Search results for "adenosine"

showing 10 items of 542 documents

Not All Floating-Harbor Syndrome Cases are Due to Mutations in Exon 34 of SRCAP

2013

International audience; Floating-Harbor syndrome (FHS) is a rare disorder characterized by short stature, delayed bone age, speech delay, and dysmorphic facial features. We report here the molecular analysis of nine cases, fulfilling the diagnostic criteria for FHS. Using exome sequencing, we identified SRCAP as the disease gene in two cases and subsequently found SRCAP truncating mutations in 6/9 cases. All mutations occurred de novo and were located in exon 34, in accordance with the recent report of Hood et al. However, the absence of SRCAP mutations in 3/9 cases supported genetic heterogeneity of FH syndrome. Importantly, no major clinical differences were observed supporting clinical h…

AdultHeart Septal Defects VentricularMaleDNA Mutational AnalysisBiologyShort statureCraniofacial Abnormalitiesgenetic heterogeneity03 medical and health sciencesExonGeneticsmedicineHumansAbnormalities MultipleGenetic Predisposition to DiseaseChildFloating-Harbor syndromeGenetics (clinical)Exome sequencingGrowth Disorders030304 developmental biologyDisease geneGeneticsAdenosine Triphosphatases0303 health sciences[SDV.GEN]Life Sciences [q-bio]/GeneticsGenetic heterogeneity030305 genetics & heredityBone ageExonsmedicine.diseaseSRCAP3. Good healthFloating–Harbor syndromeSpeech delayMutationFemalemedicine.symptom[ SDV.GEN ] Life Sciences [q-bio]/Genetics
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Effect of cordycepin on interleukin-10 production of human peripheral blood mononuclear cells

2002

Therapeutic options for controlling autoimmune diseases are still very limited. Interleukin-10 has been reported to be a promising approach to therapeutic intervention. In the search for a drug which results in the selective upregulation of interleukin-10, we investigated the immunoregulative effects of cordycepin. We have measured interleukin-10 and interleukin-2 secretion of human peripheral blood mononuclear cells that were incubated with cordycepin and assessed the influence of cordycepin on the expression of interleukin-10 mRNA, the proliferative response and the expression of surface markers on T lymphocytes. In addition, the subsets of interleukin-10-secreting cells, the influence of…

AdultInterleukin 2T-Lymphocytesmedicine.medical_treatmentPharmacologyBiologyLymphocyte ActivationPeripheral blood mononuclear cellchemistry.chemical_compoundAdjuvants ImmunologicAntigens CDmedicineHumansRNA MessengerIL-2 receptorCells CulturedPharmacologyDeoxyadenosinesDose-Response Relationship DrugCordycepinMonocyteInterleukinFlow CytometryInterleukin-10Up-RegulationInterleukin 10medicine.anatomical_structureCytokinechemistryImmunologyLeukocytes MononuclearInterleukin-2Cell Divisionmedicine.drugEuropean Journal of Pharmacology
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Cellular-free magnesium depletion in brain and muscle of normal and preeclamptic pregnancy: A nuclear magnetic resonance spectroscopic study

2004

Preeclampsia is a pregnancy disorder of unknown origin, characterized by vasospasm, elevated blood pressure, and increased neuromuscular irritability, features common to syndromes of magnesium deficiency. Evidence of serum and ionized magnesium metabolism disturbances have been observed in women with preeclampsia. This and the therapeutic utility of magnesium in preeclampsia led us to investigate the extent to which an endogenous tissue magnesium deficiency might be present in and contribute to its pathophysiology. We used 31 P nuclear magnetic resonance spectroscopy to noninvasively measure in situ intracellular-free magnesium levels in brain and skeletal muscle of fasting nonpregnant wom…

AdultIntracellular Fluidmedicine.medical_specialtyPregnancy Trimester ThirdDiastolechemistry.chemical_elementBlood PressurePreeclampsiaAdenosine TriphosphatePre-EclampsiaPregnancyMagnesium deficiency (medicine)Internal medicineInternal MedicinemedicineHumansMagnesiumIonMuscle SkeletalNuclear Magnetic Resonance BiomolecularBrain ChemistryMagnesiumbusiness.industrySkeletal muscleFastingHydrogen-Ion Concentrationmedicine.diseasePreeclampsiaPathophysiologymedicine.anatomical_structureBlood pressureEndocrinologyMetabolismchemistryFemalebusinessMagnesium DeficiencyPregnancy disorder
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Effects of allergen exposure on methacholine and AMP-induced air trapping in pollen-sensitive subjects

2011

Summary Background The effect of pro-inflammatory stimuli on bronchoconstrictor-induced air trapping has not been studied. Objective To determine the effect of natural allergen exposure, a pro-inflammatory stimulus, on methacholine- and adenosine 5′-monophospate (AMP)-induced air trapping. Methods Airway responsiveness to methacholine and AMP before and during the pollen season was obtained in 25 subjects with pollen allergy and in 10 healthy controls. The response was expressed by the sensitivity (PC 20 value) and by the slope and intercept of the FVC values recorded at each step of the challenge against the corresponding FEV 1 values. Results The slope and intercept FVC versus FEV 1 value…

AdultMaleAdenosine monophosphatePulmonary and Respiratory Medicinemedicine.medical_specialtyAllergyBronchoconstrictionVital Capacitymedicine.disease_causeAir trappingBronchial Provocation TestsAirway responsivenesschemistry.chemical_compoundFEV1/FVC ratioAllergenForced Expiratory VolumeInternal medicinePollenotorhinolaryngologic diseasesmedicineHumansMethacholinebusiness.industryRhinitis Allergic SeasonalMiddle AgedAllergensrespiratory systemmedicine.diseaseAdenosine MonophosphateAsthmaAir trappingrespiratory tract diseasesEndocrinologyAdenosine 5’-monophosphatechemistrySpirometryImmunologyPollenFemaleMethacholineBronchial Hyperreactivitymedicine.symptomALLERGEN EXPOSUREbusinesscirculatory and respiratory physiologymedicine.drugRespiratory Medicine
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Safety and Ergogenic Properties of Combined Aminophylline and Ambrisentan in Hypoxia.

2017

We hypothesized that concomitant pharmacological inhibition of the endothelin and adenosine pathway is safe and improves exercise performance in hypoxic humans, via a mechanism that does not involve augmentation of blood oxygenation. To test this hypothesis, we established safety and drug interactions for aminophylline (500 mg) plus ambrisentan (5 mg) in normoxic volunteers. Subsequently, a placebo‐controlled study was employed to test the combination in healthy resting and exercising volunteers at simulated altitude (4,267 m). No serious adverse events occurred. Drug interaction was minimal or absent. Aminophylline alleviated hypoxia‐induced headaches. Aminophylline, ambrisentan, and their…

AdultMaleAdenosineAmbrisentanAdolescent030204 cardiovascular system & hematologyPharmacologyPlaceboHypoxemia03 medical and health sciencesYoung Adult0302 clinical medicineDouble-Blind MethodmedicineHumansPharmacology (medical)Adverse effectHypoxiaExercisePharmacologyPhenylpropionatesbusiness.industryAltitudeEndothelinsResearchArticlesHypoxia (medical)Drug interactionMiddle AgedAminophylline3. Good healthPyridazinesAnesthesiaAminophyllineDrug Therapy CombinationFemalemedicine.symptomEndothelin receptorbusiness030217 neurology & neurosurgerymedicine.drugSignal TransductionClinical pharmacology and therapeutics
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A Highly Decreased Binding of Cyclic Adenosine Monophosphate to Protein Kinase A in Erythrocyte Membranes is Specific for Active Psoriasis

2002

A cyclic adenosine monophosphate binding abnormality in psoriatic erythrocytes that could be corrected by retinoid treatment has been reported. It was tested whether this binding abnormality is specific for psoriasis and the effects of treatment were compared with etretinate, cyclosporine A, or anthralin on 2-(3)H-8-N(3)-cyclic adenosine monophosphate binding to the regulatory subunit of protein kinase A in erythrocyte membranes. One hundred and fifteen individuals were evaluated, including: (i) 34 healthy persons; (ii) 15 patients with nonatopic inflammatory skin diseases (eczema, erythroderma, tinea, Grover's disease, erysipelas, urticaria); (iii) eight with other dermatoses mediated by i…

AdultMaleAzidesmedicine.medical_specialtyAdolescentmedicine.drug_classAdministration TopicalAnti-Inflammatory AgentsErythrodermaEtretinateDermatologySeverity of Illness IndexBiochemistryRetinoidschemistry.chemical_compoundPsoriasis Area and Severity IndexKeratolytic AgentsPsoriasis Area and Severity IndexPsoriasisInternal medicineCyclic AMPmedicineHumansPsoriasisCyclic adenosine monophosphateRetinoidMolecular BiologydermatitisAgedErythema nodosumbusiness.industryErythrocyte MembraneAffinity LabelsCell BiologyAtopic dermatitisAnthralinMiddle Agedmedicine.diseaseCyclic AMP-Dependent Protein KinasesEndocrinologychemistryEtretinateCyclosporineFemaleDermatologic Agentsbusinesscyclosporine AProtein Bindingmedicine.drugJournal of Investigative Dermatology
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Biologic relevance of elevated red cell adenosine deaminase activity in myelodysplastic syndromes and paroxysmal nocturnal hemoglobinuria.

1992

Red cell adenosine deaminase (ADA-RBC) activity in patients with myelodysplastic syndromes and paroxysmal nocturnal hemoglobinuria is significantly increased compared to that observed in normal controls. ADA-RBC activity is not related to fetal hemoglobin concentration, but it is significantly correlated with hemoglobin concentration at diagnosis and with the degree of morphologic dysplasia in the erythroid lineage. The results of our study suggest that the observed enzymatic abnormality may constitute a non-specific manifestation of the stem cell alteration that determines these disorders.

AdultMaleCancer Researchmedicine.medical_specialtyErythrocytesAdenosine DeaminaseHemoglobinuria Paroxysmal030218 nuclear medicine & medical imaging03 medical and health sciences0302 clinical medicineAdenosine deaminaseInternal medicineFetal hemoglobinmedicineHumansPreleukemiaAgedRed Cellbiologybusiness.industryMyelodysplastic syndromeshemic and immune systemsGeneral MedicineMiddle Agedmedicine.diseaseEndocrinologyOncologyDysplasia030220 oncology & carcinogenesisMyelodysplastic Syndromesbiology.proteinParoxysmal nocturnal hemoglobinuriaElevated red cell adenosine deaminase activityFemaleHemoglobinbusinessTumori
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Effect of ouabain and furosemide on erythrocyte sodium and phosphate transport.

1981

The effects of ouabain and furosemide on the unidirectional efflux of sodium and phosphate ions were studied in freshly drawn human red blood cells (RBCs). In the presence of physiologic concentrations of sodium and potassium the rate of sodium efflux was reduced by 74% due to ouabain sensitivity. Furosemide (1.0 mmol/l) reduced ouabain-insensitive sodium transport rate by a further 50%. Thus, 13% of total sodium efflux was inhibited by furosemide when ouabain was present. In the absence of ouabain, however, furosemide inhibited 31% of total sodium transport, indicating that it also affected ouabain-sensitive sodium efflux. Phosphate transfer of RBCs was almost 1.0 mmol/l RBCs per hour. Ery…

AdultMaleCell Membrane PermeabilityErythrocytesPotassiumSodiumchemistry.chemical_elementBiological Transport ActiveOuabainIon ChannelsPhosphateschemistry.chemical_compoundHydrolysisFurosemidemedicineHumansPharmacology (medical)OuabainPharmacologyChromatographyChemistrySodiumFurosemidePhosphateBiochemistryPotassiumEffluxSodium-Potassium-Exchanging ATPaseAdenosine triphosphatemedicine.drugClinical pharmacology and therapeutics
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Platelet aggregation, ATP release and cytoplasmic Ca2+ movement: the effects of cloricromene.

1994

A placebo-controlled, double-blind, randomized, cross-over study was performed in 24 healthy volunteers. 12 volunteers received Cloricromene (100mg gastroresistant capsules twice a day) for 7 days, the other volunteers received identical placebo capsules. Subsequently, after a 7-day wash-out period, at day 15, each subject received the other treatment. Blood samples were taken on days 1 and 15 (1st day of each treatment) as well as on days 7 and 21 (7th day of each treatment) before the morning drug administration and 2 and 4 hours later. Platelet aggregation and ATP secretion were studied in whole blood (WB) using ADP and collagen as stimulating agents. Ca2+ fluxes were studied in aequorin…

AdultMaleCytoplasmAdolescentPlatelet Aggregationchemistry.chemical_elementAdministration OralPharmacologyCalciumPlaceboAdenosine TriphosphateDouble-Blind MethodOral administrationHumansPlateletSecretionWhole bloodCalcium metabolismCross-Over StudiesChemistryChromonarHematologyMiddle AgedCrossover studyAdenosine DiphosphateAnesthesiaCalciumFemaleCollagenPlatelet Aggregation InhibitorsThrombosis research
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KIAA0556 is a novel ciliary basal body component mutated in Joubert syndrome

2015

Background Joubert syndrome (JBTS) and related disorders are defined by cerebellar malformation (molar tooth sign), together with neurological symptoms of variable expressivity. The ciliary basis of Joubert syndrome related disorders frequently extends the phenotype to tissues such as the eye, kidney, skeleton and craniofacial structures. Results Using autozygome and exome analyses, we identified a null mutation in KIAA0556 in a multiplex consanguineous family with hallmark features of mild Joubert syndrome. Patient-derived fibroblasts displayed reduced ciliogenesis potential and abnormally elongated cilia. Investigation of disease pathophysiology revealed that Kiaa0556-/- null mice possess…

AdultMaleK04F10.2KIAA0556MicrotubuleMicrotubulesRetinaMiceJoubert syndromeCerebellumAnimalsHumansAbnormalities MultipleExomeCiliaEye AbnormalitiesSensory disorders Radboud Institute for Molecular Life Sciences [Radboudumc 12]Caenorhabditis elegansChildCells CulturedAdenosine TriphosphatasesADP-Ribosylation FactorsResearchBrainMetabolic Disorders Radboud Institute for Molecular Life Sciences [Radboudumc 6]Kidney Diseases CysticBasal BodiesPedigreeMice Inbred C57BLRenal disorders Radboud Institute for Molecular Life Sciences [Radboudumc 11]Basal bodyChild PreschoolMutationFemaleKataninMicrotubule-Associated ProteinsProtein BindingGenome Biology
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