Search results for "adenosine"

showing 10 items of 542 documents

Selected enzyme activities in mouse cardiac muscle during training and terminated training

1984

We studied the effects of running-training, heavy exercise and termination of training on the heart weight, the ratio heart to body weight and the cardiac muscle activities of actomyosin ATPase, citrate synthase, succinate dehydrogenase, cytochrome c oxidase, malate dehydrogenase, adenylate kinase and beta-glucuronidase with adult male NMRI-mice. Stable hypertrophy (6-7%), estimated by the ratio heart or ventricle weight to body weight, was achieved by 28 exercises and it was dependent on the running speed (20 vs. 25 m X min-1). The withdrawal of training for 5-61 days did not permanently decrease the heart weight or the heart to body weight ratio to the level of sedentary controls. The act…

Malemedicine.medical_specialtyTime FactorsHeart diseasePhysiologyATPasePhysical ExertionCardiomegalyMice Inbred StrainsCitrate (si)-SynthaseBiologyMalate dehydrogenaseMitochondria HeartMuscle hypertrophyMicePhysiology (medical)Internal medicinemedicineAnimalsCitrate synthaseGlucuronidaseAdenosine TriphosphatasesCardiac muscleActomyosinmedicine.diseaseMyocardial ContractionMitochondria MuscleEndocrinologymedicine.anatomical_structureVentriclebiology.proteinHeart enlargementCardiology and Cardiovascular MedicineOxidation-ReductionBasic Research in Cardiology
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Calcium and increase excitability promote tolerance against anoxia in hippocampal slices.

1999

We have previously demonstrated that anoxic preconditioning (APC) protects against a subsequent otherwise 'lethal' anoxic insult in hippocampal slices. Tested here are two hypotheses: (a) APC requires calcium to improve electrical recovery in hippocampal slices; and (b) mild excitation promotes preconditioning neuroprotection. Control hippocampal slices were given a single 'test' anoxic insult followed by reoxygenation. Experimental slices were preconditioned by three short anoxic insults of 1 min separated by 10 min of reoxygenation. At 30 min after the third 'conditioning' insult, slices underwent a 'test' anoxic insult [1 min of anoxic depolarization (AD)], and then slices were reoxygena…

Malemedicine.medical_specialtychemistry.chemical_elementHippocampal formationCalciumIn Vitro TechniquesNeuroprotectionHippocampusPotassium ChlorideAdenosine A1 receptorInternal medicineConditioning PsychologicalExtracellularmedicineAnimalsRats WistarHypoxiaMolecular BiologyEvoked PotentialsChemistryGeneral NeuroscienceCortical Spreading DepressionDepolarizationAdaptation PhysiologicalRatsElectrophysiologyEndocrinologyCortical spreading depressionAnesthesiaXanthinesExcitatory postsynaptic potentialCalciumNeurology (clinical)Developmental BiologyBrain research
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Plasmonic Nanosensors for the Label-Free Imaging of Dynamic Protein Patterns.

2020

We introduce a new approach to monitor the dynamics and spatial patterns of biological molecular assemblies. Our molecular imaging method relies on plasmonic gold nanoparticles as point-like detectors and requires no labeling of the molecules. We show spatial resolution of up to 5 μm and 30 ms temporal resolution, which is comparable to wide-field fluorescence microscopy, while requiring only readily available gold nanoparticles and a dark-field optical microscope. We demonstrate the method on MinDE proteins attaching to and detaching from lipid membranes of different composition for 24 h. We foresee our new imaging method as an indispensable tool in advanced molecular biology and biophysic…

Materials scienceCardiolipinsLipid BilayersMetal NanoparticlesNanotechnologyCell Cycle Proteins02 engineering and technology010402 general chemistry01 natural sciencesNanosensorFluorescence microscopeEscherichia coliGeneral Materials SciencePhysical and Theoretical ChemistryImage resolutionPlasmonAdenosine TriphosphatasesMicroscopyNanotubesEscherichia coli ProteinsPhosphatidylglycerols021001 nanoscience & nanotechnology0104 chemical sciencesMembraneColloidal goldTemporal resolutionPhosphatidylcholinesGoldMolecular imaging0210 nano-technologyThe journal of physical chemistry letters
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Cytochalasin B selectively releases ovalbumin mRNA precursors but not the mature ovalbumin mRNA from hen oviduct nuclear matrix

1987

Hen oviduct nuclear matrix-bound mature ovalbumin mRNA is released from the matrix in the presence of ATP, while the ovalbumin mRNA precursors remain bound to this structure. Detachment of the mature mRNA from the matrix by ATP as well as ATP-dependent efflux of mRNA from isolated nuclei were found to be inhibited by cytochalasin B. On the other hand, in the absence of ATP, cytochalasin B exclusively caused the release (and nucleocytoplasmic efflux) of the ovalbumin messenger precursors, but not of the mature mRNA. After cytochalasin B treatment, actin could be detected in the matrix supernatant. Phalloidin which stabilizes actin filaments did not cause RNA liberation in the absence of ATP,…

Mature messenger RNACytochalasin BOvalbuminPhalloidinePhalloidinOviductsmacromolecular substancesBiologyBiochemistrychemistry.chemical_compoundAdenosine TriphosphateRNA PrecursorsAnimalsCytochalasinRNA MessengerIntermediate filamentCytochalasin BCell NucleusMessenger RNAAntibodies MonoclonalNucleic Acid PrecursorsNuclear matrixMolecular biologyOvalbuminchemistrybiology.proteinFemaleChickensEuropean Journal of Biochemistry
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Mitochondrial dynamics in type 2 diabetes: Pathophysiological implications

2017

Mitochondria play a key role in maintaining cellular metabolic homeostasis. These organelles have a high plasticity and are involved in dynamic processes such as mitochondrial fusion and fission, mitophagy and mitochondrial biogenesis. Type 2 diabetes is characterised by mitochondrial dysfunction, high production of reactive oxygen species (ROS) and low levels of ATP. Mitochondrial fusion is modulated by different proteins, including mitofusin-1 (MFN1), mitofusin-2 (MFN2) and optic atrophy (OPA-1), while fission is controlled by mitochondrial fission 1 (FIS1), dynamin-related protein 1 (DRP1) and mitochondrial fission factor (MFF). PARKIN and (PTEN)-induced putative kinase 1 (PINK1) partici…

MiD51 mitochondrial dynamics proteins of 51 kDaΔΨm mitochondrial membrane potential0301 basic medicineMitochondrial fission factorClinical BiochemistryMitochondrial DegradationMFN2Review ArticleTXNIP thioredoxin interacting proteinMitochondrial DynamicsBiochemistryAdenosine TriphosphateGRP78 78 kDa glucose-regulated proteinMFF mitochondrial fission factorMFN2 mitofusin 2TRX2 thioredoxin 2Redox biologylcsh:QH301-705.5NF-κB nuclear factor kappa Blcsh:R5-920MitophagyType 2 diabetesDRP1 dynamin-related protein 1FIS1 fission protein 1BNIP3 BCL2/adenovirus E1B 19 kDa interacting protein 3MitochondriaOPA1 optic atrophy 1SIRT1/3 sirtuin 1/3Biochemistrymitochondrial fusionTGF-β1 transforming growth factor-β1Mitochondrial fissionOMM outer mitochondrial membranelcsh:Medicine (General)MiD49 mitochondrial dynamics proteins of 49Nox 4 NADPH oxidase-4IMM inner mitochondrial membraneFIS1ATF6 activating transcription factor 6PINK1mTOR mammalian target of rapamycinCHOP C/EBP homologous proteinBiologymdivi-1 mitochondrial division inhibitor-1Mitochondrial Proteins03 medical and health sciencesROS reactive oxygen speciessXBP1 spliced X-box binding protein 1UCP-1 uncoupling protein-1MFN1 mitofusin 1SOD superoxide dismutaseLC3 1 A/1B-light chain 3HumansPINK1 (PTEN)-induced putative kinase 1S3 15-OxospiramilactoneOrganic ChemistrymtDNA mitochondrial DNAAMPK AMP-activated protein kinase030104 developmental biologyDiabetes Mellitus Type 2Mitochondrial biogenesislcsh:Biology (General)Oxidative stressp38 MAPK p38 mitogen-activated protein kinasep62/SQSTM1 ubiquitin and sequestosome-1Reactive Oxygen SpeciesRedox Biology
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Suppression of inflammation by low-dose methotrexate is mediated by adenosine A2A receptor but not A3 receptor activation in thioglycollate-induced p…

2005

Prior studies demonstrate that adenosine, acting at one or more of its receptors, mediates the anti-inflammatory effects of methotrexate in animal models of both acute and chronic inflammation. Both adenosine A2A and A3 receptors contribute to the anti-inflammatory effects of methotrexate treatment in the air pouch model of inflammation, and the regulation of inflammation by these two receptors differs at the cellular level. Because different factors may regulate inflammation at different sites we examined the effect of low-dose weekly methotrexate treatment (0.75 mg/kg/week) in a model of acute peritoneal inflammation in adenosine A2A receptor knockout mice and A3 receptor knockout mice an…

Mice KnockoutAdenosineDose-Response Relationship DrugReceptor Adenosine A2AReceptor Adenosine A3Anti-Inflammatory AgentsExudates and TransudatesPeritonitisDexamethasoneInterleukin-10MiceMethotrexateThioglycolatesAnimalsPeritoneumGlucocorticoidsResearch ArticleArthritis researchtherapy
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Use of ATP bioluminescence for assessing the cleanliness of hospital surfaces: A review of the published literature (1990–2012)

2014

Summary: Hospital cleanliness tends to be considered by patients and the public as an important indicator of the general quality of healthcare. Tests for detecting the presence of adenosine triphosphate (ATP) as a proxy of microbial contamination are increasing in popularity, and several studies have been conducted on this topic in the last few decades. The aim of the present study was to review the published literature on this topic and summarize and discuss the available results. The review focused on relevant English-language articles that were identified through searches of two databases [PubMed and Scopus (1990–2012)] by using the keywords “ATP”, “bioluminescence”, “hospital”, and “sur…

Microbiological Techniquesmedicine.medical_specialtyReviewMicrobial contaminationATP bioluminescence; Hospital; Review; SurfacesSettore MED/42 - Igiene Generale E ApplicataChemistry Techniques Analyticallcsh:Infectious and parasitic diseasesHospitalAdenosine TriphosphateEnvironmental MicrobiologyHumansMedicinelcsh:RC109-216International levelbusiness.industrylcsh:Public aspects of medicinePublic Health Environmental and Occupational HealthObjective methodlcsh:RA1-1270Housekeeping HospitalGeneral MedicineAtp bioluminescenceSurgeryATP bioluminescenceDisinfectionSurfacesInfectious DiseasesLuminescent MeasurementsEmergency medicinebusinessJournal of Infection and Public Health
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Efavirenz alters mitochondrial respiratory function in cultured neuron and glial cell lines.

2015

Abstract Background The NNRTI efavirenz is among the most widely employed antiretroviral drugs. Although it is considered safe, efavirenz has been linked with several adverse effects including neurological manifestations, which appear in the majority of the patients on efavirenz-containing regimens. The molecular mechanisms responsible for these manifestations are not understood, but mounting evidence points to altered brain bioenergetics. Methods We evaluated the effect of short-term efavirenz treatment on the mitochondrial respiratory function of cultured glioblastoma and differentiated neuroblastoma cell lines using a Seahorse Extracellular Flux Analyzer. Results Incubation with efaviren…

Microbiology (medical)CyclopropanesCell typeEfavirenzCell RespirationBiologyPharmacologyMitochondrionCell Linechemistry.chemical_compoundAdenosine TriphosphateRespirationExtracellularmedicineHumansPharmacology (medical)Respiratory functionPharmacologyNeuronsNeurotoxicityvirus diseasesmedicine.diseaseVirologyBenzoxazinesMitochondriaInfectious Diseasesmedicine.anatomical_structurechemistryAnti-Retroviral AgentsAlkynesNeurogliaEnergy MetabolismNeurogliaThe Journal of antimicrobial chemotherapy
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Mutations in KATNB1 Cause Complex Cerebral Malformations by Disrupting Asymmetrically Dividing Neural Progenitors

2014

SummaryExome sequencing analysis of over 2,000 children with complex malformations of cortical development identified five independent (four homozygous and one compound heterozygous) deleterious mutations in KATNB1, encoding the regulatory subunit of the microtubule-severing enzyme Katanin. Mitotic spindle formation is defective in patient-derived fibroblasts, a consequence of disrupted interactions of mutant KATNB1 with KATNA1, the catalytic subunit of Katanin, and other microtubule-associated proteins. Loss of KATNB1 orthologs in zebrafish (katnb1) and flies (kat80) results in microcephaly, recapitulating the human phenotype. In the developing Drosophila optic lobe, kat80 loss specificall…

Microtubule-associated proteinNeurogenesisNeuroscience(all)Cell CountKataninSpindle ApparatusBiologymedicine.disease_causeArticleMice03 medical and health sciences0302 clinical medicineNeural Stem CellsNeuroblastmedicineAnimalsDrosophila ProteinsHumansProgenitor cellZebrafishMitosisZebrafishAdenosine TriphosphatasesMutationGeneral NeuroscienceOptic Lobe NonmammalianBrainDendritesbiology.organism_classificationSpindle apparatusmedicine.anatomical_structureCentrosome030220 oncology & carcinogenesisCerebral malformationsMutationMicrocephalybiology.proteinDrosophilaNeuronKataninMicrotubule-Associated ProteinsNeuroscienceCell Division030217 neurology & neurosurgery
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Mitochondrial involvement in non-alcoholic steatohepatitis

2007

Non-alcoholic steatohepatitis (NASH) is an increasing recognized condition that may progress to end-stage liver disease. There are consistent evidences that mitochondrial dysfunction plays a central role in NASH whatever its origin. Mitochondria are the key controller of fatty acids removal and this is part of an intensive gene program that modifies hepatocytes to counteract the excessive fat storage. Mitochondrial dysfunction participates at different levels in NASH pathogenesis since it impairs fatty liver homeostasis and induces overproduction of ROS that in turn trigger lipid peroxidation, cytokines release and cell death. In this review we briefly recall the role of mitochondria in fat…

Mitochondrial DNAmedicine.medical_specialtyClinical BiochemistryBiologyMitochondrionModels BiologicalBiochemistryEnergy homeostasisAdenosine TriphosphateInternal medicinemedicineAnimalsHumansMolecular BiologyFatty liverGeneral MedicineTFAMLipid Metabolismmedicine.diseaseMitochondriaFatty LiverEndocrinologyMitochondrial respiratory chainMolecular MedicineSteatohepatitisSteatosisReactive Oxygen SpeciesMolecular Aspects of Medicine
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