Search results for "albicans"

showing 10 items of 328 documents

Chemical composition and biological activities of Artemisia judaica essential oil from southern desert of Jordan

2016

Abstract Ethnopharmacologic relevance Artemisia judaica L. (Arabic name: Beithran ), is a medicinal and aromatic plant growing in the valley bottoms of desert areas, particularly in the southern desert of Jordan nearest to the Jordan-Saudi Arabia borders and in Wadi Araba in the Southern Badia. In Jordan, A. judaica is widely used in traditional medicine being recommended by aboriginal Bedouins in the North Badia region of Jordan as calmative. Furthermore, it is used for the treatment of stomach ache, heart diseases, sexual weakness, diabetes, gastro-intestinal disorders and external wounding. Additionally, other folk medicines of the Arabic region commonly use this aromatic plant for the t…

Lipopolysaccharides0301 basic medicineAntifungal AgentsDPPHAnti-Inflammatory AgentsGerm tube01 natural scienceslaw.inventionMicechemistry.chemical_compoundCamphorlawCandida albicansDrug DiscoveryCandida albicansbiologyTraditional medicineHep G2 CellsCorpus albicansDesert ClimatePiperitoneCell SurvivalMicrobial Sensitivity TestsNitric OxideGas Chromatography-Mass SpectrometryArtemisia judaica03 medical and health sciencesOils VolatileAnimalsHumansPlant OilsEssential oilPharmacologyJordanPlants MedicinalDose-Response Relationship DrugPlant ExtractsMacrophagesMacrophage ActivationPlant Components Aerialbiology.organism_classification0104 chemical sciences010404 medicinal & biomolecular chemistryRAW 264.7 Cells030104 developmental biologyArtemisiachemistryBiofilmsCryptococcus neoformansPhytotherapyJournal of Ethnopharmacology
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TLR2, TLR4 and Dectin-1 signalling in hematopoietic stem and progenitor cells determines the antifungal phenotype of the macrophages they produce

2016

TLRs represent an attractive target for the stimulation of myeloid cell production by HSPCs. We have previously demonstrated that HSPCs use TLR2 to sense Candida albicans in vivo and induce the production of macrophages. In this work, we used an in vitro model of HSPCs differentiation to investigate the functional consequences for macrophages of exposure of HSPCs to various PAMPs and C. albicans cells. Mouse HSPCs (Lin(-) cells) were cultured with M-CSF to induce macrophage differentiation, in the presence or absence of the following PRR agonists: Pam3CSK4 (TLR2 ligand), LPS (TLR4 ligand), depleted zymosan (which only activates Dectin-1), or C. albicans yeasts (which activate several PRRs, …

Lipopolysaccharides0301 basic medicineMacrophage colony-stimulating factorCellular differentiationImmunologyBiologyMicrobiologyMicrobiologyProinflammatory cytokineLipopeptidesMice03 medical and health sciences0302 clinical medicineCandida albicansAnimalsLectins C-TypeProgenitor cellCandida albicansInnate immune systemMacrophage Colony-Stimulating FactorMacrophagesZymosanCell DifferentiationHematopoietic Stem Cellsbiology.organism_classificationToll-Like Receptor 2Cell biologyMice Inbred C57BLToll-Like Receptor 4TLR2030104 developmental biologyInfectious DiseasesTLR4Female030215 immunologyMicrobes and Infection
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Killed Candida albicans yeasts and hyphae inhibit gamma interferon release by murine natural killer cells.

2006

ABSTRACT Killed yeasts and hyphae of Candida albicans inhibit gamma interferon secretion by highly purified murine NK cells in response to the Toll-like receptor ligands lipopolysaccharide and zymosan. This effect, which is also observed in the presence of NK-activating cytokines (interleukin-2 [IL-2], IL-12, and IL-15), may represent a novel mechanism of immune evasion that contributes to the virulence of C. albicans .

LipopolysaccharidesHyphaLipopolysaccharideImmunologyHyphaeMicrobiologyNatural killer cellMicrobiologychemistry.chemical_compoundInterferon-gammaMiceImmune systemCandida albicansmedicineAnimalsInterferon gammaCandida albicansbiologyInterleukinsZymosanZymosanbiology.organism_classificationCorpus albicansToll-Like Receptor 2Killer Cells NaturalMice Inbred C57BLToll-Like Receptor 4Infectious Diseasesmedicine.anatomical_structurechemistryParasitologyFungal and Parasitic Infectionsmedicine.drugInfection and immunity
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Evaluation of the Role of Candida albicans Agglutinin-Like Sequence (Als) Proteins in Human Oral Epithelial Cell Interactions

2012

The fungus C. albicans uses adhesins to interact with human epithelial surfaces in the processes of colonization and pathogenesis. The C. albicans ALS (agglutinin-like sequence) gene family encodes eight large cell-surface glycoproteins (Als1-Als7 and Als9) that have adhesive function. This study utilized C. albicans Δals mutant strains to investigate the role of the Als family in oral epithelial cell adhesion and damage, cytokine induction and activation of a MAPK-based (MKP1/c-Fos) signaling pathway that discriminates between yeast and hyphae. Of the eight Δals mutants tested, only the Δals3 strain showed significant reductions in oral epithelial cell adhesion and damage, and cytokine pro…

MAPK/ERK pathwaySciencemedicine.medical_treatmentImmunologyBlotting WesternMycologyMicrobiologyFungal ProteinsMolecular Cell BiologymedicineGeneticsHumansPhosphorylationCandida albicansCell damageBiologyMultidisciplinarybiologyQRImmunityMouth MucosaDual Specificity Phosphatase 1Epithelial Cellsbiology.organism_classificationmedicine.diseaseCorpus albicansSignaling CascadesCell biologyBacterial adhesinCytokineImmune SystemMedicineCytokinesSignal transductionCellular TypesCandidalysinCell Adhesion MoleculesProto-Oncogene Proteins c-fosResearch ArticleDevelopmental BiologySignal Transduction
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Candida albicans Yeast and Hyphae are Discriminated by MAPK Signaling in Vaginal Epithelial Cells

2011

We previously reported that a bi-phasic innate immune MAPK response, constituting activation of the mitogen-activated protein kinase (MAPK) phosphatase MKP1 and c-Fos transcription factor, discriminates between the yeast and hyphal forms of Candida albicans in oral epithelial cells (ECs). Since the vast majority of mucosal Candida infections are vaginal, we sought to determine whether a similar bi-phasic MAPK-based immune response was activated by C. albicans in vaginal ECs. Here, we demonstrate that vaginal ECs orchestrate an innate response to C. albicans via NF-κB and MAPK signaling pathways. However, unlike in oral ECs, the first MAPK response, defined by c-Jun transcription factor acti…

MAPK/ERK pathwaylcsh:MedicineYeast and Fungal ModelsPathogenesisSignal transductionMolecular cell biologyCandida albicansGranulocyte Colony-Stimulating FactorCandida albicanslcsh:ScienceImmune Response0303 health sciencesMultidisciplinarybiologyCandidiasisNF-kappa BSignaling cascadesObstetrics and GynecologyCorpus albicansInnate ImmunityHost-Pathogen InteractionInfectious DiseasesVaginaCytokinesMedicineFemaleSignal transductionCandidalysinResearch ArticleMAPK signaling cascadesMAP Kinase Signaling SystemUrologyImmunologySexually Transmitted DiseasesHyphaeMycologyMicrobiologyMicrobiologyImmune Activation03 medical and health sciencesModel OrganismsHumansTranscription factorBiology030304 developmental biologyInnate immune systemChemokine CCL20030306 microbiologyGenitourinary InfectionsInterleukin-6lcsh:RImmunityFungiMouth MucosaImmune DefenseEpithelial Cellsbiology.organism_classificationImmunity InnateCCL20Immune Systemlcsh:QClinical ImmunologyPLoS ONE
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Oral colonization by Candida species in orthodontic patients before, during and after treatment with fixed appliances: A prospective controlled trial

2020

Este artículo se encuentra disponible en la siguiente URL: http://www.medicinaoral.com/medoralfree01/aop/57565.pdf Background: Orthodontic treatment with fixed appliances is associated with changes in oral microbiota, including increased Candida colonization. The Candida fungus can cause oral lesions and infections such as candidiasis and angular cheilitis, and is harmful to both the patient and the orthodontist. Poor hygiene facilitates the colonization of these microorganisms. The key aim was to quantify the colonization of C. albicans in patients prior to beginning orthodontic treatment, and during the treatment process. Material and Methods: A total of 124 patients (43 males and 80 fema…

MEDLINEDentistryOrthodonticsOrthodontics.law.invention03 medical and health sciences0302 clinical medicineRandomized controlled triallawOrtodoncia.MedicineColonizationGeneral Dentistry0303 health sciences030306 microbiologybusiness.industryResearch030206 dentistry:CIENCIAS MÉDICAS [UNESCO]Candida Albicans - Treatment.Fungicidas.UNESCO::CIENCIAS MÉDICASCándida albicans - Tratamiento.businessAfter treatmentFungicides.Journal of Clinical and Experimental Dentistry
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Using a Multi-Locus Microsatellite Typing method improved phylogenetic distribution of Candida albicans isolates but failed to demonstrate associatio…

2012

EA MERS CT3 Enjeu 3; International audience; The dimorphic yeast Candida albicans is a component of the normal microflora at the mucosal surfaces of healthy individuals. It possesses an array of phenotypic properties considered as virulence traits that contribute to pathogenicity of the yeast in immuno-compromised patients. We addressed the question of the pathogenicity of lineages of C. albicans with regard to their genotype in three series of C. albicans isolates (a series of commensal isolates collected in healthy individuals, a group of bloodstream isolates and a group of non-bloodstream clinical isolates) using a Multi-Locus Microsatellite Typing (MLMT) approach based on the analysis o…

MESH: Genetic MarkersMESH : Microsatellite RepeatsMESH : CandidiasisGenotypeCandida albicansMESH : Genetic MarkersDNA FungalMycological Typing TechniquesCandida albicansMESH : Mycological Typing TechniquesMESH: PhylogenyPhylogeny[ SDV.MP.MYC ] Life Sciences [q-bio]/Microbiology and Parasitology/Mycology[SDV.MP.MYC]Life Sciences [q-bio]/Microbiology and Parasitology/MycologyGenetics0303 health sciencesbiologyCandidiasisFungal geneticsAllelic frequenciesMESH: Case-Control StudiesCorpus albicansMESH: CandidiasisInfectious DiseasesMESH : Carrier StateCarrier StateMicrosatelliteMESH: Carrier StateGenetic MarkersMicrobiology (medical)MESH : Case-Control StudiesGenotypingMESH : Candida albicansGenes FungalMicrobiologyMicrobiology03 medical and health sciencesMESH: Mycological Typing TechniquesGeneticsHumansPathogenicityTypingLineagesMolecular BiologyEcology Evolution Behavior and Systematics030304 developmental biologyMESH: Humans030306 microbiologyMESH: Candida albicansMESH : HumansUPGMAMESH : Phylogenybiology.organism_classificationMESH: DNA FungalCase-Control StudiesMultilocus sequence typingMLMTMESH : Genes FungalMESH: Microsatellite RepeatsMESH : DNA FungalMESH: Genes FungalMicrosatellite Repeats
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Enhanced proinflammatory response to the Candida albicans gpi7 null mutant by murine cells.

2008

International audience; The Candida albicans gpi7/gpi7 null mutant strain (Deltagpi7), which is affected in glycosylphosphatidylinositol (GPI) anchor biosynthesis, showed a reduced virulence following systemic infection of C57BL/6 mice. In vitro production of TNF-alpha, IL-6 and IL-1beta by macrophages in response to Deltagpi7 cells was significantly increased as compared to control (wild type GPI7/GPI7 and revertant gpi7/GPI7) cells; this probably contributes to the enhanced recruitment of neutrophils to the peritoneal cavity in response to Deltagpi7 cells. Survival of knockout mice for Toll-like receptor (TLR) 2 and TLR4 following intravenous injection of Deltagpi7 cells showed no signifi…

MESH: InflammationGlycosylphosphatidylinositolsNeutrophilsmedicine.medical_treatmentInterleukin-1betasourisMESH: NeutrophilsMESH: VirulenceMESH: Mice KnockoutMiceMESH: Interleukin-1betaNull cellMESH: AnimalsCandida albicansPeritoneal CavityCells CulturedMice Knockout0303 health sciencesToll-like receptorbiologyVirulenceMESH: Toll-Like Receptor 2MESH: Peritoneal CavityMESH: Toll-Like Receptor 4MESH: GlycosylphosphatidylinositolsInfectious DiseasesCytokineMESH: Survival AnalysisTumor necrosis factor alphaMESH: Fungal Proteinsprotéine de la paroi cellulaireMESH: Macrophages PeritonealMESH: Cells CulturedVirulence FactorsImmunologyMicrobiologyMicrobiologyProinflammatory cytokineFungal Proteins03 medical and health sciencesMESH: Mice Inbred C57BLmedicineAnimalsMESH: Mice030304 developmental biologyMESH: Virulence FactorsInflammation030306 microbiologyInterleukin-6Tumor Necrosis Factor-alphaMESH: Candida albicans[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologybiology.organism_classificationSurvival AnalysispathogénicitéMESH: Interleukin-6Toll-Like Receptor 2Mice Inbred C57BLToll-Like Receptor 4TLR2GlycosylphosphatidylinositolMESH: Gene DeletionMESH: Tumor Necrosis Factor-alphaTLR4Macrophages Peritonealcandida albicansimmunitéGene Deletion
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Interleukin-17 Inhibition in Spondyloarthritis Is Associated With Subclinical Gut Microbiome Perturbations and a Distinctive Interleukin-25-Driven In…

2020

Objective To characterize the ecological effects of biologic therapies on the gut bacterial and fungal microbiome in psoriatic arthritis (PsA)/spondyloarthritis (SpA) patients. Methods Fecal samples from PsA/SpA patients pre- and posttreatment with tumor necrosis factor inhibitors (TNFi; n = 15) or an anti-interleukin-17A monoclonal antibody inhibitor (IL-17i; n = 14) underwent sequencing (16S ribosomal RNA, internal transcribed spacer and shotgun metagenomics) and computational microbiome analysis. Fecal levels of fatty acid metabolites and cytokines/proteins implicated in PsA/SpA pathogenesis or intestinal inflammation were correlated with sequence data. Additionally, ileal biopsies obtai…

Male0301 basic medicineArthritisPsoriatic0302 clinical medicineInterleukin 25Monoclonal2.1 Biological and endogenous factorsImmunology and AllergyMedicineAetiologyIntestinal MucosaCandida albicansHumanizedSubclinical infectionbiologyInterleukin-17Innate lymphoid cellMiddle AgedIntestinesPublic Health and Health ServicesFemaleTumor necrosis factor alphaInterleukin 17Clinical SciencesImmunologyAntibodies Monoclonal HumanizedAutoimmune DiseaseAntibodiesArticle03 medical and health sciencesRheumatologyClinical ResearchSpondylarthritisHumansMicrobiomeInflammation030203 arthritis & rheumatologybusiness.industryArthritisInflammatory and immune systemArthritis Psoriaticbiology.organism_classificationmedicine.diseaseArthritis & RheumatologyGastrointestinal Microbiome030104 developmental biologyImmunologyTumor Necrosis Factor InhibitorsDigestive Diseasesbusiness
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Candida arthritis: cellular immune responses of synovial fluid and peripheral blood lymphocytes to Candida albicans.

1991

A case of septic Candida albicans arthritis of the knee in a patient with systemic candidiasis is presented. Systemic and intra-articular cellular immune responses to C albicans and various bacterial antigens were monitored for 15 weeks. It is shown that the candida induced blastogenesis of synovial fluid lymphocytes was much more stimulated than that of peripheral blood lymphocytes, and that the proportion of activated cells expressing HLA class II antigens was markedly increased in the synovial fluid. Strong cellular immune responses to Candida albicans could still be shown many weeks after the synovial fluid aspirates had become sterile. For the first time synovial fluid derived, CD4 pos…

MaleCellular immunityAntigens FungalKnee JointT-LymphocytesImmunologyArthritisGeneral Biochemistry Genetics and Molecular BiologyEpitopesImmune systemRheumatologyAntigenCandida albicansSynovial FluidmedicineImmunology and AllergySynovial fluidHumansCandida albicansArthritis Infectiousbiologybusiness.industryCandidiasisAntibodies MonoclonalMiddle Agedmedicine.diseasebiology.organism_classificationLymphocyte SubsetsImmunologySystemic candidiasisBacterial antigenbusinessCell DivisionResearch Article
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