Search results for "acetylcholine receptor"

showing 10 items of 263 documents

The Non-neuronal Cholinergic System

2001

Acetylcholine, one of the most exemplary neurotransmitters, has been detected in bacteria, algae, protozoa, tubellariae and primitive plants, suggesting an extremely early appearance in the evolutionary process and a wide expression in non-neuronal cells. In plants (Urtica dioica), acetylcholine is involved in the regulation of water resorption and photosynthesis. In humans, acetylcholine and/or the synthesizing enzyme, choline acetyltransferase, have been demonstrated in epithelial (airways, alimentary tract, urogenital tract, epidermis), mesothelial (pleura, pericardium), endothelial, muscle and immune cells (granulocytes, lymphocytes, macrophages, mast cells). The widespread expression o…

Pharmacologymedicine.medical_specialtyInflammationBiologyCholine acetyltransferaseCell biologyNicotinic agonistEndocrinologyInternal medicineMuscarinic acetylcholine receptormedicinebiology.proteinSignal transductionmedicine.symptomReceptorAcetylcholinemedicine.drugCholinesteraseJapanese Journal of Pharmacology
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Non-neuronal acetylcholine, a locally acting molecule, widely distributed in biological systems: expression and function in humans.

1998

Acetylcholine acts as a neurotransmitter in the central and peripheral nervous systems in humans. However, recent experiments demonstrate a widespread expression of the cholinergic system in non-neuronal cells in humans. The synthesizing enzyme choline acetyltransferase, the signalling molecule acetylcholine, and the respective receptors (nicotinic or muscarinic) are expressed in epithelial cells (human airways, alimentary tract, epidermis). Acetylcholine is also found in mesothelial, endothelial, glial, and circulating blood cells (platelets, mononuclear cells), as well as in alveolar macrophages. The existence of non-neuronal acetylcholine explains the widespread expression of muscarinic …

Pharmacologymedicine.medical_specialtyMuscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2BiologyAcetylcholineCell biologyCholine O-AcetyltransferaseCircadian RhythmEndocrinologyNicotinic agonistInternal medicineMuscarinic acetylcholine receptor M5Muscarinic acetylcholine receptormedicineMuscarinic acetylcholine receptor M4CholinergicHumansPharmacology (medical)Acetylcholinemedicine.drugPharmacologytherapeutics
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Chapter 19 Muscarinic activation of phosphatidylcholine hydrolysis

1996

Publisher Summary This chapter focuses on the muscarinic activation of phosphatidylcholine hydrolysis. The release of choline from tissues or cells is a sensitive indicator of an enhanced hydrolysis of phosphatidylcholine (PtdCho) and is easily determined by chemiluminescence. In certain cells, choline release may reflect the activity of a specific receptor-activated enzyme catalyzing PtdCho hydrolysis. A physiological role of the receptor-mediated release of choline in the brain is given by its role as biosynthetic precursor for acetylcholine (ACh) and phospholipids. When PtdCho hydrolysis is investigated to identify the phospholipase involved, the sole determination of enzymatic products …

Phospholipase DPhosphatidic acidPhospholipasechemistry.chemical_compoundchemistryBiochemistryPhosphatidylcholineMuscarinic acetylcholine receptormedicineCholinelipids (amino acids peptides and proteins)Protein kinase CAcetylcholinemedicine.drug
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MICROWAVE EFFECTS ON ACETYLCHOLINE-INDUCED CHANNELS IN CULTURED CHICK MYOTUBES

1988

The behavior of cultured myotubes from chick embryos exposed to microwaves has been experimentally analyzed. Recordings of acetylcholine-induced currents have been obtained via patch-clamp techniques using both cell-attached (single-channel current recording) and whole-cell (total current recording) configurations. During the exposure to low-power microwaves the frequency of the ACh-activated single channel openings decreased, while the ACh-induced total current showed a faster falling phase. Channel open time and conductance were not affected by microwave irradiation. It is concluded that the exposure to microwaves increases the rate of desensitization and decreases the channel opening pro…

PhysiologyMyogenesisChemistryMusclesBiophysicsPhase (waves)ConductanceChick EmbryoGeneral MedicineAnatomyIn Vitro TechniquesElectromagnetic radiationAcetylcholineIon ChannelsDesensitization (telecommunications)medicineBiophysicsAnimalsRadiology Nuclear Medicine and imagingacetylcholine receptor; microwave irradiationCurrent (fluid)MicrowavesMicrowaveAcetylcholinemedicine.drug
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Manipulation of HSP70-SOD1 Expression Modulates SH-SY5Y Differentiation and Susceptibility to Oxidative Stress-Dependent Cell Damage: Involvement in …

2023

The differentiation of neural progenitors is a complex process that integrates different signals to drive transcriptional changes, which mediate metabolic, electrophysiological, and morphological cellular specializations. Understanding these adjustments is essential within the framework of stem cell and cancer research and therapy. Human neuroblastoma SH-SY5Y cells, widely used in neurobiology research, can be differentiated into neuronal-like cells through serum deprivation and retinoic acid (RA) supplementation. In our study, we observed that the differentiation process triggers the expression of Heat Shock Protein 70 (HSP70). Notably, inhibition of HSP70 expression by KNK437 causes a dra…

Physiologyoxotremorine; muscarinic acetylcholine receptor; KNK437; neuroprotection; heat shock proteins; superoxide dismutaseClinical Biochemistryheat shock proteinmuscarinic acetylcholine receptorKNK437neuroprotectionCell BiologyMolecular BiologyBiochemistrysuperoxide dismutaseoxotremorine
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Physostigmine and Neuromuscular Transmission

1993

Single channel studies carried out in cultured rat myoballs and cultured hippocampal neurons, and ion flux studies performed on Torpedo electrocyte membrane vesicles, showed that physostigmine (Phy), a well-established acetylcholinesterase inhibitor, interacts directly with nicotinic acetylcholine receptors (nAChR). Low concentrations (0.1 microM) of Phy activate the receptor integral channel, whereas higher concentrations blocked the channel in its opened state. In contrast to channel activation by acetylcholine (ACh) and classical cholinergic agonists, however, Phy was capable of activating the nAChR channel even when the ACh binding sites were blocked by competitive antagonists, such as …

PhysostigmineMolecular Sequence DataNeuromuscular JunctionNeuromuscular transmissionIn Vitro TechniquesReceptors NicotinicTorpedoHippocampusSynaptic TransmissionGeneral Biochemistry Genetics and Molecular BiologyNeuromuscular junctionHistory and Philosophy of SciencemedicineAnimalsAmino Acid SequencePatch clampBinding siteCells CulturedAcetylcholine receptorBinding SitesChemistryGeneral NeuroscienceAcetylcholineRatsQuaternary Ammonium CompoundsNicotinic agonistmedicine.anatomical_structureBiophysicsCholinergicIon Channel GatingNeuroscienceAcetylcholinemedicine.drugAnnals of the New York Academy of Sciences
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Photoaffinity labeling of Torpedo acetylcholine receptor by physostigmine.

1993

The plant alkaloid physostigmine, an established anti-cholinesterase agent of the carbamate type, has recently been shown to bind to the nicotinic acetylcholine receptor from Torpedo marmorata electrocytes [Okonjo, K. O., Kuhlmann, J.Maelicke, A. (1991) Eur. J. Biochem. 200, 671-677]. Pharmacological studies of physostigmine-induced ion flux into nicotinic-acetylcholine-receptor-rich membrane vesicles, indicated distinct binding sites for physostigmine and acetylcholine. As shown in this study by photoaffinity labeling with [phenyl-(n)-3H](-)physostigmine, the physostigmine-binding site is located within the same subunit (alpha polypeptide) of the receptor as the acetylcholine-binding site.…

PhysostigmineStereochemistryPhotochemistryUltraviolet RaysPhysostigmineMolecular Sequence DataReceptors NicotinicTorpedoTritiumBiochemistrylaw.inventionlawmedicineAnimalsAmino Acid SequenceAcetylcholine receptorBinding SitesPhotoaffinity labelingChemistryAffinity LabelsBungarotoxinLigand (biochemistry)Nicotinic acetylcholine receptorBiochemistryTorpedoAcetylcholinemedicine.drugEuropean journal of biochemistry
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Expression and renaturation of the N-terminal extracellular domain of torpedo nicotinic acetylcholine receptor alpha-subunit.

1998

The N-terminal extracellular region (amino acids 1-209) of the alpha-subunit of the nicotinic acetylcholine receptor (nAChR) from Torpedo marmorata electric tissue was expressed as inclusion bodies in Escherichia coli using the pET 3a vector. Employing a novel protocol of unfolding and refolding, in the absence of detergent, a water-soluble globular protein of 25 kDa was obtained displaying approximately 15% alpha-helical and 45% beta-structure. The fragment bound alpha-[3H]bungarotoxin in 1:1 stoichiometry with a KD value of 0.5 nM as determined from kinetic measurements (4 nM from equilibrium binding). The kinetics of association of toxin and fragment were of second order, with a similar …

Protein DenaturationProtein FoldingMolecular Sequence DataReceptors NicotinicTorpedoBiochemistrylaw.inventionchemistry.chemical_compoundGanglion type nicotinic receptorlawExtracellularmedicineEscherichia coliAnimalsAmino Acid SequenceCloning MolecularMolecular BiologyMethyllycaconitineCell BiologyBungarotoxinBungarotoxinsRecombinant ProteinsNicotinic acetylcholine receptorBiochemistrychemistryAlpha-4 beta-2 nicotinic receptorTorpedoAcetylcholinemedicine.drugProtein BindingThe Journal of biological chemistry
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The Non-neuronal cholinergic system: an emerging drug target in the airways.

2001

The non-neuronal cholinergic system is widely expressed in human airways. Choline acetyltransferase (ChAT) and/or acetylcholine are demonstrated in more or less all epithelial surface cells (goblet cells, ciliated cells, basal cells), submucosal glands and airway smooth muscle fibres. Acetylcholine is also demonstrated in the effector cells of the immune system (lymphocytes, macrophages, mast cells). Epithelial, endothelial and immune cells express nicotinic and muscarinic receptors. Thus the cytomolecule acetylcholine can contribute to the regulation of basic cell functions via auto-/paracrine mechanisms (proliferation, differentiation, ciliary activity, secretion of water, ions and mucus,…

Pulmonary and Respiratory MedicineLung Diseasesmedicine.medical_specialtyInflammationBiologyReceptors NicotinicCholine O-AcetyltransferaseImmune systemInternal medicineMuscarinic acetylcholine receptorMuscarinic acetylcholine receptor M5medicineHomeostasisHumansPharmacology (medical)InflammationImmunity CellularBiochemistry (medical)Muscarinic acetylcholine receptor M3Epithelial CellsMuscle SmoothCholine acetyltransferaseReceptors MuscarinicAcetylcholineCell biologyNicotinic agonistEndocrinologyAntibody Formationmedicine.symptomAcetylcholinemedicine.drugPulmonary pharmacologytherapeutics
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Muscarinic M2 receptors in acetylcholine-isoproterenol functional antagonism in human isolated bronchus

2002

The muscarinic functional antagonism of isoproterenol relaxation and the contribution of muscarinic M2 receptors were examined in human isolated bronchus. In intact tissues, acetylcholine (ACh) precontraction decreased isoproterenol potency and maximal relaxation (−log EC50 shift = −1.49 ± 0.16 and Emax inhibition for 100 μM ACh = 30%) more than the same levels of histamine contraction. The M2receptor-selective antagonist methoctramine (1 μM) reduced this antagonism in ACh- but not histamine-contracted tissues. Similar results were obtained for forskolin-induced relaxation. After selective inactivation of M3 receptors with 4-diphenylacetoxy- N-(2-chloroethyl)piperadine hydrochloric acid (3…

Pulmonary and Respiratory Medicinemedicine.medical_specialtyLung NeoplasmsPhysiologyMuscle RelaxationBronchiMuscarinic AntagonistsIn Vitro Techniqueschemistry.chemical_compoundPiperidinesPhysiology (medical)Internal medicineIsoprenalineMuscarinic acetylcholine receptorCyclic AMPmedicineMethoctramineHumansNeurotransmitterAcetylcholine receptorReceptor Muscarinic M2BronchusColforsinIsoproterenolMuscle SmoothMuscarinic acetylcholine receptor M2Cell BiologyReceptors MuscarinicAcetylcholinemedicine.anatomical_structureEndocrinologychemistryAcetylcholineMuscle Contractionmedicine.drugAmerican Journal of Physiology-Lung Cellular and Molecular Physiology
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