Search results for "cerebral cortex"

showing 10 items of 529 documents

Acute electrocorticographic changes following localized cortical freezing in the cat. Preliminary report.

1968

Pathologymedicine.medical_specialtyClinical BiochemistryElectroencephalographyText miningPreliminary reportSeizuresFreezingmedicineAnimalsAnesthesiaElectrodesCerebral CortexCATSmedicine.diagnostic_testbusiness.industryElectroencephalographyGeneral MedicineCerebrovascular Circulationmedicine.anatomical_structureCerebral cortexRegional Blood FlowAnesthesiaCerebrovascular CirculationCatsbusinessScandinavian journal of clinical and laboratory investigation. Supplementum
researchProduct

Assessment of cortical damage in early multiple sclerosis with quantitative T 2 relaxometry

2016

T2 relaxation time is a quantitative MRI in vivo surrogate of cerebral tissue damage in multiple sclerosis (MS) patients. Cortical T2 prolongation is a known feature in later disease stages, but has not been demonstrated in the cortical normal appearing gray matter (NAGM) in early MS. This study centers on the quantitative evaluation of the tissue parameter T2 in cortical NAGM in a collective of early MS and clinically isolated syndrome (CIS) patients, hypothesizing that T2 prolongation is already present at early disease stages and variable over space, in line with global and focal inflammatory processes in MS. Additionally, magnetization transfer ratio (MTR) mapping was performed for furt…

Pathologymedicine.medical_specialtyClinically isolated syndromemedicine.diagnostic_testbusiness.industryMultiple sclerosisMagnetic resonance imagingmedicine.disease030218 nuclear medicine & medical imaging03 medical and health sciences0302 clinical medicinemedicine.anatomical_structureCerebral cortexIn vivoRegion of interestCortex (anatomy)medicineMolecular MedicineRadiology Nuclear Medicine and imagingMagnetization transferbusiness030217 neurology & neurosurgerySpectroscopyNMR in Biomedicine
researchProduct

Multimodal quantitative MRI assessment of cortical damage in relapsing-remitting multiple sclerosis

2016

Purpose To investigate magnetization transfer ratio (MTR), T1 relaxation time, and proton density (PD) as indicators of gray matter damage in relapsing-remitting multiple sclerosis (RRMS), reflecting different aspects of microstructural damage and as imaging correlates of clinical disability. We aimed to determine which of these parameters may optimally quantify cortical damage, and serve as an imaging surrogate of clinical disability. In this study, cortical values of MTR, a surrogate for demyelination in MS, of PD, reflecting replacement of neural tissue by water, and of T1 , indicating a complex array of microstructural changes, were assessed in a group of RRMS patients in comparison to …

Pathologymedicine.medical_specialtyExpanded Disability Status Scalemedicine.diagnostic_testbusiness.industryMultiple sclerosisArea under the curveMagnetic resonance imagingmedicine.disease030218 nuclear medicine & medical imaging03 medical and health sciences0302 clinical medicinemedicine.anatomical_structureRelapsing remittingCerebral cortexmedicineRadiology Nuclear Medicine and imagingMagnetization transferNuclear medicinebusinessProton density030217 neurology & neurosurgeryJournal of Magnetic Resonance Imaging
researchProduct

Preservation of glial cytoarchitecture from ex vivo human tumor and non-tumor cerebral cortical explants: A human model to study neurological diseases

2007

For the human brain, in vitro models that accurately represent what occurs in vivo are lacking. Organotypic models may be the closest parallel to human brain tissue outside of a live patient. However, this model has been limited primarily to rodent-derived tissue. We present an organotypic model to maintain intraoperatively collected human tumor and non-tumor explants ex vivo for a prolonged period of time (similar to 11 days) without any significant changes to the tissue cytoarchitecture as evidenced through immunohistochemistry and electron microscopy analyses. The ability to establish and reliably predict the cytoarchitectural changes that occur with time in an organotypic model of tumor…

Pathologymedicine.medical_specialtyIndolesTime FactorsbrainMatrix (biology)BiologyModels BiologicalStatistics NonparametricArticleOrgan Culture TechniquesMicroscopy Electron TransmissionIn vivoGlial Fibrillary Acidic ProteinmedicineHumanshumanorganotypicCerebral Cortexelectron microscopyBrain NeoplasmsGeneral NeuroscienceexplantReproducibility of ResultsCell migrationHuman brainMiddle AgedImmunohistochemistrymedicine.anatomical_structureCytoarchitectureImmunohistochemistryFemaleTissue PreservationNervous System DiseasesNeurogliaEx vivoExplant culture
researchProduct

Postischemic Brain Infiltration of Leukocyte Subpopulations Differs among Murine Permanent and Transient Focal Cerebral Ischemia Models

2012

Cellular and humoral inflammations play important roles in ischemic brain injury. The effectiveness of immunomodulatory therapies may critically depend on the chosen experimental model. Our purpose was to compare the post-ischemic neuroinflammation among murine permanent and transient middle cerebral artery occlusion (MCAO) models. Permanent MCAO was induced by transtemporal electrocoagulation and 30 minutes or 90 minutes transient MCAO was induced by intraluminal filament in C57BL/6 mice. Infiltration of leukocyte subpopulations was quantified by immunohistochemistry and fluorescence-activated cell sorting. Cerebral cytokine and adhesion molecule expression was measured by real-time polyme…

Pathologymedicine.medical_specialtyMicrogliabusiness.industryCell adhesion moleculeanimal diseasesGeneral Neurosciencemedicine.medical_treatmentIschemiaInflammationmedicine.diseasenervous system diseasesPathology and Forensic Medicinemedicine.anatomical_structureCytokinenervous systemCerebral cortexcardiovascular systemMedicinecardiovascular diseasesNeurology (clinical)medicine.symptombusinessInfiltration (medical)NeuroinflammationBrain Pathology
researchProduct

2013

Cortical function is impaired in various disorders of the central nervous system including Alzheimer’s disease, autism and schizophrenia. Some of these disorders are speculated to be associated with insults in early brain development. Pericytes have been shown to regulate neurovascular integrity in development, health and disease. Hence, precisely controlled mechanisms must have evolved in evolution to operate pericyte proliferation, repair and cell fate within the neurovascular unit (NVU). It is well established that pericyte deficiency leads to NVU injury resulting in cognitive decline and neuroinflammation in cortical layers. However, little is known about the role of pericytes in pathop…

Pathologymedicine.medical_specialtyMultidisciplinaryCentral nervous systemBrain damageCell fate determinationBiologymedicine.anatomical_structureCerebral cortexmedicinePericytemedicine.symptomCognitive declineReprogrammingNeuroscienceNeuroinflammationPLOS ONE
researchProduct

2015

AbstractAccumulating evidence suggests a pivotal role of PDGFRß positive cells, a specific marker for central nervous system (CNS) pericytes, in tissue scarring. Identification of cells that contribute to tissue reorganization in the CNS upon injury is a crucial step to develop novel treatment strategies in regenerative medicine. It has been shown that pericytes contribute to scar formation in the spinal cord. It is further known that ischemia initially triggers pericyte loss in vivo, whilst brain trauma is capable of inducing pericyte detachment from cerebral vessels. These data point towards a significant role of pericytes in CNS injury. The temporal and spatial dynamics of PDGFRß cells a…

Pathologymedicine.medical_specialtyMultidisciplinaryTraumatic brain injurybusiness.industryCentral nervous systemIschemiamedicine.diseaseSpinal cordRegenerative medicinemedicine.anatomical_structureCerebral cortexmedicinePericytebusinessPathologicalScientific Reports
researchProduct

Reactive neurogenesis during regeneration of the lesioned medial cerebral cortex of lizards

1995

Abstract This study reports that lesion of the adult lizard medial cortex (lizard hipocampal fascia dentata) induces a short period of intensive neurogenesis which we have termed reactive neurogenesis; a cell proliferation event that occurs in the subjacent ependyma. Specific lesion of the medial cortex was achieved by intraperitoneal injection of the neurotoxin 3-acetylpyridine and proliferating cells were detected using tritiated thymidine or 5-bromodeoxiuridine pulse labelling. After lesion, granule neurons in the lizard medial cortex cell layer appeared pyknotic and died; they were then removed and progressively replaced by a set of new neurons. These neurons were mostly generated from …

Pathologymedicine.medical_specialtyPyridinesMedial cortexNeurotoxinsPodarcis hispanicaLesionNeuroblastmedicineAnimalsCerebral CortexbiologyGeneral NeuroscienceNeurogenesisLizardsDNAAnatomybiology.organism_classificationImmunohistochemistryNerve RegenerationMicroscopy Electronmedicine.anatomical_structureCerebral cortexAutoradiographyFascia dentatamedicine.symptomEpendymaCell DivisionNeuroscience
researchProduct

Extracellular space and electrolyte distribution in cortex and white matter of dog brain in cold induced oedema

1973

24 hours after a circumscribed cold injury of the cortex dog brains were perfused from the lateral ventricle and the frontal subarachnoidal space to the cisterna magna with an artificial CSF containing trace amounts of35S-labelled thiosulphate. Simultaneously the extracellular tracer was administered intravenously. Extracellular fluid volume was estimated and found to be increased from 10 to 15% in the oedematous cortex and from 10 to 27% in the oedematous white matter. The actual size of ECS in oedematous white matter, however, must be larger as indicated by the relative alterations of thiosulphate distribution, tissue water, sodium and chloride. Apparently a small part of the fluid accumu…

Pathologymedicine.medical_specialtySodiumThiosulfateschemistry.chemical_elementBrain EdemaCisterna magnaIodine RadioisotopesWhite matterchemistry.chemical_compoundDogsChloridesCortex (anatomy)Sulfur IsotopesExtracellular fluidExtracellularAnimalsMedicineSerum Albumin Radio-IodinatedEvans BlueCerebral CortexStaining and Labelingbusiness.industrySodiumWater-Electrolyte BalanceCold TemperatureDisease Models Animalmedicine.anatomical_structurechemistryBlood-Brain BarrierCerebral cortexPotassiumSurgeryNeurology (clinical)Extracellular SpacebusinessActa Neurochirurgica
researchProduct

Novel modes of rhythmic burst firing at cognitively-relevant frequencies in thalamocortical neurons.

2008

It is now widely accepted that certain types of cognitive functions are intimately related to synchronized neuronal oscillations at both low (alpha/theta) (4-7/8-13 Hz) and high (beta/gamma) (18-35/30-70 Hz) frequencies. The thalamus is a key participant in many of these oscillations, yet the cellular mechanisms by which this participation occurs are poorly understood. Here we describe how, under appropriate conditions, thalamocortical (TC) neurons from different nuclei can exhibit a wide array of largely unrecognised intrinsic oscillatory activities at a range of cognitively-relevant frequencies. For example, both metabotropic glutamate receptor (mGluR) and muscarinic Ach receptor (mAchR) …

Periodicity* Cognition* Acetylcholine; * Metabotropic glutamate receptor; * Lateral geniculate nucleus; * Intralaminar nucleus; * Oscillations; * EEG; * Cognition; * Perception; * Memory* EEGAction PotentialsSettore BIO/09 - FisiologiaIon ChannelsArticle* PerceptionBurstingThalamusBiological Clocks* Lateral geniculate nucleuMuscarinic acetylcholine receptorNeural PathwaysmedicineAnimalsHumans* Metabotropic glutamate receptorMolecular BiologyCerebral CortexNeurons* OscillationChemistryGeneral Neuroscience* Intralaminar nucleuGlutamate receptorReceptors NeurotransmitterElectrophysiology* MemoryMetabotropic receptormedicine.anatomical_structure* AcetylcholineMetabotropic glutamate receptorWakefulnessNeurology (clinical)NeuronNeuroscienceDevelopmental Biology
researchProduct