Search results for "codon"

showing 10 items of 196 documents

Epidemiological study of nonsyndromic hearing loss in Sicilian newborns

2007

Deafness is caused by a variety of facts, genetic and environmental. Regarding the acquired causes, deafness can be the consequence of prenatal infections, acoustic or cerebral trauma, and the use of ototoxic drugs. Deafness can be the only manifestation (nonsyndromic forms) or it may occur together with other phenotypic findings (syndromic forms). The majority of nonsyndromicdeafness has a genetic basis [Van Camp et al., 1997]. In recent years, deafness and hearing loss have assumed a clinical importance in the study of congenital disorders [Morton et al., 1991]. The clinical interest for hearing loss is supported by the social impact that this disorder has; if not treated, delays in the d…

GenotypeHearing lossHearing Loss SensorineuralDNA Mutational AnalysisNonsense mutationBiologyGene mutationConnexinsneonate deafness geneticExonNeonatal ScreeningGene Frequencyotorhinolaryngologic diseasesGeneticsmedicineHumansGenetic TestingSicilyGeneGenetics (clinical)Chromosome 13GeneticsSplice site mutationInfant NewbornGenetic VariationStop codonConnexin 26PhenotypeMutationmedicine.symptomAmerican Journal of Medical Genetics Part A
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Use and misuse in the application of the phytoplankton functional classification: a critical review with updates

2008

Since its publication, the article ‘Towards a functional classification of the freshwater phytoplankton’ (Reynolds et al., J Plankton Res 24: 417–428, 2002), has attracted the attention of dozens of phytoplankton ecologists worldwide. These numerous applications of the functional classification to describe phytoplankton patterns in various aquatic ecosystems allowed the recognition of some uncertain features of this concept originating from various reasons. In this article, we attempt to facilitate the application of the functional classification, by providing a detailed description of the typical misplacements and by modifying some of the original habitat templates and species allocations.…

GeographyWater Framework DirectiveHabitatEcologyAquatic ecosystemEcology (disciplines)PhytoplanktonAquatic SciencePlanktonFunctional groups Codon Phytoplankton Water Framework DirectiveFunctional group (ecology)Hydrobiology
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Characterization of a novel open reading frame, urf a, in the mitochondrial genome of fission yeast: correlation of urf a mutations with a mitochondr…

1991

Between the genes for tRNA(gin) and tRNA(ile) an open reading frame of 227 amino acids has been identified which is unique among known mitochondrial genomes and which has been termed urf a (Lang et al. 1983; Kornrumpf et al. 1984). It uses the "mitochondrial" genetic code, i.e., it contains a TGA codon, whereas all other protein-encoding genes, and all but one intronic open reading frame, use the "standard" genetic code (UGG for tryptophan). A previous paper has demonstrated that "mutator" strains show an increased formation of mitochondrial drug-resistant and respiration-deficient mutants (including deletions). In this paper we show that the mutator activity is correlated with mutations in…

GlycerolMitochondrial DNAMutantMolecular Sequence DataExtrachromosomal InheritanceBiologymedicine.disease_causeDNA MitochondrialFrameshift mutationFungal ProteinsMitochondrial ProteinsOpen Reading FramesGene Expression Regulation FungalSchizosaccharomycesGeneticsmedicineAmino Acid SequenceCodonDNA FungalFrameshift MutationGeneGeneticsMutationTranslational frameshiftBase SequenceGeneral MedicineGenetic codeOpen reading framePhenotypeMutationSchizosaccharomyces pombe ProteinsCurrent genetics
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Mutations in KIAA0586 Cause Lethal Ciliopathies Ranging from a Hydrolethalus Phenotype to Short-Rib Polydactyly Syndrome

2015

KIAA0586, the human ortholog of chicken TALPID3, is a centrosomal protein that is essential for primary ciliogenesis. Its disruption in animal models causes defects attributed to abnormal hedgehog signaling; these defects include polydactyly and abnormal dorsoventral patterning of the neural tube. Here, we report homozygous mutations of KIAA0586 in four families affected by lethal ciliopathies ranging from a hydrolethalus phenotype to short-rib polydactyly. We show defective ciliogenesis, as well as abnormal response to SHH-signaling activation in cells derived from affected individuals, consistent with a role of KIAA0586 in primary cilia biogenesis. Whereas centriolar maturation seemed una…

Heart Defects CongenitalMolecular Sequence DataCell Cycle ProteinsBiologyShort Rib-Polydactyly SyndromeCiliopathies03 medical and health sciencesFatal OutcomeCiliogenesisReportGLI3GeneticsmedicineHumansGenetics(clinical)Europe EasternGenetics (clinical)030304 developmental biologyGenetics0303 health sciencesLikelihood FunctionsShort rib – polydactyly syndromePolydactylyBase SequenceCilium030305 genetics & hereditySequence Analysis DNAmedicine.diseasePhenotypeHuman geneticsHedgehog signaling pathwayFounder EffectPedigreePhenotypeCodon NonsenseCentriolar satelliteErratumHand Deformities CongenitalCiliary Motility DisordersHydrocephalus
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Mutation specific PCR and direct solid phase sequencing assay for the detection of hepatitis B virus pre-C/C mutants in anti-HBe-positive, chronic he…

1994

Sequence analysis of the HBV DNA from patients with anti-HBe+, chronic hepatitis B revealed that the lack of HBeAg is mostly due to a single GA transition at nucleotide position 1896, resulting in a translational stop codon. A point mutation-specific polymerase chain reaction (msPCR) for the detection of this genetic variant was established. Two serologically defined groups of patients with symptomatic chronic hepatitis B (HBeAg+ n = 14, anti-HBe+ n = 11) were included in this study. Viral DNA from 43 sera (26 eAg+/17 anti-HBe+) was amplified twice, using two different sets of PCR primers. Each set contained the same — strand primer, but the + strand primers differed at their 3′-end, thus b…

Hepatitis B virusHepatitis B virus DNA polymeraseMolecular Sequence DataBiologymedicine.disease_causePolymerase Chain Reactionlaw.inventionlawVirologymedicineHumansPoint MutationHepatitis B e AntigensHepatitis B AntibodiesPolymerase chain reactionDNA PrimersHepatitis B virusBase SequencePoint mutationvirus diseasesGenetic Variationbiology.organism_classificationHepatitis BVirologyMolecular biologydigestive system diseasesStop codonInfectious DiseasesHepadnaviridaeHBeAgDNA ViralPrimer (molecular biology)Journal of medical virology
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Stop codon insertion restores the particle formation ability of hepatitis B virus core-hantavirus nucleocapsid protein fusions.

2003

In recent years, epitopes of various origin have been inserted into the core protein of hepatitis B virus (HBc), allowing the formation of chimeric HBc particles. Although the C-terminus of a C-terminally truncated HBc (HBcΔ) tolerates the insertion of extended foreign sequences, the insertion capacity is still a limiting factor for the construction of multivalent vaccines. Previously, we described a new system to generate HBcΔ mosaic particles based on a read-through mechanism in an <i>Escherichia coli</i> suppressor strain [J Gen Virol 1997;78:2049–2053]. Those mosaic particles allowed the insertion of a 114-amino acid (aa)-long segment of a Puumala hantavirus (PUUV) nucleocap…

Hepatitis B virusHepatitis B virus DNA polymerasevirusesRecombinant Fusion ProteinsMolecular Sequence Datamedicine.disease_causeEpitopeHepatitis B virus PRE betaMiceVirologyparasitic diseasesmedicineAnimalsNucleocapsidHantavirusHepatitis B virusMice Inbred BALB CBase SequenceChemistryHepatitis B virus coreVirionvirus diseasesNucleocapsid ProteinsVirologyMolecular biologyHepatitis B Core Antigensdigestive system diseasesStop codonNS2-3 proteaseInfectious DiseasesCodon TerminatorImmunizationIntervirology
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Semiquantitative assessment of pre-core stop-codon mutant and wildtype hepatitis B virus during the course of chronic hepatitis B using a new PCR-bas…

1996

In most patients with chronic hepatitis B positive for antibodies (anti-HBe) to HBe antigen (HBeAg), a pre-core mutant hepatitis B virus (HBV) with a point-mutation at nt. 1896 can be isolated. Clinical significance of the mutant virus in chronic hepatitis B is not proven yet, and screening of large numbers of sera during different clinical courses of numerous patients is necessary. We therefore aimed to develop a fast and reliable assay, that allows to discriminate wildtype from nt. 1896 G-->A mutant HBV and to determine the ratio of mutant and wildtype HBV in patients' sera. A mutation specific polymerase chain reaction (ms PCR) with new primers served to distinguish nt. 1896 G-->A mutant…

Hepatitis B virusMutantPopulationBiologymedicine.disease_causePolymerase Chain ReactionSensitivity and SpecificityViruslaw.inventionlawVirologymedicineHumansPoint MutationHepatitis B e AntigenseducationPolymerase chain reactionHepatitis B viruseducation.field_of_studyWild typevirus diseasesGeneral Medicinebiology.organism_classificationHepatitis BVirologydigestive system diseasesHBeAgHepadnaviridaeEvaluation Studies as TopicChronic DiseaseCodon TerminatorFollow-Up Studies
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Presence and coding properties of 2'-O-methyl-5-carbamoylmethyluridine (ncm5Um) in the wobble position of the anticodon of tRNA(Leu) (U*AA) from brew…

1992

AbstractThe unknown modified nucleoside U* has been isolated by enzymatic and HPLC protocols from tRNALeu(U*AA) recently discovered in brewer's yeast. The pure U* nucleoside has been characterized by electron impact mass spectroscopy, and comparison of its chromatographic and UV-absorption properties with those of appropriate synthetic compounds. The structure of U* was established as 2′-O-methyl-5-carbamoylmethyluridine (ncm5Um). The yeast tRNALeu (U*AA) is the only tRNA so far sequenced which has been shown to contain ncm5Um. The location of such a modified uridine at the first position of the anticodon restricts the decoding property to A of the leucine UUA codon.

IdentificationRNA Transfer LeuStereochemistryBiophysicsAminoacylationWobble base pairModified nucleosideSaccharomyces cerevisiaeBiochemistryMass SpectrometryFungal Proteinschemistry.chemical_compoundStructural BiologyGeneticsAnticodonMolecular BiologyUridineChromatography High Pressure Liquidchemistry.chemical_classificationMolecular StructureRNA FungalCell BiologyUridineYeastYeastEnzymechemistryBiochemistryTransfer RNAtRNALeu (U*AA)Spectrophotometry UltravioletLeucineNucleosideFEBS letters
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Deciphering the Nonsense Readthrough Mechanism of Action of Ataluren: An in Silico Compared Study

2019

Ataluren was reported to suppress nonsense mutations by promoting the readthrough of premature stop codons, although its mechanism of action (MOA) is still debated. The likely interaction of Ataluren with CFTR-mRNA has been previously studied by molecular dynamics. In this work we extended the modeling of Ataluren's MOA by complementary computational approaches such as induced fit docking (IFD), quantum polarized ligand docking (QPLD), MM-GBSA free-energy calculations, and computational mutagenesis. In addition to CFTR-mRNA, this study considered other model targets implicated in the translation process, such as eukaryotic rRNA 18S, prokaryotic rRNA 16S, and eukaryotic Release Factor 1 (eRF…

In silicoNonsense mutationComputational biology01 natural sciencesRibosomeBiochemistrychemistry.chemical_compoundDrug DiscoveryQPLDcomputational mutagenesiMM-GBSA010405 organic chemistryChemistryDrug Discovery3003 Pharmaceutical ScienceOrganic Chemistrypremature termination codonSettore CHIM/06 - Chimica OrganicaSettore CHIM/08 - Chimica FarmaceuticaStop codon0104 chemical sciencesAtalurenInduced fit docking010404 medicinal & biomolecular chemistrySettore BIO/18 - GeneticaDocking (molecular)ProofreadingRelease factoroxadiazole
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Rapid Titration With Intravenous Oxycodone for Severe Cancer Pain and Oral Conversion Ratio.

2022

to assess a dose titration with intravenous oxycodone to achieve rapid pain relief of cancer pain of severe intensity. The second objective was to provide a conversion ratio with the oral route.Cancer patients admitted for severe pain were prospectively assessed. At admission (T0) previous opioid doses were recorded. Edmonton symptom assessment scale (ESAS) was collected from T0 until the conclusion of the observation. Intravenous boluses of oxycodone were given until the initial signs of significant analgesia were detected. The effective dose was multiplied for six and given as intravenous continuous infusion. When the patient was considered stabilized the intravenous daily dose was conver…

Intravenous oxycodonePainOpioidCancer PainDose titrationAnalgesics OpioidAnesthesiology and Pain MedicineNeoplasmsPalliative careHumansNeurology (clinical)Neoplasms.General NursingOxycodoneHumanJournal of pain and symptom management
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