Search results for "bowel"

showing 10 items of 637 documents

Clinical benefit of vedolizumab on articular manifestations in patients with active spondyloarthritis associated with inflammatory bowel disease.

2017

Vedolizumab (VDZ) is a new biological agent which was recently approved for the treatment of inflammatory bowel disease (IBD)1 following the good clinical responses reported by clinical trials for both Crohn's disease2 and ulcerative colitis.3 However, the effects of VDZ on extraintestinal manifestations were not reported in these trials, and the ‘real life’ experience is still limited. On these premises, we read with interest the recent work by Varkas et al 4 reporting a series of five patients with IBD who were treated with VDZ and promptly developed new onset or exacerbation of spondyloarthritis (SpA), irrespective of the response to treatment on intestinal symptoms. Although the hypothe…

Genetics and Molecular Biology (all)medicine.medical_specialtyExacerbationT CellImmunologyPremisesAntibodies Monoclonal HumanizedBiochemistryInflammatory bowel diseaseGastroenterologyGeneral Biochemistry Genetics and Molecular BiologyNew onsetVedolizumab03 medical and health sciences0302 clinical medicineRheumatologyInternal medicineSpondylarthritismedicineImmunology and AllergyHumansIn patientSacroiliitis030203 arthritis & rheumatologyBiochemistry Genetics and Molecular Biology (all)business.industrymedicine.diseaseInflammatory Bowel Diseasesdigestive system diseasesRheumatologyTreatmentClinical trial030211 gastroenterology & hepatologySpondyloarthritis; T Cells; Treatment; Rheumatology; Immunology and Allergy; Immunology; Biochemistry Genetics and Molecular Biology (all)Spondyloarthritibusinessmedicine.drugAnnals of the rheumatic diseases
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Genotype-Phenotype Analysis across 130,422 Genetic Variants Identifies Rspo3 as the First Genome-Wide Significant Modifier Gene in Primary Sclerosing…

2016

Background Ulcerative colitis (UC), a complex polygenic disorder, is one of the main subphenotypes of inflammatory bowel disease. A comprehensive dissection of the genetic etiology of UC needs to assess the contribution of rare genetic variants including copy number variations (CNVs) to disease risk. In this study, we performed a multi-step genome-wide case-control analysis to interrogate the presence of disease-relevant rare copy number variants. Methods One thousand one hundred twenty-one German UC patients and 1770 healthy controls were initially screened for rare deletions and duplications employing SNP-array data. Quantitative PCR and high density custom array-CGH were used for validat…

GeneticsHepatologyIn silicoBiologymedicine.diseaseGenomeInflammatory bowel diseasePrimary sclerosing cholangitis03 medical and health sciences0302 clinical medicine030220 oncology & carcinogenesisGene duplicationGenotypemedicine030211 gastroenterology & hepatologyCopy-number variationGeneJournal of Hepatology
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Telemedicina en el manejo de pacientes con enfermedad inflamatoria intestinal

2017

Inflammatory bowel disease (IBD) is a chronic and relapsing disorder with significant medical, social and financial impacts. IBD patients require continuous follow-up, and healthcare resource use in this context increases over time. In the last decade, telemedicine has influenced the treatment of chronic diseases like IBD via the application of information and communication technologies to provide healthcare services remotely. Telemedicine and its various applications (telemanagement, teleconsulting and tele-education) enable closer follow-up and provide education resources that promote patient empowerment, encouraging treatment optimisation over the entire course of the disease. We describ…

Gerontologymedicine.medical_specialtyTelemedicineHepatologybusiness.industryPatient EmpowermentGastroenterologyContext (language use)Diseasemedicine.diseaseHealth outcomesInflammatory bowel diseasedigestive system diseases03 medical and health sciences0302 clinical medicine030220 oncology & carcinogenesisHealth caremedicineResource use030211 gastroenterology & hepatologybusinessIntensive care medicineGastroenterología y Hepatología
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Co-factors, Microbes, and Immunogenetics in Celiac Disease to Guide Novel Approaches for Diagnosis and Treatment.

2021

Celiac disease (CeD) is a frequent immune-mediated disease that affects not only the small intestine but also many extraintestinal sites. The role of gluten proteins as dietary triggers, HLA-DQ2 or -DQ8 as major necessary genetic predisposition, and tissue transglutaminase (TG2) as mechanistically involved autoantigen, are unique features of CeD. Recent research implicates many cofactors working in synergism with these key triggers, including the intestinal microbiota and their metabolites, nongluten dietary triggers, intestinal barrier defects, novel immune cell phenotypes, and mediators and cytokines. In addition, apart from HLA-DQ2 and -DQ8, multiple and complex predisposing genetic fact…

GlutensTissue transglutaminaseHuman leukocyte antigenDiseaseGut floraImmunologic Testsmedicine.disease_causeBioinformaticsAutoimmunityImmune systemPredictive Value of TestsRisk FactorsGenetic predispositionMedicineAnimalsHumansGenetic Predisposition to DiseaseImmunogenetic PhenomenaIrritable bowel syndromeHepatologybiologyBacteriabusiness.industryfungiGastroenterologynutritional and metabolic diseasesmedicine.diseasebiology.organism_classificationPrognosisGastrointestinal MicrobiomeIntestinesCeliac DiseaseDisease Models AnimalPhenotypeHost-Pathogen Interactionsbiology.proteinbusinessGastroenterology
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Possible links between intestinal permeablity and food processing: a potential therapeutic niche for glutamine

2010

Increased intestinal permeability is a likely cause of various pathologies, such as allergies and metabolic or even cardiovascular disturbances. Intestinal permeability is found in many severe clinical situations and in common disorders such as irritable bowel syndrome. In these conditions, substances that are normally unable to cross the epithelial barrier gain access to the systemic circulation. To illustrate the potential harmfulness of leaky gut, we present an argument based on examples linked to protein or lipid glycation induced by modern food processing. Increased intestinal permeability should be largely improved by dietary addition of compounds, such as glutamine or curcumin, which…

Glycation End Products AdvancedCurcuminAllergyFood HandlingGlutamineInflammationBiologyIntestinal permeabilityIntestinal absorptionPermeabilitychemistry.chemical_compoundGlycationmedicineHumansIrritable bowel syndromeInflammationMetabolic Syndromelcsh:R5-920GlycationIntestinal permeabilityTight junctionGeneral Medicinemedicine.diseaseMetabolic syndromeDietGlutaminechemistryIntestinal AbsorptionBringing Ideas TogetherImmunologyDietary SupplementsCurcuminmedicine.symptomlcsh:Medicine (General)Gastrointestinal MotilityFood Hypersensitivity
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Progress Report The ItalianSocietyofGastroenterology(SIGE)andtheItalianGroupforthestudy of InflammatoryBowelDisease(IG-IBD)ClinicalPracticeGuidelines…

2011

Biological therapiesareanimportantstepinthemanagementofInflammatoryBowelDiseases.Incon- sideration ofhighcostandsafetyissuesthereistheneedtohaveclearrecommendationsfortheiruse. Despite theAmericanGastroenterologicalAssociationandtheEuropeanCrohn’sandColitisOrganisation have publishedexhaustiveInflammatoryBowelDiseaseguidelines,nationalguidelinesmaybeneces- sary asculturalvalues,economicalandlegalissuesmaydifferbetweencountries.Forthesereasonsthe Italian SocietyofGastroenterologyandtheItalianGroupforthestudyofInflammatoryBowelDisease have decidedtoelaboratetheItalianguidelinesontheuseofbiologicsinInflammatoryBowelDisease. The followingitemshavebeenchosen:definitionsofactive,inactive,steroidd…

Guidelines.Biologics.Inflammatory bowel disease.
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The many roads to inflammatory bowel diseases.

2006

Two independent studies by Rakoff-Nahoum et al. (2006) and Uhlig et al. (2006) in this issue of Immunity have illuminated a unique pathogenic role of innate immunity via Toll-like receptor and interleukin-23 signaling, respectively, in intestinal inflammation. These data define new roads to gut inflammation and future avenues for therapy.

Gut inflammationInnate immune systemInterleukinsImmunologyToll-Like ReceptorsInflammatory Bowel DiseasesBiologyInflammatory Bowel DiseasesInterleukin-12Interleukin-23Immunity InnateInfectious DiseasesImmunityIntestinal inflammationImmunologyImmunology and AllergyAnimalsSignal TransductionImmunity
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Zur Immunpathogenese der chronisch entz�ndlichen Darmerkrankungen

2000

Die chronisch entzundlichen Darmerkrankungen (CED: Morbus Crohn, Colitis ulcerosa) sind durch schubartig verlaufende destruierende Entzundungsreaktionen der Darmschleimhaut gekennzeichnet. In den letzten Jahren konnte gezeigt werden, das bei diesen Erkrankungen eine pathologische Aktivierung des intestinalen Immunsystems durch mucosale Antigene auftritt. In der chronischen Phase der Erkankungen sowie bei der Entwicklung postoperativer Rezidive sind Anderungen in der Zellmigration sowie der Cytokinproduktion intestinaler Zellen wahrscheinlich von entscheidender Bedeutung. Basierend auf diesen neuen pathogenetischen Erkenntnissen werden zur Zeit innovative klinische Behandlungsstrategien gete…

Gynecologymedicine.medical_specialtyTransplant surgeryIntestinal mucosabusiness.industryCrohn diseasemedicineInflammatory Bowel DiseasesSurgerybusinessGenetic therapyDer Chirurg
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Ileus following spontaneous jejunum intramural hematoma: case report and review of the literature.

2013

Anticoagulant therapy may cause the onset of a spontaneous intramural hema- toma of the small bowel, in the jejunum, ileum or duodenum. A 53-year-old woman on therapy with heparin for previous pulmonary embolism was admitted for abdominal pain and vomit. Computed tomography scan visualized an intramural hematoma of the jejunum causing subtotal obstruction of the intestinal lumen. The patient underwent resection of a part of the jejunum, securing intestinal continuity by a mechanical side-to-side anastomosis. The postoperative course was regular, but the initial anticoagulant therapy was reduced to prevent recurrence. In conclusion, spontaneous hema- toma of small bowel can occur as a compli…

HematomaHeparinSpontaneous intramural hematomaAnticoagulantsJejunal DiseasesMiddle AgedSettore MED/15 - Malattie Del SangueSettore MED/18 - Chirurgia GeneraleIleusHumansFemaleSpontaneous intramural hematoma; Anticoagulant therapy; Bowel obstructionAnticoagulant therapyBowel obstruction
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Letter: switching from one to another anti-tumour necrosis factor alpha agent, and the risks of an overlap of exposure

2016

Hepatologybiologybusiness.industryImmunoglobulin Fab FragmentsGastroenterologyInflammatory Bowel DiseasesToxicology03 medical and health sciences0302 clinical medicineAntibodies monoclonal030220 oncology & carcinogenesisMonoclonalbiology.proteinCancer researchMedicine030211 gastroenterology & hepatologyPharmacology (medical)Tumor necrosis factor alphaAnti-tumour necrosis factor alpha agentAntibodybusinessAlimentary Pharmacology & Therapeutics
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