Search results for "androgeni"

showing 10 items of 94 documents

Does metformin induce ovulation in normoandrogenic anovulatory women?

2004

Objective: This study was undertaken to evaluate the efficacy of metformin in women with anovulation who do not have evidence for hyperandrogenism and classic polycystic ovary syndrome. Study design: A randomized trial of metformin (1500 mg daily) and placebo in 24 anovulatory women was undertaken for 3 months. Assessments of changes in hormone levels and insulin sensitivity were carried out. Abnormal ormonal values were defined by levels exceeding the range in normal ovulatory controls. Results: Anovulatory women had normal androgen levels and luteinizing hormone but had higher serum insulin and lower insulin sensitivity compared with controls. Over 3 months, there were 16 ovulatory cycles…

AdultOvulationmedicine.medical_specialtymedia_common.quotation_subjectmedicine.medical_treatmentDrug Administration ScheduleAnovulationInternal medicinemedicineHumansHypoglycemic AgentsInsulinOvulationMenstrual CycleMenstrual cyclemedia_commonbusiness.industryInsulinHyperandrogenismObstetrics and Gynecologymedicine.diseasePolycystic ovaryMetforminMetforminTreatment OutcomeEndocrinologyInfertilityFemaleHyperandrogenismLuteinizing hormonebusinessAnovulationPolycystic Ovary Syndromemedicine.drugAmerican Journal of Obstetrics and Gynecology
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Clinical and endocrine characteristics of the main polycystic ovary syndrome phenotypes

2009

Abstract OBJECTIVE: To evaluate the clinical and endocrine differences between main polycystic ovary syndrome (PCOS) phenotypes. DESIGN: To evaluate clinical and hormone parameters in a large group of consecutive women with PCOS diagnosed according Rotterdam criteria and divided according their phenotype. SETTING: University department of medicine. PATIENT(S): Three hundred eighty-two consecutive women with PCOS and 85 ovulatory controls. INTERVENTION(S): Evaluation of clinical and hormone parameters. MAIN OUTCOME MEASURE(S): Blood levels of gonadotropins, testosterone, sex-hormone-binding globulin, dehydroepiandrosterone sulfate, 17α-hydroxyprogesterone, progesterone, glucose, and insulin,…

AdultPCOS Lipids Insulin resistance Fertility Androgens Obesitymedicine.medical_specialtySettore MED/09 - Medicina Internaendocrine system diseasesEndocrine SystemSettore MED/13 - EndocrinologiaAnovulationYoung Adultchemistry.chemical_compoundDehydroepiandrosterone sulfateSex Hormone-Binding GlobulinInternal medicinePrevalencemedicineHumansTestosteroneRetrospective StudiesDehydroepiandrosterone SulfateFree androgen indexbusiness.industryHyperandrogenismObstetrics and GynecologyLuteinizing Hormonemedicine.diseaseSettore MED/40 - Ginecologia E OstetriciaPolycystic ovaryfemale genital diseases and pregnancy complicationsPhenotypeEndocrinologyReproductive MedicinechemistryCase-Control StudiesAndrogensFemaleFollicle Stimulating HormoneHyperandrogenismLuteinizing hormonebusinessPolycystic Ovary SyndromeHormoneFertility and Sterility
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Long-term consequences of polycystic ovary syndrome on cardiovascular risk

2009

Most available data suggest that the prevalence of cardiovascular diseases in women with polycystic ovary syndrome (PCOS) is smaller than expected based on risk calculations during fertile years; therefore, more studies are needed on long-term cardiovascular consequences. Evidence is accumulating that postmenopausal women with PCOS have an increased risk of cerebrovascular events and cardiovascular morbidity. These events are partially related to persisting hyperandrogenism but are mostly correlated with excessive body weight (mainly visceral obesity); this suggests that our best long-term strategy is to ensure that women with PCOS are informed about their high risk for metabolic and cardio…

AdultPolycystic ovary syndrome cardiovascular risk menopause eventsAgingPediatricsmedicine.medical_specialty10265 Clinic for Endocrinology and Diabetology610 Medicine & healthBody weightDiabetes ComplicationsRisk FactorsmedicineHumansCystObesityAgedAged 80 and overGynecologyPostmenopausal womenbusiness.industryHyperandrogenismObstetrics and Gynecology2729 Obstetrics and Gynecology2743 Reproductive MedicineMiddle Agedmedicine.diseasePolycystic ovaryMenopauseC-Reactive ProteinIncreased riskReproductive MedicineCardiovascular DiseasesAndrogensFemaleAdiponectinHyperandrogenismbusinessBiomarkersVisceral ObesityPolycystic Ovary Syndrome
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PCOS: metabolic impact and long-term management.

2012

Polycystic ovary syndrome (PCOS) is the most frequent endocrinopathy in women, affecting up to 10% of those in reproductive age. Furthermore, PCOS presents a lifetime risk of type II diabetes, cardiovascular diseases and endometrial cancer. Women with PCOS have increased cardiovascular risk; however, the risk is not the same in all patients and it is necessary to assess an individual risk profile. There is a discrepancy between increased cardiovascular risk at young age and postmenopausal number of cardiovascular events, probablu depending on changes in androgen ovarian function after the forties. However, changes with age of metabolic profile in women with PCOS have not been studied yet an…

AdultSettore MED/09 - Medicina InternaAdolescentComorbiditySettore MED/11 - Malattie Dell'Apparato CardiovascolareMetforminSettore MED/13 - EndocrinologiaDiabetes Mellitus Type 2Cardiovascular DiseasesRisk FactorsObesity AbdominalPCOS Metabolic Syndrome Cardiovascular risk aging insulin resistance hyperandrogenismHypertensionHumansFemaleHydroxymethylglutaryl-CoA Reductase InhibitorsInsulin ResistanceChildAntihypertensive AgentsDyslipidemiasPolycystic Ovary SyndromeMinerva ginecologica
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Increased DHEAs levels in PCO syndrome: evidence for the existence of two subgroups of patients

1986

In 49 patients affected by PCO syndrome the serum levels of dehydroepiandroster-one-sulphate (DHEAs) were determined and correlated with the cfinical presentation and the endocrine pattern. Twenty-three patients (47%) had high DHEAs levels (h-DHEAs patients). They presented a milder clinical presentation (low incidence of amenorrhea) than PCO patients with normal DHEAs levels (n-DHEAs patients). In h-DHEAs patients the finding of a normal DHEAs. response to ACTH and of slightly increased 17OHP serum levels suggested that the elevation of serum DHEAs was not due to an adrenal enzymatic deficiency but to a tonic hyperstimulation of the adrenals. Two subgroups of h-DHEAs patients were identifi…

Adultendocrine systemmedicine.medical_specialtyAdolescentEndocrinology Diabetes and MetabolismDehydroepiandrosteroneEstroneAdrenocorticotropic hormoneBiologychemistry.chemical_compoundEndocrinologyDehydroepiandrosterone sulfateAdrenocorticotropic HormoneInternal medicinepolycyclic compoundsmedicineHumansEndocrine systemskin and connective tissue diseasesAmenorrheaThyrotropin-Releasing HormoneDehydroepiandrosterone SulfateAdrenal glandHyperandrogenismDehydroepiandrosteronemedicine.diseasePolycystic ovaryProlactinmedicine.anatomical_structureEndocrinologychemistryFemalehuman activitieshormones hormone substitutes and hormone antagonistsPolycystic Ovary SyndromeJournal of Endocrinological Investigation
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Refractory Acne and 21-Hydroxylase Deficiency in a Selected Group of Female Patients.

2009

<i>Background:</i> Excessive androgen production, suspected in women when acne is accompanied by hirsutism and menstrual irregularities, may be due to congenital adrenal hyperplasia. This inherited disorder of cortisol biosynthesis is caused in more than 90–95% of all cases by 21-hydroxylase deficiency (21-OHD). The steroid 21-hydroxylase gene <i>(CYP21)</i> has a high degree of variability. <i>Objective:</i> This study was conducted to evaluate <i>CYP21 </i>gene mutations in a selected group of women with papulopustular and comedonal acne refractory to treatment, irregular menses and hirsutism. <i>Methods:</i> 30 out of 61 women e…

Adultmedicine.medical_specialtyAdolescentDrug ResistancePhysiologyDermatologyAdrenocorticotropic hormoneYoung AdultSettore MED/38 - Pediatria Generale E SpecialisticaPapulopustularInternal medicineAcne VulgarismedicineHumansPoint MutationCongenital adrenal hyperplasiaGenetic TestingRefractory acne Excessive androgen production Non-classical 21-hydroxylase deficiency CYP21 gene mutations.AcnehirsutismAdrenal Hyperplasia Congenitalmedicine.diagnostic_testbiologybusiness.industry17-alpha-HydroxyprogesteroneACTH stimulation test21-Hydroxylasemedicine.diseasePolycystic ovaryEndocrinologybiology.proteinFemaleSteroid 21-HydroxylaseHyperandrogenismbusinessPolycystic Ovary Syndrome
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Low estradiol-to-testosterone ratio is associated with oligo-anovulatory cycles and atherogenic lipidic pattern in women with polycystic ovary syndro…

2010

The estradiol-to-testosterone (E2/T) ratio has been investigated in different diseases but few in vivo data are available with regard to its role in women with ovary syndrome (PCOS). The aim of this study was to evaluate the role of the E2/T ratio in the ovulatory function and metabolic pattern in such women.We retrospectively evaluated hyperandrogenemia, clinical hyperandrogenism, ovarian morphology, hypothalamo-hypophyseal axis and metabolic syndrome parameters in a cohort of 202 consecutive women affected by PCOS. An oral glucose tolerance test measured areas under the curve for insulin (AUC(2hIRI)), for glucose (AUC(2hglucose)), and the HOMA-IR and Matsuda index of insulin resistance we…

Adultmedicine.medical_specialtyAdolescentEndocrinology Diabetes and Metabolismmedia_common.quotation_subjectOvarySettore MED/13 - EndocrinologiaCohort StudiesYoung AdultEndocrinologyInsulin resistanceRisk FactorsInternal medicineFollicular phasePCOSmedicineHumansTestosteroneSicilyOvulationProgesteroneMenstrual cycleRetrospective Studiesmedia_commonEstradiolbusiness.industryOvaryHyperandrogenismObstetrics and GynecologyAtherosclerosismedicine.diseasePolycystic ovaryCholesterolEndocrinologymedicine.anatomical_structureFollicular PhaseFemaleMetabolic syndromeHyperandrogenismbusinessAlgorithmsAnovulationPolycystic Ovary SyndromeGynecological Endocrinology
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The oligomenorrhoic phenotypes of polycystic ovary syndrome are characterized by a high visceral adiposity index: a likely condition of cardiometabol…

2011

BACKGROUND: Women with polycystic ovary syndrome (PCOS) frequently exhibit central obesity, glucose intolerance, atherogenic dys- lipidemia and hypertension, which are characteristic features of a condition of cardiometabolic risk. Our objective was to investigate the relationship between visceral adiposity index (VAI) and phenotypic characteristics in women with PCOS. METHODS: We conducted a cross-sectional case-control study in our Endocrinology Outpatients Clinic. A total of 220 women with PCOS (Rotterdam definition) and I44 age- and BMI-matched healthy women were studied. We evaluated hyperandrogenemia and clinical hyperandrogenism, ovarian morphology, hypothalamic-hypophyseal axis and …

Adultmedicine.medical_specialtyAdolescentPopulationIntra-Abdominal FatSettore MED/13 - EndocrinologiaInsulin resistanceRisk FactorsInternal medicinemedicinePCOSHumanseducationAdiposityRetrospective StudiesMetabolic Syndromeeducation.field_of_studyGlucose tolerance testmedicine.diagnostic_testbusiness.industryRehabilitationHyperandrogenismCase-control studyObstetrics and GynecologyOdds ratioGlucose Tolerance Testmedicine.diseasePolycystic ovaryOligomenorrheaPhenotypeEndocrinologyReproductive MedicineCardiovascular DiseasesCase-Control StudiesFemaleInsulin ResistanceMetabolic syndromebusinessPolycystic Ovary Syndrome
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The ratio of androstenedione: 11β-hydroxyandrostenedione is an important marker of adrenal androgen excess in women

1992

To determine if the ratio of serum androstenedione (A):11 beta-hydroxyandrostenedione (OHA) would be helpful in differentiating adrenal from ovarian hyperandrogenism.Prospective study of outpatients being evaluated for hyperandrogenism.Normal women (n = 27), those with hyperandrogenic chronic anovulation (n = 25), and 7 with adult onset of congenital adrenal hyperplasia (CAH) because of 21-hydroxylase deficiency.Fasting serum between 8:00 A.M. and 9:00 A.M. Patients with hyperandrogenic chronic anovulation and CAH received dexamethasone (DEX) 2 mg for 7 days.Serum testosterone (T), unbound T, dehydroepiandrosterone sulfate (DHEAS), A, and 11 beta-OHA by radioimmunoassay.Serum 11 beta-OHA an…

Adultmedicine.medical_specialtyAdolescentmedicine.drug_classAdrenal Gland DiseasesRadioimmunoassayDexamethasoneAnovulationchemistry.chemical_compoundDehydroepiandrosterone sulfateInternal medicineAdrenal GlandsmedicineHumansCongenital adrenal hyperplasiaProspective StudiesAndrostenedioneDexamethasoneHyperplasiaAdrenal Hyperplasia Congenitalbusiness.industryHyperandrogenismAndrostenedioneObstetrics and GynecologyRadioimmunoassaymedicine.diseaseAndrogenEndocrinologyReproductive MedicinechemistryAndrogensFemalebusinessBiomarkersmedicine.drugFertility and Sterility
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Atherogenic lipoprotein phenotype and low-density lipoproteins size and subclasses in women with polycystic ovary syndrome.

2006

Abstract Context: An altered lipid profile is common in polycystic ovary syndrome (PCOS) and is usually characterized by increased triglycerides and low high-density lipoprotein (HDL)-cholesterol levels. In the general population, these alterations are often associated with the increase of small low-density lipoproteins (LDLs) in the so-called “atherogenic lipoprotein phenotype” (ALP) that determines a further increase of cardiovascular risk. In this study, we evaluated the presence of ALP in the plasma of women with PCOS. Setting: Measurements and analysis of LDL size were performed at the Clinic of Endocrinology, Diabetes, and Clinical Nutrition, University Hospital, Zurich. PCOS patients…

Adultmedicine.medical_specialtyEndocrinology Diabetes and MetabolismClinical BiochemistryPopulationClinical nutritionBiologyBiochemistryAnovulationEndocrinologyDiabetes mellitusInternal medicinemedicineHumansInsulinParticle SizeeducationTriglyceridesAtherogenic lipoprotein low-density lipoproteins polycystic ovary syndromeeducation.field_of_studymedicine.diagnostic_testBiochemistry (medical)HyperandrogenismCholesterol HDLnutritional and metabolic diseasesmedicine.diseasePolycystic ovaryLipoproteins LDLEndocrinologyPhenotypelipids (amino acids peptides and proteins)FemaleLipid profileLipoproteinPolycystic Ovary SyndromeThe Journal of clinical endocrinology and metabolism
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