Search results for "acute pancreatiti"

showing 10 items of 66 documents

The management of abdominal hydatidosis after the rupture of a pancreatic hydatid cyst: a case report

2015

Introduction Echinococcosis or hydatidosis is a zoonosis caused by cestodes from the genus Echinococcus; its habitat is the small intestine of the definitive host, represented by dogs/carnivorous animals, where it produces eggs which are eliminated in the environment. Cystic echinococcosis represents more than 95% of the hydatidosis cases registered annually. The most frequent localization is the hepatic one, followed by the pulmonary localization with a ratio of 2.5:1. A pancreatic localization represents 0.2% of hydatidosis cases with a higher possibility of disseminating intra-abdominally. The incidence of hydatidosis in Romania has not been investigated yet through national studies. Cas…

Malemedicine.medical_specialtyCase ReportAlbendazoleAlbendazoleEchinococcosisSurgical oncologyAnimalsHumansMedicineAnti-parasite treatmentAnthelminticsEchinococcus genusMedicine(all)Rupture SpontaneousRomaniabusiness.industryIncidence (epidemiology)ZoonosisPancreatic DiseasesPancreatic cystGeneral MedicineMiddle Agedmedicine.diseaseEchinococcosisSurgeryPraziquantelAcute abdomenIntraabdominal InfectionsAcute pancreatitismedicine.symptombusinessmedicine.drugJournal of Medical Case Reports
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Multiple intrahepatic pseudocysts in acute pancreatitis

2007

Liver pseudocysts are a very rare complication in acute pancreatitis with only a few cases previously described. The lack of experience and literature on this condition leads to difficulties in the differential diagnosis and management. We report herein a case of acute pancreatitis who developed multiple intrahepatic pseudocysts. After complete imaging evaluation, the diagnosis was still unclear and the patient was operated on. The presence of liver lesions in patients with acute pancreatitis should raise the possibility of intrahepatic pseudocysts.

Malemedicine.medical_specialtyCase ReportSeverity of Illness IndexGastroenterologyDiagnosis DifferentialInternal medicineSeverity of illnessmedicineHumansIn patientAgedmedicine.diagnostic_testCystsbusiness.industryLiver DiseasesGastroenterologyMagnetic resonance imagingGeneral Medicinemedicine.diseaseMagnetic Resonance Imagingdigestive system diseasesTreatment OutcomeTomography x ray computedPancreatitisAcute DiseasePancreatitisAcute pancreatitisDifferential diagnosisTomography X-Ray ComputedbusinessComplicationWorld Journal of Gastroenterology
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Circulating TNF-alpha and its soluble receptors during experimental acute pancreatitis.

2004

Clinical and experimental studies have shown increased concentrations of TNF-α and its soluble receptors in serum of patients with acute pancreatitis. In this work, we have investigated the time-course of TNF-α and its soluble receptors during taurocholate-induced acute pancreatitis. In addition, since TNF-α itself could mediate the shedding of its receptors, we have assessed the effect of inhibiting TNF-α production on the release of soluble TNF-α receptors in experimental acute pancreatitis. Our results indicate that soluble receptors are released in the early stages of the disease and this increase is concomitant with the release of TNF-α, which is mainly bound to specific proteins. The …

Malemedicine.medical_specialtyImmunologyInflammationBiochemistryDNA-binding proteinReceptors Tumor Necrosis FactorPentoxifyllineInternal medicinemedicineSIRSImmunology and AllergyAnimalsPentoxifyllineRats WistarReceptorMolecular BiologyInflammationbusiness.industryTumor Necrosis Factor-alphaHematologymedicine.diseasesTNF-αRRatsDisease Models AnimalEndocrinologyPancreatitisSolubilityTNF-αAcute DiseasePancreatitisAcute pancreatitisTumor necrosis factor alphamedicine.symptombusinessmedicine.drugCytokine
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Pancreatic T cell protein-tyrosine phosphatase deficiency ameliorates cerulein-induced acute pancreatitis.

2014

Background Acute pancreatitis (AP) is a common clinical problem whose incidence has been progressively increasing in recent years. Onset of the disease is trigged by intra-acinar cell activation of digestive enzyme zymogens that induce autodigestion, release of pro-inflammatory cytokines and acinar cell injury. T-cell protein tyrosine phosphatase (TCPTP) is implicated in inflammatory signaling but its significance in AP remains unclear. Results In this study we assessed the role of pancreatic TCPTP in cerulein-induced AP. TCPTP expression was increased at the protein and messenger RNA levels in the early phase of AP in mice and rats. To directly determine whether TCPTP may have a causal rol…

MessengerWistarProtein tyrosine phosphataseInbred C57BLBiochemistryOral and gastrointestinalSTAT3Mice2.1 Biological and endogenous factorsPhosphorylationAetiologySTAT3Non-Receptor Type 2CeruletideCancerMice KnockoutProtein Tyrosine Phosphatase Non-Receptor Type 2Pancreatitis Acute NecrotizingNF-kappa B3. Good healthAcute NecrotizingAmylasesTumor necrosis factor alphaTCPTPCell activationCeruletideSTAT3 Transcription Factormedicine.medical_specialtyBiochemistry & Molecular BiologyKnockoutBiologyProinflammatory cytokinePancreatic CancerRare DiseasesInternal medicineAcinar cellmedicineGeneticsAnimalsRNA MessengerRats WistarMolecular BiologyInflammationTumor Necrosis Factor-alphaInterleukin-6ResearchCell BiologyLipaseNFKB1RatsAcute pancreatitisMice Inbred C57BLEndocrinologyPancreatitisbiology.proteinRNAProtein Tyrosine PhosphataseBiochemistry and Cell BiologyDigestive DiseasesKnockout mice
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Redox signaling in acute pancreatitis

2015

Acute pancreatitis is an inflammatory process of the pancreatic gland that eventually may lead to a severe systemic inflammatory response. A key event in pancreatic damage is the intracellular activation of NF-κB and zymogens, involving also calcium, cathepsins, pH disorders, autophagy, and cell death, particularly necrosis. This review focuses on the new role of redox signaling in acute pancreatitis. Oxidative stress and redox status are involved in the onset of acute pancreatitis and also in the development of the systemic inflammatory response, being glutathione depletion, xanthine oxidase activation, and thiol oxidation in proteins critical features of the disease in the pancreas. On th…

NecrosisGSH reduced glutathioneSTAT3 signal transducer and activator of transcription 3ERK extracellular signal-regulated kinasesClinical BiochemistryCCK cholecystokininTRAFs TNF receptor associated factorsReview ArticleIκB kinasePharmacologymedicine.disease_causeBiochemistrySHP small heterodimer partnerSTIM1 stromal interaction molecule 1chemistry.chemical_compoundHATs histone acetyltransferasesMedicineASK1GCL glutamate cysteine ligaseTNF-α tumor necrosis factor alphaIKK IκB kinaseNOS nitric oxide synthaseAcute inflammationHIF hypoxia inducible factorlcsh:QH301-705.5NF-κB nuclear factor kappa BDAMPs damage-associated molecular pattern moleculeslcsh:R5-920biologyGSSG oxidized glutathioneNF-kappa BNLRs nucleotide-binding oligomerization domain (NOD) like receptorsTRADD tumor necrosis factor receptor type 1-associated DEATH domain proteinTRPC3 transient receptor potential channel 3VEGF vascular endothelial growth factorGlutathioneTNFR tumor necrosis factor receptorHMGB1 high-mobility group Box 1 proteinIP3R inositol 145-trisphosphate receptor type 3VCAM-1 Vascular Cell adhesion protein 1Acute DiseaseJNK c-Jun N-terminal kinaseAcute pancreatitisTLRs toll-like receptorsmedicine.symptomlcsh:Medicine (General)Oxidation-ReductionAP-1 activator protein-1Signal TransductionmRNA messenger ribonucleic acidHMGB1ASC apoptosis-associated speck-like protein containing a carboxy-terminal CARDRNS reactive nitrogen speciesPTPs protein tyrosine phosphatasesROS reactive oxygen speciesNADH nicotinamide adenine dinucleotidepHe extracellular pHFAEE fatty acid ethyl estersAP acute pancreatitisHumansXanthine oxidaseCBP CREB-binding proteinRyR endoplasmic reticulum membrane ryanodine receptorsMDA malondialdehydeNO nitric oxideXO xanthine oxidaseASK1 apoptosis signal-regulating kinase-1business.industryOrganic ChemistryAutophagyNADPH nicotinamide adenine dinucleotide phosphateHDACs histone deacetylasesmedicine.diseaseCARS compensatory anti-inflammatory response syndromeXDH xanthine dehydrogenaseIL interleukinIκB inhibitor of kappa BAcute pancreatitisETC Electron transport chainPancreatitisMKPs MAPK phosphatasesSAP severe acute pancreatitischemistrylcsh:Biology (General)DTT dithiothreitolOxidative stressNAC N-acetyl cysteineImmunologybiology.proteinCalciumLysosomesReactive Oxygen SpeciesbusinessMAPK mitogen-activated protein kinaseOxidative stressERCP endoscopic retrograde cholangiopancreatographyRedox Biology
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Impairment of PGC-1 Alpha Up-Regulation Enhances Nitrosative Stress in the Liver during Acute Pancreatitis in Obese Mice

2020

Acute pancreatitis is an inflammatory process of the pancreatic tissue that often leads to distant organ dysfunction. Although liver injury is uncommon in acute pancreatitis, obesity is a risk factor for the development of hepatic complications. The aim of this work was to evaluate the role of PGC-1&alpha

PGC-1&#9450301 basic medicineobesitymedicine.medical_specialtyAntioxidantacute pancreatitisPhysiologymedicine.medical_treatmentClinical BiochemistryPGC-1αAlpha (ethology)Nitrosative stressliverHepatic ComplicationBiochemistryArticle03 medical and health sciences0302 clinical medicineDownregulation and upregulationInternal medicinemedicineObesityMolecular BiologyLiver injurybusiness.industrylcsh:RM1-950Organ dysfunctionCell BiologyBiología y Biomedicina / Biologíamedicine.diseasenitrosative stressAcute pancreatitislcsh:Therapeutics. Pharmacology030104 developmental biologyEndocrinologyLiver030220 oncology & carcinogenesisPancreatitisAcute pancreatitismedicine.symptombusinessAntioxidants
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Biliary Pancreatitis: Endoscopic Diagnostic and Therapeutic Techniques

1987

Acute pancreatitis constitutes one of the most hazardous emergencies in clinical gastroenterology. Although the diagnosis “biliary pancreatitis” is often used in clinical practice for patients presenting with concrements in the biliary tree or impacted in the papillary orifice [17, 32], the common phrase “biliary pancreatitis” has not so far been exactly defined. Patients with cholecystolithiasis may be included in this group, as well as patients with dysfunction of the sphincter of Oddi. Some authors have reported that juxtapapillary diverticula increase the risk for development of biliary pancreatitis [28].

Pancreatic ductmedicine.medical_specialtybusiness.industryBile ductmedicine.diseasedigestive systemGastroenterologyClinical Practicemedicine.anatomical_structureInternal medicineSphincter of OddimedicineAcute cholecystitisAcute pancreatitisCommon bile duct stoneBiliary pancreatitisbusiness
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Pancreas and Cystic Fibrosis: The Implications of Increased Survival in Cystic Fibrosis

2007

Background: In acute pancreatitis (AP), pancreatic necrosis (PN) is an important local complication that can be identified by means of contrast-enhanced computed tomography (CECT).

Pathologymedicine.medical_specialtyNecrosisHepatologybiologybusiness.industryEndocrinology Diabetes and MetabolismfungiC-reactive proteinGastroenterologyfood and beveragesmedicine.diseasemedicinebiology.proteinTechnetium Tc 99m ExametazimePancreatitisAcute pancreatitisIn patientmedicine.symptombusinessComplicationTechnetium-99mPancreatology
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Pentoxifylline and Oxypurinol: Potential Drugs to Prevent the “Cytokine Release (Storm) Syndrome” Caused by SARS-CoV-2?

2020

Background: COVID-19, caused by SARS-CoV-2, is a potentially lethal, rapidly-expanding pandemic and many efforts are being carried out worldwide to understand and control the disease. COVID-19 patients may display a cytokine release syndrome, which causes severe lung inflammation, leading, in many instances, to death. Objective: This paper is intended to explore the possibilities of controlling the COVID-19-associated hyperinflammation by using licensed drugs with anti-inflammatory effects. Hypothesis: We have previously described that pentoxifylline alone, or in combination with oxypurinol, reduces the systemic inflammation caused by experimentally-induced pancreatitis in rats. Pentoxifyl…

Pharmacology0303 health sciencesbusiness.industryInflammationmedicine.diseaseSystemic inflammationPentoxifyllineProinflammatory cytokine03 medical and health scienceschemistry.chemical_compoundCytokine release syndrome0302 clinical medicinechemistry030220 oncology & carcinogenesisDrug DiscoveryImmunologyMedicineAcute pancreatitisPancreatitismedicine.symptomXanthine oxidasebusiness030304 developmental biologymedicine.drugCurrent Pharmaceutical Design
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Co-administration of pentoxifylline and thiopental causes death by acute pulmonary oedema in rats

2006

Background and purpose: Pentoxifylline exhibits rheological properties that improve microvascular flow and it is widely used in vascular perfusion disorders. It also exhibits marked anti-inflammatory properties by inhibiting tumour necrosis factor a production. Thiopental is one of the most widely used drugs for rapid induction of anaesthesia. During experimental studies on the treatment of acute pancreatitis, we observed that when pentoxifylline was administered after anaesthesia with thiopental, most of the rats exhibited dyspnea, signs of pulmonary oedema and died. The aim of the work described here was to investigate the cause of the unexpected toxic effect of the combined treatment wit…

PharmacologyLungbusiness.industryVascular permeabilitymedicine.diseasePulmonary edemaPulmonary hypertensionHypoxemiaPentoxifyllinemedicine.anatomical_structureAnesthesiaMedicineAcute pancreatitisPancreatitismedicine.symptombusinessmedicine.drugBritish Journal of Pharmacology
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