Search results for "caffold"

showing 10 items of 470 documents

3D printing novel in vitro cancer cell culture model systems for lung cancer stem cell study

2021

Two-dimensional (2D) in vitro cell cultures and laboratory animals have been used traditionally as the gold-standard preclinical cancer model systems. However, for cancer stem cell (CSC) studies, they exhibit notable limitations on simulating native environment, which depreciate their translatability for clinical development purposes. In this study, different three-dimensional (3D) printing platforms were used to establish novel 3D cell cultures enriched in CSCs from non-small cell lung cancer (NSCLC) patients and cell lines. Rigid scaffolds with an elevated compressive modulus and uniform pores and channels were produced using different filaments. Hydrogel-based scaffolds were printed with…

Lung NeoplasmsStereolithographyMaterials scienceCell Culture TechniquesBioengineeringFused deposition modeling02 engineering and technology010402 general chemistry01 natural sciencesBiomaterialsCancer stem cellIn vivoCarcinoma Non-Small-Cell LungAnimalsHumansCancer modelLungTissue ScaffoldsCancer stem cellsSpheroidHydrogels3D printing021001 nanoscience & nanotechnologyIn vitro0104 chemical sciencesCell biologyMechanics of MaterialsCell culturePrinting Three-DimensionalSelf-healing hydrogelsCancer cellNeoplastic Stem CellsLung cancerStem cell0210 nano-technologyMaterials Science and Engineering: C
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Dimerization of the kinase ARAF promotes MAPK pathway activation and cell migration.

2014

The RAF family of kinases mediates RAS signaling, and RAF inhibitors can be effective for treating tumors with BRAF V600E mutant protein. However, RAF inhibitors paradoxically accelerate metastasis in RAS -mutant tumors and become ineffective in BRAF V600E tumors because of reactivation of downstream mitogen-activated protein kinase (MAPK) signaling. We found that the RAF isoform ARAF has an obligatory role in promoting MAPK activity and cell migration in a cell type–dependent manner. Knocking down ARAF prevented the activation of MAPK kinase 1 (MEK1) and extracellular signal–regulated kinase 1 and 2 (ERK1/2) and decreased the number of protrusions from tumor cell spheroids in three-dimensi…

MAPK/ERK pathwayScaffold proteinModels MolecularNiacinamideProto-Oncogene Proteins B-rafMAP Kinase Signaling SystemBlotting WesternMAP Kinase Kinase 1MAPK cascadeBiologyKSR1BiochemistryBinding CompetitiveProto-Oncogene Proteins A-rafTime-Lapse ImagingMutant proteinCell MovementTumor Cells CulturedHumansNeoplasm InvasivenessRNA Small InterferingProtein kinase AMolecular BiologyAnalysis of VarianceKinasePhenylurea CompoundsCell BiologySorafenibCell biologyEnzyme ActivationProto-Oncogene Proteins c-rafHEK293 CellsIndenesGene Knockdown TechniquesCancer researchPyrazolesElectrophoresis Polyacrylamide GelARAFDimerizationScience signaling
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Regulating TRAIL Receptor-Induced Cell Death at the Membrane: A Deadly Discussion

2011

Article Open access plus; International audience; The use of TRAIL/APO2L and monoclonal antibodies targeting TRAIL receptors for cancer therapy holds great promise, due to their ability to restore cancer cell sensitivity to apoptosis in association with conventional chemotherapeutic drugs in a large variety of tumors. TRAIL-induced cell death is tightly regulated right from the membrane and at the DISC (Death-Inducing Signaling Complex) level. The following patent and literature review aims to present and highlight recent findings of the deadly discussion that determines tumor cell fate upon TRAIL engagement.

MESH: Cell DeathMESH: Signal TransductionCancer ResearchApoptosisTRAILMESH : Models BiologicalscaffoldCell membrane0302 clinical medicineDrug DiscoveryMESH: AnimalsPharmacology (medical)Receptordeath effector domain0303 health sciencesCell DeathGeneral MedicineTRAIL-R4.3. Good healthCell biologymedicine.anatomical_structureOncology030220 oncology & carcinogenesisSignal transductionMESH : Apoptosis Regulatory ProteinsSignal TransductionProgrammed cell deathc-FLIPdeath domainmedicine.drug_classMESH : Cell MembraneCancer therapyBiologyMonoclonal antibodyModels BiologicalArticle03 medical and health sciencesmedicineAnimalsHumansChemotherapy[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyMESH: Receptors TNF-Related Apoptosis-Inducing LigandMESH : Receptors TNF-Related Apoptosis-Inducing Ligand[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular Biology030304 developmental biologyMESH : Signal TransductionMESH: HumansMESH: Apoptosis Regulatory ProteinsMESH: ApoptosisMESH : HumansCell MembraneMESH: Models BiologicalDISCReceptors TNF-Related Apoptosis-Inducing LigandApoptosisMESH : Cell DeathFADDCancer cellMESH : AnimalsApoptosis Regulatory ProteinsMESH : ApoptosisMESH: Cell MembraneRecent Patents on Anti-Cancer Drug Discovery
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Symplekin, a polyadenylation factor, prevents MOZ and MLL activity on HOXA9 in hematopoietic cells

2013

International audience; MOZ and MLL encoding a histone acetyltransferase and a histone methyltransferase, respectively, are targets for recurrent chromosomal translocations found in acute myeloblastic or lymphoblastic leukemia. We have previously shown that MOZ and MLL cooperate to activate HOXA9 gene expression in hematopoietic stem/progenitors cells. To dissect the mechanism of action of this complex, we decided to identify new proteins interacting with MOZ. We found that the scaffold protein Symplekin that supports the assembly of polyadenylation machinery was identified by mass spectrometry. Symplekin interacts and co-localizes with both MOZ and MLL in immature hematopoietic cells. Its …

MLLScaffold proteinPolyadenylationHematopoietic System[SDV]Life Sciences [q-bio]PolyadenylationCell Line03 medical and health scienceschemistry.chemical_compound0302 clinical medicinehemic and lymphatic diseasesGene expressionTranscriptional regulationHumansRNA MessengerPromoter Regions GeneticSymplekinHSF1neoplasmsMolecular BiologyHistone Acetyltransferases030304 developmental biologyHomeodomain ProteinsmRNA Cleavage and Polyadenylation Factors0303 health sciences[ SDV ] Life Sciences [q-bio]biologyNuclear ProteinsHistone-Lysine N-MethyltransferaseHOXA9Transcription regulationCell BiologyHistone acetyltransferaseMOZCell biology[SDV] Life Sciences [q-bio]Protein TransportRUNX1chemistry030220 oncology & carcinogenesisHistone methyltransferaseCancer researchbiology.proteinMyeloid-Lymphoid Leukemia ProteinProtein BindingBiochimica et Biophysica Acta (BBA) - Molecular Cell Research
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β1 integrin signaling promotes neuronal migration along vascular scaffolds in the post-stroke brain

2017

Cerebral ischemic stroke is a main cause of chronic disability. However, there is currently no effective treatment to promote recovery from stroke-induced neurological symptoms. Recent studies suggest that after stroke, immature neurons, referred to as neuroblasts, generated in a neurogenic niche, the ventricular-subventricular zone, migrate toward the injured area, where they differentiate into mature neurons. Interventions that increase the number of neuroblasts distributed at and around the lesion facilitate neuronal repair in rodent models for ischemic stroke, suggesting that promoting neuroblast migration in the post-stroke brain could improve efficient neuronal regeneration. To move t…

Male0301 basic medicineChain migrationlcsh:MedicineExtracellular matrixNeural Stem CellsCell MovementLamininCells CulturedMice KnockoutNeuronslcsh:R5-920Mice Inbred ICRMicroscopy ConfocalTissue ScaffoldsbiologyIntegrin beta1BrainCell migrationGeneral MedicineCell biologyStrokeVasculature-guided migrationFemaleBlood vesselmedicine.symptomlcsh:Medicine (General)Signal TransductionResearch Paperanimal structuresIntegrinMice TransgenicGeneral Biochemistry Genetics and Molecular BiologyLesion03 medical and health sciencesNeuroblastβ1 integrinNeuroblast migrationmedicineAnimalsRegeneration (biology)lcsh:RCoculture TechniquesMicroscopy Electron030104 developmental biologynervous systemAstrocytesImmunologybiology.proteinBlood VesselsLamininEBioMedicine
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Tissue engineered pre-vascularized buccal mucosa equivalents utilizing a primary triculture of epithelial cells, endothelial cells and fibroblasts

2015

Artificial generated buccal mucosa equivalents are a promising approach for the reconstruction of urethral defects. Limiting in this approach is a poor blood vessel supply after transplantation, resulting in increased morbidity and necrosis. We generated a pre-vascularized buccal mucosa equivalent in a tri-culture of primary buccal epithelial cells, fibroblasts and microvascular endothelial cells, using a native collagen membrane as a scaffold. A successful pre-vascularization and dense formation of capillary-like structures at superficial areas was demonstrated. The lumen size of pre-formed blood vessels corresponded to the capillary size in vivo (10-30 μm). Comparing native with a highly …

Male0301 basic medicinePathologymedicine.medical_specialtyNecrosisForeskinGingivaBiophysicsMice NudeTransplantsBioengineeringBiologyBiomaterialsAngiopoietinMice03 medical and health sciencesForeskinTissue engineeringmedicineAnimalsHumansSecretionCells CulturedTissue EngineeringTissue ScaffoldsMouth MucosaEndothelial CellsEpithelial CellsMembranes ArtificialBuccal administrationFibroblastsCoculture TechniquesCapillariesOrganoidsPlatelet Endothelial Cell Adhesion Molecule-1Transplantation030104 developmental biologymedicine.anatomical_structureMechanics of MaterialsCeramics and CompositesHeterograftsAngiogenesis Inducing AgentsCollagenmedicine.symptomBlood vesselBiomaterials
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Application of nano-hydroxyapatite/chitosan scaffolds on rat calvarial critical-sized defects: A pilot study

2018

Background The purpose of this pilot study was to evaluate for the first time the effect of 75/25 w/w nano-Hydroxyapatite/Chitosan (nHAp/CS) scaffolds on Guided Bone Regeneration (GBR) in rat calvarial critical-sized defects (CSDs). Material and Methods Six adult Sprague Dawley rats, 3 males and 3 females, were used. Two CSDs, full thickness and 5mm in diameter, were trephined in both sides of the parietal bone. The right CSD was filled with nHAp/CS scaffold, while the left CSD remained empty, as the control group. The wound was sutured in layers. Rats were euthanized with diethyl ether inhalation at 2, 4 and 8 weeks after surgical procedure. Histological and histomorphometric analysis was …

MaleBiocompatible MaterialsPilot Projects02 engineering and technologyChitosanRats Sprague-Dawley03 medical and health scienceschemistry.chemical_compound0302 clinical medicineSprague dawley ratsmedicineAnimalsBone regenerationGeneral DentistryChitosanBone DevelopmentTissue ScaffoldsChemistryResearchSkullBiomaterial030206 dentistryAnatomy021001 nanoscience & nanotechnology:CIENCIAS MÉDICAS [UNESCO]Sagittal planeRatsmedicine.anatomical_structureOtorhinolaryngologyNano hydroxyapatiteUNESCO::CIENCIAS MÉDICASNanoparticlesSurgeryFull thicknessFemaleHydroxyapatitesOral Surgery0210 nano-technologyParietal boneMedicina Oral, Patología Oral y Cirugía Bucal
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Absorb Bioresorbable Scaffold Versus Xience Metallic Stent for Prevention of Restenosis Following Percutaneous Coronary Intervention in Patients at H…

2019

Abstract Background The advent of bioresorbable vascular scaffolds (BVS) was considered as a potential improvement in percutaneous coronary intervention (PCI) after the groundbreaking development of drug eluting stents (DES). However, the clinical performance, long-term safety and efficacy of BVS in complex coronary lesions remain uncertain. COMPARE ABSORB, a multicenter, single blind, prospective randomized trial, aims to compare the clinical outcomes between the Absorb BVS and Xience everolimus-eluting metallic stent (EES) in patients with coronary artery disease and a high risk of restenosis. Design COMPARE ABSORB is designed to enroll 2100 patients at up to 45 European sites. Enrolled p…

MaleBioresorbable scaffoldTime Factorsmedicine.medical_treatmentVascular damage Radboud Institute for Health Sciences [Radboudumc 16]030204 cardiovascular system & hematologyAbsorbCoronary artery diseaseCoronary artery disease0302 clinical medicineRestenosisRisk FactorsAbsorbable ImplantsClinical endpointMulticenter Studies as TopicMedicineSingle-Blind MethodProspective Studies030212 general & internal medicineMyocardial infarctionAngioplasty Balloon CoronaryGeneral MedicineMiddle AgedEuropeTreatment OutcomeMetalsCardiologyFemaleStentsCardiology and Cardiovascular MedicineAdultmedicine.medical_specialtyAdolescentProsthesis DesignRisk AssessmentCoronary RestenosisYoung Adult03 medical and health sciencesAll institutes and research themes of the Radboud University Medical CenterInternal medicineHumansAgedbusiness.industryStentPercutaneous coronary interventionCoronary Lesion ComplexityProtective Factorsmedicine.diseaseConventional PCIbusiness
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Evaluation of the tissue reaction to a new bilayered collagen matrix in vivo and its translation to the clinic.

2011

This study evaluates a new collagen matrix that is designed with a bilayered structure in order to promote guided tissue regeneration and integration within the host tissue. This material induced a mild tissue reaction when assessed in a murine model and was well integrated within the host tissue, persisting in the implantation bed throughout the in vivo study. A more porous layer was rapidly infiltrated by host mesenchymal cells, while a layer designed to be a barrier allowed cell attachment and host tissue integration, but at the same time remained impermeable to invading cells for the first 30 days of the study. The tissue reaction was favorable, and unlike a typical foreign body respons…

MaleMaterials scienceBiomedical EngineeringConnective tissueNeovascularization PhysiologicBioengineeringContext (language use)Pilot ProjectsMatrix (biology)BiomaterialsMiceMaterials TestingmedicineAnimalsHumansTissue ScaffoldsRegeneration (biology)Foreign-Body ReactionMesenchymal stem cellGranulation tissueSoft tissueBiomaterialCell biologymedicine.anatomical_structureGuided Tissue Regeneration PeriodontalMicroscopy Electron ScanningFemaleCollagenPorosityBiomedical engineeringBiomedical materials (Bristol, England)
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Neomyogenesis in 3D Dynamic Responsive Prosthesis for Inguinal Hernia Repair

2018

Despite undisputed improvements, prosthetics hernia repair continues to be affected by unacceptable rates of complications. Postoperative adverse events such as discomfort and chronic pain represent a subject of increasing concern among the surgical community. Poor quality biologic response, stiff scar plates, and mesh shrinkage, a typical effect of conventional static meshes and plugs, are all indicated as the main reasons for many of the complications related to inguinal hernia repair. Even the conventional concept of treatment based upon a supposed reinforcement of the groin consequent to the fibrotic incorporation of meshes, would appear to be inadequate in the light of the latest scien…

MaleMuscle CellsInguinal herniaBiomedical EngineeringTissue scaffoldMuscle CellMedicine (miscellaneous)Hernia InguinalBioengineeringMiddle AgedMuscle DevelopmentBiomaterialFollow-Up StudieProsthesis ImplantationProstheseTissue regenerationHumansMuscular tissueHerniorrhaphyFollow-Up StudiesAgedHuman
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