Search results for "caspase"

showing 10 items of 390 documents

Triclosan activates aryl hydrocarbon receptor (AhR)-dependent apoptosis and affects Cyp1a1 and Cyp1b1 expression in mouse neocortical neurons.

2016

Triclosan (TCS) is an antimicrobial agent that is used extensively in personal care and in sanitizing products, such as soaps, toothpastes, and hair products. A number of studies have revealed the presence of TCS in human tissues, such as fat, liver and brain, in addition to blood and breast milk. The aim of the present study was to investigate the impact of TCS on AhR and Cyp1a1/Cyp1b1 signaling in mouse neocortical neurons in primary cultures. In addition to the use of selective ligands and siRNAs, expression levels of mRNA and proteins as well as caspase-3 activity, reactive oxygen species (ROS) formation, and lactate dehydrogenase (LDH) release have been measured. We also studied the in…

0301 basic medicinemedicine.medical_specialtySmall interfering RNAStimulationCaspase 3ApoptosisNeocortex010501 environmental sciencesBiology01 natural sciencesBiochemistry03 medical and health sciencesMiceInternal medicinemedicineCytochrome P-450 CYP1A1Cyp1a1AnimalsRNA MessengerCells Cultured0105 earth and related environmental sciencesGeneral Environmental Sciencechemistry.chemical_classificationNeuronsReactive oxygen speciesCaspase 3fungiAhRNeurotoxicityCyp1b1respiratory systemNeuronmedicine.diseaseAryl hydrocarbon receptorTriclosanCell biology030104 developmental biologyEndocrinologyMechanism of actionchemistryReceptors Aryl HydrocarbonApoptosisCytochrome P-450 CYP1B1biology.proteinAnti-Infective Agents LocalFemalemedicine.symptomReactive Oxygen SpeciesEnvironmental research
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Synthesis, antiproliferative activity and possible mechanism of action of novel 2-acetamidobenzamides bearing the 2-phenoxy functionality.

2015

Several new 2-(2-phenoxyacetamido)benzamides 17a-v, 21 and 22 were synthesized by stirring in pyridine the acid chlorides 16a-e and the appropriate5-R-4-R1-2-aminobenzamide 15a-e and initially evaluated in vitro for antiproliferative activity against the K562 (human chronic myelogenous leukemia) cell line. Some of synthesized compounds were evaluated for their in vitro antiproliferative activity against the full NCI tumor cell line panel derived from nine clinically isolated cancer types (leukemia, non-small cell lung, colon, CNS, melanoma, ovarian, renal, prostate and breast). The most active compounds caused an arrest of K562 cells in the G0-G1 phase of cell cycle and induction of apoptos…

3003Clinical BiochemistryCellPharmaceutical ScienceAntineoplastic AgentsApoptosisAntiproliferative activityPharmacologyG0/G1 arrestBiochemistryArticle2-(2-Phenoxyacetamido)benzamideAntineoplastic AgentStructure-Activity RelationshipBenzamideSettore BIO/10 - BiochimicaCell Line TumorDrug DiscoveryG1 Phase Cell Cycle CheckpointK562 CellmedicineHumansMolecular BiologyCell ProliferationCell growthChemistryDrug Discovery3003 Pharmaceutical ScienceOrganic ChemistryApoptosiCell cyclemedicine.diseaseCaspaseSettore CHIM/08 - Chimica FarmaceuticaG1 Phase Cell Cycle CheckpointsLeukemiamedicine.anatomical_structureMicroscopy FluorescenceCell cultureApoptosisCaspasesBenzamidesMolecular MedicineDrug Screening Assays AntitumorK562 CellsPro-caspase 3HumanK562 cellsChronic myelogenous leukemiaBioorganicmedicinal chemistry
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Proeryptotic Activity of 4-Hydroxynonenal: A New Potential Physiopathological Role for Lipid Peroxidation Products

2020

Background: Eryptosis is a physiological, apoptosis-like death of injured erythrocytes crucial to prevent premature haemolysis and the pathological sequalae generated by cell-free haemoglobin. When dysregulated, the process is associated to several inflammatory-based pathologies. 4-Hydroxy-trans-2-nonenal (HNE) is an endogenous signalling molecule at physiological levels and, at higher concentrations, is involved in the pathogenesis of several inflammatory-based diseases. This work evaluated whether HNE could induce eryptosis in human erythrocytes. Methods: Measurements of phosphatidylserine, cell volume, intracellular oxidants, Ca++, glutathione, ICAM-1, and ceramide were assessed by flow …

Adult0301 basic medicineCeramideErythrocyteslcsh:QR1-502PhosphatidylserinesBiochemistryArticleRBClcsh:Microbiology4-HydroxynonenalLipid peroxidationprostaglandins03 medical and health scienceschemistry.chemical_compound0302 clinical medicineeryptosisCell AdhesionHuman Umbilical Vein Endothelial CellsHumansMolecular BiologyCells CulturedCaspaseAldehydesbiologyGlutathionePhosphatidylserineMiddle AgedIntercellular Adhesion Molecule-1Haemolysislipid peroxidation productsGlutathione4-hydroxynonenalCell biology030104 developmental biologychemistryinflammation030220 oncology & carcinogenesisbiology.proteinCalciumLipid PeroxidationIntracellularBiomolecules
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Involvement of caspase-3 and GD3 ganglioside in ceramide-induced apoptosis in Farber disease.

2000

Farber's disease (FD) is a rare genetic disorder caused by ceramidase deficiency, which results in ceramide accumulation in lung, liver, colon, skeletal muscle, cartilage, and bone. Although this disease has been symptomatically characterized, little is known about its molecular pathogenetic process. Because recent studies reported that ceramide accumulation induces GD3 ganglioside formation and apoptosis, we investigated, in tissue obtained via colonoscopy from seriously involved patients, the possible involvement of ceramide in FD colonocyte destruction. Histochemical and TUNEL analyses of paraffin-embedded sections revealed that 45 ± 4.3% of FD colonocytes showed morphological signs of …

AdultCeramidePathologymedicine.medical_specialtyHistologyColonCaspase 3ApoptosisCeramideschemistry.chemical_compoundGangliosidesmedicineGD3 gangliosideHumansIntestinal MucosaCaspaseFarber diseaseFarber diseaseTUNEL assaybiologyCaspase 3ApoptosiCell Biologymedicine.diseaseCeramidaseCaspaseK18EpitheliumActive caspase-3Lysosomal Storage Diseasesmedicine.anatomical_structurechemistryApoptosisCaspasesCancer researchbiology.proteinAnatomyActive caspase-3; Apoptosis; Caspases; Farber disease; GD3 ganglioside; K18; Anatomy; Cell BiologyThe journal of histochemistry and cytochemistry : official journal of the Histochemistry Society
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Haplotypes of the caspase-1 gene, plasma caspase-1 levels, and cardiovascular risk.

2006

Caspase-1 processes the interleukin (IL)-1β and IL-18 inactive precursors to the biologically active cytokines that are known to have proatherogenic effects. The present study investigated the genetic variability of the CASP1 gene and plasma levels of caspase-1 in relation to cardiovascular risk. In Europeans, 3 tag SNPs captured 4 common haplotypes of the CASP1 gene. Among these, the A in6 allele of the G+7/in6A polymorphism was less frequent in 246 cases with myocardial infarction and a parental history of disease than in 253 controls free of familial history of disease (0.13±0.02 versus 0.20±0.02; P =0.005). However, in a larger case/control study (n=1774), these effects are borderline …

AdultMale/dk/atira/pure/subjectarea/asjc/1300/1314medicine.medical_specialtyPathologyGenotypePhysiologyPopulationMyocardial Infarction/dk/atira/pure/subjectarea/asjc/2700/2705Single-nucleotide polymorphismCoronary Artery DiseaseBiologyPolymorphism Single NucleotideRisk AssessmentCoronary artery diseaseCohort StudiesGene FrequencyPolymorphism (computer science)Internal medicinemedicineHumansGenetic Predisposition to DiseaseProspective StudiesAlleleeducationProspective cohort studyAgededucation.field_of_studyVascular diseaseHazard ratioCaspase 1Interleukin-18Genetic VariationMiddle Agedmedicine.diseaseEndocrinologyHaplotypesCardiovascular DiseasesCase-Control StudiesFemaleCardiology and Cardiovascular MedicineFollow-Up StudiesCirculation research
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GDF 15 as an anti-apoptotic, diagnostic and prognostic marker in oral squamous cell carcinoma

2011

Growth-differentiation factor 15 (GDF 15) is involved in tumor pathogenesis and its expression is increased in many types of cancers. Functional effects of GDF 15 on oncogenesis of oral squamous cell carcinoma (OSCC) remain unclear. Therefore, the aim of this study was to examine the apoptotic characteristics of GDF 15 in OSCC cell lines in vitro and to analyze serum GDF 15 concentrations as a diagnostic and prognostic tumor marker for OSCC in vivo. Caspase activity was assessed in OSCC cell lines with the Caspase-Glo 3/7 system. Serum GDF 15 concentrations from 64 patients with histopathological proven OSCC and from 30 healthy volunteers were measured using an enzyme-linked immunosorbent a…

AdultMaleCancer ResearchPathologymedicine.medical_specialtyGrowth Differentiation Factor 15Enzyme-Linked Immunosorbent AssayCaspase 3Biologymedicine.disease_causePathogenesisIn vivoCell Line TumorBiomarkers TumorCarcinomamedicineHumansAgedTumor markerAged 80 and overMiddle AgedPrognosismedicine.diseasestomatognathic diseasesOncologyApoptosisCase-Control StudiesCaspasesembryonic structuresCarcinoma Squamous CellCancer researchFemaleMouth NeoplasmsGDF15Oral SurgeryCarcinogenesisOral Oncology
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Poorly differentiated synovial sarcoma: A case report

2001

Poorly differentiated synovial sarcoma is a rare soft tissue tumor. We studied a case arising in the pleural cavity of a young subject, characterised by the presence of spindle cell, small cell, and large epithelioid cell areas. We performed stains for mucosubstances and analysed the expression of cytokeratins 5/6, 7, 8, 18, 19, CEA, CD34, Ber-Ep4 and calretinin to characterize the phenotype of this neoplasm. We furthermore assessed immunohistochemically the presence of p53, Bcl-2, Bax and caspase 3, four apoptotic markers, to evaluate a relationship between apoptotic activity and the behaviour of this tumor. Our findings showed a strong presence of calretinin, p53 and Bcl-2 in all three ar…

AdultMaleCancer ResearchPathologymedicine.medical_specialtyPleural Neoplasms2734CD34Caspase 3Pathology and Forensic MedicineSynovial sarcomaImmunoenzyme TechniquesSarcoma SynovialS100 Calcium Binding Protein GBcl-2-associated X proteinCalretininProto-Oncogene ProteinsBiomarkers TumormedicineHumansCaspasebcl-2-Associated X ProteinbiologyCaspase 3ApoptosiSoft tissue tumorGeneral MedicinePoorly differentiated synovial sarcomamedicine.diseaseSynovial sarcomaNeoplasm ProteinsApoptosis; Calretinin; Poorly differentiated synovial sarcoma; Soft tissue tumors; Synovial sarcoma; Cancer Research; Oncology; 2734Proto-Oncogene Proteins c-bcl-2OncologyCalbindin 2Caspasesbiology.proteinSarcomaTumor Suppressor Protein p53CalretininEpithelioid cell
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The role of intrinsic pathway in apoptosis activation and progression in Peyronie's Disease.

2014

Peyronie’s disease (PD) is characterized with formation of fibrous plaques which result in penile deformity, pain, and erectile dysfunction. The aim of this study was to investigate the activation of the intrinsic apoptotic pathway in plaques from PD patients. Tunica albuginea from either PD or control patients was assessed for the expression of bax, bcl-2 and caspases 9 and 3 using immunohistochemistry and by measurement of apoptotic cells using TUNEL assay. Bax overexpression was observed in metaplastic bone tissue, in fibroblasts, and in myofibroblast of plaques from PD patients. Little or no bcl-2 immunostaining was detected in samples from either patients or controls. Caspase 3 immunos…

AdultMalePathologymedicine.medical_specialtyArticle SubjectPenile Indurationlcsh:MedicineCaspase 3Apoptosisapoptosis peironie's disease urethraBone tissueGeneral Biochemistry Genetics and Molecular BiologySettore MED/24 - UrologiaYoung AdultBcl-2-associated X proteinPeyronie’s diseasemedicineIn Situ Nick-End LabelingHumansCaspaseAgedbcl-2-Associated X ProteinGeneral Immunology and MicrobiologybiologyStaining and LabelingOssificationCaspase 3Settore BIO/16 - Anatomia Umanalcsh:RGeneral MedicineMiddle Agedmedicine.diseaseImmunohistochemistryCaspase 9medicine.anatomical_structurePeyronie’s disease. ; Apoptosis; Immunohistochemistrybiology.proteinDisease Progressionmedicine.symptomPeyronie's diseaseMyofibroblastImmunostainingSignal TransductionResearch Article
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Expression of the tumor necrosis factor receptor—associated factors 1 and 2 and regulation of the nuclear factor—kB antiapoptotic activity in human g…

2005

Object. Tumor necrosis factor receptor (TNFR)—associated factors (TRAFs) are a recently established group of proteins involved in the intracellular signaling of the TNFR superfamily members. The TRAFs have been implicated in promoting cell survival through the activation of transcription factor nuclear factor (NF)—κB. The authors investigated the expression of NF-κB, caspase 3, TRAF1, TRAF2, and TRAF-associated NF-κB activator/TRAF—interacting protein (TANK/I-TRAF), a regulator of TRAF activity, in human gliomas. Methods. Tumor samples were obtained in 27 adult patients harboring seven low-grade gliomas, nine anaplastic astrocytomas, and 11 glioblastomas multiforme. The NF-κB activation was…

AdultMalePathologymedicine.medical_specialtyBlotting WesternTRAF1Apoptosis Blotting Brain Neoplasms/metabolism Brain Neoplasms/pathology Caspase GliomaApoptosisCaspase 3BiologyGliomamedicineHumansTranscription factorAgedAged 80 and overTankyrasesBrain NeoplasmsCaspase 3NF-kappa BGliomaMiddle AgedTNF Receptor-Associated Factor 2medicine.diseaseTNF Receptor-Associated Factor 1Up-RegulationIntracellular signal transductionBlotTumor Necrosis Factor Receptor-Associated FactorsCaspasesCancer researchFemaleTumor necrosis factor alphaSignal TransductionJournal of Neurosurgery
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Cigarette smoke causes caspase-independent apoptosis of bronchial epithelial cells from asthmatic donors

2015

BackgroundEpidemiologic studies have demonstrated important links between air pollution and asthma. Amongst these pollutants, environmental cigarette smoke is a risk factor both for asthma pathogenesis and exacerbation. As the barrier to the inhaled environment, the bronchial epithelium is a key structure that is exposed to cigarette smoke.ObjectivesSince primary bronchial epithelial cells (PBECs) from asthmatic donors are more susceptible to oxidant-induced apoptosis, we hypothesized that they would be susceptible to cigarette smoke-induced cell death.MethodsPBECs from normal and asthmatic donors were exposed to cigarette smoke extract (CSE); cell survival and apoptosis were assessed by fl…

AdultMaleProgrammed cell deathDNA damageScienceCaspase 3ApoptosisBronchiBiologyTobacco smokeAntioxidantschemistry.chemical_compoundYoung Adultparasitic diseasesHumansAgricultural and Biological Sciences (all); Biochemistry Genetics and Molecular Biology (all); Medicine (all)MultidisciplinaryBiochemistry Genetics and Molecular Biology (all)Caspase-Independent ApoptosisCaspase 3Medicine (all)QSmokingREpithelial CellsGlutathioneMiddle AgedAscorbic acid3. Good healthchemistryAgricultural and Biological Sciences (all)13. Climate actionApoptosisImmunologyMedicineFemaleResearch Article
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