Search results for "aorta"

showing 10 items of 458 documents

Modulatory role of magnesium on the contractile response of rat aorta to several agonists in normal and calcium-free medium.

1993

Abstract Acute withdrawal of external Mg2+ increased basal tone of rat isolated aorta incubated in the presence of Ca2+. Above normal levels of Mg2+ (1–4 Mm) inhibited basal tone while much higher levels of the divalent cation (64–256 Mm) evoked contractile responses regardless of the presence of Ca2+. Contractile responses to noradrenaline (1μm) and KCl (80 Mm) were inhibited by addition of cumulative concentrations of Mg2+. Acetylcholine-induced contractions in the presence of physiological concentrations of Mg2+ (1 Mm) decreased gradually to the basal tone, but a sustained contraction was observed in the absence of this ion. In Ca2+-free medium, acetylcholine-induced phasic responses ind…

Malemedicine.medical_specialtyContraction (grammar)Muscle RelaxationPharmaceutical Sciencechemistry.chemical_elementAorta ThoracicCalciumIn Vitro TechniquesMuscle Smooth VascularDivalentPotassium ChlorideNorepinephrineInternal medicinemedicineExtracellularAnimalsMagnesiumRats WistarPharmacologychemistry.chemical_classificationAcetylcholineCulture MediaRatsKineticsMuscle relaxationEndocrinologychemistryCalciummedicine.symptomIsotonic SolutionsExtracellular SpaceMagnesium DeficiencyVasoconstrictionAcetylcholinemedicine.drugMuscle contractionMuscle ContractionThe Journal of pharmacy and pharmacology
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β-Adrenoceptors differentially regulate vascular tone and angiogenesis of rat aorta via ERK1/2 and p38

2014

β-Adrenoceptors (β-ARs) modulate ERK1/2 and p38 in different cells, but little is known about the contribution of these signaling pathways to the function of β-ARs in vascular tissue. Immunoblotting analysis of rat aortic rings, primary endothelial (ECs) and smooth muscle cells (SMCs) isolated from aorta showed that β-AR stimulation with isoprenaline activated p38 in aortic rings and in both cultured cell types, whereas it had a dual effect on ERK1/2 phosphorylation, decreasing it in ECs while increasing it in SMCs. These effects were reversed by propranolol, which by itself increased p-ERK1/2 in ECs. Isoprenaline β-AR mediated vasodilation of aortic rings was potentiated by the ERK1/2 inhi…

Malemedicine.medical_specialtyEndotheliumPhysiologyAngiogenesisVasodilator AgentsAdrenergic beta-AntagonistsMyocytes Smooth MuscleNeovascularization PhysiologicAorta ThoracicStimulationVasodilationFibroblast growth factorp38 Mitogen-Activated Protein KinasesMuscle Smooth VascularInternal medicineIsoprenalinemedicine.arteryReceptors Adrenergic betamedicineAnimalsHumansRats WistarMitogen-Activated Protein Kinase 1PharmacologyMatrigelAortaMitogen-Activated Protein Kinase 3business.industryAdrenergic beta-AgonistsPropranololRatsVasodilationHEK293 Cellsmedicine.anatomical_structureEndocrinologycardiovascular systemMolecular MedicineEndothelium Vascularbusinessmedicine.drugVascular Pharmacology
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Troponin I concentrations of shed blood might influence monitoring of myocardial injury after coronary operations.

2002

Abstract In a prospective study we evaluated the concentration of cardiac troponin I (cTnI) and creatine kinase activities (CK) in shed mediastinal blood in the early postoperative period after coronary artery bypass grafting (CABG). Forty seven patients who underwent first time elective CABG were studied. CTnI levels and CK activities in arterial blood and shed mediastinal blood were measured after admission to the intensive care unit (ICU) and 6 h after unclamping the aorta. Mediastinal shed blood samples were drawn from 23 patients (group A) before the filter of the cardiotomy reservoir and from 24 patients (group B) behind. Additionally, both markers were measured in blood samples colle…

Malemedicine.medical_specialtyExtracorporeal Circulationlaw.inventionlawmedicine.arteryInternal medicineMonitoring IntraoperativeTroponin ImedicineHumansMyocardial infarctionCoronary Artery BypassCreatine KinaseAgedAortabiologybusiness.industryMyocardiumTroponin IHematologyPerioperativemedicine.diseaseIntensive care unitmedicine.anatomical_structurecardiovascular systembiology.proteinCardiologyArterial bloodCreatine kinaseFemalebusinessBiomarkersArteryTransfusion and apheresis science : official journal of the World Apheresis Association : official journal of the European Society for Haemapheresis
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Early distal remodeling after elephant trunk repair of thoraco-abdominal aortic aneurysms

2016

Hemodynamic alterations occur when the elephant trunk (ET) technique is adopted to treat extensive aortic aneurysms. In planning the 2nd stage operation to complete ET repair, surgeons must weigh an adequate recovery time after initial surgery against the risk of postoperative ET-related complications. The purpose of this study was to understand the mechanistic link between the flow alteration caused by the ET graft and the development of premature aortic rupture before the 2nd stage operation. Specifically, fluid-structure interaction (FSI) analysis was performed using the CT imaging data of aorta at different stages of ET repair, and then computational variables were compared to those obs…

Malemedicine.medical_specialtyFluid-structure interaction analysiElephant trunksAortic RuptureBiomedical EngineeringBiophysicsDiastoleHemodynamics030204 cardiovascular system & hematologyAnastomosisModels Biological03 medical and health sciencesAortic aneurysmPostoperative Complications0302 clinical medicinemedicine.arterymedicineHumansOrthopedics and Sports MedicineAortic ruptureAgedAortaAortic aneurysmTEVARAortic Aneurysm Thoracicbusiness.industryRehabilitationHemodynamicsmedicine.diseaseSurgeryBiophysic030228 respiratory systemDescending aortacardiovascular systemFemaleTomography X-Ray ComputedbusinessVascular Surgical ProceduresElephant trunk techniqueJournal of Biomechanics
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Arrhythmic sudden cardiac death in a 3-year-old child with intimal fibroplasia of coronary arteries, aorta, and its branches

2001

Abstract We report an unusual case of “arrhythmic” sudden cardiac death in a 3-year-old child who died of ischemic myocardial lesions as a result of intimal fibroplasia of the coronary arteries. Also affected were the aorta and its major branches, whereas renal and mesenteric arteries, celiac trunk, and systemic veins were normal. Histopathologic examination showed severe concentric thickening of intima because of a proliferation of spindle-shaped cells (mesenchymal cells) set in an abundant extracellular matrix. In some vascular segments the intima was densely fibrotic and hyalinized. No significant inflammation, foam cells, cholesterol clefts, or other evidence of atheroma were present. T…

Malemedicine.medical_specialtyHeart VentriclesFibromuscular dysplasiaPathology and Forensic MedicineSudden cardiac deathPolypsAdventitiaInternal medicinemedicine.arterymedicineFibromuscular DysplasiaHumansVimentinMesenteric arteriesAortaAortabusiness.industryMyocardiumArrhythmias CardiacGeneral Medicinemedicine.diseaseCoronary VesselsImmunohistochemistryActinsCoronary arteriesDeath Sudden CardiacAtheromamedicine.anatomical_structureChild Preschoolcardiovascular systemCardiologyDesminTunica IntimaCardiology and Cardiovascular MedicinebusinessCardiovascular Pathology
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The distinct gene expression of the pro-hormone convertases in the rat heart suggests potential substrates

1995

The present study examined the distribution of the pro-hormone convertases PC1, PC2, furin, PACE4 and PC5 in the rat heart. Northern blot analysis of RNA extracted from cardiac tissues showed high levels of furin and PACE4 mRNA in the atria and ventricles, while PC5 mRNA was found to be expressed at high levels in the dorsal aorta. Although undetectable by Northern blot analysis, both PC1 and PC2 mRNA were detected by in situ hybridization and immunohistochemistry in discrete regions of the intracardiac para-aortic ganglia. In situ hybridization studies also showed that furin mRNA was observed in all cardiac tissues and cells, consistent with the previously reported ubiquitous expression of…

Malemedicine.medical_specialtyHistologyMolecular Sequence DataGene ExpressionIn situ hybridizationSubstrate SpecificityPathology and Forensic MedicineRats Sprague-DawleyDorsal aortaInternal medicineGene expressionmedicineAnimalsAspartic Acid EndopeptidasesAmino Acid SequenceNorthern blotFurinIn Situ HybridizationMessenger RNAbiologyMyocardiumSerine EndopeptidasesRNACell BiologyBlotting NorthernImmunohistochemistryMolecular biologyHormonesRatsEndocrinologycardiovascular systembiology.proteinImmunohistochemistryCell and Tissue Research
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Characterization of two different Ca2+ entry pathways dependent on depletion of internal Ca2+ pools in rat aorta

1998

Ryanodine (10 microM), thapsigargin (1 microM) and cyclopiazonic acid (10 microM) produced a slow, sustained contractile response in rat aorta that only can be observed in Ca2+-containing solution. In Ca2+-free medium, no response to the drugs was obtained, which suggests that the contraction elicited in presence of Ca2+ is mainly due to the contribution of extracellular influx. This Ca2+ entry does not depend on the opening of dihydropyridine-dependent Ca2+-channels for nimodipine does not affect this. Noradrenaline (1 microM) induced a biphasic response in Ca2+-free medium that was mediated by two different Ca2+ compartments, one of which is common to caffeine (10 mM), and is also deplete…

Malemedicine.medical_specialtyIndolesThapsigarginContraction (grammar)Phosphodiesterase InhibitorsVasodilator AgentsAorta ThoracicIn Vitro TechniquesMuscle Smooth VascularNorepinephrinechemistry.chemical_compoundCaffeineInternal medicinemedicine.arterymedicineExtracellularAnimalsVasoconstrictor AgentsRats WistarCa2 entryNimodipinePharmacologyAortaRyanodineRyanodine receptorGeneral MedicineRatsEndocrinologychemistryBiophysicsThapsigarginCalciumCalcium ChannelsCyclopiazonic acidMuscle Contractionmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Effect of Divalent Cations on the Contractile Response of Rat Aorta to Depolarization before and after Nifedipine Treatment

1996

The influence of the divalent cations, Ca2+, Mg2+ and Ba2+, on the contractile response of the rat aorta to KCl and on the recovery of this response after nifedipine treatment was analyzed. KCl (80 mmol/l) promoted a two-phase (phasic and tonic) contractile response in Krebs solution but, as expected, no contractile response in Ca(2+)-free medium. In Mg(2+)-free medium, the phasic response to KCl was unaffected but the tonic one decreased slowly, suggesting that a long incubation time in the absence of Mg2+ (65 min) promotes a loss of or a change in the intracellular distribution of this ion that modifies Ca2+ entry through L channels or Ca2+ handling. Ba2+ (1.8 mmol/l) contracted the rat a…

Malemedicine.medical_specialtyNifedipineCations DivalentAorta ThoracicMuscle Smooth VascularPotassium ChlorideDivalentTonic (physiology)NifedipineInternal medicinemedicine.arterymedicineAnimalsRats WistarPharmacologychemistry.chemical_classificationAnalysis of VarianceAortaChemistryContractile responseDepolarizationGeneral MedicineCalcium Channel BlockersRatsEndocrinologyKrebs solutionIntracellularMuscle Contractionmedicine.drugPharmacology
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Down-regulation of the expression of endothelial NO synthase is likely to contribute to glucocorticoid-mediated hypertension.

1999

Hypertension is a side effect of systemically administered glucocorticoids, but the underlying molecular mechanism remains poorly understood. Ingestion of dexamethasone by rats telemetrically instrumented increased blood pressure progressively over 7 days. Plasma concentrations of Na + and K + and urinary Na + and K + excretion remained constant, excluding a mineralocorticoid-mediated mechanism. Plasma NO 2 − /NO 3 − (the oxidation products of NO) decreased to 40%, and the expression of endothelial NO synthase (NOS III) was found down-regulated in the aorta and several other tissues of glucocorticoid-treated rats. The vasodilator response of resistance arterioles was tested by intravital m…

Malemedicine.medical_specialtyNitric Oxide Synthase Type IIIDown-RegulationVasodilationBiologyEndothelial NOSRats Inbred WKYUmbilical veinDexamethasonechemistry.chemical_compoundInternal medicinemedicineAnimalsRNA MessengerPromoter Regions GeneticAortaCells CulturedNitritesDNA PrimersMultidisciplinaryNitratesBase SequenceAntiglucocorticoidNitric Oxide Synthase Type IIIBiological SciencesRatsNitric oxide synthaseVasodilationEndocrinologychemistryHypertensionbiology.proteinEndothelium VascularNitric Oxide SynthaseGlucocorticoidIntravital microscopymedicine.drugTranscription FactorsProceedings of the National Academy of Sciences of the United States of America
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Chronic Therapy With Isosorbide-5-Mononitrate Causes Endothelial Dysfunction, Oxidative Stress and a Marked Increase in Vascular Endothelin-1 Express…

2011

Aims Isosorbide-5-mononitrate (ISMN) is one of the most frequently used compounds in the treatment of coronary artery disease predominantly in the USA. However, ISMN was reported to induce endothelial dysfunction, which was corrected by vitamin C pointing to a crucial role of reactive oxygen species (ROS) in causing this phenomenon. We sought to elucidate the mechanism how ISMN causes endothelial dysfunction and oxidative stress in vascular tissue. Methods and results Male Wistar rats ( n = 69 in total) were treated with ISMN (75 mg/kg/day) or placebo for 7 days. Endothelin (ET) expression was determined by immunohistochemistry in aortic sections. Isosorbide-5-mononitrate infusion caused si…

Malemedicine.medical_specialtyNitric Oxide Synthase Type IIIIsosorbide DinitratePharmacologymedicine.disease_causeBiochemistryMicechemistry.chemical_compoundSuperoxidesEnosInternal medicinePhysiology (medical)medicineAnimalsNitric Oxide DonorsEnzyme InhibitorsRats WistarEndothelial dysfunctionCyclic GMPAortaMice KnockoutNADPH oxidaseEndothelin-1biologybusiness.industryNADPH Oxidasesmedicine.diseasebiology.organism_classificationEndothelin 1BosentanRatsNitric oxide synthaseEndothelial stem cellOxidative StressNG-Nitroarginine Methyl EsterEndocrinologychemistryApocyninbiology.proteinEndothelium VascularCardiology and Cardiovascular MedicineEndothelin receptorbusinessOxidative stressSignal Transductionmedicine.drugFree Radical Biology and Medicine
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