Search results for "binding proteins"

showing 10 items of 911 documents

Retinol-Binding Protein 4 Levels Are Associated with Measures of Liver and Renal Function and Oxidant/Antioxidant Status in Obese Children

2013

Serum retinol-binding protein 4 (RBP4) has been proposed as a metabolic risk factor in obesity. We found that RBP4 levels also were associated with liver enzymes and cystatin C. Oxidant stress is a significant feature in obese children with greater values of RBP4 that can mediate the development of comorbidities.

Malemedicine.medical_specialtyAdolescentRenal functionKidneyKidney Function TestsAntioxidantschemistry.chemical_compoundLiver Function TestsInternal medicinemedicineHumansObesityChildRetinol binding protein 4biologybusiness.industryMetabolic riskOxidant antioxidantGlutathioneOxidantsmedicine.diseaseObesityOxidative StressEndocrinologyLiverchemistryCystatin CPediatrics Perinatology and Child Healthbiology.proteinFemaleCystatinbusinessRetinol-Binding Proteins PlasmaThe Journal of Pediatrics
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Mesenchymal Stem Cells Improve Motor Functions and Decrease Neurodegeneration in Ataxic Mice

2014

The main objective of this work is to demonstrate the feasibility of using bone marrow-derived stem cells in treating a neurodegenerative disorder such as Friedreich's ataxia. In this disease, the dorsal root ganglia of the spinal cord are the first to degenerate. Two groups of mice were injected intrathecally with mesenchymal stem cells isolated from either wild-type or Fxntm1Mkn/Tg(FXN)YG8Pook (YG8) mice. As a result, both groups presented improved motor skills compared to nontreated mice. Also, frataxin expression was increased in the dorsal root ganglia of the treated groups, along with lower expression of the apoptotic markers analyzed. Furthermore, the injected stem cells expressed th…

Malemedicine.medical_specialtyAtaxiaCellular differentiationGene ExpressionBone Marrow CellsMice TransgenicMotor ActivityMesenchymal Stem Cell TransplantationTransplantation AutologousMiceGlutathione Peroxidase GPX1Neurotrophin 3Internal medicineGanglia SpinalIron-Binding ProteinsDrug DiscoverymedicineGeneticsAnimalsTransplantation HomologousNerve Growth FactorsMolecular BiologyInjections SpinalPharmacologyGlutathione PeroxidasebiologyBrain-Derived Neurotrophic FactorMesenchymal stem cellCell DifferentiationMesenchymal Stem CellsAnatomySpinal cordCatalaseDisease Models AnimalEndocrinologymedicine.anatomical_structureFriedreich AtaxiaFrataxinbiology.proteinMolecular MedicineOriginal ArticleFemaleBone marrowmedicine.symptomStem cellAdult stem cell
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Infrequent co-existence of nitric oxide synthase and parvalbumin, calbindin and calretinin immunoreactivity in rat pontine neurons.

1995

Neurons in the laterodorsal tegmental nucleus (LDTg), ventrolateral dorsal tegmental nucleus (LDTgV), pedunculopontine tegmental nucleus (PPTg), lateral and medial parabrachial nuclei (LPB and MPB) were immunoreactive to brain nitric oxide synthase (NOS) or isoform I. Double-labeling experiments showed that very few NOS-containing neurons in the pons were immunoreactive to any of the three calcium-binding proteins: calbindin-D 28K (CB-IR), parvalbumin (PV-IR) and calretinin (CR-IR). These findings extend our previous observation in the neocortex and suggest that a population of central NOS-containing neurons can be neurochemically characterized as CB/CR/PV deficient.

Malemedicine.medical_specialtyCalbindinsNerve Tissue ProteinsCalbindinRats Sprague-DawleyS100 Calcium Binding Protein GInternal medicinePonsTegmentummedicineAnimalsPedunculopontine Tegmental NucleusNeuronsParabrachial NucleusbiologyStaining and LabelingChemistryGeneral NeuroscienceCalcium-Binding ProteinsPonsRatsLaterodorsal tegmental nucleusmedicine.anatomical_structureEndocrinologyParvalbuminsnervous systemCalbindin 2biology.proteinImmunologic TechniquesCalmodulin-Binding ProteinsFemaleAmino Acid OxidoreductasesCalretininNitric Oxide SynthaseParvalbuminNeuroscience letters
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Altered expression of neuropeptides in the primary somatosensory cortex of the Down syndrome model Ts65Dn.

2011

Down syndrome is the most common genetic disorder associated with mental retardation. Subjects and mice models for Down syndrome (such as Ts65Dn) show defects in the formation of neuronal networks in both the hippocampus and the cerebral cortex. The principal neurons display alterations in the morphology, density and distribution of dendritic spines in the cortex as well as in the hippocampus. Several evidences point to the possibility that the atrophy observed in principal neurons could be mediated by changes in their inhibitory inputs and, in fact, an imbalance between excitation and inhibition has been observed in Ts65Dn mice in these regions, which are crucial for learning and informati…

Malemedicine.medical_specialtyDendritic spineHippocampusBiologySomatosensory systemCalbindinHippocampusCellular and Molecular NeuroscienceMiceEndocrinologyInterneuronsCortex (anatomy)Internal medicinemedicineAnimalsNeuronsEndocrine and Autonomic SystemsCalcium-Binding ProteinsNeuropeptidesGeneral MedicineSomatosensory CortexDisease Models Animalmedicine.anatomical_structureEndocrinologySomatostatinnervous systemNeurologyCerebral cortexCalretininDown SyndromeSomatostatinNeuroscienceNeuropeptides
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Evaluation of the therapeutic potential of PPARalpha agonists for X-linked adrenoleukodystrophy.

2003

Adrenoleukodystrophy protein (ABCD1), a peroxisomal membrane protein, is mutated in patients affected by X-linked adrenoleukodystrophy (X-ALD). Adrenoleukodystrophy-related protein (ABCD2) is the closest relative of ABCD1. Pharmacological induction of ABCD2 gene expression has been proposed as a novel therapy strategy for X-ALD. Fibrates induce peroxisome proliferation and Abcd2 expression in rodent liver. Here we evaluate the possibility of using peroxisome proliferator-activated receptor alpha (PPARalpha) agonists for pharmacological induction of ABCD2 expression. In the liver of PPARalpha-deficient mice, both the constitutive and the fenofibrate-inducible Abcd2 gene expression was found …

Malemedicine.medical_specialtyEndocrinology Diabetes and MetabolismMolecular Sequence DataDrug Evaluation PreclinicalPeroxisome ProliferationReceptors Cytoplasmic and NuclearBiologySulfidesATP Binding Cassette Transporter Subfamily DResponse ElementsBiochemistrychemistry.chemical_compoundMiceEndocrinologyInternal medicineGene expressionGeneticsmedicineAnimalsAdrenoleukodystrophyMolecular BiologyGenePhenylurea CompoundsTetradecylthioacetic acidBrainmedicine.diseaseMolecular biologyIntronsMice Mutant StrainsSterol regulatory element-binding proteinDNA-Binding ProteinsMice Inbred C57BLButyratesSterolsEndocrinologychemistryGene Expression RegulationLiverCCAAT-Enhancer-Binding ProteinsSterol Regulatory Element Binding Protein 1AdrenoleukodystrophyATP-Binding Cassette TransportersSterol regulatory element-binding protein 2Sterol Regulatory Element Binding Protein 1Sterol Regulatory Element Binding Protein 2Transcription FactorsMolecular genetics and metabolism
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Low Prevalence of Celiac Disease among Patients with Functional Gastrointestinal Disorders in Latvia.

2020

Background and Aims: Studies suggest that the prevalence of celiac disease (CD) is increased in individuals with functional gastrointestinal disorders (FGIDs), in particular, irritable bowel syndrome (IBS); however, the evidence is conflicting. We aimed to analyze the prevalence of CD in patients with FGIDs in Latvia.
 Methods: This retrospective study included patients with FGIDs, referred for a gastroenterologist consultation in a secondary gastroenterology practice unit. Patients were divided into three groups – patients only with IBS (IBS group), patients only with functional dyspepsia (FD) (FD group), patients with mixed symptoms IBS and FD (Mixed group). Patient levels of tissue …

Malemedicine.medical_specialtyGastrointestinal DiseasesDiseaseGastroenterologySerologyIrritable Bowel SyndromeGTP-Binding ProteinsInternal medicineBiopsymedicinePrevalenceHumansMixed groupIn patientProtein Glutamine gamma Glutamyltransferase 2Serologic TestsDuodenoscopyIrritable bowel syndromeAutoantibodiesRetrospective StudiesRoutine screeningTransglutaminasesmedicine.diagnostic_testbusiness.industryGastroenterologyRetrospective cohort studyMiddle Agedmedicine.diseaseLatviaImmunoglobulin ACeliac DiseaseFemaleSymptom AssessmentbusinessJournal of gastrointestinal and liver diseases : JGLD
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Muscarinic receptors, leukotriene B4 production and neutrophilic inflammation in COPD patients.

2005

Background:  Acetylcholine (ACh) plays an important role in smooth muscle contraction and in the development of airway narrowing; preliminary evidences led us to hypothesize that ACh might also play a role in the development of airways inflammation in chronic obstructive pulmonary disease (COPD). Methods:  We evaluated the concentrations of leukotriene B4 (LTB4) in induced sputum, and the expression of Ach M1, M2, and M3 receptors in sputum cells (SC) obtained from 16 patients with COPD, 11 smokers, and 14 control subjects. The SC were also treated with ACh and the production of LTB4 assessed in the presence or absence of a muscarinic antagonist (oxitropium). In blood monocytes, we evaluate…

Malemedicine.medical_specialtyLeukotriene B4NeutrophilsImmunologyProtozoan ProteinsLeukotriene B4chemistry.chemical_compoundPulmonary Disease Chronic ObstructiveInternal medicineMuscarinic acetylcholine receptorImmunology and AllergyMedicineHumansReceptorCells CulturedAgedFlavonoidsCOPDMitogen-Activated Protein Kinase 3business.industryCalcium-Binding ProteinsSputumMuscarinic acetylcholine receptor M3Muscarinic antagonistSmooth muscle contractionMiddle Agedmedicine.diseaseImmunohistochemistryReceptors MuscarinicAcetylcholinerespiratory tract diseasesChemotaxis LeukocyteEndocrinologychemistryLeukocytes MononuclearFemalebusinessAcetylcholinemedicine.drugAllergy
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Left ventricular diastolic function and cardiometabolic factors in obese normotensive children

2014

Abstract Background and aim Left ventricular (LV) hypertrophy and diastolic function have been found to be associated with obesity and hypertension in adults. However, there are scarce data about the association of obesity itself to cardiac alteration in children. The aim of this study was to detect early changes in LV structure and function in obese children and whether they are associated with the biomarkers of metabolic risk and endothelial activation. Methods and results A total of 130 children aged 7–16 years (88 obese and 42 normal-weight children) were studied. All children had normal resting blood pressure. Two-dimensional ultrasound with M-mode imaging was performed to assess the L…

Malemedicine.medical_specialtyMean arterial pressureAdolescentHeart VentriclesEndocrinology Diabetes and MetabolismDiastoleVascular Cell Adhesion Molecule-1Medicine (miscellaneous)Left ventricular hypertrophyBody Mass IndexMuscle hypertrophyEndothelial activationVentricular Dysfunction LeftRisk FactorsInternal medicinemedicineHumansMass indexObesityProspective StudiesChildUltrasonographyMetabolic SyndromeNutrition and DieteticsApolipoprotein A-Ibusiness.industrymedicine.diseaseCross-Sectional StudiesEarly DiagnosisBlood pressureSolubilityCardiovascular DiseasesSpainCardiologyFemaleCardiology and Cardiovascular MedicinebusinessRetinol-Binding Proteins PlasmaBody mass indexBiomarkersNutrition, Metabolism and Cardiovascular Diseases
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Glucocorticoids as modulators of expression and activity of Antithrombin (At): potential clinical relevance.

2014

Abstract Introduction An inverse relationship has been reported between decreased postoperative Antithrombin (AT) plasmatic levels and the incidence of complications. We hypothesized that Nuclear Hormone Receptors could modulate the expression of SERPINC1 , encoding AT, through a Hormone Regulatory Element present in its promoter, and thus hormone analogs could be a pharmacological complement in surgical procedures to activate endogenous AT synthesis. Materials and Methods The expression of SERPINC1 was analyzed in HepG2 cells by quantitative RT-PCR and Western Blot. Two studies were conducted with (a) patients submitted to cardiac surgery with cardiopulmonary bypass receiving (n =17) or no…

Malemedicine.medical_specialtyMolecular Sequence DataReceptors Cytoplasmic and NuclearRetinoid X receptorLigandsAntithrombinsCohort StudiesRetinoidsInternal medicinemedicineHumansGlucocorticoidsDexamethasoneAgedCardiopulmonary BypassBase Sequencebusiness.industryAntithrombinRNA-Binding ProteinsHematologyHep G2 CellsIsoxazolesMiddle AgedEndocrinologyRetinoid X ReceptorsTreatment OutcomeMethylprednisoloneNuclear receptorHemostasisFemaleCortisonebusinessHormonemedicine.drugThrombosis research
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TET2 gene mutation is a frequent and adverse event in chronic myelomonocytic leukemia

2009

Background Acquired somatic deletions and loss-of-function mutations in one or several codons of the TET2 ( Ten-Eleven Translocation-2 ) gene were recently identified in hematopoietic cells from patients with myeloid malignancies, including myeloproliferative disorders and myelodys-plastic syndromes. The present study was designed to determine the prevalence of TET2 gene alterations in chronic myelomonocytic leukemias. Design and Methods Blood and bone marrow cells were collected from 88 patients with chronic phase chronic myelomonocytic leukemia and from 14 with acute transformation of a previously identified disease. Polymerase chain reaction analysis and direct sequencing were used to se…

Malemedicine.medical_specialtyMyeloidDNA Mutational AnalysisChronic myelomonocytic leukemiaSingle-nucleotide polymorphismKaplan-Meier EstimateGene mutationBiologymedicine.disease_causeDioxygenasesGene FrequencyMonocytosisInternal medicinehemic and lymphatic diseasesProto-Oncogene ProteinsmedicineHumansGenetic Predisposition to DiseaseLetters to the EditorAgedProportional Hazards ModelsAged 80 and overComparative Genomic HybridizationMutationHematologyLeukemia Myelomonocytic ChronicHematologyMiddle Agedmedicine.diseaseMyelodysplastic-Myeloproliferative DiseasesDNA-Binding ProteinsLeukemiamedicine.anatomical_structureImmunologyMutationFemaleOriginal Article
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