Search results for "active"

showing 10 items of 5361 documents

Twitter as a tool for teaching and communicating microbiology: the #micromoocsem initiative

2016

López-Goñi, Ignacio et al.

0301 basic medicineComputer scienceHuman immunodeficiency virus (HIV)medicine.disease_causeMicrobiologíaSocial networksMultidisciplinary approachScience communicationDuration (project management)Biology (General)lcsh:QH301-705.5X300Centro Oceanográfico de Gijónmedia_commoneducation.field_of_studylcsh:LC8-66914. Education05 social sciences050301 educationC500Special aspects of educationsocial networkGeneral Agricultural and Biological SciencesP990AcuiculturaQH301-705.5media_common.quotation_subject030106 microbiologyPopulationTwitterAcademic practiceTips & Toolscollaborative teachingMOOCMicrobiologyGeneral Biochemistry Genetics and Molecular BiologyEducationMicrobiology03 medical and health sciencesactive learningmedicineInstitutioneducationGeneral Immunology and MicrobiologyLC8-6691lcsh:Special aspects of educationTeachingmicrobiologySocial learningsocial learningMicroMOOCSEMlcsh:Biology (General)0503 education
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The Problem of Mental Action: Predictive Control Without Sensory Sheets by Metzinger, T. (2017). In Philosophy and Predictive Processing, eds T. Metz…

2018

A growing number of studies on the acquisition of lexical tone by adult learners have revealed that factors such as language background, musical experience, cognitive abilities, and neuroanatomy all play a role in determining tone learning success. On the basis of these findings, it has been argued that the effectiveness of tone learning in adulthood depends on individual differences in these factors. However, it is not clear whether similar individual differences play an analogous role in tone learning in childhood. Indeed, relatively few studies have made comparisons between how adults and children learn lexical tones. Here, we review recent developments for tone learning in both adults a…

0301 basic medicineControl theory (sociology)General Commentarylcsh:BF1-990predictive processingSensory systemmental actioncontrol theory03 medical and health sciencesModel predictive controllcsh:Psychology030104 developmental biology0302 clinical medicineactive inferenceaction-oriented cognitionPsychologyPsychologyMental action030217 neurology & neurosurgeryGeneral PsychologyCognitive psychologyFrontiers in Psychology
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Prevention from radiation damage by natural products

2018

Abstract Background Radiotherapy is a mainstay of cancer treatment since decades. Ionizing radiation (IR) is used for destruction of cancer cells and shrinkage of tumors. However, the increase of radioresistance in cancer cells and radiation toxicity to normal tissues are severe concerns. The exposure to radiation generates intracellular reactive oxygen species (ROS), which leads to DNA damage by lipid peroxidation, removal of thiol groups from cellular and membrane proteins, strand breaks and base alterations. Hypothesis Plants have to deal with radiation-induced damage (UV-light of sun, other natural radiation sources). Therefore, it is worth speculating that radioprotective mechanisms ha…

0301 basic medicineCurcuminAntioxidantDNA damagemedicine.medical_treatmentPhytochemicalsPharmaceutical ScienceSilibininRadiation-Protective AgentsAscorbic AcidBiologyPharmacologyAntioxidantsLipid peroxidation03 medical and health scienceschemistry.chemical_compound0302 clinical medicineNeoplasmsRadioresistanceStilbenesDrug DiscoverymedicineHumansRadiation InjuriesFlavonoidsPharmacologyRadiotherapyRadiochemistryPolyphenolsPlantsAscorbic acid030104 developmental biologyComplementary and alternative medicinechemistryResveratrol030220 oncology & carcinogenesisCancer cellCurcuminMolecular MedicineLipid PeroxidationReactive Oxygen SpeciesDNA DamagePhytomedicine
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Oxidative damage and disturbance of antioxidant capacity by zearalenone and its metabolites in human cells.

2017

Mycotoxin contamination of foods and feeds represent a serious problem worldwide. Zearalenone (ZEA) is a secondary metabolite produced by Fusarium species. This study explores oxidative cellular damage and intracellular defense mechanisms (enzymatic and non-enzymatic) in the hepatoma cell line HepG2 after exposure to ZEA and its metabolites (α-zearalenol, α-ZOL; β-zearalenol, β-ZOL). Our results demonstrated that HepG2 cells exposed to ZEA, α-ZOL or β-ZOL at different concentrations (0, 6.25, 12.5 and 25μM) showed: (i) elevated ROS levels (1.5- to 7-fold) based on the formation of the highly fluorescent 2',7'-dichlorofluorescein (DCF), (ii) increased DNA damage measured by the comet assay (…

0301 basic medicineDNA damage010501 environmental sciencesSecondary metaboliteToxicologymedicine.disease_cause01 natural sciencesAntioxidantsSuperoxide dismutase03 medical and health scienceschemistry.chemical_compoundDichlorofluoresceinmedicineHumans0105 earth and related environmental sciencesbiologySuperoxide Dismutasefood and beveragesGeneral MedicineGlutathioneHep G2 CellsMycotoxinsCatalaseGlutathioneComet assayOxidative Stress030104 developmental biologychemistryBiochemistryCatalasebiology.proteinta1181ZearalenoneComet AssayReactive Oxygen SpeciesOxidative stressmedicine.drugDNA DamageToxicology in vitro : an international journal published in association with BIBRA
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Anisakis pegreffii (Nematoda: Anisakidae) products modulate oxidative stress and apoptosis-related biomarkers in human cell lines

2016

Background In countries with elevated prevalence of zoonotic anisakiasis and high awareness of this parasitosis, a considerable number of cases that associate Anisakis sp. (Nematoda, Anisakidae) and different bowel carcinomas have been described. Although neoplasia and embedded larvae were observed sharing the common site affected by chronic inflammation, no association between the nematode and malignancy were directly proved. Similarly, no data are available about the effect of secretory and excretory products of infecting larvae at the host’s cellular level, except in respect to allergenic interaction. Methods To test the mechanisms by which human non-immune cells respond to the larvae, w…

0301 basic medicineDNA damageCell SurvivalApoptosismedicine.disease_causeAnisakisFibroblast cell lines HS-68lcsh:Infectious and parasitic diseasesCell Line03 medical and health sciences0302 clinical medicineSettore AGR/20 - ZoocolturemedicineAnisakis pegreffii ; Apoptosis ; Fibroblast cell lines HS-68 ; Inflammation ; Oxidative stressAnimalsHumanslcsh:RC109-216Viability assayAnisakis pegreffii Apoptosis Fibroblast cell lines HS-68 Inflammation Oxidative stressSettore BIO/06 - Anatomia Comparata E Citologiachemistry.chemical_classificationInflammationReactive oxygen speciesBiological ProductsbiologyKinaseCell growthResearchbiology.organism_classificationMolecular biologyAnisakisOxidative Stress030104 developmental biologyInfectious DiseaseschemistryApoptosis030220 oncology & carcinogenesisLarvaAnisakis pegreffiiImmunologyParasitologyInflammation MediatorsReactive Oxygen SpeciesOxidative stressBiomarkersParasites & Vectors
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Modification of DNA structure by reactive nitrogen species as a result of 2-methoxyestradiol–induced neuronal nitric oxide synthase uncoupling in met…

2020

Abstract 2-methoxyestradiol (2-ME) is a physiological anticancer compound, metabolite of 17β-estradiol. Previously, our group evidenced that from mechanistic point of view one of anticancer mechanisms of action of 2-ME is specific induction and nuclear hijacking of neuronal nitric oxide synthase (nNOS), resulting in local generation of nitro-oxidative stress and finally, cancer cell death. The current study aims to establish the substantial mechanism of generation of reactive nitrogen species by 2-ME. We further achieved to identify the specific reactive nitrogen species involved in DNA-damaging mechanism of 2-ME. The study was performed using metastatic osteosarcoma 143B cells. We detected…

0301 basic medicineDNA damageClinical BiochemistryBone NeoplasmsNitric Oxide Synthase Type INitric OxideBiochemistryNitric oxide03 medical and health scienceschemistry.chemical_compound0302 clinical medicinePeroxynitrous AcidHumansMTT assayViability assaylcsh:QH301-705.5Reactive nitrogen speciesSettore CHIM/02 - Chimica FisicaOsteosarcomalcsh:R5-920Settore BIO/16 - Anatomia UmanaOrganic ChemistryDNAReactive Nitrogen Species2-MethoxyestradiolPeroxynitrous acid030104 developmental biologychemistrylcsh:Biology (General)Settore CHIM/03 - Chimica Generale E InorganicaCancer cellBiophysicslcsh:Medicine (General)030217 neurology & neurosurgeryPeroxynitrite2 methoxyestradiol nitric oxide chemotherapyResearch PaperRedox Biology
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Preventive effects of guanosine on intestinal inflammation in 2, 4-dinitrobenzene sulfonic acid (DNBS)-induced colitis in rats

2018

Background: Guanosine, a guanine-based purine, is an extracellular signaling molecule exerting anti-inflammatory and antioxidative effects in several in vivo and in vitro injury models. We aimed to investigate its protective effects on 2, 4-dinitrobenzene sulfonic acid (DNBS)-induced colitis in rat. Methods: Rats were divided into five groups and colitis was induced by intracolonic instillation of DNBS (15 mg/rat). Guanosine (4 or 8 mg/kg) was administered for 6 days i.p. starting the day of the colitis induction. Body weight loss, stool consistency, colon weight/length, histological analysis, myeloperoxidase activity (MPO) and pro-inflammatory cytokine levels were assessed. Immunoblotting …

0301 basic medicineDNBS ratColonmedicine.medical_treatmentInterleukin-1betaImmunologyAnti-Inflammatory AgentsGuanosineInflammationPharmacologySettore BIO/09 - FisiologiaInflammatory bowel diseaseAntioxidantsInflammatory bowel disease03 medical and health scienceschemistry.chemical_compound0302 clinical medicineIn vivomedicineAnimalsPharmacology (medical)Intestinal MucosaRats WistarColitisPurineInflammationPharmacologychemistry.chemical_classificationReactive oxygen speciesGuanosineInterleukin-6Tumor Necrosis Factor-alphaNF-kappa BColitismedicine.diseaseRats030104 developmental biologyCytokinechemistryCytokinesDinitrofluorobenzeneTumor necrosis factor alphamedicine.symptomReactive Oxygen Species030217 neurology & neurosurgeryInflammopharmacology
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Tetrabromobisphenol A (TBBPA)-stimulated reactive oxygen species (ROS) production in cell-free model using the 2′,7′-dichlorodihydrofluorescein diace…

2016

t Tetrabromobisphenol A (TBBPA) is a widely used brominated flame retardant, applied in a variety of commercial and household products, mainly electronic ones. Since the production of reactive oxygen species (ROS) is considered one of the principal cytotoxicity mechanisms, numerous studies undertake that aspect of TBBPA’s mechanism of action. The present study verifies if the fluorogenic substrate 2′,7′- dichlorodihydrofluorescein diacetate (H2DCFDA) should be used to detect ROS production induced by TBBPA. To determine the ability of TBBPA alone to stimulate the conversion of H2DCFDA to its fluorescent product 2’, 7’- dichlorofluorescein (DCF), we used a cell-free model. In the experiments…

0301 basic medicineDPPHHealth Toxicology and MutagenesisPolybrominated BiphenylsCell-free system03 medical and health scienceschemistry.chemical_compound0302 clinical medicineH2DCFDAFree radicalDichlorofluoresceinEnvironmental ChemistryOrganic chemistryCytotoxicitychemistry.chemical_classificationReactive oxygen speciesCell-Free SystemROSFree Radical ScavengersGeneral MedicineFluoresceinsFree radical scavengerPollutionTBBPA030104 developmental biologychemistryBrominated flame retardantTetrabromobisphenol AReactive Oxygen Species030217 neurology & neurosurgeryResearch ArticleDPPHNuclear chemistryEnvironmental Science and Pollution Research
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Customised in vitro model to detect human metabolism-dependent idiosyncratic drug-induced liver injury

2017

Drug-induced liver injury (DILI) has a considerable impact on human health and is a major challenge in drug safety assessments. DILI is a frequent cause of liver injury and a leading reason for post-approval drug regulatory actions. Considerable variations in the expression levels of both cytochrome P450 (CYP) and conjugating enzymes have been described in humans, which could be responsible for increased susceptibility to DILI in some individuals. We herein explored the feasibility of the combined use of HepG2 cells co-transduced with multiple adenoviruses that encode drug-metabolising enzymes, and a high-content screening assay to evaluate metabolism-dependent drug toxicity and to identify…

0301 basic medicineDrugCYP2B6Drug-induced liver injuryHealth Toxicology and Mutagenesismedia_common.quotation_subjectPopulationDrug Evaluation PreclinicalPharmacologyToxicologyHepatotoxicity mechanismsGene Expression Regulation EnzymologicOrgan Toxicity and MechanismsAdenoviridae03 medical and health sciences0302 clinical medicineCYPToxicity TestsHumansCytochrome P450 Family 2educationmedia_commonMembrane Potential Mitochondrialeducation.field_of_studyCYP3A4biologyCytochrome P450IdiosyncrasyHep G2 CellsGeneral MedicineCYP2E1Recombinant ProteinsHigh-Throughput Screening Assays030104 developmental biology030220 oncology & carcinogenesisInactivation MetabolicToxicityCell modelbiology.proteinChemical and Drug Induced Liver InjuryReactive Oxygen SpeciesDrug metabolism
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A Multi-Parametric Fluorescent Assay for the Screening and Mechanistic Study of Drug-Induced Steatosis in Liver Cells in Culture.

2017

Human hepatic cells have been used for drug safety risk evaluations throughout early development phases. They provide rapid, cost-effective early feedback to identify drug candidates with potential hepatotoxicity. This unit presents a cell-based assay to evaluate the risk of liver damage associated with steatogenic drugs. Detailed protocols for cell exposure to test compounds and for the assessment of steatosis-related cell parameters (intracellular lipid content, reactive oxygen species production, mitochondrial impairment, and cell death) are provided. A few representative results that illustrate the utility of this procedure for the screening of drug-induced steatosis are shown. © 2017 b…

0301 basic medicineDrugProgrammed cell deathmedia_common.quotation_subjectCellMitochondria LiverBiologyToxicology03 medical and health sciencesmedicineHumansCells Culturedmedia_commonchemistry.chemical_classificationReactive oxygen speciesCell Deathmedicine.diseaseLipid MetabolismFatty Liver030104 developmental biologymedicine.anatomical_structurechemistryBiochemistryLiverHigh-content screeningCancer researchHepatic stellate cellHepatocytesSteatosisChemical and Drug Induced Liver InjuryReactive Oxygen SpeciesIntracellularCurrent protocols in toxicologyLiterature Cited
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