Search results for "c12"

showing 10 items of 382 documents

Effects of ions-releasing restorative materials on the dentine bonding longevity of modern universal adhesives after load-cycle and prolonged artific…

2019

This study aimed at evaluating the microtensile bond strength (MTBS) and fractographic features of dentine-bonded specimens created using universal adhesives applied in etch-and-rinse (ER) or self-etching (SE) mode in combination with modern ion-releasing resin-modified glass-ionomer cement (RMGIC)-based materials after load cycling and artificial saliva aging. Two universal adhesives (FTB: Futurabond M+, VOCO, Germany

SalivaComposite numberGlass ionomer cement02 engineering and technologyCementos dentales.lcsh:TechnologyAdhesivos dentales.dentineDental adhesives.0302 clinical medicineGeneral Materials ScienceComposite materiallcsh:QC120-168.85Bond strengthGlass-ionomer cements021001 nanoscience & nanotechnologyDentin.adhesionRestorative materialAdhesionDental materials.0210 nano-technologylcsh:TK1-9971cycling mechanical stressMaterials scienceLongevityArticle03 medical and health sciencesstomatognathic systemlongevityOdontología restauradora.Materiales dentales.lcsh:MicroscopyDentina.Cementlcsh:QH201-278.5Dental cements.lcsh:Tglass-ionomer cementstechnology industry and agricultureCycling mechanical stressDentine030206 dentistryUniversal adhesiveslcsh:TA1-2040lcsh:Descriptive and experimental mechanicslcsh:Electrical engineering. Electronics. Nuclear engineeringAdhesiveuniversal adhesiveslcsh:Engineering (General). Civil engineering (General)Dentistry Operative.
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Validation of the Marathon Motivation Scale in Chile

2021

The objective of this work was to validate the Marathon Motivation Scale (MOMS) for its application in the Chilean context. The study used a non-experimental, descriptive and transversal design. The sample was non-probability and intentional and was composed of 250 subjects (143 male and 104 female) runners who participated in the International Marathon of Viña del Mar with an average age of 29.8 (TD = 10.1) years. For the analysis of the data, an exploratory factorial analysis was carried out with free parameters, another one with seven factors fixed to the theoretical dimensions and a confirmatory factorial analysis with the items corresponding to the MOMS model. The validity and reliabil…

Scale (ratio)ValidityPhysical Therapy Sports Therapy and RehabilitationSample (statistics)Context (language use)010501 environmental sciences01 natural sciencesMotivation scale0502 economics and businessStatisticsEducación Física y Deportivalcsh:Sports medicinechilean validation0105 earth and related environmental sciencesFactorial model05 social sciencesSpanish versionmotivation scaleChilean validationTransversal designmarathonersFactorial analysislcsh:RC1200-1245PsychologyMarathoners050203 business & managementJournal of Human Sport and Exercise
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Vibration effect on ball score test in international vs. national level table tennis

2018

In table tennis, motor skills are crucial for discriminating player level. However, there is a dearth of studies exploring the impact of a vibrational stimulus on performance. Thirty-four male players (age 25 +/- 2 years ; body mass index, BMI 23.4 +/- 1.2 kg.m(-2)) participated in the study. Seventeen played at international level (IL), while the remaining 17 played at national level (NL). The participants underwent a ball-handling test, the ball score, before (PRE) and after (POST) a vibrational stimulus. Intra-class correlation (ICC) for the ball score result showed good reliability (ICC 0.87 for IL and 0.80 for NL). Repeated measures ANOVA showed differences between groups for ball scor…

Score testmedicine.medical_specialtyPhysical Therapy Sports Therapy and Rehabilitation03 medical and health sciences0302 clinical medicineMotor controlFatigued conditionPhysiology (medical)Muscle activity ; Motor control ; Reaction time ; Fatigued conditionsMedicineOrthopedics and Sports MedicineNational levellcsh:Sports medicinelcsh:QH301-705.5Reaction timeInternational levelOriginal PaperSettore M-EDF/02 - Metodi E Didattiche Delle Attivita' Sportivebusiness.industryRepeated measures design030229 sport sciencesfatigued conditions; motor control; muscle activity; reaction timeFatigued conditionslcsh:Biology (General)Physical therapylcsh:RC1200-1245businessBody mass index030217 neurology & neurosurgeryMuscle activityBiology of Sport
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THE ANABOLIC-ANDROGENIC STEROIDS EXERT DIRECT TOXIC EFFECTS ON NEURON-LIKE CELLS

2012

INTRODUCTION Intake of AAS by atheletes and others in an attempt to gain muscle strength and improve perfor- mance is often associated with toxic effects on the liver, the cardiovascular system, the male and female reproductive systems, and the central nervous system (Trifunovic et al., 1995). In vivo administration of high doses of androgens has been linked to neurobehavioral changes that could be the outward manifestation of neuronal damage. METHODS PC12 cells are a cell line that have been widely used as a model in neurobiological investigations (Fujita et al., 1989; Vaudry et al., 2002). Following exposure to nerve growth factor (NGF), PC12 cells differentiate into a sympathetic-like ne…

Settore BIO/10 - BiochimicaAASPC12 cellsToxic effect
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Evaluation of a Cell-Free Collagen Type I-Based Scaffold for Articular Cartilage Regeneration in an Orthotopic Rat Model.

2020

The management of chondral defects represents a big challenge because of the limited self-healing capacity of cartilage. Many approaches in this field obtained partial satisfactory results. Cartilage tissue engineering, combining innovative scaffolds and stem cells from different sources, emerges as a promising strategy for cartilage regeneration. The aim of this study was to evaluate the capability of a cell-free collagen I-based scaffold to promote cartilaginous repair after orthotopic implantation in vivo. Articular cartilage lesions (ACL) were created at the femoropatellar groove in rat knees and cell free collagen I-based scaffolds (S) were then implanted into right knee defect for the…

Settore BIO/17 - IstologiaPathologymedicine.medical_specialtyScaffoldcartilage tissue engineeringcollagen I-based scaffold02 engineering and technologySOX9lcsh:TechnologyArticle03 medical and health sciencesIn vivoarticular cartilage lesionmedicineGeneral Materials Sciencelcsh:Microscopycartilage regenerationAggrecan03 Chemical Sciences 09 Engineering030304 developmental biologylcsh:QC120-168.850303 health scienceslcsh:QH201-278.5Chemistrylcsh:TCartilageRegeneration (biology)021001 nanoscience & nanotechnologymusculoskeletal systemmedicine.anatomical_structurelcsh:TA1-2040ImmunohistochemistryArticular cartilage lesion; Cartilage regeneration; Cartilage tissue engineering; Collagen i-based scaffold; Orthotopic implantationlcsh:Descriptive and experimental mechanicslcsh:Electrical engineering. Electronics. Nuclear engineeringStem cellorthotopic implantation0210 nano-technologylcsh:Engineering (General). Civil engineering (General)lcsh:TK1-9971
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PTC124 derivatives as a novel approach to improve the readthrough of premature stop codons in the CFTR gene.

2011

Background Cystic fibrosis (CF) is caused by mutations in the gene encoding the cystic fibrosis transmembrane conductance regulator (CFTR). Approximately 10% (worldwide) of patients have in-frame nonsense mutations (UAA, UAG or UGA class I mutations) in the CF trans-membrane regulator (CFTR) gene that result in premature stop codons (PTCs) in the messenger RNA (mRNA) generating truncated CFTR protein responsible for a severe CF phenotype. Pharmacological approaches have been proposed to directly overcome PTCs. Ataluren (PTC124) a small molecule that mimics the activity of aminoglycosides has been suggested to allow PTCs readthrough and to partially restore the protein function. However, des…

Settore BIO/18 - GeneticaCystic fibrosis Nonsense mutation PTC124Settore CHIM/06 - Chimica OrganicaPTC124 Cystic fibrosis.
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AZIONE READTHROUGH DI DERIVATI DEL PTC124 SU SISTEMI MODELLO CELLULARI E IN CELLULE DI EPITELIO BRONCHIALE-FC IB3.1 (CFTR F508/W1282X )

2013

Obiettivi specifici: Le mutazioni nonsenso (mutazioni STOP), un difetto genetico frequente negli individui affetti da Fibrosi Cistica (CF), causano la sintesi di proteine CFTR tronche e non funzionanti che sono associate ad un fenotipo più severo della CF (McKone EF. et al., Chest 2006). L’obiettivo del nostro studio è stato quello di disegnare derivati dell’Ataluren (PTC124), una ‘small molecule’ a cui è stata attribuita attività readthrough, e valutarne l’attività su tre differenti sistemi modello sperimentali contenenti codoni di STOP prematuri (UGA, UAG, UAA). Materiali e metodi: Sono state sintetizzate 24 molecole derivate dal PTC124 e analizzate mediante tecniche spettroscopiche per v…

Settore BIO/18 - GeneticaMutazioni non senso PTC124 fibrosi cisticaSettore CHIM/06 - Chimica Organica
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Valutazione dell'azione readthorough della molecola PTC124 su sistemi modello cellulari contenenti mutazioni non senso e in cellule epiteliali bronch…

2013

Circa il 10% dei pazienti affetti da fibrosi cistica (FC) presenta nel gene CFTR mutazioni non senso (o 'stop': UGA, UAG or UAA, mutazioni di classe I) che bloccano prematuramente la sintesi della proteina. Attualmente non esiste una cura per questo tipo di mutazioni ma si sta cercando di individuare delle molecole che siano in grado di indurre la traduzione di codoni di stop prematuri (readthrough) che, rispetto a molecole già note come il G418, abbiano effetti collaterali ridotti ed una maggiore specificità per uno specifico codone. Una piccola molecola che sembra possedere una tale attività è il PTC124 (Welch, 2007). Ad oggi però non c’è ancora un consenso generale sul meccanismo di azio…

Settore BIO/18 - GeneticaPTC124 readthrough mutazioni di stop
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X CONVENTION OF INVESTIGATORS IN CYSTIC FIBROSIS.

2012

Background Cystic fibrosis (CF) is caused by mutations in the gene encoding the cystic fibrosis transmembrane conductance regulator (CFTR). Approximately 10% (worldwide) of patients have in-frame nonsense mutations (UAA, UAG or UGA class I mutations) in the CF trans-membrane regulator (CFTR) gene that result in premature stop codons (PTCs) in the messenger RNA (mRNA) generating truncated CFTR protein responsible for a severe CF phenotype. Pharmacological approaches have been proposed to directly overcome PTCs. Ataluren (PTC124) a small molecule that mimics the activity of aminoglycosides has been suggested to allow PTCs readthrough and to partially restore the protein function. However, des…

Settore BIO/18 - GeneticaSettore CHIM/06 - Chimica OrganicaCystic fibrosis PTC124 Nonsense mutation
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PTC124 DERIVATIVES AS A NOVEL APPROACH TO IMPROVE THE READTHROUGH OF PREMATURE AMBER AND OCHRE STOP CODONS

2013

Nucleotide changes within an exon may alter the trinucleotide normally encoding a particular amino acid, such that a new “stop” signal is transcribed into the mRNA open reading frame. This causes the ribosome to prematurely terminate its reading of the mRNA, leading to the lack of production of a normal full-length protein. Such premature termination codon (PTC) mutations occur in an estimated 10% to 15% of many genetically based disorders (1). Pathological nonsense mutations resulting in TAG (40.4%), TGA (38.5%), and TAA (21.1%) occur in different proportions to naturally occurring stop codons (2). Several genetic disorders are characterized by opal (TGA; Cystic fibrosis, Duchenne/Becker m…

Settore BIO/18 - Geneticareadthrough PTC124 Cystic fibrosis
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