Search results for "carcinogenic"

showing 10 items of 48 documents

Modulation of aflatoxin B1 carcinogenicity, genotoxicity and metabolism in rat liver by dietary carotenoids: evidence for a protective effect of CYP1…

1997

The effects of several carotenoids of vitamin A and of 3-methylcholanthrene have been tested on the initiation of hepatocarcinogenesis by aflatoxin B1, using the sequential protocol of Solt and Farber. AFB1-induced DNA single-strand breaks and AFB1-metabolism were also assessed. The P4501A inducer carotenoids (canthaxanthin, astaxanthin, beta-apo-8'-carotenal) and 3-methylcholanthrene reduce the carcinogenicity of AFB1, divert AFB1-metabolism into the less genotoxic aflatoxin M1 and reduce AFB1-induced DNA single-strand breaks: we conclude that these carotenoids exert their protective effect through the deviation of AFB1 metabolism towards detoxification pathways. beta-Carotene decreased AF…

MaleVitaminCancer ResearchAflatoxinAflatoxin B1[SDV]Life Sciences [q-bio]MutagenBiologymedicine.disease_causeAntioxidants03 medical and health scienceschemistry.chemical_compound0404 agricultural biotechnologyAstaxanthinmedicineAnimalsAnticarcinogenic AgentsCanthaxanthinRats WistarVitamin ACarotenoidCarcinogenComputingMilieux_MISCELLANEOUS030304 developmental biologychemistry.chemical_classification0303 health sciencesINDUCTIONfood and beverages04 agricultural and veterinary sciencesCarotenoids040401 food scienceDietRats3. Good health[SDV] Life Sciences [q-bio]LiverOncologychemistryBiochemistryCarcinogensRATCARCINOGENESEGenotoxicityDNA DamageMethylcholanthrene
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Dietary carotenoids inhibit aflatoxin B1-induced liver preneoplastic foci and DNA damage in the rat: Role of the modulation of aflatoxin B1 metabolism

1998

To study the effects of carotenoids on the initiation of liver carcinogenesis by aflatoxin B1 (AFB1), male weanling rats were fed beta-carotene, beta-apo-8'-carotenal, canthaxanthin, astaxanthin or lycopene (300 mg/kg diet), or an excess of vitamin A (21000 RE/kg diet), or were injected i.p. with 3-methylcholanthrene (3-MC) (6 x 20 mg/kg body wt) before and during i.p. treatment with AFB1 (2 x 1 mg/kg body wt). The rats were later submitted to 2-acetylaminofluorene treatment and partial hepatectomy, and placental glutathione S-transferase-positive liver foci were detected and quantified. The in vivo effects of carotenoids or of 3-MC on AFB1-induced liver DNA damage were evaluated using diff…

MaleVitaminCancer Researchmedicine.medical_specialtyAflatoxinAflatoxin B1DNA damage[SDV]Life Sciences [q-bio]Biology03 medical and health scienceschemistry.chemical_compoundLiver Neoplasms Experimental0302 clinical medicineAstaxanthinInternal medicinemedicineAnimalsAnticarcinogenic AgentsCanthaxanthinRats WistarVitamin ACYTOCHROME P 450CarotenoidSerum Albumin030304 developmental biologychemistry.chemical_classification0303 health sciencesRetinolGeneral MedicineCarotenoidsDietRats3. Good health[SDV] Life Sciences [q-bio]Endocrinologychemistry030220 oncology & carcinogenesisCarcinogensMicrosomes LiverRAT2-AcetylaminofluoreneCARCINOGENESEPrecancerous ConditionsDNA DamageMethylcholanthrene
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Dietary lycopene decreases the initiation of liver preneoplastic foci by diethylnitrosamine in the rat

1997

To test whether carotenoids can modulate the initiation of liver preneoplasia by diethylnitrosamine (DEN) or by 2-nitropropane (2-NP) in a sequential protocol of hepatocarcinogenesis, male weanling rats were fed for three or four weeks (respectively) diets containing beta-carotene, canthaxanthin, astaxanthin, or lycopene (300 mg/kg diet) or an excess of vitamin A (15,000 retinol equivalents/kg diet) or were treated intraperitoneally with 3-methylcholanthrene. During this period, all rats were injected intraperitoneally with the initiator carcinogen, either 2-NP (6 times at 100 mg/kg body wt) or DEN (once at 100 mg/kg body wt). Three weeks after the termination of carotenoid or vitamin A fee…

MaleVitaminCancer Researchmedicine.medical_specialtySTRUCTURE[SDV]Life Sciences [q-bio]Medicine (miscellaneous)WeanlingBiologyNitroparaffinsPropane03 medical and health scienceschemistry.chemical_compoundLycopene0302 clinical medicinebeta-CaroteneAstaxanthinInternal medicinemedicineAnimalsAnticarcinogenic AgentsDiethylnitrosamineCanthaxanthinRats Wistar030304 developmental biology0303 health sciencesNutrition and DieteticsLiver NeoplasmsRetinolRetinol EquivalentCarotenoidsLycopeneRats3. Good health[SDV] Life Sciences [q-bio]EndocrinologyOncologychemistry030220 oncology & carcinogenesisCarcinogensRATPrecancerous Conditions
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Post-initiation modulating effects of allyl sulfides in rat hepatocarcinogenesis

2004

Effects of administration of diallyl sulfide (DAS) and diallyl disulfide (DADS) on the promotion stage of hepatocarcinogenesis were investigated in rats using the Ito model. They were compared with those of phenobarbital (PB), a well-known liver promoter in rats. Initiation was induced by a single dose of N-nitrosodiethylamine (NDEA) and 3 weeks later, a partial hepatectomy was conducted. Two weeks after the NDEA injection, rats received either 0.05% allyl sulfides, PB or both in their diet for 8 weeks. Feeding with DAS increased the number of liver preneoplastic foci by 63% with respect to the untreated group. However, rats fed DAS showed a lower foci development than rats fed PB. The DADS…

Malemedicine.medical_treatment[SDV]Life Sciences [q-bio]Toxicologymedicine.disease_causechemistry.chemical_compound0302 clinical medicineLiver Neoplasms ExperimentalDiethylnitrosamineDrug InteractionsDisulfidesComputingMilieux_MISCELLANEOUS0303 health sciencesDiallyl disulfideGeneral MedicineCANCER3. Good healthSpecific Pathogen-Free OrganismsAllyl Compounds[SDV] Life Sciences [q-bio]BiochemistryDiallyl sulfide030220 oncology & carcinogenesismedicine.drugmedicine.medical_specialtySulfidesDIALLYL DISULFITEChemopreventionPreneoplastic foci03 medical and health sciencesInternal medicinemedicineAnimalsAnticarcinogenic AgentsHepatectomyDIALLYL SULFITERats Wistar030304 developmental biologyRatsEndocrinologychemistryRat liverCarcinogensRATPhenobarbitalHepatectomyCarcinogenesisAllyl SulfidePrecancerous ConditionsFood Science
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Problems Associated with the Use of Chemical Class Controls in Absence of Information on the Underlying Mechanism

1982

It is simply not possible to test all chemicals in adequately performed whole animal carcinogenicity tests. Short-term tests are, therefore, frequently used as a substitute. None of these short-term tests give results which correspond in all cases to the results obtained in whole animal carcinogenicity tests. Certain short-term tests have a notoriously poor predictibility for whole animal carcinogenicity for certain classes of chemicals. Therefore, if a chemical, whose carcinogenicity is unknown, is tested in a short-term test, frequently related compounds of known carcinogenicity are also tested simultaneously as chemical class controls.

Mechanism (biology)Carcinogenicity TestWHOLE ANIMALBiologyBioinformaticsMathematical economics
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Assessing the Differential Affinity of Small Molecules for Noncanonical DNA Structures

2012

The targeting of higher-order DNA structures has been thoroughly developed with G-quadruplex DNA but not with other structures like branched DNA (also known as DNA junctions). Because these alternative higher-order DNA architectures might be of high biological relevance, we implemented a high-throughput version of the FRET melting assay that enabled us to map the interactions of a candidate with four different DNA structures (duplex- and quadruplex DNA, three- and four-way junctions) in a rapid and reliable manner. We also introduce a novel index, the BONDS (branched and other noncanonical DNA selectivity) index, to conveniently quantify this differential affinity.

Models MolecularBase pairBiologyG-quadruplex01 natural sciencesBiochemistrySmall Molecule Libraries03 medical and health scienceschemistry.chemical_compoundCaffeineFluorescence Resonance Energy TransferAnticarcinogenic AgentsMolecular BiologyComputingMilieux_MISCELLANEOUS030304 developmental biology0303 health sciencesBase Sequence010405 organic chemistryOrganic ChemistrySmall Molecule LibrariesDNAMolecular biologySmall molecule0104 chemical sciencesG-Quadruplexes[SDV.BBM.BP]Life Sciences [q-bio]/Biochemistry Molecular Biology/BiophysicsQuadruplex DNAFörster resonance energy transferchemistryDuplex (building)BiophysicsNucleic Acid ConformationThermodynamicsMolecular MedicineOrganogold CompoundsDNAChemBioChem
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Bioinformatic and experimental fishing for artemisinin-interacting proteins from human nasopharyngeal cancer cells.

2012

Determining interacting cellular partners of drugs by chemical proteomic techniques is complex and tedious. Most approaches rely on activity-based probe profiling and compound-centric chemical proteomics. The anti-malarial artemisinin also exerts profound anti-cancer activity, but the mechanisms of action are incompletely understood. In the present investigation, we present a novel approach to identify artemisinin-interacting target proteins. Our approach overcomes usual problems in traditional fishing procedures, because the drug was attached to a surface without further chemical modification. The proteins identified effect among others, cell cycle arrest, apoptosis, inhibition of angiogen…

ProteomicsCell cycle checkpointCell growthAngiogenesisComputational BiologyCell migrationApoptosisNasopharyngeal NeoplasmsCell Cycle CheckpointsBiologyProteomicsArtemisininsCell biologyNeoplasm ProteinsNuclear receptorCell cultureCell Line TumormedicineAnticarcinogenic AgentsHumansArtemisininMolecular BiologyBiotechnologymedicine.drugCell ProliferationMolecular bioSystems
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Establishment and comparative characterization of novel squamous cell non-small cell lung cancer cell lines and their corresponding tumor tissue.

2010

Abstract Background Cell lines play an important role for studying tumor biology and novel therapeutic agents. Particularly in pulmonary squamous cell carcinoma (SCC) the availability of cell lines is limited and knowledge about their representativeness for corresponding tumor tissue is scanty. Materials and methods We established three novel SCC cell lines from fresh tumor tissue of 28 donors, including 8 SCC. Two cell lines were derived from different localizations of the same donor, i.e. primary tumor and lymph node metastasis. This represents a so far unique combination in lung cancer. The genotypes, gene expression profiles and mutational status of epidermal growth factor receptor ( EG…

Pulmonary and Respiratory MedicineCancer ResearchLung NeoplasmsAngiogenesisCarcinogenicity TestsCellIn situ hybridizationCell Growth ProcessesBiologymedicine.disease_causeMiceCell MovementCarcinoma Non-Small-Cell LungCell Line TumormedicineCell AdhesionAnimalsHumansCell LineageIn Situ Hybridization FluorescenceMutationComparative Genomic Hybridizationmedicine.diagnostic_testNeovascularization PathologicGene Expression ProfilingCell Differentiationmedicine.diseasePrimary tumorMolecular biologyDNA FingerprintingGene expression profilingErbB Receptorsmedicine.anatomical_structureGenes rasOncologyCell cultureTandem Repeat SequencesLymphatic MetastasisMutationCarcinoma Squamous CellFluorescence in situ hybridizationLung cancer (Amsterdam, Netherlands)
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Resveratrol as a Chemopreventive Agent: A Promising Molecule for Fighting Cancer

2006

International audience; Resveratrol (3,4',5 tri-hydroxystilbene) is a phytoalexin produced in hudge amount in grapevine skin in response to infection by Bothrytis cinerea. This production of resveratrol blocks the proliferation of the pathogen, thereby acting as a natural antibiotic. Numerous studies have reported interesting properties of trans-resveratrol as a preventive agent against important pathologies i.e. vascular diseases, cancers, viral infection or neurodegenerative processes. Moreover, several epidemiological studies have revealed that resveratrol is probably one of the main microcomponents of wine responsible for its health benefits such as prevention of vaso-coronary diseases …

Radiation-Sensitizing AgentsMESH : Radiation-Sensitizing AgentsAngiogenesisClinical BiochemistryTumor initiationPharmacologyResveratrolBiologyMESH : Antineoplastic Agents Phytogenicmedicine.disease_causeMESH : Anticarcinogenic AgentsMESH : Stilbeneschemistry.chemical_compoundNeoplasmsMESH : Cell CycleStilbenesDrug DiscoverymedicineAnimalsAnticarcinogenic AgentsHumansCytotoxicity[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyPharmacologychemistry.chemical_classificationPhytoalexinMESH : HumansCell Cyclefood and beveragesCancerCell cyclemedicine.diseaseMESH : NeoplasmsAntineoplastic Agents PhytogenicchemistryResveratrolMolecular MedicineMESH : AnimalsCarcinogenesisCurrent Drug Targets
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Effects of garlic powders with varying alliin contents on hepatic drug metabolizing enzymes in rats

2003

International audience; The anticarcinogenic effect of garlic has been demonstrated in both epidemiologic and experimental studies. In this study, possible mechanisms involved in the anticarcinogenic effect of garlic consumption were assessed by determining its capacity to alter drug metabolizing enzymes, in relation with its alliin content. Rats were fed a diet for 2 weeks containing 5% garlic powders produced from bulbs grown on soils with different levels of sulfate fertilization and therefore containing differing amounts of alliin. Activities of several hepatic enzymes, which are important in carcinogen metabolism such cytochromes P450 (CYP) and phase II enzymes, were determined. Garlic…

S01 - Nutrition humaine - Considérations généralesMaleDiallyl disulfideAlliinPharmacognosyhttp://aims.fao.org/aos/agrovoc/c_11091chemistry.chemical_compound0302 clinical medicinehttp://aims.fao.org/aos/agrovoc/c_4395[SDV.IDA]Life Sciences [q-bio]/Food engineeringGlucuronosyltransferaseComputingMilieux_MISCELLANEOUSAilGlutathione Transferasechemistry.chemical_classification0303 health sciencesbiologyDiallyl disulfidehttp://aims.fao.org/aos/agrovoc/c_2603food and beveragesBiological activityCytochrome P-450 CYP2E1[SDV.IDA] Life Sciences [q-bio]/Food engineering3. Good healthBiochemistryLiver030220 oncology & carcinogenesisGeneral Agricultural and Biological SciencesAllium sativumDrug-metabolizing enzymesFoiehttp://aims.fao.org/aos/agrovoc/c_290Médicamenthttp://aims.fao.org/aos/agrovoc/c_25197Alliin03 medical and health scienceshttp://aims.fao.org/aos/agrovoc/c_2395Cytochrome P-450 CYP1A2Cytochrome P-450 CYP1A1AnimalsAnticarcinogenic AgentsCysteineRats WistarQ04 - Composition des produits alimentairesGarlic030304 developmental biologySantéCytochrome P450General ChemistryGlutathioneAllium sativumPropriété pharmacologiqueDietRatsEnzymechemistryEnzymebiology.proteinRAThttp://aims.fao.org/aos/agrovoc/c_3511http://aims.fao.org/aos/agrovoc/c_6464
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