Search results for "cimo"

showing 10 items of 65 documents

Neuroprotective effect of ceftriaxone on the penumbra in a rat venous ischemia model.

2012

Glutamate transporter-1 (GLT-1) maintains low concentrations of extracellular glutamate by removing glutamate from the extracellular space. It is controversial, however, whether upregulation of GLT-1 is neuroprotective under all ischemic/hypoxic conditions. Recently, a neuroprotective effect of preconditioning with a β-lactam antibiotic ceftriaxone (CTX) that increases expression of GLT-1 has been reported in animal models of focal ischemia. On the other hand, it is said that CTX does not play a neuroprotective role in an in vitro study. Thus, we examined the effect of CTX on ischemic injury in a rat model of two-vein occlusion (2VO). This model mimics venous ischemia during, e.g. tumor sur…

MaleIschemiaAMPA receptorPharmacologyNeuroprotectionReceptors N-Methyl-D-AspartateBrain IschemiaPotassium Chloridechemistry.chemical_compoundMedicineAnimalsDrug InteractionsReceptors AMPAKainic Acidbusiness.industryGABAA receptorGeneral NeuroscienceCeftriaxoneCortical Spreading DepressionGlutamate receptorCerebral Infarctionmedicine.diseaseReceptors GABA-AAnti-Bacterial AgentsRatsNeuroprotective AgentsMuscimolchemistryExcitatory Amino Acid Transporter 2Cortical spreading depressionAnesthesiaNMDA receptorbusinessNeuroscience
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NMDA-GABA interactions in an animal model of behaviour: a gating mechanism from motivation toward psychotic-like symptoms

1994

We studied the effects of desipramine, alprazolam, muscimol and dizocilpine (MK-801) (alone or associated with desipramine) in the forced swimming test in rats after long-lasting termination of chronic exposure to vehicle and pentylenetetrazol. Sensitisation with pentylenetetrazol was ineffective in changing immobility time in the forced swimming test compared to vehicle treatment; pentylenetetrazol enhanced the anti-immobility effect of desipramine, abolished the anti-immobility effect of alprazolam and did not affect the anti-immobility effect of muscimol. MK-801 at the dose that did not modify immobility time in vehicle-treated rats and in pentylenetetrazol-treated animals strongly poten…

MaleN-MethylaspartatePoison controlGatingMotor ActivityPharmacologybehavioral disciplines and activitieschemistry.chemical_compoundDesipramineKindling NeurologicmedicineAnimalsPharmacology (medical)Rats WistarPentylenetetrazolSwimminggamma-Aminobutyric AcidBiological PsychiatryPharmacologyMotivationAlprazolamBehavior AnimalMuscimolDesipramineRatsnervous system diseasesDizocilpineDisease Models AnimalPsychiatry and Mental healthPsychotic DisordersNeurologyAlprazolamMuscimolchemistryAnesthesiaPentylenetetrazoleNeurology (clinical)Dizocilpine MaleatePsychologymedicine.drugBehavioural despair testEuropean Neuropsychopharmacology
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The gamma(2)-MSH peptide mediates a central analgesic effect via a GABA-ergic mechanism that is independent from activation of melanocortin receptors.

2001

Using the latency for tail-flick after thermal stimulation we have assessed the effects of alpha-, gamma(1)- and gamma(2)-MSH on nociceptive threshold in the mice. Intracisternal injections of gamma(2)-MSH induced a distinct analgesia, while gamma(1)-MSH in the same doses gave only a minor analgesia. Intracisternal alpha-MSH instead gave a short-term hyperalgesia. The effect of gamma(2)-MSH was not blocked by any of the MC(4)/MC(3)receptor antagonist HS014, naloxone or by the prior intracisternal administrations of gamma(1)-MSH. However, the gamma(2)-MSH analgesic response was completely attenuated by treating animals with the GABA(A)antagonist bicuculline. The gamma(2)-MSH analgesic effect…

MaleNarcotic Antagonists(+)-NaloxonePharmacologyGABA Antagonistschemistry.chemical_compoundMiceEndocrinologyDrug Interactionsgamma-Aminobutyric AcidAnalgesicsMice Inbred BALB Cintegumentary systemMuscimolNaloxoneReceptors MelanocortinNociceptorsGeneral MedicineReceptor antagonistNeurologyHyperalgesiamedicine.symptomhormones hormone substitutes and hormone antagonistsmedicine.drugPain ThresholdTailendocrine systemmedicine.medical_specialtyanimal structuresmedicine.drug_classCatalepsyBicucullinePeptides CyclicCellular and Molecular Neurosciencegamma-MSHMelanocortin receptorInternal medicinemedicineAnimalsGABA ModulatorsGABA AgonistsCatalepsyDiazepamEthanolEndocrine and Autonomic SystemsAntagonistCentral Nervous System DepressantsBicucullinemedicine.diseaseEndocrinologyMuscimolchemistryReceptors Corticotropinalpha-MSHNeuropeptides
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Brain-Derived Neurotrophic Factor But Not Forced Arm Use Improves Long-Term Outcome After Photothrombotic Stroke and Transiently Upregulates Binding …

2008

Background and Purpose— Both application of neurotrophic factors like brain-derived neurotrophic factor (BDNF) and constraint-induced movement therapy like forced arm use have been shown to potentially improve outcome after stroke. The aim of the present study was to check whether postischemic long-term outcome correlates to specific modifications in the abundance of various neurotransmitter receptors. Methods— Adult male Wistar rats were subjected to photothrombotic ischemia and assigned to various treatment groups (n=5 each) with end points at 3 and 6 weeks: (1) ischemic control (saline); (2) BDNF (ischemia, 20 μg BDNF); (3) forced arm use (ischemia, saline, and ipsilateral plaster cast …

MaleRestraint PhysicalMotor ActivityLigandsDownregulation and upregulationNeurotrophic factorsForelimbmedicineAnimalsRats WistarReceptorGABA Agonistsalpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic AcidStrokePhysical Therapy ModalitiesAdvanced and Specialized NursingBrain-derived neurotrophic factorMuscimolbusiness.industryBrain-Derived Neurotrophic FactorGlutamate receptorBrainCerebral Infarctionmedicine.diseaseRatsUp-RegulationStrokemedicine.anatomical_structureReceptors Glutamatenervous systemAnesthesiaExcitatory postsynaptic potentialAutoradiographyNeurology (clinical)Dizocilpine MaleateIntracranial ThrombosisForelimbCardiology and Cardiovascular MedicinebusinessExcitatory Amino Acid AntagonistsNeuroscienceStroke
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Sex differences in GABAA receptor binding in rat brain measured by an improved in vitro binding assay.

1990

The distribution of GABAA receptor sites was studied in female and male rat brain by an improved in vitro binding assay using 3H-muscimol and carefully washed membrane fractions. The binding studies revealed a single class of binding sites exhibiting the properties of GABAA receptors. The specific binding of 3H-muscimol was measured in four brain areas: the preoptic brain area, mediobasal hypothalamus, corticomedial amygdala and cerebral cortex. The equilibrium dissociation constants (KD) ranged from 11.2 to 23.3 nM in diestrous females and from 13.1 to 50.4 nM in males, the maximal number of binding sites (Bmax) from 3290 to 10240 fmol/mg protein in females and from 5495 to 17449 fmol/mg p…

Maleendocrine systemmedicine.medical_specialtychemistry.chemical_compoundSex FactorsInternal medicinemedicineAnimalsBinding siteReceptorChemistryGABAA receptorMuscimolGeneral NeuroscienceCell MembraneBrainRats Inbred StrainsReceptors GABA-AGonadotropin secretionRatsPreoptic areaDissociation constantKineticsmedicine.anatomical_structureEndocrinologynervous systemMuscimolCerebral cortexOrgan SpecificityFemaleExperimental brain research
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Cholinergic and GABAergic regulation of nitric oxide synthesis in the guinea pig ileum.

1999

Nitric oxide (NO) synthesis was examined in intact longitudinal muscle-myenteric plexus preparations of the guinea pig ileum by determining the formation of [3H]citrulline during incubation with [3H]arginine. Spontaneous [3H]citrulline production after 30 min was 80–90 dpm/mg, which constituted ∼1% of the tissue radioactivity. Electrical stimulation (10 Hz) led to a threefold increase in [3H]citrulline formation. Removal of calcium from the medium or addition of N G-nitro-l-arginine strongly inhibited both spontaneous and electrically induced production of [3H]citrulline. TTX reduced the electrically induced but not spontaneous [3H]citrulline formation. The electrically induced formation o…

Malemedicine.medical_specialtyBaclofenArgininePhysiologyGuinea PigsScopolamineMyenteric PlexusTubocurarineTetrodotoxinBiologyCholinergic AgonistsIn Vitro TechniquesMecamylamineBicucullineNitric OxideNitroarginineCholinergic Antagonistschemistry.chemical_compoundIleumPhysiology (medical)Internal medicineIsometric ContractionMuscarinic acetylcholine receptorMecamylaminemedicineCitrullineAnimalsEgtazic AcidGABA AgonistsMyenteric plexusgamma-Aminobutyric AcidHepatologyGABAA receptorMuscimolOxotremorineGastroenterologyMuscle SmoothBicucullineElectric StimulationEndocrinologychemistryMuscimolCitrullineFemalemedicine.drugThe American journal of physiology
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Blunted furosemide action on cerebellar GABAA receptors in ANT rats selectively bred for high alcohol sensitivity.

1996

Furosemide specifically reverses the inhibition by gamma-aminobutyric acid (GABA) of t-[35S]-butylbicyclophosphorothionate ([35S]TBPS) binding and increases the basal [35S]TBPS binding to the cerebellar granule cell layer GABAA receptors. For the selectivity of furosemide, an interplay between GABAA receptor alpha 6 and beta 2 or beta 3 subunits is needed. We have now investigated the furosemide sensitivity of cerebellar [35S]TBPS binding in the alcohol-sensitive (ANT) rat line that harbors a pharmacologically critical point mutation in the alpha 6 subunit [alpha 6 (Q1000)], increasing benzodiazepine affinity of the normally insensitive alpha 6-containing receptors. ANT receptors were less …

Malemedicine.medical_specialtyCerebellumPharmacologyLigandsTransfectionGABAA-rho receptorCell LineCellular and Molecular Neurosciencechemistry.chemical_compoundFurosemideInternal medicineCerebellummedicineAnimalsHumansGABA-A Receptor AntagonistsReceptorDiureticsGABA AgonistsIn Situ HybridizationPharmacologyEthanolGABAA receptorAntagonistFurosemideCentral Nervous System DepressantsRats Inbred StrainsReceptors GABA-AANTRecombinant ProteinsRatsEndocrinologymedicine.anatomical_structurenervous systemMuscimolchemistryAutoradiographyFemalemedicine.drugNeuropharmacology
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Opposite role played by GABAA and GABAB receptors in the modulation of peristaltic activity in mouse distal colon.

2014

We investigated the role of GABA on intestinal motility using as model the murine distal colon. Effects induced by GABA receptors recruitment were examined in whole colonic segments and isolated circular muscle preparations to analyze their influence on peristaltic reflex and on spontaneous and neurally-evoked contractions. Using a modified Trendelenburg set-up, rhythmic peristaltic contractions were evoked by gradual distension of the colonic segments. Spontaneous and neurally-evoked mechanical activity of circular muscle strips were recorded in vitro as changes in isometric tension. GABA, at low concentrations (10-50 µM), potentiated peristaltic activity and the neural cholinergic contrac…

Malemedicine.medical_specialtyMouseColonGABAB receptorGABAA-rho receptorMicechemistry.chemical_compoundPhaclofenInternal medicinemedicineAnimalsPeristaltic activityCholinergic contractiongamma-Aminobutyric AcidPharmacologyGABAA receptorGABAA receptorBicucullineReceptors GABA-AElectric StimulationMice Inbred C57BLEndocrinologyGABA AgentsReceptors GABA-Bnervous systemchemistryMuscimolPeristalsisHexamethoniumDistal colonMuscle Contractionmedicine.drugGABAB receptor
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gamma-Aminobutyric acid type A/benzodiazepine receptors regulate rat retina neurosteroidogenesis.

1995

Abstract It has been previously shown that retinal ganglion cells have the ability to synthesize steroids including neuroactive steroids such as pregnenolone sulfate. Since ganglion cells possess GABAA/benzodiazepine (BZ) receptors and neurosteroids modulate retinal GABAA receptor function, we investigated the role of these receptors in isolated rat retina neurosteroidogenesis. Ligands for central-type BZ receptors stimulated retinal pregnenolone synthesis. Clonazepam was the most potent ligand examined acting at nanomolar concentrations. Moreover, the effective steroidogenesis stimulatory dose (ED50) for these ligands and theKi to inhibit [3-H]flunitrazepam binding showed a coefficient of …

Malemedicine.medical_specialtyNeuroactive steroidFlunitrazepamBiologyPharmacologyIn Vitro TechniquesRetinal ganglionSynaptic TransmissionRetinaGABAA-rho receptorchemistry.chemical_compoundInternal medicinemedicineAnimalsGABA-A Receptor AgonistsGABA-A Receptor AntagonistsRats WistarMolecular BiologyNeurotransmitter AgentsGABAA receptorGeneral NeuroscienceBicucullineIsoquinolinesReceptors GABA-ARatsKineticsEndocrinologychemistryMuscimolPregnenolonePregnenoloneSteroidsNeurology (clinical)Pregnenolone sulfateNeurogliaDevelopmental Biologymedicine.drugBrain research
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GABAergic inhibition of nitric oxide-mediated relaxation of guinea-pig ileum

1999

The effects of GABA receptor agonists were investigated on guinea-pig isolated ileum longitudinal muscle with intact myenteric plexus. Electrical field stimulation (1 Hz, 10 s) of the histamine (1 microM)-precontracted preparation caused a contraction followed by a relaxation. Relaxations were inhibited by L-N(G)-nitroarginine (L-NA; EC50 3 microM) in a concentration-dependent manner. The inhibitory action of 10 microM L-NA was blocked by 10 microM L-arginine but not by D-arginine, which indicates that the relaxation was largely mediated by endogenous nitric oxide (NO). Tetrodotoxin (1 microM) reduced the relaxation only by about 50%. GABA and the GABA(B) agonist, baclofen, inhibited the fi…

Malemedicine.medical_specialtyNitric Oxide Synthase Type IIIMuscle RelaxationGuinea PigsTetrodotoxinIn Vitro TechniquesGABAB receptorBicucullineNitric OxideNitroargininechemistry.chemical_compoundGABA receptorIleumInternal medicinemedicineAnimalsGABA-A Receptor AgonistsEnzyme InhibitorsGABA Agonistsgamma-Aminobutyric AcidMyenteric plexusPharmacologyMuscimolGABAA receptorMuscle SmoothGeneral MedicineBicucullineElectric StimulationBaclofenEndocrinologynervous systemchemistryMuscimolGABA-B Receptor AgonistsSaclofenBiophysicsFemaleNitric Oxide SynthaseHistaminemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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