Search results for "central nervous system"

showing 10 items of 898 documents

Charting the Drosophila neuropile: a strategy for the standardised characterisation of genetically amenable neurites

2003

Insect neurons are individually identifiable and have been used successfully to study principles of the formation and function of neuronal circuits. In the fruitfly Drosophila, studies on identifiable neurons can be combined with efficient genetic approaches. However, to capitalise on this potential for studies of circuit formation in the CNS of Drosophila embryos or larvae, we need to identify pre- and postsynaptic elements of such circuits and describe the neuropilar territories they occupy. Here, we present a strategy for neurite mapping, using a set of evenly distributed landmarks labelled by commercially available anti-Fasciclin2 antibodies which remain comparatively constant between s…

Central Nervous SystemEmbryo NonmammalianNeuropilTime FactorsNeuritePeriod (gene)CD8 AntigensModels BiologicalSynapseNeurons EfferentPostsynaptic potentialNeuritesAnimalsDrosophila ProteinsDrosophilaMolecular BiologybiologyfungiNeurogenesisGene Expression Regulation DevelopmentalAnatomyCell Biologybiology.organism_classificationNeuronal circuitsLarvaGene TargetingDrosophilaNeuroscienceDevelopmental BiologyDevelopmental Biology
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Abnormal Somatosensory Evoked Potentials Indicate Compressive Cervical Myelopathy in Mucopolysaccharidoses

2000

Compressive myelopathy at the cranio-cervical junction is a complication of mucopolysaccharidoses (MPS). To detect cervical myelopathy we recorded median and posterior tibial nerve SEPs in 15 patients aged 2.4 - 33.4 years (median 8.8 years) with MPS I-S (n = 3), MPS IVA (n = 8) and MPS VI (n = 4). In addition to the cortical waveforms we recorded the subcortical median nerve SEPs N13b and P13 generated near the cranio-cervical junction and the lemniscal P30 after posterior tibial nerve stimulation. MRI studies in 13 subjects revealed spinal cord compression at the cranio-cervical junction in 10 patients; 5 patients had an increased signal intensity on the T2-weighted initial MRI indicating…

AdultMalecongenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyAdolescentSensitivity and SpecificityCentral nervous system diseaseMyelopathySpinal cord compressionEvoked Potentials SomatosensorymedicineHumansChildbusiness.industryGeneral MedicineCervical cord compressionMucopolysaccharidosesmedicine.diseaseSpinal cordMagnetic Resonance ImagingMedian nerveMedian NerveSurgerybody regionsmedicine.anatomical_structureSpinal CordSomatosensory evoked potentialChild PreschoolPediatrics Perinatology and Child HealthFemaleNeurology (clinical)RadiologyTibial NervebusinessSpinal Cord CompressionMyelomalaciaNeckNeuropediatrics
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Novel <b><i>VANGL1</i></b> Gene Mutations in 144 Slovakian, Romanian and German Patients with Neural Tube Defects

2012

Neural tube defects (NTDs) are a group of congenital malformations of the central nervous system occurring at an average rate of 1 per 1,000 human pregnancies worldwide. Numerous genetic and environmental factors are discussed to be relevant in their etiology. In mice, mutants in >200 genes including the planar cell polarity (PCP) pathway are known to cause NTDs, and recently, heterozygous mutations in the human <i>VANGL1</i> gene have been described in a small subset of patients with NTDs. We performed a <i>VANGL1</i> mutation analysis in 144 unrelated individuals with NTDs from Slovakia, Romania and Germany and identified 3 heterozygous missense mutations: c.613…

Geneticscongenital hereditary and neonatal diseases and abnormalitiesMutationbusiness.industryMutantCentral nervous systemNeural tubeGene mutationmedicine.disease_causemedicine.anatomical_structureGeneticsMutation testingmedicineMissense mutationbusinessGeneGenetics (clinical)Molecular Syndromology
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Spinal Intradural Intramedullary Dissemination in the Absence of Intracranial Relapse of a Previously Radically Treated Temporal Lobe Glioblastoma Mu…

2017

Intracranial glioblastoma multiforme (GBM) constitutes the most frequent and unfortunately aggressive primary central nervous system malignancy. Despite the high tendency of these tumors to show local relapse within the brain after primary therapy, dissemination into the spinal axis is an infrequent event. If spinal metastases occur they are leptomeningeal in the vast majority of cases and always in the context of intracranial progressive disease. Spinal intramedullary metastases of intracranial GBM have rarely been described to date. We report the unique case of a young woman with subacute progressive paraparesis due to spinal intramedullary metastases of a temporal lobe GBM despite the re…

medicine.medical_specialtyPathologymedicine.medical_treatmentCentral nervous systemSpinal intradural intramedullary disseminationContext (language use)Case ReportMalignancylcsh:RC254-282Temporal lobelaw.inventionIntramedullary rod03 medical and health sciences0302 clinical medicinelawmedicineChemotherapybusiness.industrymedicine.diseaselcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.anatomical_structureOncology030220 oncology & carcinogenesisTemporal lobe glioblastoma multiformeRadiologybusiness030217 neurology & neurosurgeryProgressive diseaseGlioblastomaCase Reports in Oncology
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A modeling study suggesting how a reduction in the context-dependent input on CA1 pyramidal neurons could generate schizophrenic behavior.

2011

The neural mechanisms underlying schizophrenic behavior are unknown and very difficult to investigate experimentally, although a few experimental and modeling studies suggested possible causes for some of the typical psychotic symptoms related to this disease. The brain region most involved in these processes seems to be the hippocampus, because of its critical role in establishing memories for objects or events in the context in which they occur. In particular, a hypofunction of the N-methyl-D-aspartate (NMDA) component of the synaptic input on the distal dendrites of CA1 pyramidal neurons has been suggested to play an important role for the emergence of schizophrenic behavior. Modeling st…

Computer scienceCognitive Neurosciencemedia_common.quotation_subjectSchizophrenia Realistic model CA1 Hippocampus Object recognition Synaptic integrationCentral nervous systemModels NeurologicalCa1 neuronHippocampusHippocampal formationSynapse03 medical and health sciences0302 clinical medicineArtificial IntelligencePerceptionmedicineAnimalsHumansInvariant (mathematics)CA1 Region Hippocampal030304 developmental biologymedia_common0303 health sciencesRecallArtificial neural networkPyramidal NeuronSynaptic integrationPyramidal CellsCognitive neuroscience of visual object recognitionDendritesmedicine.diseasemedicine.anatomical_structurenervous systemSchizophreniaSynapsesSchizophreniaNMDA receptorNeuronNerve NetNeuroscience030217 neurology & neurosurgeryNeural networks : the official journal of the International Neural Network Society
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TLR4 elimination prevents synaptic and myelin alterations and long-term cognitive dysfunctions in adolescent mice with intermittent ethanol treatment.

2015

The adolescent brain undergoes important dynamic and plastic cell changes, including overproduction of axons and synapses, followed by rapid pruning along with ongoing axon myelination. These developmental changes make the adolescent brain particularly vulnerable to neurotoxic and behavioral effects of alcohol. Although the mechanisms of these effects are largely unknown, we demonstrated that ethanol by activating innate immune receptors toll-like receptor 4 (TLR4), induces neuroinflammation and brain damage in adult mice. The present study aims to evaluate whether intermittent ethanol treatment in adolescence promotes TLR4-dependent pro-inflammatory processes, leading to myelin and synapti…

MAPK/ERK pathwaySynaptic dysfunctionImmunologyNitric Oxide Synthase Type IIBrain damageHMGB1Behavioral NeuroscienceMyelinMiceCognitionmedicineAnimalsTLR4AxonHMGB1 ProteinReceptorNeuroinflammationMyelin SheathMice KnockoutMitogen-Activated Protein Kinase KinasesbiologyBinge ethanol treatmentEthanolEndocrine and Autonomic SystemsNF-kappa BCentral Nervous System DepressantsMyelin alterationsAdolescenceToll-Like Receptor 4medicine.anatomical_structureCyclooxygenase 2SynapsesTLR4biology.proteinmedicine.symptomPsychologyCognition DisordersNeuroscienceCognitive behaviorAlcohol-Related DisordersMyelin ProteinsSignal TransductionBrain, behavior, and immunity
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Evaluation of the acute toxicity, analgesic and CNS activities of different species ofTeucrium genus

1995

Methanol and dichloromethanol extracts of the leaves and stems of four Teucrium species (T. cartaginenses, T. flavum, T. pumillum and T. buxifolium) have been tested for their toxicity, analgesic and central depressor effects. The intraperitoneal administration of the different extracts showed a CNS depressant activity in mice, but they lacked anticonvulsive effects. When tested for analgesic activity none of the extracts increased the threshold of pain thermal stimulus. However, the methanol and dichloromethanol extracts of T. cartaginenses and T. buxifolium species showed a significant analgesic effect in models of pain induced by chemical or mechanical stimulation.

PharmacologybiologyChemistryTEUCRIUM CARTAGINENSES EXTRACTSTEUCRIUM FLAVUM EXTRACTSAnalgesicCentral nervous systemTEUCRIUM BUXIFOLIUM EXTRACTSStimulationBiological activityEFFECTS ON CNSPharmacologybiology.organism_classificationAcute toxicityTeucriumANALGESIAmedicine.anatomical_structureThreshold of painToxicitymedicineTEUCRIUM PUMILLUM EXTRACTSPhytotherapy Research
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Cannabinoid modulation of hippocampal long-term memory is mediated by mTOR signaling.

2009

Cognitive impairment is one of the most important negative consequences associated with cannabis consumption. We found that CB1 cannabinoid receptor (CB1R) activation transiently modulated the mammalian target of rapamycin (mTOR)/p70S6K pathway and the protein synthesis machinery in the mouse hippocampus, which correlated with the amnesic properties of delta9-tetrahydrocannabinol (THC). In addition, non-amnesic doses of either the mTOR blocker rapamycin or the protein synthesis inhibitor anisomycin abrogated the amnesic-like effects of THC, pointing to a mechanism involving new protein synthesis. Moreover, using pharmacological and genetic tools, we found that THC long-term memory deficits …

MaleCannabinoid receptormedicine.medical_treatmentGlutamic AcidHippocampusReceptors N-Methyl-D-AspartateGlutamatergicchemistry.chemical_compoundMiceCognitionReceptor Cannabinoid CB1Memorymental disordersmedicineAnimalsDronabinolPI3K/AKT/mTOR pathwayAnisomycingamma-Aminobutyric AcidMice KnockoutNeuronsProtein Synthesis InhibitorsSirolimusMemory DisordersChemistryGeneral NeuroscienceTOR Serine-Threonine KinasesRibosomal Protein S6 Kinases 70-kDanervous systemKnockout mouseNMDA receptorPhosphorylationCannabinoidNeuroscienceProtein KinasesAnisomycinCentral Nervous System AgentsSignal TransductionNature neuroscience
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Choice of reference area in studies of Alzheimer's disease using positron emission tomography with fluorodeoxyglucose-F18

2007

At present, there is still no consensus on the choice of the reference area in positron emission tomography (PET) studies of Alzheimer's disease (AD). In this study, PET scans with fluorodeoxyglucose-F18 were carried out in the following groups of subjects: 47 patients with probable AD, 8 patients with mild cognitive impairment, and 15 age-similar healthy subjects. Scans normalized to the cerebral global mean (CGM), cerebellum (CBL), and the primary sensorimotor cortex (SMC). We evaluated the effect of the different count normalization procedures on the accuracy of (18)F-FDG PET to detect AD-specific metabolic abnormalities (voxel-based group comparison) and to differentiate between patient…

MaleNormalization (statistics)Pathologymedicine.medical_specialtyAdolescentNeuroscience (miscellaneous)Neuropsychological TestsStatistical parametric mappingGyrus CinguliSeverity of Illness IndexCentral nervous system diseaseAlzheimer DiseaseFluorodeoxyglucose F18CerebellumParietal LobemedicineHumansDementiaRadiology Nuclear Medicine and imagingAgedFluorodeoxyglucosemedicine.diagnostic_testbusiness.industryCognitive disorderMotor CortexSomatosensory Cortexmedicine.diseaseTemporal LobeFrontal LobePsychiatry and Mental healthPositron emission tomographyPositron-Emission TomographyFemaleRadiopharmaceuticalsAlzheimer's diseaseCognition DisordersNuclear medicinebusinessPsychologymedicine.drugPsychiatry Research: Neuroimaging
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Homeostasis of Microglia in the Adult Brain: Review of Novel Microglia Depletion Systems.

2015

Microglia are brain macrophages that emerge from early erythro-myeloid precursors in the embryonic yolk sac and migrate to the brain mesenchyme before the blood brain barrier is formed. They seed the brain, and proliferate until they have formed a grid-like distribution in the central nervous system that is maintained throughout lifespan. The mechanisms through which these embryonic-derived cells contribute to microglia homoeostasis at steady state and upon inflammation are still not entirely clear. Here we review recent studies that provided insight into the contribution of embryonically-derived microglia and of adult 'microglia-like' cells derived from monocytes during inflammation. We ex…

NeuroimmunomodulationCellular differentiationMesenchymeImmunologyCentral nervous systemEmbryonic DevelopmentInflammation610 Medicine & healthBiologyBlood–brain barrier10263 Institute of Experimental ImmunologymedicineImmunology and AllergyAnimalsHomeostasisHumansNeuroinflammationInflammation2403 ImmunologyMicrogliaMacrophagesBrainCell DifferentiationEmbryonic stem cellDisease Models Animalmedicine.anatomical_structureImmunologyModels Animal2723 Immunology and Allergy570 Life sciences; biologyMicrogliamedicine.symptomTrends in immunology
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