Search results for "adipocyte"

showing 10 items of 89 documents

Time course of changes in serum glucose, insulin, lipids and tissue lipase activities in macrosomic offspring of rats with streptozotocin-induced dia…

1999

The aim of this investigation was to determine the time course of changes in serum glucose, insulin and lipid levels, as well as lipid and protein content and lipolytic activities in insulin target organs (liver, adipose tissue and muscle), in macrosomic offspring of streptozotocin-induced mildly hyperglycaemic rats. Food intake and nutritional efficiency were also evaluated. Mild hyperglycaemia in pregnant rats was induced by intraperitoneal injection of streptozotocin (40 mg/kg body weight) on day 5 of gestation. Control pregnant rats were injected with citrate buffer. At birth, macrosomic pups (birth weight > 1.7 S.D. greater than the mean value for the control pups) had higher serum …

Blood GlucoseMalemedicine.medical_specialtyTime Factorsmedicine.medical_treatmentLipolysisAdipose tissueBiologyDiabetes Mellitus ExperimentalFetal Macrosomiachemistry.chemical_compoundEatingInsulin resistanceSex FactorsDiabetes mellitusAdipocyteInternal medicinemedicineFetal macrosomiaAnimalsInsulinObesityRats WistarMuscle SkeletalInsulinLipid metabolismGeneral MedicineLipasemedicine.diseaseStreptozotocinLipidsRatsEndocrinologychemistryAdipose TissueLiverFemalemedicine.drugClinical science (London, England : 1979)
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Myoglobin, expressed in brown adipose tissue of mice, regulates the content and activity of mitochondria and lipid droplets

2021

Abstract The identification of novel physiological regulators that stimulate energy expenditure through brown adipose tissue (BAT) activity in substrate catalysis is of utmost importance to understand and treat metabolic diseases. Myoglobin (MB), known to store or transport oxygen in heart and skeletal muscles, has recently been found to bind fatty acids with physiological constants in its oxygenated form (i.e., MBO2). Here, we investigated the in vivo effect of MB expression on BAT activity. In particular, we studied mitochondrial function and lipid metabolism as essential determinants of energy expenditure in this tissue. We show in a MB-null (MBko) mouse model that MB expression in BAT i…

PalmitatesOxidative phosphorylationMitochondrion1307 Cell BiologyMiceAdipose Tissue BrownLipid dropletBrown adipose tissueRespiration1312 Molecular BiologymedicineAnimalsHumansPPAR alpha11434 Center for Clinical StudiesMuscle SkeletalMolecular BiologyUncoupling Protein 1Mice KnockoutMyoglobinChemistryProteinsThermogenesisLipid metabolismLipid DropletsCell BiologyMetabolism10081 Institute of Veterinary PhysiologyPeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaMitochondriaCell biologyOxygenDisease Models AnimalAdipocytes Brownmedicine.anatomical_structure10076 Center for Integrative Human Physiology570 Life sciences; biologyApoptosis Regulatory ProteinsEnergy MetabolismThermogenesisBiochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids
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Multipotential nestin and Isl-1 positive mesenchymal stem cells isolated from human pancreatic islets.

2006

Mesenchymal cells in the developing pancreas express the neural stem cell marker nestin and the transcription factor islet-1 (Isl-1). Using defined culture conditions we isolated on a single cell basis nestin producing cells from human pancreatic islets. These cells were immortalized with lentiviral vectors coding for telomerase and mBmi. They are positive for Isl-1 and nestin and have the potential to adopt a pancreatic endocrine phenotype with expression of critical transcription factors including Ipf-1, Isl-1, Ngn-3, Pax4, Pax6, Nkx2.2, and Nkx6.1 as well as the islet hormones insulin, glucagon, and somatostatin. In addition, they can be differentiated into human albumin producing cells …

endocrine systemLIM-Homeodomain ProteinsBiophysicsCell Culture TechniquesNerve Tissue ProteinsBiologyBiochemistryNestinIslets of LangerhansIntermediate Filament ProteinsNeurosphereAlbuminsmedicineAdipocytesATP Binding Cassette Transporter Subfamily G Member 2HumansMolecular BiologyStem cell transplantation for articular cartilage repairHomeodomain ProteinsNeuronsOsteoblastsPancreatic isletsMesenchymal stem cellLentivirusNuclear ProteinsCell DifferentiationMesenchymal Stem CellsCell BiologyNestinNeural stem cellNeoplasm Proteinsmedicine.anatomical_structureHomeobox Protein Nkx-2.2Cancer researchPAX4ATP-Binding Cassette TransportersPancreasTranscription FactorsBiochemical and biophysical research communications
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Immune Cell Toll-like Receptor 4 Mediates the Development of Obesity- and Endotoxemia-Associated Adipose Tissue Fibrosis

2014

International audience; Adipose tissue fibrosis development blocks adipocyte hypertrophy and favors ectopic lipid accumulation. Here, we show that adipose tissue fibrosis is associated with obesity and insulin resistance in humans and mice. Kinetic studies in C3H mice fed a high-fat diet show activation of macrophages and progression of fibrosis along with adipocyte metabolic dysfunction and death. Adipose tissue fibrosis is attenuated by macrophage depletion. Impairment of Toll-like receptor 4 signaling protects mice from obesity-induced fibrosis. The presence of a functional Toll-like receptor 4 on adipose tissue hematopoietic cells is necessary for the initiation of adipose tissue fibros…

LipopolysaccharidesMESH: Signal TransductionMESH: InflammationMESH : Toll-Like Receptor 4Adipose tissueMESH : AdipocytesMESH : LipopolysaccharidesMicechemistry.chemical_compoundFibrosisAdipocyteAdipocytes[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyMESH: ObesityMESH: Animalslcsh:QH301-705.5Mice Inbred C3HToll-like receptorMESH : Diet High-FatMESH: Toll-Like Receptor 43. Good healthMESH: Insulin ResistanceAdipose TissueMESH: FibrosisMESH : Fibrosis[SDV.IMM]Life Sciences [q-bio]/ImmunologyMESH : ObesityMESH : Insulin ResistanceMESH: Adipose TissueSignal Transductionmedicine.medical_specialty[SDV.IMM] Life Sciences [q-bio]/ImmunologyAdipose tissue macrophagesBiologyDiet High-FatMESH : Adipose TissueGeneral Biochemistry Genetics and Molecular BiologyImmune systemMESH : Mice Inbred C3HInternal medicineMESH : MicemedicineAnimalsHumansObesityMESH: Mice Inbred C3HMESH: MiceMESH: AdipocytesInflammationMESH : Signal TransductionMESH : InflammationMESH: HumansMESH : EndotoxemiaMESH : Humans3T3-L1medicine.diseaseFibrosisMESH : Disease Models AnimalEndotoxemiaToll-Like Receptor 4Disease Models AnimalMESH: Diet High-FatEndocrinologylcsh:Biology (General)chemistryMESH: EndotoxemiaMESH : AnimalsInsulin ResistanceMESH: Disease Models AnimalMESH: LipopolysaccharidesAdipocyte hypertrophyCell Reports
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NT3/TrkC pathway modulates the expression of UCP-1 and adipocyte size in human and murine adipose tissue

2020

ABSTRACTNT3, through activation of its tropomyosin-related kinase receptor C (TrkC), modulates neuronal survival and neural stem cell differentiation. It is widely distributed in peripheral tissues (specially vessels and pancreas) and this ubiquitous pattern suggests a role for NT3, outside the nervous system and related to metabolic functions. The presence of the NT3/TrkC pathway in the adipose tissue (AT) has never been investigated. Present work studies in human and murine adipose tissue (AT) the presence of elements of the NT3/TrkC pathway and its role on lipolysis and adipocyte differentiation. qRT-PCR and immunoblot indicate that NT3 was present in human retroperitoneal AT and decreas…

Genetically modified mousechemistry.chemical_compoundanimal structureschemistryAdipocyteembryonic structuresLipolysisAdipose tissueReceptorThermogenesisTropomyosin receptor kinase CNeural stem cellCell biology
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The human liver fatty acid binding protein (FABP1) gene is activated by FOXA1 and PPARα; and repressed by C/EBPα: Implications in FABP1 down-regulati…

2013

Liver fatty acid binding protein (FABP1) prevents lipotoxicity of free fatty acids and regulates fatty acid trafficking and partition. Our objective is to investigate the transcription factors controlling the human FABP1 gene and their regulation in nonalcoholic fatty liver disease (NAFLD). Adenovirus-mediated expression of multiple transcription factors in HepG2 cells and cultured human hepatocytes demonstrated that FOXA1 and PPARα are among the most effective activators of human FABP1, whereas C/EBPα is a major dominant repressor. Moreover, FOXA1 and PPARα induced re-distribution of FABP1 protein and increased cytoplasmic expression. Reporter assays demonstrated that the major basal activ…

Hepatocyte Nuclear Factor 3-alphaMaleRepressorBiologyFatty Acid-Binding ProteinsFatty acid-binding proteinMiceTransactivationNon-alcoholic Fatty Liver DiseaseNonalcoholic fatty liver diseaseCCAAT-Enhancer-Binding Protein-alphamedicineAnimalsHumansPPAR alphaadipocyte protein 2Molecular BiologyTranscription factorCells Culturedchemistry.chemical_classificationFatty acidHep G2 CellsCell Biologymedicine.diseaseMolecular biologyFatty LiverMice Inbred C57BLLipotoxicitychemistrybiology.proteinProtein BindingBiochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids
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Morphological description of hypothetical cellulite starting unit. The first step to explain cellulite development

2017

Introduction: Cellulite is a complex pathology that affects about 85% of women. The origin of cellulite is poorly understood, even if some studies describe different hypotheses based on the observation of subcutaneous architecture, vascular and lymphatic patterns. Material and methods: in this work an ultra-structural description of cellulite specimens, harvested from fifteen healthy women affected by first degree cellulite, was provided. Specimens were harvested from trochanter, after local anaesthesia using punch biopsy. Samples were fixated, immediately after biopsy, in glutaraldehyde 2% and processed for electron microscopy. Results: with scanning and transmission electron microscopy it…

Settore MED/18 - Chirurgia GeneraleCellulite mature adipocyte sweat gland TEM SEM cellulite starting unit
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Relationship between endocrine disruptors and obesity with a focus on bisphenol A: a narrative review

2021

International audience; Introduction: Scientific data suggest that early exposure to endocrine-disrupting chemicals (EDCs) affect -repro, -neuro, -metabolic systems, to which are added other notions such as mixtures, window and duration of exposure, trans-generational effects, and epigenetic mechanisms. Methods: In the present narrative review, we studied the relationship between exposure to EDCs with the appearance and development of obesity. Results: Exposure to EDCs like Bisphenol A during the early stages of development has been shown to lead to weight gain and obesity. EDCs can interfere with endocrine signaling, affect adipocytes differentiation and endocrine function and disrupt meta…

Medicine (General)obesityFuture studiesQH301-705.5bisphenol APharmaceutical Science030209 endocrinology & metabolismReviewBiologyBioinformaticsadipocyteGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences0302 clinical medicineR5-920medicineEndocrine systemmetabolic disordersEpigeneticsBiology (General)030304 developmental biology0303 health sciencesfat tissuesLow doseGeneral Medicine[SDV.SP]Life Sciences [q-bio]/Pharmaceutical sciencesmedicine.diseaseObesity3. Good healthperoxisome proliferator-activated receptor-γNarrative review
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Enterobacter cloacae administration induces hepatic damage and subcutaneous fat accumulation in high-fat diet fed mice.

2018

Accumulating evidence indicates that gut microbiota plays a significant role in obesity, insulin resistance and associated liver disorders. Family Enterobacteriaceae and especially Enterobacter cloacae strain B29 have been previously linked to obesity and hepatic damage. The underlying mechanisms, however, remain unclear. Therefore, we comprehensively examined the effects of E. cloacae subsp. cloacae (ATCC® 13047™) administration on host metabolism of mice fed with high-fat diet (HFD). C57BL/6N mice were randomly divided into HFD control, chow control, and E. cloacae treatment groups. The E. cloacae treatment group received live bacterial cells in PBS intragastrically twice a week, every ot…

rasvahapotPathology and Laboratory Medicinerasvat (orgaaniset yhdisteet)ruokavaliotBiochemistryMiceAnimal CellsFibrosislcsh:ScienceImmune ResponseConnective Tissue CellsChemical Reactionsta3141ta3142Lipids3. Good healthPhysical sciencesAdiponectinCellular Typesmedicine.medical_specialtyfatsLipolysisImmunologySubcutaneous FatrasvakudoksetMonomers (Chemistry)glycerolDiet High-Fatta311103 medical and health sciencesSigns and SymptomsEnterobacter cloacaeLipolysisPolymer chemistrylcsh:RBiology and Life SciencesHypertrophymedicine.diseaseReceptor InsulinMice Inbred C57BLBiological Tissue030104 developmental biologyEndocrinologylihavuuslcsh:QGlycerol0301 basic medicinePhysiologyLiver cytologysuolistomikrobistolcsh:MedicineAdipose tissueGut floraMedicine and Health SciencesAdipocytesenterobakteerit2. Zero hungerrasvatMultidisciplinarygastrointestinal microbiotatulehdusbiologyHydrolysisadipose tissueChemistryPhysiological ParametersLiverConnective Tissueembryonic structuresFemaleAnatomymedicine.symptomResearch Articleanimal structuresadipocytesInflammationInsulin resistanceEnterobacteriaceaeDiagnostic MedicineInternal medicinemedicineAnimalsObesityTriglyceridesNutritionurogenital systembusiness.industryBody WeightCell Biologybiology.organism_classificationDietToll-Like Receptor 5Gene Expression RegulationinflammationlipolysisdietbusinessEnterobacter cloacaePLoS ONE
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In vivo triglyceride synthesis in subcutaneous adipose tissue of humans correlates with plasma HDL parameters

2016

Backgrounds and aims: Low concentrations of plasma HDL-C are associated with the development of atherosclerotic cardiovascular diseases and type 2 diabetes. Here we aimed to explore the relationship between the in vivo fractional synthesis of triglycerides (fTG) in subcutaneous (s.q.) abdominal adipose tissue (AT), HDL-C concentrations and HDL particle size composition in non-diabetic humans. Methods: The fTG in s.q. abdominal AT was measured in 16 non-diabetic volunteers (7 women, 9 men; Age: 49 ± 20 years; BMI: 31 ± 5 kg/m; Fasting Plasma Glucose: 90 ± 10 mg/dl) after 2H2O labeling. HDL-C concentration and subclasses, large (L-HDL), intermediate (I-HDL) and small (S-HDL) were measured. Re…

Blood GlucoseMale0301 basic medicinemedicine.medical_specialtyAdipocyte; Atheroprotective; Gender-related; HDL-C metabolism; Mass spectrometry; Stable isotope tracer kinetics; Triglycerides; Cardiology and Cardiovascular MedicineAdipose tissueWhite adipose tissueType 2 diabetes030204 cardiovascular system & hematologyTriglycerideArticleAtheroprotective03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInsulin resistanceIn vivoInternal medicineAdipocytemedicineHumansObesityTriglyceridesAgedHDL-C metabolismAdipocyteStable isotope tracer kineticMass spectrometrybusiness.industryLipogenesisCholesterol HDLnutritional and metabolic diseasesMiddle AgedAtherosclerosismedicine.diseaseLipidsObesitySubcutaneous Fat AbdominalGender-related030104 developmental biologyEndocrinologychemistryLipogenesisFemalelipids (amino acids peptides and proteins)Insulin ResistanceCardiology and Cardiovascular MedicinebusinessAtherosclerosis
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