Search results for "blood vessels"

showing 10 items of 94 documents

Progressive Development of Renal Vascular Dysfunction in Brain Death Implicates Reversible Alterations of Nitric Oxide Metabolism

2011

Vascular endothelial dysfunction occurs in the kidney graft from marginal brain death (BD) donors and may be responsible for a low success rate after transplantation.BD was induced in 16 dogs for 6 hours. Immediately after the inflation of the intracranial balloon, the treated group (n = 8) received 40 mg/kg bolus followed by 3 mg/kg/min infusion of L-arginine for 30 minutes. Renal vascular function and hemodynamic and biochemical parameters were determined.BD caused vasoconstriction, increase in renal venous nitrite (4.9 ± 0.8 versus 2.6 ± 0.1, P.05) and myeloperoxidase levels (1.43 ± 0.04 versus 2.43 ± 0.23, P.001), and reduced vasodilatation of renal artery to acetylcholine. Larginine di…

Brain Deathmedicine.medical_specialtyHemodynamicsKidneyNitric Oxidechemistry.chemical_compoundDogsBolus (medicine)Internal medicinemedicineAnimalsNitriteEndothelial dysfunctionTransplantationKidneybusiness.industryHemodynamicsNitric oxide metabolismmedicine.diseaseTransplantationmedicine.anatomical_structureEndocrinologychemistryAnesthesiaDisease ProgressionBlood VesselsSurgerymedicine.symptombusinessVasoconstrictionTransplantation Proceedings
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Collagen-embedded hydroxylapatite–beta-tricalcium phosphate–silicon dioxide bone substitute granules assist rapid vascularization and promote cell gr…

2010

In the present study we assessed the biocompatibility in vitro and in vivo of a low-temperature sol-gel-manufactured SiO(2)-based bone graft substitute. Human primary osteoblasts and the osteoblastic cell line, MG63, cultured on the SiO(2) biomatrix in monoculture retained their osteoblastic morphology and cellular functionality in vitro. The effect of the biomaterial in vivo and its vascularization potential was tested subcutaneously in Wistar rats and demonstrated both rapid vascularization and good integration within the peri-implant tissue. Scaffold degradation was progressive during the first month after implantation, with tartrate-resistant acid phosphatase-positive macrophages being …

Calcium PhosphatesScaffoldMaterials scienceBiocompatibilityBiomedical EngineeringNeovascularization PhysiologicBioengineeringCell LineBiomaterialschemistry.chemical_compoundVasculogenesisIn vivoMaterials TestingHumansCell ProliferationOsteoblastsCell growthBiomaterialHydroxylapatiteSilicon DioxideIn vitroCell biologychemistryBone SubstitutesBlood VesselsCollagenBiomedical engineeringBiomedical Materials
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Ceramide Mediates Acute Oxygen Sensing in Vascular Tissues

2014

AbstractAims: A variety of vessels, such as resistance pulmonary arteries (PA) and fetoplacental arteries and the ductus arteriosus (DA) are specialized in sensing and responding to changes in oxygen tension. Despite opposite stimuli, normoxic DA contraction and hypoxic fetoplacental and PA vasoconstriction share some mechanistic features. Activation of neutral sphingomyelinase (nSMase) and subsequent ceramide production has been involved in hypoxic pulmonary vasoconstriction (HPV). Herein we aimed to study the possible role of nSMase-derived ceramide as a common factor in the acute oxygen-sensing function of specialized vascular tissues. Results: The nSMase inhibitor GW4869 and an anticera…

CeramideContraction (grammar)PhysiologyClinical BiochemistryGene ExpressionChick EmbryoPulmonary ArterySphingomyelin phosphodiesteraseBiologyCeramidesBenzylidene CompoundsBiochemistrychemistry.chemical_compoundHypoxic pulmonary vasoconstrictionmedicineAnimalsHumansHypoxiaMolecular BiologyGeneral Environmental Sciencechemistry.chemical_classificationReactive oxygen speciesAniline CompoundsDuctus ArteriosusCell BiologyCell biologyOxygen tensionOxygenOriginal Research CommunicationsSphingomyelin PhosphodiesteraseShaw Potassium ChannelsBiochemistrychemistryVasoconstrictionBlood VesselsGeneral Earth and Planetary Sciencesmedicine.symptomReactive Oxygen SpeciesSphingomyelinVasoconstrictionAntioxidants & Redox Signaling
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Reinterpreting the Magnetic Resonance Signs of Hemodynamic Impairment in the Brains of Multiple Sclerosis Patients From the Perspective of a Recent D…

2010

Multiple sclerosis patients examined with perfusion magnetic resonance (MR) imaging techniques have been found to have patterns of abnormal blood flow. These include prolonged mean transit time, a trend toward decreased cerebral blood flow in the area of plaques, and decreased cerebral blood flow and prolonged mean transit time within normal-appearing white matter. In-creased cerebral blood flow and volume and decreased mean transit time (compared with the baseline values before the relapse) were found to precede the development of plaques. In addition, susceptibility-weighted imaging utilizing deoxyhemoglobin as the contrast has revealed that venous blood in cerebral veins of multiple scle…

Cerebral veinsmedicine.medical_specialtymedicine.diagnostic_testbusiness.industryMultiple sclerosisHemodynamicsBrainMagnetic resonance imagingVenous bloodmedicine.diseasemultiple sclerosisVeinsWhite matterblood vesselsCellular and Molecular Neurosciencemedicine.anatomical_structureCerebral blood flowCerebrovascular CirculationmedicineHumansmagnetic resonance imagingRadiologybusinessPerfusionJournal of Neuroscience Research
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Thromboinflammation and Vascular Dysfunction.

2018

Thromboinflammation and vascular dysfunction are inseparably combined with the innate and the adaptive immune response. While the role of this interplay has gained considerable attention in the arena of atherosclerosis/atherothrombosis and in deep vein thrombosis, its role in other forms of vascular disease and risk factors is currently emerging. In this brief review, we will focus on thromboinflammation with regard to cytokine signalling as well as on the novel role of a vascular coagulation-inflammatory circuit in arterial hypertension.Thrombo-Inflammation und vaskuläre Dysfunktion sind untrennbar mit dem angeborenen und dem erworbenen Immunsystem verbunden. Dieses Zusammenspiel wurde im …

Deep veinInflammation030204 cardiovascular system & hematologyBioinformatics03 medical and health sciences0302 clinical medicineImmune systemCoagulation cascademedicineAnimalsHumansCytokine signallingBlood CoagulationInflammationbusiness.industryVascular diseaseThrombosisHematologymedicine.diseaseAcquired immune systemThrombosismedicine.anatomical_structureBlood Vesselsmedicine.symptombusiness030215 immunologyHamostaseologie
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Caffeine and cardiovascular diseases: critical review of current research.

2015

Caffeine is a most widely consumed physiological stimulant worldwide, which is consumed via natural sources, such as coffee and tea, and now marketed sources such as energy drinks and other dietary supplements. This wide use has led to concerns regarding the safety of caffeine and its proposed beneficial role in alertness, performance and energy expenditure and side effects in the cardiovascular system. The question remains "Which dose is safe?", as the population does not appear to adhere to the strict guidelines listed on caffeine consumption. Studies in humans and animal models yield controversial results, which can be explained by population, type and dose of caffeine and low statistica…

DrugSettore BIO/17 - Istologiamedia_common.quotation_subjectmedicine.medical_treatmentPopulationMedicine (miscellaneous)030204 cardiovascular system & hematologyCoffeeToxicology03 medical and health scienceschemistry.chemical_compound0302 clinical medicineMeta-Analysis as TopicEnvironmental healthCaffeinemedicineAnimalsHumanseducationmedia_commonMetabolic Syndromeeducation.field_of_studyNutrition and DieteticsDose-Response Relationship Drugbusiness.industryClinical Studies as TopicInsulin sensitivityHeartStimulantAlertnessDisease Models AnimalEnergy expenditurechemistryCaffeine consumptionCardiovascular diseases Caffeine Cardioprotective effects Pathogenesis Clinical studies Experimental studiesCardiovascular DiseasesBlood VesselsCaffeinebusiness030217 neurology & neurosurgeryEuropean journal of nutrition
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EGFL7 ligates αvβ3 integrin to enhance vessel formation

2013

Angiogenesis, defined as blood vessel formation from a preexisting vasculature, is governed by multiple signal cascades including integrin receptors, in particular integrin αVβ3. Here we identify the endothelial cell (EC)-secreted factor epidermal growth factor-like protein 7 (EGFL7) as a novel specific ligand of integrin αVβ3, thus providing mechanistic insight into its proangiogenic actions in vitro and in vivo. Specifically, EGFL7 attaches to the extracellular matrix and by its interaction with integrin αVβ3 increases the motility of EC, which allows EC to move on a sticky underground during vessel remodeling. We provide evidence that the deregulation of EGFL7 in zebrafish embryos leads …

EGF Family of ProteinsEmbryo NonmammalianAngiogenesisAmino Acid MotifsImmunologyIntegrinGene ExpressionMice NudeEndothelial Growth FactorsBiochemistryCollagen receptorMiceCell MovementCell AdhesionHuman Umbilical Vein Endothelial CellsmedicineAnimalsHumansImmunoprecipitationPhosphorylationZebrafishbiologyReverse Transcriptase Polymerase Chain ReactionChemistryCalcium-Binding ProteinsInfarction Middle Cerebral ArteryVenous plexusCell BiologyHematologyIntegrin alphaVbeta3ImmunohistochemistryExtracellular MatrixCell biologyEndothelial stem cellHEK293 Cellsmedicine.anatomical_structureIntegrin alpha MImmunologybiology.proteinBlood VesselsRNA InterferenceIntegrin beta 6Protein BindingBlood vesselBlood
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The effects of ergot and non-ergot-derived dopamine agonists in an experimental mouse model of endometriosis

2011

Implantation of a retrogradely shed endometrium during menstruation requires an adequate blood supply, which allows the growth of endometriotic lesions. This suggests that the development of endometriosis can be impaired by inhibiting angiogenesis. The growth of endometriotic foci is impaired by commercial oncological antiangiogenic drugs used to block vascular endothelial growth factor (VEGF) signaling. The dopamine agonist cabergoline (Cb2) inhibits the growth of established endometriosis lesions by exerting antiangiogenic effects through VEGFR2 inactivation. However, the use of ergot-derived Cb2 is associated with an increased incidence of cardiac valve regurgitation. To evaluate the pot…

Embryologymedicine.medical_specialtyCabergolineProliferation indexAngiogenesisEndometriosisEndometriosisCell CountPharmacologyDopamine agonistClavicepsNeovascularizationEndometriumMicechemistry.chemical_compoundEndocrinologyInternal medicineCabergolinemedicineAnimalsHumansErgolinesCell ProliferationUterine DiseasesNeovascularization PathologicReceptors Dopamine D2business.industryQuinagolideObstetrics and GynecologyCell Biologymedicine.diseaseVascular Endothelial Growth Factor Receptor-2Vascular endothelial growth factorDisease Models AnimalEndocrinologyReproductive MedicinechemistryDopamine AgonistsBlood VesselsFemalemedicine.symptombusinessmedicine.drugREPRODUCTION
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Anti-angiogenic drug loaded liposomes: Nanotherapy for early atherosclerotic lesions in mice.

2018

Este artículo se encuentra disponible en la página web de la revista en la siguiente URL: https://journals.plos.org/plosone/article?id=10.1371/journal.pone.0190540 También participan en la elaboración de este artículo científico: Aracely Calatayud-Pascual, Alicia López-Castellano, Elena P. Albelda, Enrique García-España, Luis Martí-Bonmatí, Juan C. Frias y M. Teresa Albelda. Fumagillin-loaded liposomes were injected into ApoE-KO mice. The animals were divided into several groups to test the efficacy of this anti-angiogenic drug for early treatment of atherosclerotic lesions. Statistical analysis of the lesions revealed a decrease in the lesion size after 5 weeks of treatment.

Fluorescence-lifetime imaging microscopyPathologylcsh:MedicineAngiogenesis Inhibitors02 engineering and technology030204 cardiovascular system & hematologyVascular MedicineBiochemistryArteriosclerosis - Chemotherapy.Diagnostic RadiologyAteroesclerosis - Farmacoterapia.MiceWhite Blood Cells0302 clinical medicineAnimal CellsArteriosclerosis - Farmacoterapia.Medicine and Health SciencesArteries - Diseases - Treatment.Nanotechnologylcsh:ScienceAortaPhospholipidsmedia_commonMice KnockoutLiposomeDrug CarriersMultidisciplinarymedicine.diagnostic_testRadiology and Imaging021001 nanoscience & nanotechnologyMagnetic Resonance ImagingLipidsFatty Acids UnsaturatedEngineering and Technologymedicine.symptomCellular Structures and OrganellesCellular TypesAnatomy0210 nano-technologySesquiterpenesResearch ArticleDrugmedicine.medical_specialtyImaging Techniquesmedia_common.quotation_subjectImmune CellsImmunologyLiposomes.Research and Analysis MethodsLiposomas.Lesion03 medical and health sciencesText miningApolipoproteins ECyclohexanesDiagnostic Medicinemedicine.arteryFluorescence ImagingmedicineAnimalsArterias - Enfermedades - Tratamiento.VesiclesAortaBlood Cellsbusiness.industryMacrophageslcsh:RAnti angiogenicBiology and Life SciencesMagnetic resonance imagingCell BiologyAtherosclerosisFumagillin - Therapeutic use.Atherosclerosis - Chemotherapy.Disease Models AnimalFumagilina - Uso terapéutico.LiposomesCardiovascular AnatomyNanoparticlesBlood Vesselslcsh:QbusinessPloS one
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Hsp70 is required for optimal cell proliferation in mouse A6 mesoangioblast stem cells.

2009

Mouse Hsp70 (70 kDa heat shock protein) is preferentially induced by heat or stress stimuli. We previously found that Hsp70 is constitutively expressed in A6 mouse mesoangioblast stem cells, but its possible role in these cells and the control of its basal transcription remained unexplored. Here we report that in the absence of stress, Ku factor is able to bind the HSE (heat shock element) consensus sequence in vitro, and in vivo it is bound to the proximal hsp70 promoter. In addition, we show that constitutive hsp70 transcription depends on the co-operative interaction of different factors such as Sp1 (specificity protein 1) and GAGA-binding protein with Ku factor, which binds the HSE cons…

Gene knockdownMesoangioblastBinding SitesGeneral transcription factorCell growthStem CellsCell BiologyBiologyFlow CytometryBiochemistryMolecular biologyHsp70MiceTranscription (biology)Heat shock proteinAnimalsBlood VesselsHSP70 Heat-Shock ProteinsRNA InterferenceStem cellmesoangioblast RNAi doubling timePromoter Regions GeneticMolecular BiologyCell ProliferationTranscription FactorsThe Biochemical journal
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