Search results for "cell count"

showing 10 items of 270 documents

Catalase-independent early-gene expression in rat brain following acute ethanol exposure

2004

Early-gene expression evoked by acute ethanol treatment was studied in rat brain by quantitative immunocytochemistry, with reference to ethanol metabolism by the enzyme catalase. Colocalization with mu-opioid receptor (MOR) sites was also examined. Ethanol challenges [1, 2.5, and 4 g/kg intraperitoneally (i.p.)] evoked dose-dependent increases in c-Fos expression in several brain regions, but overlap with MOR-rich sites was only partial. Strong inhibition of brain catalase activity (ca. 60%) with 3-amino-1,2,4-triazole (AT, 1 g/kg i.p.) did not alter ethanol-induced c-Fos nor Krox-24 expression in any of the brain regions analyzed. This evidence demonstrates that catalase-mediated metabolis…

MaleNervous systemmedicine.medical_specialtyCentral nervous systemReceptors Opioid muGene ExpressionCell Countc-FosRats Sprague-DawleyInternal medicinemedicineAnimalsEnzyme InhibitorsEthanol metabolismMolecular BiologyAmitroleBrain ChemistryEthanolbiologyGeneral NeuroscienceBrainCentral Nervous System DepressantsColocalizationCatalaseImmunohistochemistryRatsmedicine.anatomical_structureEndocrinologyCatalasebiology.proteinNeurology (clinical)μ-opioid receptorProto-Oncogene Proteins c-fosImmediate early geneDevelopmental BiologyBrain Research
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Mepolizumab effectiveness on small airway obstruction, corticosteroid sparing and maintenance therapy step-down in real life

2020

Background: Mepolizumab (MEP) has been recently introduced to treat severe eosinophilic asthma. Trials have demonstrated a significant effectiveness in this asthma phenotype. We evaluated MEP efficacy on lung function, symptoms, asthma exacerbations, biologic markers, steroid dependence and controller treatment level in real-life. Methods: We retrospectively analyzed 134 severe asthmatics (61 males; mean age 58.3 ± 11; mean FEV1%:72 ± 21), treated with MEP for at least 6 months (mean duration:10.9 ± 3.7 months). Results: FEV1% improved significantly after MEP. Mean FEF25-75 also increased from 37.4 ± 25.4% to 47.2 ± 27.2% (p < 0.0001). Mean baseline blood eosinophil level was 712 ± 731/μ…

MaleOmalizumabFEF(25-75)step-down0302 clinical medicineMaintenance therapyAdrenal Cortex HormonesSettore MED/10Pharmacology (medical)Anti-Asthmatic Agents030212 general & internal medicinesmall airwaysAsthma exacerbationsmepolizumabMiddle AgedAnesthesiaFEF25-75CorticosteroidDrug Therapy CombinationFemalemedicine.drugPulmonary and Respiratory Medicinesevere asthmamedicine.drug_classMepolizumab Severe asthma Real-life Small airways FEF25-75 Oral corticosteroids Step-down EffectiveneseffectivenessSettore MED/10 - Malattie Dell'Apparato Respiratoriooral corticosteroidsAntibodies Monoclonal HumanizedNO03 medical and health sciencesEffectiveness FEF 25-75 Mepolizumab Oral corticosteroids Real-life Severe asthma Small airways Step-downmedicineHumansIn real lifeFEFreal-lifeAgedRetrospective StudiesBiologic markerbusiness.industryEffectivenesBiochemistry (medical)Effectiveness; FEF; 25-75; Mepolizumab; Oral corticosteroids; Real-life; Severe asthma; Small airways; Step-downAirway obstructionmedicine.diseaseAsthmaBlood Cell Countrespiratory tract diseasesAirway ObstructionEosinophils030228 respiratory systemeffectiveness; FEF(25-75); mepolizumab; oral corticosteroids; real-life; severe asthma; small airways; step-down25-75businessMepolizumab
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Chronic fluoxetine treatment alters the structure, connectivity and plasticity of cortical interneurons

2014

Novel hypotheses suggest that antidepressants, such as the selective serotonin reuptake inhibitor fluoxetine, induce neuronal structural plasticity, resembling that of the juvenile brain, although the underlying mechanisms of this reopening of the critical periods still remain unclear. However, recent studies suggest that inhibitory networks play an important role in this structural plasticity induced by fluoxetine. For this reason we have analysed the effects of a chronic fluoxetine treatment in the hippocampus and medial prefrontal cortex (mPFC) of transgenic mice displaying eGFP labelled interneurons. We have found an increase in the expression of molecules related to critical period pla…

MalePERINEURONAL NET EXPRESSIONTime FactorsDendritic spinePSA-NCAMCritical period plasticityHippocampusCell CountADULT BRAIN PLASTICITYTREATMENT INCREASESHippocampusMice0302 clinical medicinePharmacology (medical)Prefrontal cortexCerebral Cortex0303 health sciencesNeuronal PlasticitybiologyGlutamate DecarboxylaseMEDIAL PREFRONTAL CORTEXPOLYSIALIC ACIDmusculoskeletal neural and ocular physiologyPerineuronal net3. Good healthPsychiatry and Mental healthParvalbuminsmedicine.anatomical_structureCerebral cortexCELL-ADHESION MOLECULEAntidepressive Agents Second-GenerationDendritic SpinesGreen Fluorescent ProteinseducationMice TransgenicNerve Tissue ProteinsNeural Cell Adhesion Molecule L1Inhibitory postsynaptic potentialRAT HIPPOCAMPUS03 medical and health sciencesmedicineAnimalsPSA-NCAM EXPRESSION030304 developmental biologyPharmacologyperineuronal netsinterneuronsCENTRAL-NERVOUS-SYSTEMfluoxetine3112 NeurosciencesGene Expression Regulationnervous systemVesicular Glutamate Transport Protein 1Sialic Acidsbiology.proteinNeural cell adhesion moleculeNerve NetNeuroscience030217 neurology & neurosurgeryParvalbuminThe International Journal of Neuropsychopharmacology
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Upregulation of activin-B and follistatin in pulmonary fibrosis: a translational study using human biopsies and a specific inhibitor in mouse fibrosi…

2014

Background: Activins are members of the TGF-ß superfamily of growth factors. First, we identified by expression array screening that activin-B and follistatin are upregulated in human idiopathic pulmonary fibrosis (IPF). Next, we wanted to clarify their specific role in lung fibrosis formation. Methods: We used specific antibodies for activin-A and -B subunits and follistatin to measure and localize their levels in idiopathic pulmonary fibrosis and control lung biopsies. To inhibit activin signaling, we used soluble activin type IIB receptor fused to the Fc portion of human IgG1 (sActRIIB-Fc) in two different mouse models of pulmonary fibrosis. Results: Activin-B and follistatin mRNA levels…

MalePathologyFollistatinPulmonary FibrosisPROTEINCell CountQuadriceps MuscleACTIVATIONIdiopathic pulmonary fibrosisMiceBMP-7FibrosisPulmonary fibrosisfollistatinInhibin-beta SubunitsGREMLINImmunity Cellularmedicine.diagnostic_testbiologyactivinsPIRFENIDONEPirfenidonerespiratory systemidiopathic pulmonary fibrosisMouse fibrosis model3. Good healthUp-RegulationActivinsmedicine.anatomical_structureACUTE EXACERBATIONmouse fibrosis modelembryonic structuresGROWTHBronchoalveolar Lavage Fluidhormones hormone substitutes and hormone antagonistsmedicine.drugResearch ArticleSignal TransductionPulmonary and Respiratory MedicineEXPRESSIONmedicine.medical_specialtyendocrine systemRecombinant Fusion ProteinseducationIdiopathic pulmonary fibrosisRespiratory MucosaAlveolar cellsINFLAMMATIONmedicineAnimalsHumansRNA MessengerLungbusiness.industrymedicine.diseaserespiratory tract diseasesMice Inbred C57BLPulmonary AlveoliDisease Models AnimalBronchoalveolar lavageProtein Biosynthesis3121 General medicine internal medicine and other clinical medicinebiology.proteinbusinessFollistatin
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BLBP-expression in astrocytes during experimental demyelination and in human multiple sclerosis lesions

2011

Several lines of evidence indicate that remyelination represents one of the most effective mechanisms to achieve axonal protection. For reasons that are not yet understood, this process is often incomplete or fails in multiple sclerosis (MS). Activated astrocytes appear to be able to boost or inhibit endogenous repair processes. A better understanding of remyelination in MS and possible reasons for its failure is needed. Using the well-established toxic demyelination cuprizone model, we created lesions with either robust or impaired endogenous remyelination capacity. Lesions were analyzed for mRNA expression levels by Affymetrix GeneChip® arrays. One finding was the predominance of immune a…

MalePathologyPlatelet-derived growth factormedicine.medical_treatmentCell CountBehavioral Neurosciencechemistry.chemical_compoundMice0302 clinical medicineFluorescent Antibody Technique IndirectOligonucleotide Array Sequence AnalysisPlatelet-Derived Growth Factor0303 health sciencesGlial fibrillary acidic proteinbiologyExperimental autoimmune encephalomyelitisAstrocytomaMiddle AgedImmunohistochemistrymedicine.anatomical_structureFemaleFibroblast Growth Factor 2Fatty Acid-Binding Protein 7Adultmedicine.medical_specialtyEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisImmunologyBlotting WesternNerve Tissue ProteinsFatty Acid-Binding ProteinsReal-Time Polymerase Chain ReactionTransfection03 medical and health sciencesCuprizoneCell Line TumorGlial Fibrillary Acidic ProteinmedicineAnimalsHumansRNA MessengerRemyelination030304 developmental biologyAgedEndocrine and Autonomic SystemsMultiple sclerosisGrowth factorTumor Suppressor Proteinsmedicine.diseaseOligodendrocyteMice Inbred C57BLchemistryAstrocytesbiology.proteinOsteopontinCarrier Proteins030217 neurology & neurosurgeryDemyelinating Diseases
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Distribution and density of CD1a+ and CD83+ dendritic cells in HPV-associated laryngeal papillomas.

2009

Summary Background Respiratory papillomatosis associated with human papilloma virus (HPV) infection is the most common benign laryngeal neoplasm. The age of patients at disease onset, HPV type, number of surgeries are well known prognostic factors of the disease course. The correlation between dendritic cell (DC) density in tumor tissue and clinical prognosis was established. Aim The aim of our study was to estimate the density of DC in laryngeal papillomas associated with HPV types 6/11 infection and to evaluate the relationship between the number of DC and the disease severity. Materials and methods Our study included 40 randomly selected biopsy specimens from patients with HPV-positive l…

MalePathologymedicine.medical_specialtyAdolescentImmunoglobulinsCell CountAntigens CD1Young AdultAntigens CDImmunopathologyBiopsymedicineHumansTissue DistributionChildLaryngeal NeoplasmsRetrospective StudiesLamina propriaMembrane Glycoproteinsmedicine.diagnostic_testPapillomaBenign Laryngeal Neoplasmbusiness.industryHuman papillomavirus 11Papillomavirus InfectionsInfantGeneral MedicineDendritic Cellsmedicine.diseaseHuman papillomavirus 6Epitheliummedicine.anatomical_structureOtorhinolaryngologyChild PreschoolPediatrics Perinatology and Child HealthPapillomaFemaleRecurrent Respiratory PapillomatosisLarynxbusinessLaryngeal papillomatosisInternational journal of pediatric otorhinolaryngology
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Stage-dependent and sector-specific neuronal loss in hippocampus during Alzheimer's disease

2000

Recent stereological studies documented a severe loss of hippocampal neurons in end-stage Alzheimer's disease. The development of the disease, however, is progressive and slow, over clinically inconspicuous decades. The Braak-staging system distinguishes six histopathological stages some of which are not accompanied by clinical symptoms. We analyzed hippocampal cell loss in correlation to Braak stages. Neuron numbers were determined with unbiased stereological principles in a defined subportion of the hippocampus of 28 subjects. There were no age-dependent neuronal losses in any of the hippocampal subdivisions examined. Compared to stage I, pyramidal cell loss in CA1 was reduced by 33% in s…

MalePathologymedicine.medical_specialtyHippocampusCell CountStereologyHippocampal formationBiologyHippocampusPathology and Forensic MedicineCentral nervous system diseaseCellular and Molecular NeuroscienceAlzheimer DiseasemedicineHumansAgedAged 80 and overNeuronsSubiculumMiddle Agedmedicine.diseasemedicine.anatomical_structurenervous systemFemaleNeurology (clinical)NeuronPyramidal cellAlzheimer's diseaseActa Neuropathologica
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Tissue evacuated during joint replacement procedure as a source of mononuclear cells.

2017

Background Different cell populations from bone marrow were used in various clinical trials for cardiac diseases during last decade. Four clinical studies are ongoing in our institution and enroll patients with cardiac diseases, coronary disease, type 2 diabetes, and osteoarthritis. The density gradient is used to separate bone marrow mononuclear cells. Joint replacement procedures were associated with significant loss of tissue. Usually, excess tissue as bone marrow, peripheral blood and fat are removed to clean operation site. The aim of this study is to prove whether removed tissue during joint replacement procedure can be considered as a significant source of mononuclear cells. Methods …

MalePathologymedicine.medical_specialtyJoint replacementmedicine.medical_treatmentArthroplasty Replacement HipPopulationCD34Antigens CD34OsteoarthritisPunctures030204 cardiovascular system & hematologyPeripheral blood mononuclear cellIliac crestTransplantation AutologousMonocytesOsteoarthritis HipIlium03 medical and health sciences0302 clinical medicinemedicineCentrifugation Density GradientHumansOrthopedics and Sports MedicineBone marrow030212 general & internal medicineExcised tissueeducationArthroplasty Replacement Kneeeducation.field_of_studybusiness.industryCell extractionJoint replacementTissue ProcessingMiddle AgedOsteoarthritis Kneemedicine.diseaseFlow CytometryCell countCell yieldmedicine.anatomical_structureMononuclear cellsCase-Control StudiesTissue and Organ HarvestingSurgeryFemaleBone marrowbusinessOriginal Article • HIP - ARTHROPLASTYEuropean journal of orthopaedic surgerytraumatology : orthopedie traumatologie
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Differential distribution of inflammatory cells in large and small airways in smokers

2007

BACKGROUND: Smoking induces structural changes in the airways, and is considered a major factor in the development of airflow obstruction in chronic obstructive pulmonary disease. However, differences in inflammatory cell distribution between large airways (LA) and small airways (SA) have not been systematically explored in smokers. Hypothesis: The content of cells infiltrating the airway wall differs between LA and SA. AIMS: To compare the content of neutrophils, macrophages, lymphocytes and mast cells infiltrating LA and SA in smokers who underwent surgery for lung cancer. METHODS: Lung tissue from 15 smokers was analysed. Inflammatory cells in the lamina propria were identified by immuno…

MalePathologymedicine.medical_specialtyLung NeoplasmsNeutrophilsCOPD inflammationCell CountInflammationRespiratory MucosaSettore MED/10 - Malattie Dell'Apparato RespiratorioLung injuryPathology and Forensic MedicineSmokeHumansMedicineLymphocytesMast CellsRespiratory systemLung cancerLungPhagocytesLamina propriaLungbusiness.industryMacrophagesSmokingGeneral MedicineMiddle Agedrespiratory systemmedicine.diseaseImmunohistochemistryrespiratory tract diseasesmedicine.anatomical_structureImmunologyImmunohistochemistryOriginal ArticleFemalemedicine.symptombusinessRespiratory tractJournal of Clinical Pathology
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Comparison of different quantification methods to determine hippocampal damage after cerebral ischemia

2014

Abstract Background Experimental stroke studies use multiple techniques to evaluate histopathological damage. Unfortunately, sensitivity and reproducibility of these techniques are poorly characterized despite pivotal influence on results. Method The present study compared several quantification methods to differentiate between two severities of global cerebral ischemia and reperfusion. Male Sprague-Dawley rats were randomized to moderate (10 min) or severe (14 min) ischemia by bilateral carotid occlusion (BCAO) with hemorrhagic hypotension. Neuronal cell count was determined in hippocampus at bregma −3.14 mm and −3.8 mm on day 3 and 28 post insult by counting neurons in the whole CA1 or in…

MalePathologymedicine.medical_specialtyTime FactorsIntracranial HypotensionIschemiaHippocampusCell CountNerve Tissue ProteinsBrain damageHippocampal formationSeverity of Illness IndexBrain IschemiaRats Sprague-DawleyBrain ischemiaRandom AllocationAnimalsMedicineColoring AgentsCA1 Region HippocampalStrokeFluorescent DyesNeuronsbiologyHistocytochemistrybusiness.industryGeneral NeuroscienceReproducibility of ResultsAntigens NuclearBregmaFluoresceinsmedicine.diseaseBenzoxazinesDisease Models Animalnervous systembiology.proteinmedicine.symptomNeuNbusinessIntracranial HemorrhagesJournal of Neuroscience Methods
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