial disorder in a Latvian population. Additional aims were to determine the heteroplasmy status of all non-synonymous polymorphisms identified in the current study and to identify the mitochondrial haplogroups of the studied participants because these factors may contribute to the manifestation of LHON. Methods Twelve patients, including patients in two families, were enrolled in the current study. LHON was suspected based on the findings of ophthalmologic examinations. In clinically affected individuals, the presence of all previously reported LHON-associated m…

AdultMalecongenital hereditary and neonatal diseases and abnormalitiesPolymorphism Geneticgenetic structuresnutritional and metabolic diseasesOptic Atrophy Hereditary LeberSequence Analysis DNAMiddle AgedDNA MitochondrialLatviaeye diseasesWhite PeopleMitochondriaPedigreeHaplotypesHumansPoint MutationFemaleResearch Article
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