Search results for "cerebral cortex"

showing 10 items of 529 documents

Nicotinic cholinoceptors in the rat pineal gland as analyzed by Western blot, light- and electron microscopy

1992

Abstract The monoclonal antibody WF6, raised against purified Torpedo nicotinic acetylcholine receptor (nAChR) was used to study the distribution of cholinoceptors in the rat pineal gland by means of Western blot analysis, light- and electron microscopy. The immunoblot analysis using homogenized pineal gland revealed a labeled protein band of apparent molecular weight 40 kDa which was identified as α-subunits of a nAChR. In the light microscope, approximately one-fourth of the pinealocytes exhibited cytoplasmic immunoreactivity (IR) of varying density. In the electron microscope, IR was seen as patchy staining of cell membranes of pinealocyte somata and processes. Presynaptic IR material wa…

Malemedicine.medical_specialtyBlotting WesternImmunocytochemistryReceptors NicotinicBiologyPineal GlandPinealocytelaw.inventionPineal glandWestern blotAntibody SpecificitylawInternal medicinemedicineAnimalsMolecular BiologyCerebral Cortexmedicine.diagnostic_testGeneral NeuroscienceAntibodies MonoclonalRats Inbred StrainsImmunohistochemistryMolecular biologyRatsMicroscopy ElectronNicotinic acetylcholine receptormedicine.anatomical_structureNicotinic agonistEndocrinologynervous systemElectrophoresis Polyacrylamide GelFemaleNeurology (clinical)Electron microscopeDevelopmental BiologyEndocrine glandBrain Research
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Effects of levosimendan on hemodynamics, local cerebral blood flow, neuronal injury, and neuroinflammation after asphyctic cardiac arrest in rats.

2014

Despite advances in cardiac arrest treatment, high mortality and morbidity rates after successful cardiopulmonary resuscitation are still a major clinical relevant problem. The post cardiac arrest syndrome subsumes myocardial dysfunction, impaired microcirculation, systemic inflammatory response, and neurological impairment. The calcium-sensitizer levosimendan was able to improve myocardial function and initial resuscitation success after experimental cardiac arrest/cardiopulmonary resuscitation. We hypothesized that levosimendan exerts beneficial effects on cerebral blood flow, neuronal injury, neurological outcome, and inflammation 24 hours after experimental cardiac arrest/cardiopulmonar…

Malemedicine.medical_specialtyCardiotonic AgentsHemodynamicsGene ExpressionEnzyme-Linked Immunosorbent AssayCritical Care and Intensive Care MedicineHippocampusRats Sprague-DawleyInternal medicineMedicineAnimalsNeuroinflammationSimendanCerebral CortexNeuronsAnalysis of Variancebusiness.industryInterleukin-6Reverse Transcriptase Polymerase Chain ReactionHemodynamicsHydrazonesLevosimendanCardiopulmonary ResuscitationHeart ArrestRatsSprague dawleyPyridazinesCerebral blood flowAnesthesiaCerebrovascular CirculationCardiologybusinessmedicine.drugCritical care medicine
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Steady state concentrations of clomipramine and its major metabolite desmethylclomipramine in rat brain and serum after oral administration of clomip…

2000

Twenty male Sprague-Dawley rats received five oral doses of clomipramine 20 mg/kg at 4-h intervals. The animals were decapitated 1, 2, 3, 5 and 12 h after the last dose for determination of clomipramine and desmethylclomipramine in serum and frontal cerebral cortex. Time dependent concentrations of clomipramine and desmethylclomipramine paralleled in serum and brain. Half-lives were similar in serum and brain with 7.8 versus 6.2 h and 5.5 versus 5.0 h for clomipramine and desmethylclomipramine, respectively. Absolute concentrations, however, were markedly higher in brain than in serum - 12.5 fold for clomipramine and 7.4 fold for desmethylclomipramine. The data indicate that serum and brain…

Malemedicine.medical_specialtyClomipramineMetaboliteAdministration OralPharmacologyRats Sprague-Dawleychemistry.chemical_compoundOral administrationInternal medicinemedicineAnimalsPharmacology (medical)Biological PsychiatryChromatography High Pressure LiquidPharmacologyHalf-lifeBrainRat brainRatsSprague dawleyPsychiatry and Mental healthKineticsmedicine.anatomical_structureEndocrinologyNeurologychemistryCerebral cortexBrain concentrationsClomipramineNeurology (clinical)medicine.drugHalf-LifeEuropean neuropsychopharmacology : the journal of the European College of Neuropsychopharmacology
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Association between Cognitive Performance and Cortical Glucose Metabolism in Patients with Mild Alzheimer’s Disease

2004

<i>Background:</i> Neuronal and synaptic function in Alzheimer’s disease (AD) is measured in vivo by glucose metabolism using positron emission tomography (PET). <i>Objective:</i> We hypothesized that neuronal activation as measured by PET is a more sensitive index of neuronal dysfunction than activity during rest. We investigated if the correlations between dementia severity as measured with the Mini Mental State Examination (MMSE) and glucose metabolism are an artifact of brain atrophy. <i>Method:</i> Glucose metabolism was measured using [<sup>18</sup>F]fluorodeoxyglucose PET during rest and activation due to audiovisual stimulation in 13 m…

Malemedicine.medical_specialtyCognitive NeuroscienceCentral nervous system diseaseCognitionDegenerative diseaseAtrophyAlzheimer DiseaseFluorodeoxyglucose F18Internal medicinemedicineHumansDementiaBrain atrophy;Partial volume effects;Alzheimer’s disease;Metabolism;Brain imaging techniquesAgedCerebral CortexMini–Mental State Examinationmedicine.diagnostic_testBrainMiddle Agedmedicine.diseaseddc:Psychiatry and Mental healthGlucoseEndocrinologyPositron emission tomographyPositron-Emission TomographyPosterior cingulateFemaleAtrophyGeriatrics and GerontologyAlzheimer's diseaseMental Status SchedulePsychologyDementia and Geriatric Cognitive Disorders
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Altered expression of neuropeptides in the primary somatosensory cortex of the Down syndrome model Ts65Dn.

2011

Down syndrome is the most common genetic disorder associated with mental retardation. Subjects and mice models for Down syndrome (such as Ts65Dn) show defects in the formation of neuronal networks in both the hippocampus and the cerebral cortex. The principal neurons display alterations in the morphology, density and distribution of dendritic spines in the cortex as well as in the hippocampus. Several evidences point to the possibility that the atrophy observed in principal neurons could be mediated by changes in their inhibitory inputs and, in fact, an imbalance between excitation and inhibition has been observed in Ts65Dn mice in these regions, which are crucial for learning and informati…

Malemedicine.medical_specialtyDendritic spineHippocampusBiologySomatosensory systemCalbindinHippocampusCellular and Molecular NeuroscienceMiceEndocrinologyInterneuronsCortex (anatomy)Internal medicinemedicineAnimalsNeuronsEndocrine and Autonomic SystemsCalcium-Binding ProteinsNeuropeptidesGeneral MedicineSomatosensory CortexDisease Models Animalmedicine.anatomical_structureEndocrinologySomatostatinnervous systemNeurologyCerebral cortexCalretininDown SyndromeSomatostatinNeuroscienceNeuropeptides
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Nitric oxide and glutamate interaction in the control of cortical and hippocampal excitability.

1999

Summary: Purpose: We investigated the role of nitric oxide (NO) as a new neurotransmitter in the control of excitability of the hippocampus and the cerebral cortex, as well as the possible functional interaction between NO and the glutamate systems. Methods: The experiments were performed on anesthetized rats. The bioelectrical activities of the somatosensory cortex and the CA1 region of the hippocampus of these rats were recorded. Pharmacologic inhibition of NO synthase (NOS) through the nonselective and brain-selective inhibitors, N-nitro-L-arginine methyl ester (l-NAME) and 7-nitroindazole (7-NI), was performed. Results: The treatments caused the appearance of an interictal discharge act…

Malemedicine.medical_specialtyHippocampusGlutamic AcidHippocampal formationNeurotransmissionBiologyNitric OxideHippocampusReceptors N-Methyl-D-Aspartatechemistry.chemical_compoundInternal medicinemedicineAnimalsRats WistarNeurotransmitterCerebral CortexEpilepsyGlutamate receptorSomatosensory CortexRatsEndocrinologyNeurologychemistryCNQXExcitatory postsynaptic potentialNMDA receptorNeurology (clinical)Epilepsia
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Role of dopamine neurotransmission in the long-term effects of repeated social defeat on the conditioned rewarding effects of cocaine

2016

Numerous studies report that social defeat stress alters dopamine (DA) neurotransmission in several areas of the brain. Alterations of the mesolimbic dopaminergic pathway are believed to be responsible for the increased vulnerability to drug use observed as a result of social stress. In the present study, we evaluated the influence of DA receptors on the long-term effect of repeated social defeat (RSD) on the conditioned rewarding and reinstating effects of cocaine. For this purpose, the D1R antagonist SCH 23390 and the D1R antagonist raclopride were administered 30 min before each social defeat and a cocaine-induced CPP procedure was initiated three weeks later. The expression of the D1R a…

Malemedicine.medical_specialtyHippocampusStatistics NonparametricReceptors DopamineSocial defeatMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDopamine Uptake InhibitorsRewardCocaineInternal medicineDopamine receptor D2medicineAnimalsDopamine receptorsBiological PsychiatryCerebral CortexPharmacologyRacloprideSocial stressSCH-23390Dose-Response Relationship DrugDopaminergicAge FactorsBenzazepinesConditioned place preferenceConditioned place preference030227 psychiatryDisease Models AnimalEndocrinologychemistryRacloprideDopamine receptorAnesthesiaConditioning OperantDopamine AntagonistsPsychologySocial defeat stressStress Psychological030217 neurology & neurosurgerymedicine.drug
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Effect of acidosis on lipid peroxidation in brain slices.

1991

Acidification of the incubation medium markedly increased lipid peroxidation of cortical brain slices. Lactic acidosis caused a more extensive lipid peroxidation than did phosphoric acidosis (+35% at pH 6 and +81% at pH 5), probably due to the rapid diffusion of the protonated form of lactic acid across cell membranes. These results support the hypothesis that free radical mechanisms may be involved in the cytotoxicity of acidosis.

Malemedicine.medical_specialtyIn Vitro TechniquesPhosphatesLipid peroxidationchemistry.chemical_compoundSlice preparationInternal medicinemedicineAnimalsCytotoxicityMolecular BiologyIncubationAcidosisCerebral CortexChemistryGeneral NeuroscienceBrainRats Inbred StrainsMetabolismHydrogen-Ion Concentrationmedicine.diseaseLactic acidRatsKineticsEndocrinologyBiochemistryLactic acidosisLactatesNeurology (clinical)Lipid Peroxidationmedicine.symptomAcidosisDevelopmental BiologyBrain research
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The neuroprotective effect of lactate is not due to improved glutamate uptake after controlled cortical impact in rats.

2012

For many years lactate was considered to be a waste product of glycolysis. Data are accumulating that suggest that lactate is an important energy substrate for neurons during activation. In severe traumatic brain injury (TBI) glutamate release and ischemic cerebral blood flow (CBF) are major factors for a mismatch between energy demand and supply and for neuronal cell death. Although ATP and behavior could be improved by lactate treatment after TBI, no histological correlate nor any linkage to better astrocytic glutamate uptake or CBF as possible mechanisms have been described. We subjected male rats to a controlled cortical impact (CCI; 5 m/sec, 2.5 mm). To study the effects of lactate tre…

Malemedicine.medical_specialtyMicrodialysisCoumaric AcidsMicrodialysisGlutamic AcidNeuroprotectionRats Sprague-DawleyStereotaxic TechniquesOxygen ConsumptionInternal medicinemedicineAnimalsGlycolysisLactic AcidChromatography High Pressure LiquidBrain ChemistryCerebral CortexSkull FracturesChemistryGlutamate receptorGlutamic acidRatsmedicine.anatomical_structureEndocrinologyNeuroprotective AgentsCerebral blood flowCerebral cortexAnesthesiaBrain InjuriesCerebrovascular CirculationStereotaxic techniqueNeurology (clinical)Extracellular SpaceJournal of neurotrauma
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Clinical presentation of strokes confined to the insula: a systematic review of literature

2021

Abstract Background and purpose The insular cortex serves a wide variety of functions in humans, ranging from sensory and affective processing to high-level cognition. Hence, insular dysfunction may result in several different presentations. Ischemic strokes limited to the insular territory are rare and deserve a better characterization, to be quickly recognized and to receive appropriate treatment (e.g. thrombolysis). Methods We reviewed studies on patients with a first-ever acute stroke restricted to the insula. We searched in the Medline database the keywords “insular stroke” and “insular infarction”, to identify previously published cases. Afterwards, the results were divided depending …

Malemedicine.medical_specialtyNeurologySensory systemReview ArticleDermatology030204 cardiovascular system & hematologyInsular cortexbehavioral disciplines and activities03 medical and health sciencesDysarthria0302 clinical medicinePhysical medicine and rehabilitationAphasiaAphasiamedicineHumansSpeechCerebrovascular diseaseStrokeNeuroradiologyInsular syndromesCerebral CortexInsular stroke Insular dysfunction Insular syndromes Cerebrovascular diseasebusiness.industryDysarthriaGeneral Medicinemedicine.diseaseStrokePsychiatry and Mental healthnervous systembehavior and behavior mechanismsInsular strokeSettore MED/26 - NeurologiaFemaleNeurology (clinical)medicine.symptomInsular dysfunctionbusinessInsulapsychological phenomena and processes030217 neurology & neurosurgeryNeurological Sciences
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