Search results for "Wistar"

showing 10 items of 1094 documents

Effects of cerivastatin on adrenergic pathways, hypertrophic growth and TGFbeta expression in adult ventricular cardiomyocytes.

2010

Abstract The effects of statin treatment in the setting of heart failure have already been shown. Nevertheless, there is little knowledge about its influence on adrenergic pathways in cardiomyocytes. Therefore, this study investigated the impact of cerivastatin on adrenoceptor-mediated signalling pathways in isolated adult ventricular cardiomyocytes. It focused on two endpoints: hypertrophic growth and TGFbeta expression. Cultured cardiomyocytes were used to study rac activation (analysed by its translocation into the membrane fraction), ROS formation (H 2 DCF fluorescence) and hypertrophic growth ( 14 C-phenylalanine incorporation). Alpha- and beta-adrenoceptor stimulation showed significa…

Malemedicine.medical_specialtyHistologyAdrenergic receptorMAP Kinase Signaling SystemPyridinesp38 mitogen-activated protein kinasesHeart VentriclesAdrenergicAlpha (ethology)StimulationPharmacologyp38 Mitogen-Activated Protein KinasesPathology and Forensic MedicineTransforming Growth Factor betaInternal medicineReceptors Adrenergic betamedicineAnimalsMyocytes CardiacRats WistarCells CulturedHeart FailurebiologyCerivastatinCell BiologyGeneral MedicineReceptors Adrenergic alphaRatsEnzyme ActivationEndocrinologyGene Expression RegulationNAD(P)H oxidaseMitogen-activated protein kinasebiology.proteinHydroxymethylglutaryl-CoA Reductase InhibitorsReactive Oxygen SpeciesProto-Oncogene Proteins c-aktmedicine.drugEuropean journal of cell biology
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α4-1 Subunit mRNA of the nicotinic acetylcholine receptor in the rat olfactory bulb: cellular expression in adult, pre- and postnatal stages

1996

In addition to their role in signal transduction, nicotinic acetylcholine receptors have been shown in vi-tro to be involved in neuronal growth cone regulation during development. This idea is supported by recent histochemical findings showing that iso- and archicortical nicotinic alpha4-1 receptor mRNA expression precedes cholinergic fiber ingrowth. To test whether this also holds true for rhinencephalic parts of the telencephalon, we have studied the olfactory bulb by digoxigenin-mediated in situ hybridization, using an alpha4-1 isoform-specific riboprobe and an alkaline-phosphatase-based detection system. Development is characterized by early intense alpha4-1 mRNA expression (embryonic d…

Malemedicine.medical_specialtyHistologyIn situ hybridizationReceptors NicotinicBiologyPathology and Forensic MedicinePregnancyInternal medicinemedicineAnimalsRNA MessengerRats WistarIn Situ HybridizationAcetylcholine receptorCerebrumRNA ProbesCell BiologyOlfactory BulbRatsOlfactory bulbNicotinic acetylcholine receptormedicine.anatomical_structureNicotinic agonistEndocrinologyCholinergicFemaleSignal transductionDigoxigeninCell and Tissue Research
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Evidence for the neuronal origin of immunoreactive interleukin-1 beta released by rat hypothalamic explants.

1996

In this study, we have investigated the release of immunoreactive interleukin-1 beta (irIL-1 beta) from the rat hypothalamus in vitro. It was found that (1) tissue explants release sizable amounts of irIL-1 beta (ranging from 0.43 to 0.52 pg/mg of wet tissue) in 20 min incubations; (2) basal release in significantly increased by depolarization induced with 56 mM KCl; (3) K(+)-induced irIL-1 beta release is inhibited by the specific blocker of N-type calcium channels, omega-conotoxin, and by verapamil, but not by nifedipine; (4) K(+)-induced release is also inhibited by the Na+ channel blockers tetrodotoxin and lidocaine; (5) irIL-1 beta release is significantly increased by noradrenalin; su…

Malemedicine.medical_specialtyHypothalamusRadioimmunoassaychemistry.chemical_elementPropranololCalciumIn Vitro TechniquesPotassium ChlorideNorepinephrinePhentolamineNifedipineInternal medicinemedicineAnimalsChannel blockerRats WistarNeuronsVoltage-dependent calcium channelGeneral NeuroscienceDepolarizationCalcium Channel BlockersRatsElectrophysiologyEndocrinologychemistryVerapamilmedicine.drugInterleukin-1Sodium Channel BlockersNeuroscience letters
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Circulating TNF-alpha and its soluble receptors during experimental acute pancreatitis.

2004

Clinical and experimental studies have shown increased concentrations of TNF-α and its soluble receptors in serum of patients with acute pancreatitis. In this work, we have investigated the time-course of TNF-α and its soluble receptors during taurocholate-induced acute pancreatitis. In addition, since TNF-α itself could mediate the shedding of its receptors, we have assessed the effect of inhibiting TNF-α production on the release of soluble TNF-α receptors in experimental acute pancreatitis. Our results indicate that soluble receptors are released in the early stages of the disease and this increase is concomitant with the release of TNF-α, which is mainly bound to specific proteins. The …

Malemedicine.medical_specialtyImmunologyInflammationBiochemistryDNA-binding proteinReceptors Tumor Necrosis FactorPentoxifyllineInternal medicinemedicineSIRSImmunology and AllergyAnimalsPentoxifyllineRats WistarReceptorMolecular BiologyInflammationbusiness.industryTumor Necrosis Factor-alphaHematologymedicine.diseasesTNF-αRRatsDisease Models AnimalEndocrinologyPancreatitisSolubilityTNF-αAcute DiseasePancreatitisAcute pancreatitisTumor necrosis factor alphamedicine.symptombusinessmedicine.drugCytokine
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Carnitine transport into muscular cells. inhibition of transport and cell growth by mildronate

2000

Carnitine is involved in the transfer of fatty acids across mitochondrial membranes. Carnitine is found in dairy and meat products, but is also biosynthesized from lysine and methionine via a process that, in rat, takes place essentially in the liver. After intestinal absorption or hepatic biosynthesis, carnitine is transferred to organs whose metabolism is dependent on fatty acid oxidation, such as heart and skeletal muscle. In skeletal muscle, carnitine concentration was found to be 50 times higher than in the plasma, implicating an active transport system for carnitine. In this study, we characterized this transport in isolated rat myotubes, established mouse C2C12 myoblastic cells, and …

Malemedicine.medical_specialtyIn Vitro TechniquesBiologyBiochemistryIntestinal absorptionCarnitine transportMicechemistry.chemical_compoundCarnitineInternal medicinemedicineAnimalsMyocyteCarnitineRats WistarMuscle SkeletalBeta oxidationCells CulturedPharmacologyMethionineCell MembraneSkeletal muscleBiological TransportMembrane transportRatsEndocrinologymedicine.anatomical_structureBiochemistrychemistryCell DivisionMethylhydrazinesmedicine.drugBiochemical Pharmacology
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Inhibition of nitric oxide synthase influences the activity of striatal neurons in the rat

2002

The activity of single units in the striatum of urethane-anesthetized rats was recorded before and after the systemic administration of 7-nitro-indazole (7-NI; 50 mg/kg intraperitoneally), a selective inhibitor of neuronal nitric oxide (NO) synthase. Two neuronal types were clearly distinguishable electrophysiologically, on the basis of either discharge frequency pattern or features of the individual spike waveform (spike duration, negative phase/total duration ratio, and negative phase/total amplitude ratio). Only sporadically discharging neurons (basal firing rate, <0.1 spikes/s) were influenced by 7-NI, which caused a statistically significant increase in their firing rate. In contrast, …

Malemedicine.medical_specialtyIndazolesAction PotentialsNerve Tissue ProteinsNitric Oxide Synthase Type IStriatumNeurotransmissionBiologyNitric OxideInhibitory postsynaptic potentialTonic (physiology)Nitric oxidechemistry.chemical_compoundInternal medicineBasal gangliamedicineAnimalsEnzyme InhibitorsRats WistarGeneral NeuroscienceCorpus StriatumRatsElectrophysiologyNitric oxide synthaseElectrophysiologyEndocrinologynervous systemchemistrybiology.proteinNitric Oxide SynthaseNeuroscienceNeuroscience Letters
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Involvement of nitric oxide-soluble guanylyl cyclase pathway in the control of maximal dentate gyrus activation in the rat.

2006

Summary Nitric oxide=soluble Guanylyl cyclase (NO=sGC) pathway on the maximal dentate gyrus activation (MDA) was studied in rats. The cerebral NO levels were modified by administrating 7-Nitroindazole (7-NI), a selective inhibitor of neuronal NOS, and L-arginine, a precursor of the synthesis of NO. 1H-[1,2,4]Oxadiazole[4,3-a]quinoxalin-1-one (ODQ), a specific inhibitor of the NO-sGC pathway, was administered to study the involvement of cGMP pathway. The epileptic activity of the dentate gyrus was obtained through the repetitive stimulation of the angular bundle; MDA parameters studied were: onset time, MDA duration and post-stimulus afterdischarge (AD) duration. 7-NI caused an increase of M…

Malemedicine.medical_specialtyIndazolesArginineNitric Oxide Synthase Type IIIReceptors Cytoplasmic and NuclearKeywords: Maximal dentate activation nitric oxide cGMP modulationArginineNitric OxideNitric oxidechemistry.chemical_compoundSoluble Guanylyl CyclaseInternal medicineMalondialdehydeQuinoxalinesmedicineAnimalsEnzyme InhibitorsRats WistarReceptorBiological PsychiatryOxadiazolesDentate gyrusNitric Oxide Synthase Type IIIIontophoresisRatsElectrophysiologyPsychiatry and Mental healthMetabolic pathwayEndocrinologyNeurologychemistryGuanylate CyclaseDentate GyrusNeurology (clinical)Signal transductionSoluble guanylyl cyclaseSignal TransductionJournal of neural transmission (Vienna, Austria : 1996)
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Effects of Lycopene, Indole-3-Carbinol, and Luteolin on Nitric Oxide Production and iNOS Expression are Organ-Specific in Rats

2010

Effects of Lycopene, Indole-3-Carbinol, and Luteolin on Nitric Oxide Production and iNOS Expression are Organ-Specific in RatsNatural compounds are known to modify NO content in tissues; however, the biological activity of polyphenol-rich food often does not correspond to the effects of individual polyphenols on NO synthase activity. The aim of this study was to see how natural compounds luteolin, indole-3-carbinol, and lycopene modify NO production in rat tissues and change the expression of the iNOS gene and protein. Indole-3-carbinol produced multiple effects on the NO level; it significantly decreased NO concentration in blood, lungs, and skeletal muscles and increased it in the liver. …

Malemedicine.medical_specialtyIndolesNitric Oxide Synthase Type IIInflammationNitric OxideToxicologyNitric oxidechemistry.chemical_compoundLycopeneInternal medicineGene expressionmedicineIndole-3-carbinolAnimalsRats WistarLuteolinMuscle SkeletalLungPublic Health Environmental and Occupational HealthBrainSkeletal muscleBiological activityCarotenoidsLycopeneRatsmedicine.anatomical_structureEndocrinologyLiverchemistrymedicine.symptomLuteolinArchives of Industrial Hygiene and Toxicology
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Characterization of two different Ca2+ entry pathways dependent on depletion of internal Ca2+ pools in rat aorta

1998

Ryanodine (10 microM), thapsigargin (1 microM) and cyclopiazonic acid (10 microM) produced a slow, sustained contractile response in rat aorta that only can be observed in Ca2+-containing solution. In Ca2+-free medium, no response to the drugs was obtained, which suggests that the contraction elicited in presence of Ca2+ is mainly due to the contribution of extracellular influx. This Ca2+ entry does not depend on the opening of dihydropyridine-dependent Ca2+-channels for nimodipine does not affect this. Noradrenaline (1 microM) induced a biphasic response in Ca2+-free medium that was mediated by two different Ca2+ compartments, one of which is common to caffeine (10 mM), and is also deplete…

Malemedicine.medical_specialtyIndolesThapsigarginContraction (grammar)Phosphodiesterase InhibitorsVasodilator AgentsAorta ThoracicIn Vitro TechniquesMuscle Smooth VascularNorepinephrinechemistry.chemical_compoundCaffeineInternal medicinemedicine.arterymedicineExtracellularAnimalsVasoconstrictor AgentsRats WistarCa2 entryNimodipinePharmacologyAortaRyanodineRyanodine receptorGeneral MedicineRatsEndocrinologychemistryBiophysicsThapsigarginCalciumCalcium ChannelsCyclopiazonic acidMuscle Contractionmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Inhibition by interleukin-1 beta of noradrenaline release in rat spleen: involvement of lymphocytes, NO and opioid receptors.

1995

Effects of indomethacin, N omega-nitro-L-arginine (NNA) and naloxone, and of pretreatment with cyclophosphamide (CY), on the interleukin (IL)-1 beta induced inhibition of exocytotic noradrenaline release were investigated in the isolated, vascularly perfused spleen of the rat. Neurotransmitter release was evoked by perivascular electrical stimulation (4 Hz) and the overflow of endogenous noradrenaline was determined by HPLC with electrochemical detection. Perfusion of the spleen with Tyrode's solution containing IL-1 beta (100 pg/ml) for 90 min caused an inhibition of the stimulation-evoked noradrenaline overflow which persisted for at least 20 min after washout of the IL. The evoked overfl…

Malemedicine.medical_specialtyIndomethacinSpleenEndogenyStimulation(+)-NaloxoneIn Vitro TechniquesArginineNitric OxideNitroargininechemistry.chemical_compoundNorepinephrineInternal medicinemedicineAnimalsLymphocytesRats WistarNeurotransmitterReceptorCyclophosphamidePharmacologyNaloxoneBody WeightInterleukinGeneral MedicineOrgan SizeElectric StimulationRatsmedicine.anatomical_structureEndocrinologychemistryOpioidReceptors OpioidSpleenmedicine.drugInterleukin-1Naunyn-Schmiedeberg's archives of pharmacology
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