Search results for "campus"

showing 10 items of 667 documents

Temporospatial expression of HSP72 and c-JUN, and DNA fragmentation in goat hippocampus after global cerebral ischemia

2001

The role of gene induction (expression of HSP72 and c-JUN proteins) and delayed ischemic cell death (in situ labeling of DNA fragmentation) have been investigated in the goat hippocampus after transient global cerebral ischemia. The animals were subjected to 20-min ischemia (bilateral occlusion of the external carotid arteries plus bilateral jugular vein compression) and allowed to reperfuse for 2 h, and then 1, 3, and 7 days. Histological signs of cell loss were not found in the hippocampus at 2 h, 1 day, or 3 days of reperfusion. However, such an ischemic insult produced extensive, selective, and delayed degeneration in the hippocampus, as 68% of the neurons in CA1 had died at 7 days, but…

Programmed cell deathCognitive NeuroscienceDentate gyrusc-junIschemiaSubiculumHippocampusBiologymedicine.diseasenervous systemApoptosismedicineDNA fragmentationNeuroscienceHippocampus
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Neuronal expression and regulation of rat inhibitor of apoptosis protein-2 by kainic acid in the rat brain

2002

Inhibitors of apoptosis proteins (IAPs) define a protein family with the ability to counteract cell death by the inhibition of different caspases activated during apoptosis. These proteins are present in different cells, however, the function and roles of IAPs in brain tissue are not fully understood. We report here that RIAP-2, the rat homologue of human cIAP-1/HIAP-2, is expressed in different areas of rat brain as shown by in situ hybridization and immunohistochemistry. Brain regions with relatively high expression of RIAP-2 mRNA included cortex, cerebellum and different subregions of rat hippocampus. Double labelling using a specific anti-RIAP antibody and markers for neurons and glial …

Programmed cell deathKainic acidbiologyGeneral NeuroscienceDentate gyrusGlutamate receptorHippocampusIn situ hybridizationInhibitor of apoptosisMolecular biologyCell biologychemistry.chemical_compoundnervous systemchemistrybiology.proteinNeuNEuropean Journal of Neuroscience
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Integrative proteomics: functional and molecular characterization of a particular glutamate-related neuregulin isoform.

2005

Glutamate is the major excitatory neurotransmitter in the mammalian brain and is related to memory by calcium-conducting receptors. Neuregulins have emerged as long-term modulating molecules of synaptic signaling by glutamate receptors, playing a role in some cognition/memory-related disorders and moreover being part of transient functional microdomains, called lipid rafts. Here we characterize one specific isoform of neuregulin as a central biomarker for glutamate-related signaling, integrating results from in vitro and in vivo models by a differential functional and proteomic approach.

ProteomicsNeuregulin-1Glutamic AcidNerve Tissue ProteinsBiochemistryHippocampusRats Sprague-DawleyAlzheimer DiseaseAnimalsHumansLearningProtein IsoformsNeuregulin 1ReceptorLipid raftCells CulturedbiologyGlutamate receptorGeneral ChemistryGlutamic acidCell biologyRatsbiology.proteinNeuregulinCalciumFemaleSynaptic signalingSignal transductionBiomarkersSignal TransductionJournal of proteome research
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In‐depth protein profiling of the postsynaptic density from mouse hippocampus using data‐independent acquisition proteomics

2014

Located at neuronal terminals, the postsynaptic density (PSD) is a highly complex network of cytoskeletal scaffolding and signaling proteins responsible for the transduction and modulation of glutamatergic signaling between neurons. Using ion-mobility enhanced data-independent label-free LC-MS/MS, we established a reference proteome of crude synaptosomes, synaptic junctions, and PSD derived from mouse hippocampus including TOP3-based absolute quantification values for identified proteins. The final dataset across all fractions comprised 49 491 peptides corresponding to 4558 protein groups. Of these, 2102 protein groups were identified in highly purified PSD in at least two biological replic…

ProteomicsPost-Synaptic DensityProteinsHippocampal formationBiologyProteomicsHippocampusBiochemistryCell biologyMiceTransduction (genetics)Glutamatergicnervous systemProteomeAnimalsData-independent acquisitionCytoskeletonMolecular BiologyPostsynaptic densityPROTEOMICS
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Systemic PaO2 oscillations cause mild brain injury in a pig model

2016

OBJECTIVE: Systemic PaO2 oscillations occur during cyclic recruitment and derecruitment of atelectasis in acute respiratory failure and might harm brain tissue integrity. DESIGN: Controlled animal study. SETTING: University research laboratory. SUBJECTS: Adult anesthetized pigs. INTERVENTIONS: Pigs were randomized to a control group (anesthesia and extracorporeal circulation for 20 hr with constant PaO2, n = 10) or an oscillation group (anesthesia and extracorporeal circulation for 20 hr with artificial PaO2 oscillations [3 cycles min⁻¹], n = 10). Five additional animals served as native group (n = 5). MEASUREMENTS AND MAIN RESULTS: Outcome following exposure to artificial PaO2 oscillations…

Pulmonary Atelectasismedicine.medical_specialtySwinemedicine.medical_treatment10208 Institute of NeuropathologyHippocampusInflammation610 Medicine & healthHippocampal formationReal-Time Polymerase Chain ReactionCritical Care and Intensive Care Medicinegamma-Aminobutyric acidRNA ComplementaryRandom Allocation03 medical and health sciencesExtracorporeal Membrane Oxygenation0302 clinical medicine030202 anesthesiologyNeurotransmitter receptorInternal medicinemedicineExtracorporeal membrane oxygenationAnimalsReceptoralpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acidgamma-Aminobutyric AcidRespiratory Distress Syndromebusiness.industryExtracorporeal circulationRespiration Artificialrespiratory tract diseasesEndocrinologyBrain Injuries570 Life sciences; biologyBlood Gas AnalysisInflammation Mediatorsmedicine.symptombusiness2706 Critical Care and Intensive Care Medicine030217 neurology & neurosurgerymedicine.drug
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Macrophage migration inhibitory factor is critically involved in basal and fluoxetine-stimulated adult hippocampal cell proliferation and in anxiety,…

2011

Intensive research is devoted to unravel the neurobiological mechanisms mediating adult hippocampal neurogenesis, its regulation by antidepressants, and its behavioral consequences. Macrophage migration inhibitory factor (MIF) is a pro-inflammatory cytokine that is expressed in the CNS, where its function is unknown. Here, we show, for the first time, the relevance of MIF expression for adult hippocampal neurogenesis. We identify MIF expression in neurogenic cells (in stem cells, cells undergoing proliferation, and in newly proliferated cells undergoing maturation) in the subgranular zone of the rodent dentate gyrus. A causal function for MIF in cell proliferation was shown using genetic (M…

Receptors SteroidStem-Cellsanimal diseasesmedicine.medical_treatmentHippocampusExpressionHippocampal formationHippocampusSubgranular zonememoryMice0302 clinical medicineConditioning PsychologicalCyclin D2Rat Dentate GyrusMice KnockoutNeurons0303 health sciencesMicroscopy ConfocalChronic StressMifNeurogenesisBrainFearrespiratory systemanxietyPsychiatry and Mental healthC-Reactive ProteinCytokinemedicine.anatomical_structuredepressionAntidepressive Agents Second-GenerationStem cellPsychologyAnimal-ModelNeurogenesisSpatial BehaviorNerve Tissue Proteinschemical and pharmacologic phenomena03 medical and health sciencesCellular and Molecular Neurosciencemedicineotorhinolaryngologic diseasesAnimalsRats WistarMaze LearningMacrophage Migration-Inhibitory FactorsMolecular BiologyCell Proliferation030304 developmental biologyMemory DisordersDentate gyrusfluoxetineFactor Mifbiological factorsRatsDisease Models AnimalAcoustic StimulationBromodeoxyuridineMacrophage migration inhibitory factorCorticosteroneNeuroscience030217 neurology & neurosurgery
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Role of interleukin-6 and soluble IL-6 receptor in region-specific induction of astrocytic differentiation and neurotrophin expression.

1999

Increasing evidence supports an essential role for interleukin-6 (IL-6) in the development, differentiation, as well as de- and re-generation of neurons in the central nervous system (CNS). Both IL-6 and its specific receptor (IL-6R) are expressed on neurons and glial cells including astrocytes. In this study, we have analyzed the responses of primary rat astrocytes of various brain regions to IL-6 with respect to morphological changes and neurotrophin expression. Since IL-6 alone failed to initiate effects on astrocytes, we have examined whether the soluble IL-6R (sIL-6R) can modulate the responsiveness of to IL-6 in these cells. For this purpose, we used a highly active fusion protein of …

Recombinant Fusion ProteinsCentral nervous systemHippocampusNeurotrophin-3HippocampusImmunoenzyme TechniquesRats Sprague-DawleyCellular and Molecular NeuroscienceNeurotrophin 3medicineAnimalsHumansNerve Growth FactorsCells CulturedCerebral CortexbiologyInterleukin-6Reverse Transcriptase Polymerase Chain ReactionCell DifferentiationImmunohistochemistryReceptors Interleukin-6RatsBlotting Southernmedicine.anatomical_structureNerve growth factornervous systemNeurologyAnimals NewbornCerebral cortexAstrocytesbiology.proteinNeurogliaNeuroscienceAstrocyteNeurotrophinGlia
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LOCALIZATION OF HIPPOCAMPAL ATROPHY IN ALZHEIMER'S DISEASE

2011

International audience; The hippocampus presents the highest rate of atrophy in the early stage of Alzheimer's disease (AD), with more pronounced neuron loss reported in CA1 and subiculum. The aim of this study is to increase the discrimination power of hippocampal shape analysis between AD and normal controls (NC) by focusing on the subregions with atrophy associated with AD and describing the localized shape changes using statistical shape models (SSMs).

SSM[INFO.INFO-TI] Computer Science [cs]/Image Processing [eess.IV]nervous system[INFO.INFO-TI]Computer Science [cs]/Image Processing [eess.IV][ INFO.INFO-TI ] Computer Science [cs]/Image ProcessingAlzheimer deseasehipocampus
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Age-related changes in the regulation of transcription factor NF-kappa B in rat brain.

1997

Aging process involves an increase in stress at cellular level. We studied whether aging affects the regulation of stress responsive transcription factor NF-kappa B in brain samples of Wistar rats. Hippocampus, cerebellum, and temporal and frontal lobes of cortex were studied. We observed a significant up-regulation in the constitutive, nucleus-located NF-kappa B binding activity in 30-month-old Wistar rats compared to young and 18-month-old rats. The increase was most prominent in cerebellum and in frontal cortex, but age-related changes did not occur in hippocampus. Inducible, cytoplasmic NF-kappa B binding activity was not affected by aging in any of the samples studied. Western blot ass…

SenescenceMalemedicine.medical_specialtyAgingP50HippocampusNerve Tissue ProteinsBiologyHippocampuschemistry.chemical_compoundWestern blotInternal medicineCerebellummedicineAnimalsRats WistarTranscription factorRegulation of gene expressionCell Nucleusmedicine.diagnostic_testGeneral NeuroscienceNF-kappa BBrainNF-κBTemporal LobeCell biologyFrontal LobeRatsUp-RegulationB vitaminsEndocrinologychemistryGene Expression RegulationFemaleProtein BindingNeuroscience letters
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Nicotine-induced FGF-2 mRNA in rat brain is preserved during aging

2004

Indirect trophic actions of nicotine on brain during aging are suggested from observations describing nicotine as a cognitive enhancer, increasing vigilance and improving learning and memory, and both in vitro and in vivo models have demonstrated neuroprotective effects of nAChR agonists. Previously, we have reported that an acute intermittent (-)nicotine treatment significantly increases fibroblast growth factor-2 (FGF-2) mRNA and protein in several brain regions of rat brain. The present study was designed to analyse if nicotine-induced FGF-2 expression in the rat brain was preserved during aging. Using in situ hybridization and quantitative RNase protection assay the present paper report…

SenescenceNicotineAgingmedicine.medical_specialtyCentral nervous systemStimulationIn situ hybridizationBiologynAChRNeuroprotectionNicotine treatmentStriatumNicotineHippocampuInternal medicineGliamedicineSubstantia nigraAnimalsTissue DistributionRNA MessengerAcetylcholine receptorGeneral NeuroscienceBrainNeuronFGF-2 expression; nicotine treatment; hippocampus; substantia nigra; striatum; brain; neurons; glia; nAChR; neurotrophism; neuroprotectionNeuroprotectionRatsmedicine.anatomical_structureEndocrinologyGene Expression RegulationFGF-2 expressionNeurotrophismFibroblast Growth Factor 2Neurology (clinical)NeuronGeriatrics and GerontologyDevelopmental Biologymedicine.drug
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