Search results for "ascorbic acid"

showing 10 items of 295 documents

Genetic and environmental contributions to serum ascorbic acid concentrations: the Stanislas Family Study.

2006

Although numerous environmental factors are documented to influence serum ascorbic concentrations, little is known about the genetic versus environmental contributions to variation of this trait. The aim of this study was to estimate family correlation and, additive genetic heritability and household effects in a variance component analysis for serum ascorbic acid concentrations. In a sample of ninety French families, information was obtained regarding serum ascorbic acid concentrations, usual dietary intake, lifestyle, and other related covariates. Spouse, parent –offspring and offspring –offspring significant correlation coefficients for serum ascorbic acid concentrations, adjusted for ag…

AdultMaleAdolescentOffspringVariance component analysisMedicine (miscellaneous)Ascorbic AcidBiologyAnimal scienceQuantitative Trait HeritableVegetablesHumansLongitudinal StudiesGeneticsFamily HealthNutrition and DieteticsDietary intakeSerum concentrationHeritabilityAscorbic acidHealth SurveysDiet RecordsDietContraceptive useFruitTraitRegression AnalysisFemaleThe British journal of nutrition
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In vivoprooxidant state in Werner syndrome (WS): Results from three WS patients and two WS heterozygotes

2005

The hypothesis was tested that Werner syndrome (WS) phenotype might be associated with an in vivo prooxidant state. A set of redox-related endpoints were measured in three WS patients, two of their parents, and 99 controls within a study of some cancer-prone and/or ageing-related genetic disorders. The following analytes were measured: (a) leukocyte 8-hydroxy-2'-deoxyguanosine; (b) glutathione from whole blood, and (c) plasma levels of glyoxal, methylglyoxal, 8-isoprostane, and some plasma antioxidants (uric acid, ascorbic acid, alpha- and gamma-tocopherol). Leukocyte 8-hydroxy-2'-deoxyguanosine levels showed a significant increase in the 3 WS patients vs. 85 controls (p<10(-7)). The disulf…

AdultMaleHeterozygotemedicine.medical_specialtyDinoprostmedicine.disease_causeBiochemistryAntioxidantschemistry.chemical_compoundIn vivoInternal medicineLeukocytesmedicineHumansDeoxyguanosineChromatography High Pressure LiquidMethylglyoxalDeoxyguanosine8-Hydroxy-2'-deoxyguanosineGlyoxalGeneral MedicineGlutathioneMiddle AgedPyruvaldehydeAscorbic acidGlutathioneEndocrinologychemistryBiochemistry8-Hydroxy-2'-DeoxyguanosineUric acidFemaleWerner SyndromeOxidation-ReductionOxidative stressFree Radical Research
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Clearance of Ascorbic Acid in Plasma in Patients Before Major Maxillofacial Surgery Compared With That in Volunteers

2002

Reduced concentration of ascorbic acid (AA) in plasma is often observed in tumor patients. Besides malnutrition, an increased AA consumption in tumor patients is discussed as a main reason for reduced AA concentration in plasma. A reduced AA concentration caused by malnutrition would not influence the total clearance (Cltot) of AA in plasma; however, a change in AA consumption would alter the Cltot. To investigate this further, a study was carried out on 20 patients hospitalized for major maxillofacial surgery and 20 volunteers. The Cltot of AA in plasma after bolus injection of AA (4 mg/kg body wt) in tumor patients was compared with that in volunteers. AA concentration in plasma (P0.05) w…

AdultMaleMaxillary NeoplasmsCancer Researchmedicine.medical_specialtyNutrition and Dieteticsbusiness.industryMedicine (miscellaneous)Ascorbic AcidMiddle AgedAscorbic acidSurgeryOncologyReduced concentrationArea Under CurveBlood plasmaArea under curveHumansMedicineFemaleIn patientFacial NeoplasmsbusinessAgedBolus injectionNutrition and Cancer
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Oxidative stress biomarkers in four Bloom syndrome (BS) patients and in their parents suggest in vivo redox abnormalities in BS phenotype.

2007

Objective: To evaluate an association of Bloom syndrome (BS) phenotype with an in vivo prooxidant state. Methods: The following endpoints were measured in 4 BS patients, their 6 parents, and 78 controls: a) leukocyte and urinary 8-hydroxy-2′-deoxyguanosine (8-OHdG); b) blood glutathione (GSSG and GSH), c) plasma levels of some plasma antioxidants (uric acid, UA, ascorbic acid, AA, α- and γ-tocopherol), and of glyoxal (Glx) and methylglyoxal (MGlx). Results: Leukocyte 8-OHdG levels were significantly increased in the 4 BS patients vs. 40 controls (p = 0.04), while the urinary 8-OHdG levels were non-significantly increased in BS patients. Glutathione disulfide levels and GSSG/GSH ratio were s…

AdultMaleParentsmedicine.medical_specialtyglyoxalAdolescentClinical Biochemistrymedicine.disease_causechemistry.chemical_compoundIn vivoInternal medicinemedicinemethylglyoxalLeukocytesHumansBloom syndromeChildoxidative streGlutathione DisulfideMethylglyoxalDeoxyguanosineGeneral MedicineGlutathioneMiddle AgedAscorbic acidmedicine.diseaseGlutathioneOxidative StressEndocrinologyPhenotypechemistryBiochemistry8-Hydroxy-2'-DeoxyguanosineUric acidGlutathione disulfideBloom syndromeFemaleOxidation-ReductionOxidative stressBiomarkersBloom SyndromeClinical biochemistry
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Oxidant and antioxidant status in mothers and their newborns according to birthweight

2008

The aim of this study is to determine the oxidant and antioxidant status in Algerian mothers and their newborns according to birth weight.Subjects for the study were consecutively recruited from Tlemcen hospital. 139 pregnant women and their newborns were included. The plasma total antioxidant activity (ORAC), vitamins A, C, E, hydroperoxides, carbonyl proteins, and erythrocyte antioxidant enzyme activities (catalase, glutathione peroxidase, glutathione reductase and superoxide dismutase) were measured on mothers and their newborns. Lipid and lipoprotein parameters were also determined. The results were assessed in accordance with small for gestational age (SGA), appropriate (AGA) and large…

AdultMalePediatricsmedicine.medical_specialtyErythrocytesAntioxidantLipoproteinsmedicine.medical_treatmentBirth weightGlutathione reductaseIntrauterine growth restrictionPhysiologyAscorbic AcidAntioxidantsPregnancymedicineBirth WeightHumansVitamin EVitamin Achemistry.chemical_classificationGlutathione PeroxidaseFetal Growth RetardationSuperoxide Dismutasebusiness.industryVitamin EGlutathione peroxidaseInfant NewbornObstetrics and GynecologyCatalaseOxidantsmedicine.diseaseAscorbic acidLipidsOxidative StressGlutathione ReductaseReproductive MedicinechemistryInfant Small for Gestational AgeSmall for gestational ageFemalebusinessEuropean Journal of Obstetrics &amp; Gynecology and Reproductive Biology
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Cigarette smoke causes caspase-independent apoptosis of bronchial epithelial cells from asthmatic donors

2015

BackgroundEpidemiologic studies have demonstrated important links between air pollution and asthma. Amongst these pollutants, environmental cigarette smoke is a risk factor both for asthma pathogenesis and exacerbation. As the barrier to the inhaled environment, the bronchial epithelium is a key structure that is exposed to cigarette smoke.ObjectivesSince primary bronchial epithelial cells (PBECs) from asthmatic donors are more susceptible to oxidant-induced apoptosis, we hypothesized that they would be susceptible to cigarette smoke-induced cell death.MethodsPBECs from normal and asthmatic donors were exposed to cigarette smoke extract (CSE); cell survival and apoptosis were assessed by fl…

AdultMaleProgrammed cell deathDNA damageScienceCaspase 3ApoptosisBronchiBiologyTobacco smokeAntioxidantschemistry.chemical_compoundYoung Adultparasitic diseasesHumansAgricultural and Biological Sciences (all); Biochemistry Genetics and Molecular Biology (all); Medicine (all)MultidisciplinaryBiochemistry Genetics and Molecular Biology (all)Caspase-Independent ApoptosisCaspase 3Medicine (all)QSmokingREpithelial CellsGlutathioneMiddle AgedAscorbic acid3. Good healthchemistryAgricultural and Biological Sciences (all)13. Climate actionApoptosisImmunologyMedicineFemaleResearch Article
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Effects of α-tocopherol, β-carotene and ascorbic acid on oxidative, hormonal and enzymatic exercise stress markers in habitual training activity of p…

2001

Background Intense physical exercise has been associated with an increase of free radical production. When the body's natural defense systems against free radicals are overwhelmed, oxidative stress increases. Aim of the study This study examined the effects of a vitamin antioxidant supplement, (composed of 600 mg α-tocopherol, 1000 mg ascorbic acid and 32 mg β-carotene) on oxidative, hormonal, and enzymatic exercise stress markers during habitual training activity over 35 days. Methods The plasma concentrations of ascorbic acid, α-tocopherol, β-carotene, testosterone, cortisol and lipid peroxides and the serum activities of lactate dehydrogenase and creatine kinase were measured at four tim…

AdultMaleVitaminmedicine.medical_specialtyAntioxidantHydrocortisoneAnabolismmedicine.medical_treatmentalpha-TocopherolMedicine (miscellaneous)Ascorbic AcidBasketballmedicine.disease_causeAntioxidantsLipid peroxidationchemistry.chemical_compoundInternal medicineLactate dehydrogenasemedicineHumansSingle-Blind MethodTestosteroneCreatine KinaseExerciseNutrition and DieteticsL-Lactate Dehydrogenasebeta CaroteneAscorbic acidOxidative StressEndocrinologychemistryDietary SupplementsLipid Peroxidationalpha-TocopherolOxidation-ReductionOxidative stressEuropean Journal of Nutrition
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Rosiglitazone Causes Endothelial Dysfunction in Humans

2011

We explored the impact of rosiglitazone on endothelial function in normal volunteers and its interaction with glyceryl trinitrate (GTN)-induced abnormalities in endothelial function. We hypothesized that rosiglitazone would have a neutral effect on endothelial function in normal volunteers and would favorably modify endothelial dysfunction induced by GTN.In this double-blind, randomized, placebo-controlled study, 44 participants were randomized to placebo, rosiglitazone (4 mg twice daily), transdermal GTN (0.6 mg/h), or both GTN and rosiglitazone. After 7 days of treatment, participants underwent measures of forearm blood flow during brachial artery infusion of acetylcholine (Ach). Serum gl…

AdultMalemedicine.medical_specialtyAdolescentEndotheliumVasodilator AgentsBlood PressureVasodilationAscorbic AcidPharmacologyPlaceboRosiglitazoneNitroglycerinYoung AdultDouble-Blind MethodHeart RateInternal medicinemedicine.arterymedicineHumansPharmacology (medical)Endothelial dysfunctionBrachial arteryPharmacologyDose-Response Relationship Drugbusiness.industrymedicine.diseaseAscorbic acidAcetylcholineVasodilationmedicine.anatomical_structureBlood pressureEndocrinologycardiovascular systemThiazolidinedionesEndothelium VascularCardiology and Cardiovascular MedicineRosiglitazonebusinesscirculatory and respiratory physiologymedicine.drugJournal of Cardiovascular Pharmacology and Therapeutics
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AZT treatment induces molecular and ultrastructural oxidative damage to muscle mitochondria. Prevention by antioxidant vitamins.

1998

AIDS patients who receive zidovudine (AZT) frequently suffer from myopathy. This has been attributed to mitochondrial (mt) damage, and specifically to the loss of mtDNA. This study examines whether AZT causes oxidative damage to DNA in patients and to skeletal muscle mitochondria in mice, and whether this damage may be prevented by supranutritional doses of antioxidant vitamins. Asymptomatic HIV-infected patients treated with AZT have a higher urinary excretion (355+/-100 pmol/kg/d) of 8-oxo-7, 8-dihydro-2'-deoxyguanosine (8-oxo-dG) (a marker of oxidative damage to DNA) than untreated controls (asymptomatic HIV-infected patients) (182+/-29 pmol/kg/d). This was prevented (110+/-79 pmol/kg/d)…

AdultMalemedicine.medical_specialtyDNA damageAnti-HIV Agentsmedicine.medical_treatmentAscorbic AcidBiologyDNA MitochondrialAntioxidantsZidovudinechemistry.chemical_compoundMiceInternal medicinemedicineDeoxyguanosineAnimalsHumansVitamin Eheterocyclic compoundsMyopathyVitamin ESkeletal musclevirus diseasesDeoxyguanosineGeneral MedicineGlutathioneHydrogen PeroxideAscorbic acidMitochondria Musclemedicine.anatomical_structureEndocrinologychemistryBiochemistry8-Hydroxy-2'-Deoxyguanosinemedicine.symptomZidovudinemedicine.drugDNA DamageResearch ArticleThe Journal of clinical investigation
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Different patterns of in vivo pro-oxidant states in a set of cancer- or aging-related genetic diseases

2008

A comparative evaluation is reported of pro-oxidant states in 82 patients with ataxia telangectasia (AT), Bloom syndrome (BS), Down syndrome (DS), Fanconi anemia (FA), Werner syndrome (WS), and xeroderma pigmentosum (XP) vs 98 control donors. These disorders display cancer proneness, and/or early aging, and/or other clinical features. The measured analytes were: (a) leukocyte and urinary 8-hydroxy-2'-deoxyguanosine (8-OHdG), (b) blood glutathione (GSSG and GSH), (c) plasma glyoxal (Glx) and methylglyoxal (MGlx), and (d) some plasma antioxidants [uric acid (UA) and ascorbic acid (AA)]. Leukocyte 8-OHdG levels ranked as follows: WS>BS approximately FA approximately XP>DS approximately AT appr…

AdultMalemedicine.medical_specialtyDown syndromeXeroderma pigmentosumAdolescentmedicine.disease_causeBiochemistrychemistry.chemical_compoundAtaxia TelangiectasiaPhysiology (medical)Internal medicinemedicineHumansBloom syndromeChildAgedXeroderma PigmentosumMethylglyoxalDeoxyguanosineGlutathioneGlyoxalMiddle AgedAscorbic acidmedicine.diseasePyruvaldehydeGlutathioneEndocrinologyFanconi AnemiaantioxidantschemistryBiochemistry8-Hydroxy-2'-DeoxyguanosineUric acidOxidative streFemaleWerner SyndromeDown SyndromeReactive Oxygen SpeciesOxidative stressBloom SyndromeDNA Damage
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