Search results for "XANTHINE OXIDASE"

showing 10 items of 79 documents

In vitro antioxidant properties, DNA damage protective activity, and xanthine oxidase inhibitory effect of cajaninstilbene acid, a stilbene compound …

2010

The antioxidant properties, DNA damage protective activities, and xanthine oxidase (XOD) inhibitory effect of cajaninstilbene acid (CSA) derived from pigeon pea leaves were studied in the present work. Compared with resveratrol, CSA showed stronger antioxidant properties, DNA damage protective activity, and XOD inhibition activity. The IC(50) values of CSA for superoxide radical scavenging, hydroxyl radical scavenging, nitric oxide scavenging, reducing power, lipid peroxidation, and XOD inhibition were 19.03, 6.36, 39.65, 20.41, 20.58, and 3.62 μM, respectively. CSA possessed good protective activity from oxidative DNA damage. Furthermore, molecular docking indicated that CSA was more poten…

DNA BacterialXanthine OxidaseAntioxidantDNA damagemedicine.medical_treatmentAllopurinolResveratrolBiologyAntioxidantsNitric oxideLipid peroxidationchemistry.chemical_compoundCajanusStilbenesmedicineHumansEnzyme InhibitorsXanthine oxidasechemistry.chemical_classificationPlant ExtractsAntimutagenic AgentsGeneral ChemistrySalicylatesPlant LeavesEnzymechemistryBiochemistryGeneral Agricultural and Biological Sciencesmedicine.drugDNA DamageJournal of agricultural and food chemistry
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Mitochondrial redox signaling: Interaction of mitochondrial reactive oxygen species with other sources of oxidative stress.

2014

Significance: Oxidative stress is a well established hallmark of cardiovascular disease and there is strong evidence for a causal role of reactive oxygen and nitrogen species (RONS) therein. Recent Advances: Improvement of cardiovascular complications by genetic deletion of RONS producing enzymes and overexpression of RONS degrading enzymes proved the involvement of these species in cardiovascular disease at a molecular level. Vice versa, overexpression of RONS producing enzymes as well as deletion of antioxidant enzymes was demonstrated to aggravate cardiovascular complications. Critical Issues: With the present overview we present and discuss different pathways how mitochondrial RONS inte…

Heart DiseasesPhysiologyClinical BiochemistryMitochondrionmedicine.disease_causeBiochemistrychemistry.chemical_compoundmedicineAnimalsHumansXanthine oxidaseMolecular BiologyGeneral Environmental Sciencechemistry.chemical_classificationInflammationReactive oxygen speciesNADPH oxidasebiologyNADPH OxidasesCell BiologyForum Review ArticlesAngiotensin IICell biologyMitochondriaNitric oxide synthaseCrosstalk (biology)Oxidative StressBiochemistrychemistrybiology.proteinGeneral Earth and Planetary SciencesReactive Oxygen SpeciesOxidation-ReductionOxidative stressSignal TransductionAntioxidantsredox signaling
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Impact of therapeutically induced reactive oxygen species and radical scavenging by α-tocopherol on tumor cell adhesion

2007

Many tumor treatment modalities such as ionizing radiation or some chemotherapy induce reactive oxygen species (ROS) resulting in therapeutic cell damage. The aim of this study was to analyze whether such ROS induction may affect the mechanical stability of solid tumor tissue by degradation of the extracellular matrix proteins or by a loss of cell adhesion molecules. Additionally, the protective impact of alpha-tocopherol treatment on these processes was studied. Experimental DS-sarcomas in rats were treated with a combination of localized 44 degrees C hyperthermia, inspiratory hyperoxia and xanthine oxidase in order to induce pronounced oxidative stress. A second group of animals were pret…

Hyperoxiachemistry.chemical_classificationCancer ResearchReactive oxygen speciesbiologyCell adhesion moleculeIntegrinGeneral Medicinemedicine.diseasemedicine.disease_causeExtracellular matrixchemistry.chemical_compoundOncologyBiochemistrychemistrymedicinebiology.proteinCancer researchmedicine.symptomXanthine oxidaseCell damageOxidative stressOncology Reports
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Conversion of biliverdin to bilirubin by biliverdin reductase contributes to endothelial cell protection by heme oxygenase-1—evidence for direct and …

2009

Heme oxygenase-1 (HO-1) is highly protective in various pathophysiological states such as cardiovascular and neurodegenerative diseases. HO-1-derived bilirubin is an efficient scavenger of reactive oxygen and nitrogen species (RONS). It remains to determine whether conversion of biliverdin to bilirubin is an essential step for HO-1-conferred protection of endothelial cells. RONS scavenging activities of biliverdin versus bilirubin were assessed by different RONS generating systems and detection techniques. We also silenced the biliverdin reductase (BVR) or HO-1 gene in cultured primary human endothelial cells (HUVECs) and measured the effect on RONS formation upon stimulation with lipopolys…

LipopolysaccharidesOxidoreductases Acting on CH-CH Group DonorsUmbilical VeinsXanthine OxidaseNeutrophilsBilirubinNitrosationModels BiologicalAntioxidantschemistry.chemical_compoundPeroxynitrous AcidLeukocytespolycyclic compoundsHumansGene SilencingMolecular BiologyHemeReactive nitrogen speciesRespiratory BurstBiliverdinAngiotensin IIBiliverdineBiliverdin reductaseEndothelial CellsBilirubinFree Radical ScavengersAngiotensin IIMitochondriaEndothelial stem cellHeme oxygenasechemistryBiochemistryCytoprotectionGene Knockdown TechniquesTyrosineReactive Oxygen SpeciesCardiology and Cardiovascular MedicineHeme Oxygenase-1Journal of Molecular and Cellular Cardiology
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Inhibition of leukocyte functions by the alkaloid isaindigotone from Isatis indigotica and some new synthetic derivatives.

2001

The alkaloid isaindigotone (1a) and seven derivatives have been synthesized to study their influence on several leukocyte functions and the generation of inflammatory mediators. Isaindigotone (1a) was found to be a scavenger of superoxide generated either by the hypoxanthine/xanthine oxidase system or stimulated human neutrophils. Isaindigotone (1a) and its acetylated derivative (1b) also inhibited 5-lipoxygenase activity and leukotriene B(4) production in these cells, whereas none of the compounds affected degranulation. In RAW 264.7 macrophages stimulated with lipopolysaccharide, synthetic derivatives exerted higher inhibitory effects on prostaglandin E(2) (PGE(2)) and nitric oxide (NO) g…

LipopolysaccharidesXanthine OxidaseMagnetic Resonance SpectroscopyLeukotriene B4StereochemistryNeutrophilsmedicine.medical_treatmentPharmaceutical ScienceLeukotriene B4DinoprostoneAnalytical ChemistryNitric oxidechemistry.chemical_compoundInhibitory Concentration 50MiceStructure-Activity RelationshipAlkaloidsDrug DiscoverymedicineLeukocytesAnimalsHumansLipoxygenase InhibitorsXanthine oxidaseHypoxanthineCells CulturedPharmacologyInflammationPlants MedicinalbiologyMolecular StructureSuperoxideAlkaloidMacrophagesOrganic ChemistryFree Radical ScavengersComplementary and alternative medicineBiochemistrychemistryArachidonate 5-lipoxygenaseBrassicaceaebiology.proteinQuinazolinesMolecular MedicineChromatography Thin LayerInflammation MediatorsNitric Oxide SynthaseProstaglandin EJournal of natural products
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Allopurinol prevents cardiac and skeletal muscle damage in professional soccer players

2014

Xanthine oxidase (XO), a free radical-generating enzyme, is involved in tissue damage produced during exhaustive exercise. Our aim was to test whether allopurinol, a powerful inhibitor of XO, may be effective in preventing exercise-induced tissue damage in soccer players. Twelve soccer players were randomized into two experimental groups. One received allopurinol, before a match of the premier Spanish Football League, and the other placebo. Allopurinol prevented the exercise-induced increase in all the markers of skeletal muscle damage analyzed: creatine kinase, lactate dehydrogenase, aspartate aminotransferase, and myoglobin. Creatine kinase-MB isoenzyme and highly sensitive troponin T, sp…

Liver injurymedicine.medical_specialtybiologybusiness.industryAllopurinolPhysical Therapy Sports Therapy and Rehabilitationmedicine.diseaseCreatineGamma-glutamyltransferase activitySurgeryLipid peroxidationchemistry.chemical_compoundEndocrinologychemistryLactate dehydrogenaseInternal medicinebiology.proteinMedicineOrthopedics and Sports MedicineCreatine kinasebusinessXanthine oxidasehuman activitiesmedicine.drugScandinavian Journal of Medicine & Science in Sports
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Effect of simultaneous inhibition of TNF-α production and xanthine oxidase in experimental acute pancreatitis: The role of mitogen activated protein …

2004

Javier Pereda et al.

MAPK/ERK pathwayMalemedicine.medical_specialtyXanthine OxidaseOxypurinolPharmacologychemistry.chemical_compoundInternal medicineMedicineAnimalsEnzyme InhibitorsPentoxifyllinePhosphorylationRats WistarXanthine oxidaseProtein kinase ALungPancreasPeroxidasebiologybusiness.industryKinasePancreatitis Acute NecrotizingTumor Necrosis Factor-alphaAscitesOriginal Articlesmedicine.diseaseRatsEnzyme ActivationOxidative StressEndocrinologychemistryMitogen-activated protein kinasebiology.proteinAcute pancreatitisPancreatitisSurgeryTumor necrosis factor alphaInflammation MediatorsMitogen-Activated Protein Kinasesbusiness
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Decreasing xanthine oxidase-mediated oxidative stress prevents useful cellular adaptations to exercise in rats

2005

Reactive oxygen or nitrogen species (RONS) are produced during exercise due, at least in part, to the activation of xanthine oxidase. When exercise is exhaustive they cause tissue damage; however, they may also act as signals inducing specific cellular adaptations to exercise. We have tested this hypothesis by studying the effects of allopurinol-induced inhibition of RONS production on cell signalling pathways in rats submitted to exhaustive exercise. Exercise caused an activation of mitogen-activated protein kinases (MAPKs: p38, ERK 1 and ERK 2), which in turn activated nuclear factor κB (NF-κB) in rat gastrocnemius muscle. This up-regulated the expression of important enzymes associated w…

MAPK/ERK pathwaymedicine.medical_specialtyPhysiologyKinasep38 mitogen-activated protein kinasesAllopurinolBiologymedicine.disease_causebiology.organism_classificationCell biologySuperoxide dismutasechemistry.chemical_compoundEndocrinologychemistryEnosInternal medicinemedicinebiology.proteinXanthine oxidaseOxidative stressmedicine.drugThe Journal of Physiology
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Anti-hyperuricemic and nephroprotective effects of extracts from Chaenomeles sinensis (Thouin) Koehne in hyperuricemic mice

2018

Clinically, Chaenomeles sinensis (Thouin) Koehne (C. sinensis) has been used to treat hyperuricemia and gout. However, the exact mechanism of action is still unknown. In the present study, the ethyl acetate fraction of C. sinensis fruit extract (CSF-E) was separated. Potassium oxonate (PO)-induced hyperuricemic mice and normal mice were administered with CSF-E at 60, 120 and 180 mg kg-1, respectively for 7 days. Serum uric acid, creatinine and BUN levels, liver oxidative damage, and serum and hepatic XOD activities were primarily measured using assay kits. The evaluation of its nephroprotective effects was carried out by renal histopathological analysis. Simultaneously, renal protein levels…

Male0301 basic medicineXanthine OxidaseOrganic anion transporter 1PhytochemicalsOrganic Anion TransportersRenal functionHyperuricemiaPharmacologyurologic and male genital diseasesExcretionMice03 medical and health scienceschemistry.chemical_compoundOrganic Anion Transport Protein 1medicineAnimalsHyperuricemiaRosaceaeCreatinineKidneybiologyPlant ExtractsGeneral Medicinemedicine.diseaseUric AcidGoutDisease Models AnimalOxonic Acid030104 developmental biologymedicine.anatomical_structureGene Expression RegulationLiverchemistryCreatinineFruitbiology.proteinUric acidPhytotherapyFood ScienceFood & Function
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Inhibition of Xanthine Oxidase by Allopurinol Prevents Skeletal Muscle Atrophy: Role of p38 MAPKinase and E3 Ubiquitin Ligases

2012

International audience; Abstract Top Alterations in muscle play an important role in common diseases and conditions. Reactive oxygen species (ROS) are generated during hindlimb unloading due, at least in part, to the activation of xanthine oxidase (XO). The major aim of this study was to determine the mechanism by which XO activation causes unloading-induced muscle atrophy in ratsand its possible prevention by allopurinol, a well-known inhibitor of this enzyme. For this purpose we studied one of the main redox sensitive signalling cascades involved in skeletal muscle atrophy i.e. p38 MAPKinaseand the expression of two well known muscle specific E3 ubiquitin ligases involved in proteolysis, …

MaleAgingAnatomy and Physiology[SDV]Life Sciences [q-bio]lcsh:MedicineMuscle ProteinsGene ExpressionHindlimbSignal transductionmedicine.disease_causep38 Mitogen-Activated Protein KinasesTripartite Motif Proteinschemistry.chemical_compound0302 clinical medicineMolecular cell biologySignaling in Cellular Processeslcsh:ScienceMusculoskeletal System0303 health sciencesMultidisciplinarySignaling cascadesMuscle BiochemistryAnimal ModelsMuscle atrophy3. Good healthMuscular Atrophymedicine.anatomical_structureBiochemistryHindlimb SuspensionMuscleMedicinemedicine.symptomCellular Typesmedicine.drugResearch Articlemedicine.medical_specialtyXanthine OxidaseMAPK signaling cascadesAllopurinolUbiquitin-Protein LigasesAllopurinolBiology03 medical and health sciencesAtrophyModel OrganismsInternal medicinemedicineAnimalsRats WistarXanthine oxidaseMuscle SkeletalBiology030304 developmental biologySoleus muscleMuscle CellsSKP Cullin F-Box Protein LigasesSuperoxide Dismutaselcsh:RSkeletal musclemedicine.diseaseRatsEnzyme ActivationOxidative StressEndocrinologychemistryRatlcsh:QPhysiological Processes030217 neurology & neurosurgeryOxidative stress
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