Search results for "choline"

showing 10 items of 1138 documents

Nitric oxide mediates abnormal responsiveness of thyroid arteries in methimazole-treated patients.

2005

Objective: We studied the intervention of nitric oxide (NO), prostacyclin and endothelium-derived hyperpolarizing factor (EDHF) in mediating responses to acetylcholine in thyroid arteries from euthyroid and methimazole-treated (MT) patients. Design and methods: Branches of the superior thyroid artery were obtained from 19 euthyroid patients and 17 MT patients (euthyroid at the time of surgery) undergoing total thyroidectomy or hemithyroidectomy. Artery rings were suspended in organ baths for isometric recording of tension. Results and conclusions: Acetylcholine caused endothelium-dependent relaxation of greater magnitude in arteries from MT patients (pD2 (−log EC50) values were 7.68±0.19 in…

AdultMalemedicine.medical_specialtyPotassium ChannelsCharybdotoxinEndocrinology Diabetes and MetabolismMuscle RelaxationThyroid GlandApaminNitric OxideHyperthyroidismMuscle Smooth VascularSuperior thyroid arterychemistry.chemical_compoundBiological FactorsNorepinephrineEndocrinologyAntithyroid AgentsInternal medicinemedicine.arterymedicinePotassium Channel BlockersHumansEuthyroidEnzyme InhibitorsMethimazoleomega-N-Methylargininebusiness.industryThyroidGeneral MedicineArteriesMiddle AgedAcetylcholinemedicine.anatomical_structureEndocrinologychemistryApaminCirculatory systemPotassiumCalciumFemaleEndothelium VascularNitric Oxide SynthasebusinessAcetylcholinemedicine.drugBlood vesselArteryEuropean journal of endocrinology
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Reduced airway responsiveness in non elite runners

2005

Med Sci Sports Exerc. 2005 Dec;37(12):2019-25. Reduced airway responsiveness in nonelite runners. Scichilone N, Morici G, Marchese R, Bonanno A, Profita M, Togias A, Bonsignore MR. SourceInstitute of Medicine and Pneumology, Respiratory Unit; University of Palermo, Italy. n.scichilone@libero.it Abstract PURPOSE: The effects of endurance training on airway responsiveness in nonasthmatic subjects are poorly defined. We hypothesized that airway responsiveness may differ between none-lite endurance athletes and sedentary subjects, and studied healthy, nonelite runners and sedentary controls by single-dose methacholine challenges carried out in the absence of deep inspirations, in that deep insp…

AdultMalemedicine.medical_specialtyRespiratory SystemVital CapacityPhysical Therapy Sports Therapy and RehabilitationSettore MED/10 - Malattie Dell'Apparato RespiratorioSports MedicineBronchial Provocation TestsRunningEndurance trainingasthma exercise lung functionMedicineHumansOrthopedics and Sports MedicineMethacholine Chloridebiologybusiness.industryAthletesAirway Resistancerespiratory systembiology.organism_classificationrespiratory tract diseasesRespiratory Function TestsCase-Control StudiesPhysical therapyPhysical EnduranceFemalebusinessAirway responsivenessInspiratory Capacity
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Ultra-long-distance running and the liver.

1990

During an ultra-long-distance race (1000 km in 20 days) the influence of running was examined on the enzymes aspartate aminotransferase (AST), alanine aminotransferase (ALT), alkaline phosphatase (AP), gamma-glutamyl-transferase (GGT), and glutamate dehydrogenase (GLDH) with regard to their release from the liver cells or their induction. Furthermore the liver synthetic capacity was assayed by measuring the enzyme activity of cholinesterase and the concentration of serum albumin during the race. Of the 110 participants, 55 finished the race and only the results of these runners were used in our study. AP increased continuously from day 0 (mean = 102 U/L) to day 19 (mean = 120 U/L). A fivefo…

AdultMalemedicine.medical_specialtySerum albuminPhysical Therapy Sports Therapy and Rehabilitationdigestive systemRunningInternal medicinemedicineHumansOrthopedics and Sports MedicineSerum AlbuminCholinesteraseAgedchemistry.chemical_classificationAnalysis of VariancebiologyLiver cellGlutamate dehydrogenaseAlbuminMiddle Ageddigestive system diseasesEnzyme assayOxygenEnzymeEndocrinologychemistryLiverEnzyme Inductionbiology.proteinPhysical EnduranceAlkaline phosphataseFemaleLiver CirculationInternational journal of sports medicine
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Changes in plasma lecithin: cholesterol acyltransferase activity, HDL2, HDL3 amounts and compositions in patients with chronic renal failure after di…

2002

The aim of this study was to investigate the effect of hemodialysis duration on HDL(2) and HDL(3) compositions and lecithin:cholesterol acyltransferase (LCAT) activity in 58 patients on acetate hemodialysis using cuprophane membrane, after different periods of dialysis. Patients were divided into three groups-GI1 year, GII 1-5 years, GIII 5-13 years of dialysis and were compared with 22 controls. Increase by 34% of triacylglycerols (TG) was noted in GI and by 36% in GII versus GI. Hypertriglyceridemia was correlated with hemodialysis duration (HD) (r=0.75, P0.05). The LCAT activity decrease by 25% was noted in GII versus GI (P0.05) and by 45% in GIII versus GI (P0.01), this activity was neg…

AdultMalemedicine.medical_specialtyTime Factorsfood.ingredientmedicine.medical_treatmentSterol O-acyltransferaseLecithinPhosphatidylcholine-Sterol O-AcyltransferasefoodRenal DialysisInternal medicinemedicineHumansTriglyceridesDialysisbusiness.industryCuprophaneLipoproteins HDL3Middle Agedmedicine.diseaseLipoproteins HDL2EndocrinologyKidney Failure ChronicChronic renal failureFemalelipids (amino acids peptides and proteins)Phosphatidylcholine—sterol O-acyltransferaseHemodialysisLipoproteins HDLCardiology and Cardiovascular MedicinebusinessKidney diseaseAtherosclerosis
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Possible Role of Butyrylcholinesterase in Fat Loss and Decreases in Inflammatory Levels in Patients with Multiple Sclerosis after Treatment with Epig…

2021

(1) Background. Multiple sclerosis (MS) is characterised by the loss of muscle throughout the course of the disease, which in many cases is accompanied by obesity and related to inflammation. Nonetheless, consuming epigallocatechin gallate (EGCG) and ketone bodies (especially β-hydroxybutyrate (βHB)) produced after metabolising coconut oil, have exhibited anti-inflammatory effects and a decrease in body fat. In addition, butyrylcholinesterase (BuChE), seems to be related to the pathogenesis of the disease associated with inflammation, and serum concentrations have been related to lipid metabolism. Objective. The aim of the study was to determine the role of BuChE in the changes caused after…

AdultMalemedicine.medical_specialtyfood.ingredientPilot ProjectsEpigallocatechin gallatemultiple sclerosisAntioxidantsCatechinArticlechemistry.chemical_compoundfoodβ-hydroxybutyrateInternal medicineWeight LossmedicineHumansTX341-641ObesityButyrylcholinesteraseNutrition and DieteticsbiologyNutrition. Foods and food supplybusiness.industryCoconut oilParaoxonaseAlbuminMiddle AgedLipid Metabolismmedicine.diseaseObesityPON1EndocrinologyAdipose TissuechemistryinflammationDietary SupplementsbutyrylcholinesteraseCoconut Oilbiology.proteinKetone bodiesFemalebusinessEGCGFood ScienceNutrients
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Immunosuppressive treatment of rippling muscles in patients with myasthenia gravis

2000

Rippling muscle disease is a rare autosomal dominant disorder that may occur sporadically. In this report two patients presenting with rippling muscles followed by myasthenia gravis are described. Our first patient developed rippling muscles about 1 month after infection with Yersinia enterocolitica. Two years later myasthenia gravis appeared. Our second patient had a 2-year history of asthma prior to the onset of rippling muscles which preceded the myasthenic symptoms by 4-8 weeks. Acetylcholine receptor and anti-skeletal muscle antibody titers were positive in both patients. In both patients the rippling phenomena worsened with pyridostigmine treatment but markedly improved after immunosu…

AdultMalemedicine.medical_specialtymedicine.medical_treatmentAzathioprineGastroenterologyMuscular DiseasesPhysical StimulationInternal medicineAzathioprineMyasthenia GravismedicineHumansGenetics (clinical)AsthmaAcetylcholine receptorbusiness.industryImmunosuppressionMiddle Agedmedicine.diseaseMyasthenia gravisNeurologyPyridostigmineRipplingPediatrics Perinatology and Child HealthImmunologyNeurology (clinical)medicine.symptombusinessImmunosuppressive AgentsMuscle ContractionPyridostigmine BromideMuscle contractionmedicine.drugNeuromuscular Disorders
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Smoking cessation and variations in nicotinic acetylcholine receptor subunits alpha-5, alpha-3, and beta-4 genes.

2009

Background Evidence has recently accumulated that single nucleotide polymorphisms in the genetic region encoding the nicotinic acetylcholine receptor subunits α-5, α-3, and β-4 are associated with smoking and nicotine dependence. We aimed to determine whether these genetic variations are also predictive of smoking cessation. Methods Lifetime history of smoking was assessed by questionnaire at enrolment into a large epidemiological study of the German elderly population (ESTHER study). Cox proportional hazards modeling was applied in a retrospective cohort approach to determine the associations of individual polymorphisms and haplotypes with smoking cessation probability in 1446 subjects who…

AdultMalemedicine.medical_treatmentPopulationPhysiologySingle-nucleotide polymorphismNerve Tissue ProteinsReceptors NicotinicPolymorphism Single NucleotideGenetic determinismCohort StudiesmedicineHumanseducationBiological PsychiatryAgedGeneticseducation.field_of_studybusiness.industryProportional hazards modelHaplotypeMiddle AgedNicotinic acetylcholine receptorGenetic epidemiologyHaplotypesSmoking cessationFemaleSmoking CessationbusinessBiological psychiatry
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Plasma and fibroblasts of Tangier disease patients are disturbed in transferring phospholipids onto apolipoprotein A-I

1998

Plasmas of patients with Tangier disease (TD) lack lipid-rich α-HDL which, in normal plasma, constitutes the majority of high density lipoprotein (HDL). Residual amounts of apolipoprotein (apo)A-I in TD plasma occur as lipid-poor or even lipid-free preβ-HDL. By contrast to normal plasma, TD plasma does not convert preβ-HDL into α-HDL. Moreover, fibroblasts of TD patients were found to be defective in secreting cholesterol or phospholipids in the presence of lipid-free apoA-I. We have therefore hypothesized that both defective conversion of preβ-HDL into α-HDL and defective lipid efflux from TD cells onto lipid-free apoA-I result from a disturbance in phospholipid transfer occurring in both …

AdultMaletransferring phospholipidsPhospholipidTangier diseasePhosphatidic AcidsQD415-436PhosphatidylinositolsBiochemistrychemistry.chemical_compoundEndocrinologyTangier diseasePhosphatidylcholinePhospholipid transfer proteinExtracellularmedicineHumansCells CulturedPhosphatidylethanolamineApolipoprotein A-ICholesterolPhosphatidylethanolaminesReverse cholesterol transportnutritional and metabolic diseasesBiological TransportCell BiologyFibroblastsmedicine.diseaseMolecular biologyfamilial HDL deficiencyreverse cholesterol transportLipoproteins LDLphospholipid transfer proteinsprebeta-HDLTangier disease; transferring phospholipidschemistryPhosphatidylcholinesFemalelipids (amino acids peptides and proteins)cholesterol efflux
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Nitric oxide production and endothelium-dependent vasorelaxation ameliorated by N1-methylnicotinamide in human blood vessels.

2012

N 1 -methylnicotinamide (MNA + ) has until recently been thought to be a biologically inactive product of nicotinamide metabolism in the pyridine nucleotides pathway. However, the latest observations imply that MNA + may exert antithrombotic and anti-inflammatory effects through direct action on the endothelium. We examined both in vivo and in vitro whether the compound might induce vasorelaxation in human blood vessels through the improvement of nitric oxide (NO) bioavailability and a reduction of oxidative stress mediated by endothelial NO synthase (eNOS) function. MNA + treatment (100 mg/m 2 orally) in healthy normocholesterolemic and hypercholesterolemic subjects increased the l-argini…

AdultNiacinamidemedicine.medical_specialtyEndotheliumBrachial ArteryNitric Oxide Synthase Type IIIHypercholesterolemiachemistry.chemical_elementCalciumIn Vitro Techniquesmedicine.disease_causeNitric OxideNitric oxidechemistry.chemical_compoundN^{1}-methylnicotinamideDouble-Blind MethodEnosnitric oxideInternal medicineInternal MedicinemedicineHumansflow-mediated dilationCalcimycinCells Culturedendothelial nitric oxide synthaseoxidized low-density lipoproteinbiologyDose-Response Relationship DrugChemistrySuperoxidebiology.organism_classificationendothelial cellsAcetylcholineOxygenVasodilationOxidative Stressmedicine.anatomical_structureEndocrinologysuperoxideEndothelium VascularAcetylcholineOxidative stressmedicine.drugLipoproteinHypertension (Dallas, Tex. : 1979)
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A quantitative study of the pancuronium antagonism at the motor endplate in human organophosphorus intoxication

1995

Nine patients with organophosphorus (OP) intoxication developing neuromuscular transmission defects were given pancuronium 1, 2, or 4 mg intravenously (IV). Thirteen patient controls with hypoxic encephalopathy received similar dosages. The responses were monitored electrophysiologically using single and repetitive nerve stimulation (20 and 50 Hz). In OP patients, pancuronium did not alter the amplitude of the single CMAP, whereas its repetitive discharges were reduced. Severe neuromuscular blocks were reversed only partially by pancuronium 4 mg. In less severe blocks, 1 and 2 mg resulted in marked improvement. In the patient controls, pancuronium 4 mg induced a severe neuromuscular block b…

AdultTime FactorsPhysiologymedicine.medical_treatmentNeuromuscular transmissionAction PotentialsElectromyographyMotor EndplateSynaptic TransmissionNeuromuscular junctionCellular and Molecular Neurosciencechemistry.chemical_compoundOrganophosphate PoisoningPhysiology (medical)medicineHumansPancuroniumRepetitive nerve stimulationAntidoteNeuromuscular BlockadeMovement DisordersDose-Response Relationship Drugmedicine.diagnostic_testbusiness.industryNeuromuscular DiseasesAcetylcholinesteraseElectric Stimulationmedicine.anatomical_structurechemistryAnesthesiaInjections IntravenousToxicityAcetylcholinesteraseNeurology (clinical)businessMuscle & Nerve
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