Search results for "adoptive"

showing 10 items of 182 documents

Prophylactic transfer of CD8-depleted donor lymphocytes after T-cell–depleted reduced-intensity transplantation

2006

AbstractAllogeneic hematopoietic stem cell transplantation (SCT) regimens incorporating the lymphocytotoxic CD52 antibody alemtuzumab demonstrate efficient engraftment and reduced graft-versus-host disease (GVHD). However, these protocols substantially impair posttransplantation antiviral and antitumor immunity. To accelerate immune reconstitution after alemtuzumab-based reduced-intensity SCT, we administered prophylactic CD8-depleted donor lymphocyte infusions (DLIs) starting on days 60 and 120 after transplantation. DLIs were processed in an immunomagnetic good manufacturing practice depletion procedure resulting in a 2.5- to 6-log reduction in CD8 T cells. Of 23 high-risk patients with h…

AdultMaleTransplantation ConditioningCD52Antibodies Neoplasmmedicine.medical_treatmentT cellImmunologyGraft vs Host DiseaseHematopoietic stem cell transplantationCD8-Positive T-LymphocytesAntibodies Monoclonal HumanizedImmunotherapy AdoptiveBiochemistryLymphocyte DepletionHLA AntigensmedicineHumansCytotoxic T cellAlemtuzumabPeripheral Blood Stem Cell TransplantationImmunomagnetic Separationbusiness.industryGraft SurvivalAntibodies MonoclonalCell BiologyHematologyMiddle AgedDonor Lymphocytesmedicine.diseaseTransplantationTreatment Outcomesurgical procedures operativeGraft-versus-host diseasemedicine.anatomical_structureHematologic NeoplasmsLangerhans CellsImmunologyAlemtuzumabFemaleK562 CellsbusinessFollow-Up Studiesmedicine.drugBlood
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Elevated levels of serum-soluble triggering receptor expressed on myeloid cells-1 in patients with IBD do not correlate with intestinal TREM-1 mRNA e…

2012

BACKGROUND AIMS Triggering receptor expressed on myeloid cells 1 (TREM 1) is a potent amplifier of pro inflammatory responses. We have previously demonstrated a substantial increase in TREM 1 expressing macrophages in the inflamed intestinal mucosa of patients with inflammatory bowel diseases (IBD). TREM 1 is also produced as a soluble receptor (sTREM 1). Here we aimed to determine whether serum sTREM 1 could be used as a surrogate marker of disease activity in patients with IBD. METHODS Intestinal biopsies and concurrently collected sera from patients with Crohn's disease (CD) and Ulcerative colitis (UC) enrolled in the Swiss IBD cohort study were analyzed for intestinal TREM 1 mRNA and se…

AdultMalemedicine.medical_specialtyMyeloidColonGastroenterologyInflammatory bowel diseaseEndoscopy GastrointestinalStatistics Nonparametric03 medical and health sciencesMice0302 clinical medicineIntestinal mucosaCrohn DiseaseIleumInternal medicinemedicineAnimalsHumansRNA MessengerColitisReceptors ImmunologicReceptor030304 developmental biology0303 health sciencesCrohn's diseaseMessenger RNAMembrane Glycoproteinsbusiness.industryGastroenterologyGeneral MedicineMiddle Agedmedicine.diseaseUlcerative colitisAdoptive Transferdigestive system diseasesTriggering Receptor Expressed on Myeloid Cells-13. Good healthmedicine.anatomical_structureROC CurveArea Under CurveImmunologyColitis UlcerativeFemalebusinessBiomarkers030215 immunologyJournal of Crohn's & colitis
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Regulation of IgE production and airway reactivity by CD4(-)CD8(-) regulatory T cells

2015

The mechanisms of tolerance induction occurring in the course of allergen-specific immunotherapy have not been elucidated in full detail. Our study aimed to characterize high zone tolerance in mouse models of type I allergy and of allergic airway inflammation induced by subcutaneous sensitization of mice with high doses of the model allergen ovalbumin (OVA) without the use of adjuvant. Mice were immunized by subcutaneous injection of high doses (HD) of OVA or, for comparison, low doses (LD) of OVA in saline. HD-mice showed lower specific IgE, but augmented IgG in sera than LD-mice. Pre-treatment of mice with HD-OVA antigen-specifically inhibited IgE production subsequently induced by LD-OVA…

AllergyAdoptive cell transferAllergyOvalbuminImmunologyGene ExpressionCD4-CD8-double-negative T cellsLymphocyte ActivationImmunoglobulin EAirway hyperreactivityT-Lymphocytes RegulatoryImmunophenotypingMouse modelImmunomodulationMiceSubcutaneous injectionAntibody SpecificityT-Lymphocyte SubsetsRespiratory HypersensitivitymedicineAnimalsImmunology and AllergyAntigen doseSensitizationbiologymedicine.diagnostic_testbusiness.industryReceptors Antigen T-Cell gamma-deltaHematologyImmunoglobulin Erespiratory systemmedicine.diseaseAdoptive TransferTolerance inductionOvalbuminImmunoglobulin (Ig)EBronchoalveolar lavagemedicine.anatomical_structureAntibody FormationImmunologybiology.proteinCytokinesFemaleImmunizationbusinessBronchoalveolar Lavage Fluid
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Mast Cells Induce Migration of Dendritic Cells in a Murine Model of Acute Allergic Airway Disease

2009

<i>Background: </i>The migration of dendritic cells (DCs) from the lungs to the regional lymph nodes is necessary for the development of allergic airway disease. Following activation, mast cells release a variety of stored or de novo-produced inflammatory mediators, several of them being capable of activating DCs. In this study, the role of mast cells on DC migration from the lungs to the thoracic lymph nodes was investigated in sensitized mice. <i>Methods:</i> Mast cell-deficient mice (Kit<sup>W-sh/W-sh</sup>) and their wild-type counterparts were sensitized intraperitoneally with ovalbumine (OVA) in saline and challenged by a single intranasal administr…

AllergyAdoptive cell transferOvalbuminImmunologyInflammationCell SeparationMiceAnimalsImmunology and AllergyMedicineMast CellsAntigen-presenting cellFollicular dendritic cellsbusiness.industryCell migrationDendritic CellsGeneral MedicineDendritic cellAllergensrespiratory systemFlow Cytometrymedicine.diseaseMast cellAdoptive Transferrespiratory tract diseasesChemotaxis Leukocytemedicine.anatomical_structureImmunologyBronchial Hyperreactivitymedicine.symptombusinessBronchoalveolar Lavage FluidInternational Archives of Allergy and Immunology
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Platelet, Not Endothelial, P-Selectin Expression Contributes to Generation of Immunity in Cutaneous Contact Hypersensitivity

2010

Leukocyte extravasation is a prerequisite for host defense and autoimmunity alike. Detailed understanding of the tightly controlled and overlapping sequences of leukocyte extravasation might aid development of novel therapeutic strategies. Leukocyte extravasation is initiated by interaction of selectins with appropriate carbohydrate ligands. Lack of P-selectin expression leads to decreased contact hypersensitivity responses. Yet, it remains unclear if this is due to inhibition of leukocyte extravasation to the skin or due to interference with initial immune activation in lymph nodes. In line with previous data, we here report a decreased contact hypersensitivity response, induced by 2,4,-di…

Blood PlateletsAdoptive cell transferP-selectinInflammationDermatitis ContactPathology and Forensic MedicineMiceImmunityMedicineAnimalsBlood Platelets/*metabolismCell ShapeSkinInflammationbusiness.industryImmunityEndothelial CellsSkin/immunology/*pathologyDermatitis Contact/complications/*immunology/*pathologyLeukocyte extravasationAdoptive TransferInflammation/complications/immunology/pathologyEndothelial stem cellMice Inbred C57BLP-Selectinmedicine.anatomical_structureImmunologyEndothelial Cells/*metabolismDinitrofluorobenzeneBone marrowmedicine.symptomImmunity/*immunologybusinessSelectinP-Selectin/*metabolismRegular Articles
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Exclusive transduction of human CD4+ T Cells upon systemic delivery of CD4-targeted lentiviral vectors

2015

Abstract Playing a central role in both innate and adaptive immunity, CD4+ T cells are a key target for genetic modifications in basic research and immunotherapy. In this article, we describe novel lentiviral vectors (CD4-LV) that have been rendered selective for human or simian CD4+ cells by surface engineering. When applied to PBMCs, CD4-LV transduced CD4+ but not CD4− cells. Notably, also unstimulated T cells were stably genetically modified. Upon systemic or intrasplenic administration into mice reconstituted with human PBMCs or hematopoietic stem cells, reporter gene expression was predominantly detected in lymphoid organs. Evaluation of GFP expression in organ-derived cells and blood …

CD4-Positive T-Lymphocytes10028 Institute of Medical VirologyCell TransplantationGenetic enhancementAdoptiveMice SCIDImmunotherapy AdoptiveInterleukin 21MiceMice Inbred NODTransduction GeneticBone MarrowLeukocytesImmunology and AllergyCytotoxic T cellIL-2 receptorLuciferasesCells CulturedMice KnockoutHeterologousTumorCulturedForkhead Transcription FactorsAcquired immune systemFlow Cytometry3. Good healthCell biologymedicine.anatomical_structure[SDV.MP.VIR]Life Sciences [q-bio]/Microbiology and Parasitology/Virology2723 Immunology and Allergy[SDV.IMM]Life Sciences [q-bio]/ImmunologyImmunotherapyRegulatory T cellCellsKnockoutTransplantation HeterologousImmunologyMononuclearGenetic VectorsGreen Fluorescent Proteins610 Medicine & healthStreptamerThymus GlandBiologySCIDCell LineTransductionGeneticCell Line TumormedicineAnimalsHumansInterleukin 3Transplantation2403 ImmunologyLentivirusGenetic TherapyMolecular biology[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/BacteriologyHEK293 CellsLeukocytes MononuclearInbred NOD570 Life sciences; biologySpleen
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Low zone tolerance induced by systemic application of allergens inhibits TC1-mediated skin inflammation

2005

Background The induction of tolerance may be a promising target of strategies aimed at preventing harmful allergic diseases. Low zone tolerance (LZT), induced by epicutaneous application of low doses of contact allergens, inhibits the development of T C 1-mediated contact hypersensitivity (CHS). Objective We evaluated the effect of systemic (oral, intravenous) administration of low amounts of haptens on specific immune reactions and tolerance induction. Methods By using the mouse model of LZT, we analyzed immune reactions in vivo (skin inflammation) and T-cell responses in vitro after oral, intravenous, or epicutaneous application of low amounts of the contact allergen 2,4,6-trinitro-1-chlo…

CD4-Positive T-LymphocytesAdoptive cell transferAllergymedicine.medical_treatmentImmunologyDose-Response Relationship ImmunologicAdministration OralInflammationPicryl ChlorideAdministration CutaneousDermatitis ContactT-Lymphocytes RegulatoryImmune toleranceMiceImmune systemImmune TolerancemedicineAnimalsImmunology and AllergyMice KnockoutChemistryCell DifferentiationImmunotherapyAllergensmedicine.diseaseMice Inbred C57BLTolerance inductionTrinitrobenzenesulfonic AcidOrgan SpecificityInjections IntravenousImmunologymedicine.symptomCD8T-Lymphocytes CytotoxicJournal of Allergy and Clinical Immunology
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Critical role of IL-10 in the induction of low zone tolerance to contact allergens

2003

The development and mechanisms of tolerance to allergens are poorly understood. Using the murine low zone tolerance (LZT) model, where contact hypersensitivity (CHS) is prevented by repeated topical low-dose applications of contact allergens, we show that LZT induction is IL-10 dependent. IL-10 is required for the generation of LZT effector cells, that is, CD8+ regulatory T cells. Only T cells from tolerized IL-10+/+ mice or IL-10-/- mice reconstituted with IL-10 during LZT induction adoptively transferred LZT to naive mice and prevented CHS, whereas T cells from IL-10-/- mice failed to do so. The IL-10 required for normal LZT development is derived from lymph node CD4+ T cells, the only sk…

CD4-Positive T-LymphocytesAdoptive cell transferPopulationPicryl ChlorideBiologyCD8-Positive T-LymphocytesDermatitis ContactArticleImmune tolerancePicryl chloridechemistry.chemical_compoundMiceImmune systemmedicineImmune ToleranceAnimalseducationLymph nodeMice Knockouteducation.field_of_studyGeneral MedicineAllergensAdoptive TransferInterleukin-10Mice Inbred C57BLInterleukin 10medicine.anatomical_structurechemistryImmunologyCD8
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T-cell receptor transfer into human T cells with ecotropic retroviral vectors

2014

Adoptive T-cell transfer for cancer immunotherapy requires genetic modification of T cells with recombinant T-cell receptors (TCRs). Amphotropic retroviral vectors (RVs) used for TCR transduction for this purpose are considered safe in principle. Despite this, TCR-coding and packaging vectors could theoretically recombine to produce replication competent vectors (RCVs), and transduced T-cell preparations must be proven free of RCV. To eliminate the need for RCV testing, we transduced human T cells with ecotropic RVs so potential RCV would be non-infectious for human cells. We show that transfection of synthetic messenger RNA encoding murine cationic amino-acid transporter 1 (mCAT-1), the re…

CD4-Positive T-LymphocytesAdoptive cell transfermedicine.medical_treatmentGenetic enhancementGenetic VectorsReceptors Antigen T-CellCD8-Positive T-LymphocytesBiologyImmunotherapy AdoptiveJurkat cellsVesicular stomatitis Indiana virusCell LineJurkat CellsMiceTransduction (genetics)Viral Envelope ProteinsCancer immunotherapyTransduction GeneticGeneticsmedicineAnimalsHumansRNA MessengerMolecular BiologyCationic Amino Acid Transporter 1Membrane GlycoproteinsHEK 293 cellsT-cell receptorTransfectionAdoptive TransferVirologyElectroporationHEK293 CellsRetroviridaeLeukemia Virus Gibbon ApeMolecular MedicinePlasmidsGene Therapy
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Adoptive transfer of ex vivo expanded SARS‐CoV‐2‐specific cytotoxic lymphocytes: A viable strategy for COVID‐19 immunosuppressed patients?

2021

Cellular and humoral response to acute respiratory syndrome coronavirus 2 (SARS‐CoV‐2) infections is on focus of research. We evaluate herein the feasibility of expanding virus‐specific T cells (VST) against SARS‐CoV‐2 ex vivo through a standard protocol proven effective for other viruses. The experiment was performed in three different donors' scenarios: (a) SARS‐CoV‐2 asymptomatic infection/negative serology, (b) SARS‐CoV‐2 symptomatic infection/positive serology, and (c) no history of SARS‐CoV‐2 infection/negative serology. We were able to obtain an expanded VST product from donors 1 and 2 (1.6x and 1.8x increase of baseline VST count, respectively) consisting in CD3 + cells (80.3% and 6…

CD4-Positive T-LymphocytesAdoptive cell transferviruses030230 surgerymedicine.disease_causevirus-specific T cellsAsymptomaticSARS‐CoV‐2Serology03 medical and health sciences0302 clinical medicineCOVID‐19medicineCytotoxic T cellHumansRespiratory systemthird‐party donorsCoronavirusTransplantationbusiness.industrySARS-CoV-2COVID-19Original Articlesvirus‐specific T cellsAdoptive Transferlymphocyte expansionrespiratory virusInfectious DiseasesImmunologyRespiratory virus030211 gastroenterology & hepatologyOriginal Articlethird-party donorsmedicine.symptombusinessadoptive immunotherapyEx vivo
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