Search results for "arginine"

showing 10 items of 389 documents

Identification of the Muscarinic Acetylcholine Receptor Subtype Mediating Cholinergic Vasodilation in Murine Retinal Arterioles

2011

To identify the muscarinic acetylcholine receptor subtype that mediates cholinergic vasodilation in murine retinal arterioles.Muscarinic receptor gene expression was determined in murine retinal arterioles using real-time PCR. To assess the functional relevance of muscarinic receptors for mediating vascular responses, retinal vascular preparations from muscarinic receptor-deficient mice were studied in vitro. Changes in luminal arteriole diameter in response to muscarinic and nonmuscarinic vasoactive substances were measured by video microscopy.Only mRNA for the M(3) receptor was detected in retinal arterioles. Thus, M(3) receptor-deficient mice (M3R(-/-)) and respective wild-type controls …

MaleNitroprussidemedicine.medical_specialtyNitric Oxide Synthase Type IIIRetinal ArteryVideo RecordingGene ExpressionBiologyReal-Time Polymerase Chain ReactionMuscle Smooth VascularMiceInternal medicineMuscarinic acetylcholine receptor M5Muscarinic acetylcholine receptormedicineMuscarinic acetylcholine receptor M4AnimalsRNA MessengerMice KnockoutReceptor Muscarinic M3Dose-Response Relationship DrugMuscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2ArticlesMuscarinic acetylcholine receptor M1AcetylcholineVasodilationArteriolesNG-Nitroarginine Methyl EsterEndocrinologyCholinergicCarbacholFemaleEndothelium VascularAcetylcholinemedicine.drugInvestigative Opthalmology & Visual Science
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Contribution of nitric oxide synthase isoforms to cholinergic vasodilation in murine retinal arterioles.

2013

Abstract Nitric oxide synthases (NOSs) are critically involved in regulation of ocular perfusion. However, the contribution of the individual NOS isoforms to vascular responses is unknown in the retina. Because some previous findings suggested an involvement of inducible nitric oxide synthase (iNOS) in the regulation of retinal vascular tone, a major goal of the present study was to examine the hypothesis that iNOS is involved in mediating cholinergic vasodilation responses of murine retinal arterioles. Another subject of this study was to test the contribution of the other two NOS isoforms, neuronal (nNOS) and endothelial NOS (eNOS), to cholinergic retinal arteriole responses. Expression o…

MaleNitroprussidemedicine.medical_specialtyNitric Oxide Synthase Type IIIVasodilator AgentsNitric Oxide Synthase Type IIVasodilationNitric Oxide Synthase Type IBiologyEndothelial NOSReal-Time Polymerase Chain ReactionGene Expression Regulation EnzymologicNitric oxideCellular and Molecular Neurosciencechemistry.chemical_compoundMiceInternal medicinemedicineAnimalsRNA MessengerEnzyme InhibitorsMice KnockoutBrainRetinal VesselsRetinalSensory SystemsAcetylcholineNitric oxide synthaseMice Inbred C57BLVasodilationOphthalmologyArteriolesEndocrinologyNG-Nitroarginine Methyl Esterchemistrybiology.proteinCholinergicmedicine.symptomVasoconstrictionAcetylcholinemedicine.drugExperimental eye research
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Demonstration of action-potential-producing cells in the rat pineal gland in vitro and their regulation by norepinephrine and nitric oxide

1998

There is evidence that sympathetically innervated mammalian pineal glands contain cells that exhibit action potentials. It is unknown whether ex vivo pineal glands deprived of their nervous input are still capable of firing. In the present study, multiple-unit recordings from rat pineals revealed spontaneously active cell clusters with a mean firing frequency of 1.5 +/- 0.3 Hz which could be abolished by tedrodotoxin. Regularly firing clusters showed no inherent periodicity in the minute range, whereas rhythmical clusters with periodically repeated bursts had period lengths of 12.6 min (day) and 9.5 min (night). Superfusion of norepinephrine reduced the firing frequency of both cluster type…

MaleNitroprussidemedicine.medical_specialtyPhysiologyPeriod (gene)8-Bromo Cyclic Adenosine MonophosphateAction PotentialsBiologyNitric OxideNitroargininePineal GlandNitric oxideRats Sprague-DawleyRat Pineal GlandNorepinephrine (medication)NorepinephrineBehavioral Neurosciencechemistry.chemical_compoundInternal medicinemedicineAnimalsSympathomimeticsCyclic GMPPhenylephrineInhibitory effectEcology Evolution Behavior and SystematicsNeuronsPenicillamineSulfhydryl ReagentsIsoproterenolIn vitroRatsElectrophysiologyEndocrinologychemistryAnimal Science and ZoologyEx vivomedicine.drugJournal of Comparative Physiology A: Sensory, Neural, and Behavioral Physiology
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Ca2+-activated K+ channels mediate relaxation of forearm veins in chronic renal failure

2003

In arteries, agonists such as acetylcholine release an endothelium-derived hyperpolarizing factor (EDHF) that is neither nitric oxide nor prostacyclin.To examine the responses to acetylcholine in segments of forearm veins from patients with chronic renal failure who either had never received dialysis or had undergone long-term dialysis, and to determine the contribution of nitric oxide and EDHF to endothelium-dependent relaxation in veins from patients with chronic renal failure.Isometric tension was recorded in rings of forearm vein from 34 non-dialysed patients, 27 dialysed patients and 14 multiorgan donors (controls).Relaxation in response to acetylcholine was reduced in veins of non-dia…

MaleNitroprussidemedicine.medical_specialtyPhysiologyVasodilator AgentsVasodilationIn Vitro TechniquesNitric OxideVeinsNitric oxideBiological FactorsPotassium Channels Calcium-Activatedchemistry.chemical_compoundForearmQuinoxalinesInternal medicineInternal MedicinemedicineHumansEnzyme InhibitorsVeinOxadiazolesomega-N-MethylarginineVascular diseasebusiness.industryMiddle Agedmedicine.diseaseAcetylcholinePotassium channelVasodilationForearmEndocrinologymedicine.anatomical_structurechemistrycardiovascular systemKidney Failure ChronicFemaleNitric Oxide SynthaseCardiology and Cardiovascular MedicinebusinessAcetylcholineKidney diseasemedicine.drugJournal of Hypertension
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Nitric oxide as neuromodulator of sympathetic transmission in rat vas deferens.

1998

Summary Electrical field stimulation (EFS) of muscle strips in vitro elicited a tetrodotoxin (TTX)-sensitive biphasic contractile response consisting of a phasic component followed by a tonic one. The amplitude of both components of the response was impaired by Nω-nitro-L-arginine and potentiated by sodium nitroprusside. Cystamine caused a reduction in amplitude of both phasic and tonic componets of the response to EFS. Neither Nω-nitro-L-arginine, sodium nitroprusside, nor cystamine induced changes in the resting muscle tone, or in the contractile response to exogenous agonists ATP and noradrenaline (NA). The nitric oxide scavenger, 2-phenyl-4,4,5,5-tetramethylimidazoline-1-oxyl-3-oxide, i…

MaleNitroprussidemedicine.medical_specialtySympathetic Nervous SystemCystamineNeurotransmissionNitric OxideNitroarginineTonic (physiology)Nitric oxideCyclic N-Oxideschemistry.chemical_compoundVas DeferensCystamineInternal medicinemedicineExtracellularAnimalsEnzyme InhibitorsRats WistarPharmacologyGeneral NeuroscienceVas deferensImidazolesElectric StimulationRatsmedicine.anatomical_structureEndocrinologychemistryGuanylate CyclaseTetrodotoxinSodium nitroprussideNitric Oxide Synthasemedicine.drugMuscle ContractionJournal of autonomic pharmacology
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Plasma Arginine/Asymmetric Dimethylarginine Ratio and Incidence of Cardiovascular Events: A Case-Cohort Study.

2017

CONTEXT: Arginine, its methylated metabolites, and other metabolites related to the urea cycle have been independently associated with cardiovascular risk, but the potential causal meaning of these associations (positive for some metabolites and negative for others) remains elusive due to a lack of studies measuring metabolite changes over time. OBJECTIVE: To examine the association between baseline and 1-year concentrations of urea cycle metabolites and cardiovascular disease (CVD) in a case-cohort setting. DESIGN: A case-cohort study was nested within the Prevención con Dieta Mediterránea trial. We used liquid chromatography-tandem mass spectrometry to assess metabolite levels at baseline…

MaleOrnithineArginineEndocrinology Diabetes and MetabolismSistema cardiovascular -- MalaltiesClinical BiochemistryMyocardial Infarction030204 cardiovascular system & hematologyBiochemistryGastroenterologyCohort Studieschemistry.chemical_compound0302 clinical medicineEndocrinologyRisk FactorsTandem Mass SpectrometryProspective StudiesProspective cohort studyeducation.field_of_studyIncidenceHazard ratioMiddle AgedStrokeCardiovascular DiseasesFemalemedicine.medical_specialtyPopulation030209 endocrinology & metabolismContext (language use)Lower riskArginine03 medical and health sciencesInternal medicinemedicineHumanscardiovascular diseaseseducationClinical Research ArticlesDieta -- Mediterrània Regió de laAgedProportional Hazards Modelsomega-N-Methylargininebusiness.industryBiochemistry (medical)Case-control studyEndocrinologychemistryCase-Control StudiesMultivariate AnalysisCitrullinebusinessAsymmetric dimethylarginineChromatography Liquid
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Changes in arginine are inversely associated with type 2 diabetes: A case-cohort study in the PREDIMED trial

2018

The associations between arginine‐based metabolites and incident type 2 diabetes (T2D) are unknown. We employed a case‐cohort design, nested within the PREDIMED trial, to examine six plasma metabolites (arginine, citrulline, ornithine, asymmetric dimethylarginine [ADMA], symmetric dimethylarginine [SDMA] and N‐monomethyl‐l‐arginine [NMMA]) among 892 individuals (251 cases) for associations with incident T2D and insulin resistance. Weighted Cox models with robust variance were used. The 1‐year changes in arginine (adjusted hazard ratio [HR] per SD 0.68, 95% confidence interval [CI] 0.49, 0.95; Q4 vs. Q1 0.46, 95% CI 0.21, 1.04; P trend = 0.02) and arginine/ADMA ratio (adjusted HR per SD 0.73…

MaleOrnithineArginineEndocrinology Diabetes and MetabolismType 2 diabetes030204 cardiovascular system & hematologyDiet MediterraneanCohort Studieschemistry.chemical_compound0302 clinical medicineEndocrinologyRisk FactorsObservational studyCitrullineDiet Fat-RestrictedAged 80 and overIncidenceType 2 diabetesMiddle AgedOrnithineHomeostatic model assessmentFemaleCohort studyPopulation studymedicine.medical_specialtypopulation study type 2 diabetes030209 endocrinology & metabolismArginineArticle03 medical and health sciencesInsulin resistanceDiabetes mellitusInternal medicineInternal MedicinemedicineHumansAgedomega-N-Methylargininebusiness.industryInsulin resistancemedicine.diseaseDietary interventionEndocrinologyDiabetes Mellitus Type 2chemistryCase-Control StudiesCitrullineAsymmetric dimethylargininebusiness
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Modulatory effects of nitric oxide-active drugs on the anticonvulsant activity of lamotrigine in an experimental model of partial complex epilepsy in…

2007

Abstract Background The effects induced by administering the anticonvulsant lamotrigine, the preferential inhibitor of neuronal nitric oxide synthase 7-nitroindazole and the precursor of NO synthesis L-arginine, alone or in combination, on an experimental model of partial complex seizures (maximal dentate gyrus activation) were studied in urethane anaesthetized rats. The epileptic activity of the dentate gyrus was obtained through the repetitive stimulation of the angular bundle and maximal dentate gyrus activation latency, duration and post-stimulus afterdischarge duration were evaluated. Results Either Lamotrigine (10 mg kg-1) or 7-nitroindazole (75 mg kg-1) i.p. administration had an ant…

MalePARTIAL COMPLEX EPILEPSYIndazolesArgininemedicine.medical_treatmentLamotriginePharmacologyArginineLamotrigineNitric OxideSettore BIO/09 - Fisiologialcsh:RC321-571Nitric oxideCellular and Molecular Neurosciencechemistry.chemical_compoundEpilepsy Complex PartialmedicineAnimalsDrug InteractionsEnzyme InhibitorsRats Wistarlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryNitric oxide Lamotrigine epilepsy controlbiologyTriazinesExperimental modelGeneral NeuroscienceDentate gyruslcsh:QP351-495BrainElectric StimulationRatsNitric oxide synthaseDisease Models Animallcsh:Neurophysiology and neuropsychologyAnticonvulsantnervous systemchemistryDentate Gyrusbiology.proteinAnticonvulsantsNitric Oxide SynthaseResearch Articlemedicine.drugBMC Neuroscience
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Local increase of arginase activity in lesions of patients with cutaneous leishmaniasis in Ethiopia.

2012

Background Cutaneous leishmaniasis is a vector-borne disease that is in Ethiopia mainly caused by the parasite Leishmania aethiopica. This neglected tropical disease is common in rural areas and causes serious morbidity. Persistent nonhealing cutaneous leishmaniasis has been associated with poor T cell mediated responses; however, the underlying mechanisms are not well understood. Methodology/Principal Findings We have recently shown in an experimental model of cutaneous leishmaniasis that arginase-induced L-arginine metabolism suppresses antigen-specific T cell responses at the site of pathology, but not in the periphery. To test whether these results translate to human disease, we recruit…

MalePathologyCD3 ComplexBiopsyAntigens CD8Antigens CD3Antigens CD40302 clinical medicineINFECTIONSUPPRESSOR-CELLSAETHIOPICAChildImmune ResponseSOUTH-WESTERN ETHIOPIAIN-VIVOSkin0303 health sciencesbiologyPARASITOLOGYlcsh:Public aspects of medicine11 Medical And Health SciencesMiddle Aged3. Good healthArginaseInfectious Diseasesmedicine.anatomical_structureCD4 AntigensMedicineFemalemedicine.symptomLife Sciences & BiomedicineResearch ArticleNeglected Tropical DiseasesEXPRESSIONAdultmedicine.medical_specialtylcsh:Arctic medicine. Tropical medicineAdolescentlcsh:RC955-962CD8 AntigensT cellImmunology030231 tropical medicineLeishmaniasis CutaneousPeripheral blood mononuclear cellImmunomodulationLesionYoung Adult03 medical and health sciencesLeishmania aethiopicaCutaneous leishmaniasisTropical MedicineParasitic DiseasesL-ARGININE METABOLISMmedicineACTIVATED GRANULOCYTESHumansBiology030304 developmental biologyScience & TechnologyNITRIC-OXIDEArginasebusiness.industryPublic Health Environmental and Occupational HealthLeishmaniasislcsh:RA1-127006 Biological Sciencesbiology.organism_classificationmedicine.diseaseMICEImmunologyLeukocytes MononuclearEthiopiabusinessCD8PLoS Neglected Tropical Diseases
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Downregulation of nNOS and synthesis of PGs associated with endotoxin-induced delay in gastric emptying

2002

A single intraperitoneal injection of endotoxin (40 μg/kg) significantly delayed gastric emptying of a solid nutrient meal. Blockade of nitric oxide synthase (NOS) with 30 mg/kg ip N G-nitro-l-arginine methyl ester or 20 mg/kg ip 7-nitroindazole [neuronal NOS (nNOS) inhibitor] significantly delayed gastric emptying in control animals but failed to modify gastric emptying in endotoxin-treated rats. Administration of 2.5, 5, and 10 mg/kg ip N 6-iminoethyl-l-lysine [inducible NOS (iNOS) inhibitor] had no effect in either experimental group. Indomethacin (5 mg/kg sc), NS-398 (cyclooxygenase-2 inhibitor; 10 mg/kg ip), and dexamethasone (10 mg/kg sc) but not quinacrine (20 mg/kg ip) significantl…

MalePhysiologymedicine.medical_treatmentIndomethacinNitric Oxide Synthase Type IINitric Oxide Synthase Type IprostaglandinsRats Sprague-Dawleychemistry.chemical_compoundPyloric AntrumEnzyme InhibitorsAntrumSulfonamidesArachidonic AcidbiologyReverse Transcriptase Polymerase Chain ReactionStomachdigestive oral and skin physiologyGastroenterologyNitric oxide synthasemedicine.anatomical_structureNG-Nitroarginine Methyl EsterQuinacrinenutrient mealsantrum. pylorusmedicine.medical_specialtyIndazolesIntraperitoneal injectionNitric OxidePhospholipases ANitric oxidenitric oxidePhysiology (medical)Internal medicinemedicineAnimalsCyclooxygenase InhibitorsRNA MessengerNitrobenzenesHepatologyGastric emptyingPylorusdigestive system diseasesRatsEndotoxinsEndocrinologyPyloric AntrumchemistryGastric EmptyingFoodbiology.proteinProstaglandinsNitric Oxide Synthase
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