Search results for "connective tissue disease"

showing 10 items of 874 documents

Postmodernistyczne przemiany w rodzinie

2018

Artykuł jest próbą syntezy przemian rodziny dokonujących się w epoce postmodernizmu. Sta-rano się pokazać przemiany w zakresie definiowania rodziny, liczby zawieranych małżeństw i mo-deli funkcjonujących w tej instytucji, przemiany w prokreacji oraz alternatywne do rodziny związki. Wnioski tych analiz prowadzą do stwierdzenia, że postmodernizm wpływa na współczesną rodziną, wywołując w niej dosyć wyraźnie zaznaczające się zmiany, ale ciągle to rodzina nuklearna pozosta-je dominującą formą życia małżeńsko-rodzinnego

Family relationsmedia_common.quotation_subjectOrganic Chemistrypostmodernizm; przemiany rodziny; małżeństwo; prokreacja; alternatywne formy życia rodzinnegosense organsSociologyskin and connective tissue diseasesPostmodernismFunction (engineering)BiochemistryNuclear familyGenealogymedia_commonRoczniki Pedagogiczne
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Climate change effects on trematodiases, with emphasis on zoonotic fascioliasis and schistosomiasis.

2009

The capacity of climatic conditions to modulate the extent and intensity of parasitism is well known since long ago. Concerning helminths, among the numerous environmental modifications giving rise to changes in infections, climate variables appear as those showing a greater influence, so that climate change may be expected to have an important impact on the diseases they cause. However, the confirmation of the impact of climate change on helminthiases has been reached very recently. Only shortly before, helminthiases were still noted as infectious diseases scarcely affected by climate change, when compared to diseases caused by microorganisms in general (viruses, bacteriae, protozoans). Th…

FascioliasisClimate ChangeSnailsHelminthiasisCercarial DermatitisClimate changeParasitismAnimals WildBiologyHost-Parasite InteractionsEffects of global warmingZoonosesmedicinePrevalenceHelminthsPopulation growthAnimalsSchistosomiasisskin and connective tissue diseasesDisease ReservoirsGeneral VeterinaryEcologyOocystsGeneral Medicinemedicine.diseaseAnimals DomesticParasitologysense organsSeasonsMicroparasiteVeterinary parasitology
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Evaluation of near-Infrared fluorescence-conjugated peptides for visualization of human epidermal receptor 2-overexpressed gastric cancer.

2021

338 Background: HER2 is highly overexpressed in many kinds of cancers with a poor prognosis. Recently, near-infrared (NIR) fluorescence-based imaging is a growing field for both pre-clinical and clinical application. In this study, we aimed to synthesize Human Epidermal Receptor2 (HER2)-specific near-infrared (NIR) fluorescence probes and evaluate their applicability in cancer-specific image-guided surgeries using an animal model. Methods: An NIR dye emitting light of 800 nm (IRDye800CW, Li-COR, USA) was conjugated to trastuzumab and HER2-specific affibody using click mechanism. HER2 affinity was assessed by the surface plasmon resonance technique. HER2 positive/negative gastric cancer cel…

Fluorescence-lifetime imaging microscopyBiodistributionCancer Researchbusiness.industryStomach neoplasmsGastroenterologyCancerSpleenmedicine.diseaseMolecular biologyFluorescenceFluorescencemedicine.anatomical_structureImage-guided surgeryOncologyGastrectomymedicineOriginal ArticleSurgerySurface plasmon resonanceReceptorbusinessskin and connective tissue diseasesneoplasmsJournal of Clinical Oncology
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Free radical-mediated systemic immunity in plants.

2014

SPE IPM; Systemic acquired resistance (SAR) is a form of defense that protects plants against a broad-spectrum of secondary infections by related or unrelated pathogens. SAR related research has witnessed considerable progress in recent years and a number of chemical signals and proteins contributing to SAR have been identified. All of these diverse constituents share their requirement for the phytohormone salicylic acid, an essential downstream component of the SAR pathway. However, recent work demonstrating the essential parallel functioning of nitric oxide (NO)-derived and reactive oxygen species (ROS)-derived signaling together with SA provides important new insights in the overlapping …

Free RadicalsSecondary infection[SDV]Life Sciences [q-bio]Plant ImmunityPlant ScienceBiologyNitric OxideNitric oxidechemistry.chemical_compoundImmune systemPlant Growth Regulators[SDV.BV]Life Sciences [q-bio]/Vegetal BiologyPlant Immunityskin and connective tissue diseaseschemistry.chemical_classificationReactive oxygen speciesfungiPlantsCell biologybody regionschemistryBiochemistry[SDE]Environmental SciencesSignal transductionReactive Oxygen SpeciesSalicylic AcidSalicylic acidSystemic acquired resistanceSignal TransductionCurrent opinion in plant biology
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LATE-BREAKING ABSTRACT: MUC1 downregulation induces corticosteroid resistance in asthma'sin vitroandin vivomodels

2016

Background: Corticosteroids are in the first-line of Asthma treatment. However a subset of asthmatic patients is insensitive to anti-inflammatory effects of corticosteroids. Our previous data demonstrated that MUC1 downregulation correlates corticosteroid resistance in Nasal Polyposis and COPD. We hypothesized that MUC1 is also involved in corticosteroid resistance in Asthma. Objective: To analyze the role of MUC1 in the corticosteroid effectiveness in Asthma9s in vitro and in vivo models. Methods: 20 patients with steroid resistant Asthma were recruited. The expression of MUC1 was determined by real-time PCR in inflammatory cells from peripheral blood. HMVEC-L and U937 cells knockdown with…

Gene knockdownCOPDmedicine.diagnostic_testmedicine.drug_classbusiness.industrymedicine.diseasedigestive systembiological factorsdigestive system diseasesBronchoalveolar lavageDownregulation and upregulationIn vivoImmunologymedicineCorticosteroidskin and connective tissue diseasesbusinessneoplasmsDexamethasonemedicine.drugAsthma5.1 Airway Pharmacology and Treatment
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Glycopeptide-functionalized gold nanoparticles for antibody induction against the tumor associated mucin-1 glycoprotein

2015

We report the preparation of gold nanoparticle (AuNP)-based vaccine candidates against the tumor-associated form of the mucin-1 (MUC1) glycoprotein. Chimeric peptides, consisting of a glycopeptide sequence derived from MUC1 and the T-cell epitope P30 sequence were immobilized on PEGylated AuNPs and the ability to induce selective antibodies in vivo was investigated. After immunization, mice showed significant MHC-II mediated immune responses and their antisera recognized human MCF-7 breast cancer cells. Nanoparticles designed according to this report may become key players in the development of anticancer vaccines.

Genes MHC Class IIMolecular Sequence DataClinical BiochemistryEpitopes T-LymphocyteMetal NanoparticlesPharmaceutical Science02 engineering and technology010402 general chemistryCancer Vaccines01 natural sciencesBiochemistryAntibodiesEpitopeMiceImmune systemNeoplasmsDrug DiscoveryAnimalsHumansAmino Acid Sequenceskin and connective tissue diseasesMolecular BiologyMUC1Cancerchemistry.chemical_classificationAntiserumVaccinesbiologyMucin-1Organic ChemistryGlycopeptides021001 nanoscience & nanotechnologyMolecular biologyGlycopeptide0104 chemical scienceschemistryColloidal goldImmunologyMCF-7 Cellsbiology.proteinNanoparticlesMolecular MedicineImmunizationGoldAntibody0210 nano-technologyGlycoproteinBioorganic & Medicinal Chemistry
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Family studies in scleroderma (systemic sclerosis) demonstrating an HLA-linked increased chromosomal breakage rate in cultured lymphocytes

1988

An increased chromosomal breakage rate (ICBR) was found in 27 of 28 patients with scleroderma (systemic sclerosis, SS) - 5 with the syndrome including calcinosis cutis, Raynaud phenomenon, esophagus hypomotility, sclerodactyly and telangiectasia (CREST), 4 incomplete CREST, 1 overlapping syndrome, 18 progressive systemic sclerosis (PSS). Not only the patients, but also about half of their first-degree relatives showed an increased chromosomal breakage rate (more than 5 breaks per 100 metaphases). This character segregated as a dominant marker in nine families of scleroderma patients. In the six informative of the nine families, the ICBR trait showed close linkage with the HLA region on chro…

Genetic MarkersMaleSystemic diseaseGenetic LinkageHuman leukocyte antigenBiologySclerodermaCalcinosis cutisHLA AntigensGeneticsmedicineHumansLymphocytesCells CulturedGenetics (clinical)Chromosome AberrationsAutoimmune diseaseScleroderma SystemicSclerodactylyChromosome Fragilitymedicine.diseaseConnective tissue diseasePedigreeHaplotypesImmunologyFemalemedicine.symptomHuman Genetics
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C1q acts in the tumour microenvironment as a cancer-promoting factor independently of complement activation

2015

Complement C1q is the activator of the classical pathway. However, it is now recognized that C1q can exert functions unrelated to complement activation. Here we show that C1q, but not C4, is expressed in the stroma and vascular endothelium of several human malignant tumours. Compared with wild-type (WT) or C3- or C5-deficient mice, C1q-deficient (C1qa−/−) mice bearing a syngeneic B16 melanoma exhibit a slower tumour growth and prolonged survival. This effect is not attributable to differences in the tumour-infiltrating immune cells. Tumours developing in WT mice display early deposition of C1q, higher vascular density and an increase in the number of lung metastases compared with C1qa−/− mi…

Genetics and Molecular Biology (all)0301 basic medicinePROTEINGeneral Physics and AstronomyMELANOMAApoptosisInbred C57BLBiochemistryDISEASEAnimals; Apoptosis; Cell Line Tumor; Cell Movement; Cell Proliferation; Complement Activation; Complement C1q; Complement C3; Complement C5; Humans; Mice; Mice Inbred C57BL; Mice Knockout; Neoplasms; Biochemistry Genetics and Molecular Biology (all); Chemistry (all); Physics and Astronomy (all)Micefluids and secretionsCell Movementimmune system diseasesNeoplasmsIMMUNE-RESPONSEskin and connective tissue diseasesComplement ActivationComplement C1qMice KnockoutComplement component 5TumorMultidisciplinaryQChemistry (all)Complement C5Complement C33. Good healthCell biologyMultidisciplinary SciencesDEFICIENCYmedicine.anatomical_structureScience & Technology - Other TopicsHumanKnockoutSciencechemical and pharmacologic phenomenaBiologyArticleGeneral Biochemistry Genetics and Molecular BiologyTROPHOBLAST INVASIONMECHANISMSCell LinePhysics and Astronomy (all)03 medical and health sciencesClassical complement pathwayImmune systemINFLAMMATIONCell Line TumormedicineAnimalsHumansCell ProliferationScience & TechnologyBiochemistry Genetics and Molecular Biology (all)AnimalCell growthEFFECTOR SYSTEMComplement C1qApoptosiGeneral ChemistryComplement systemMice Inbred C57BL030104 developmental biologyCancer cellNeoplasmBone marrowANTIBODY THERAPYNature Communications
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A framework for remission in SLE

2017

ObjectivesTreat-to-target recommendations have identified ‘remission’ as a target in systemic lupus erythematosus (SLE), but recognise that there is no universally accepted definition for this. Therefore, we initiated a process to achieve consensus on potential definitions for remission in SLE.MethodsAn international task force of 60 specialists and patient representatives participated in preparatory exercises, a face-to-face meeting and follow-up electronic voting. The level for agreement was set at 90%.ResultsThe task force agreed on eight key statements regarding remission in SLE and three principles to guide the further development of remission definitions:1. Definitions of remission wi…

Genetics and Molecular Biology (all)PediatricsAutoimmune diseasesNEPHRITIS PATIENTSDISEASE-ACTIVITYSeverity of Illness IndexBiochemistryRETROSPECTIVE ANALYSIS0302 clinical medicineQuality of lifePrednisoneAdrenal Cortex HormonesLupus Erythematosus SystemicImmunology and AllergyCHINESE PATIENTS030212 general & internal medicineSYSTEMIC-LUPUS-ERYTHEMATOSUSskin and connective tissue diseasesPREDICTORSOUTCOMESSystemic lupus erythematosusMalalties autoimmunitàriesRemission InductionSYSTEMIC-LUPUS-ERYTHEMATOSUS; DISEASE-ACTIVITY; RETROSPECTIVE ANALYSIS; INITIAL VALIDATION; NEPHRITIS PATIENTS; AMERICAN-COLLEGE; CHINESE PATIENTS; RENAL FLARES; PREDICTORS; OUTCOMESSymptom Flare UpConnective tissue diseaseManchester Institute for Collaborative Research on AgeingEstudi de casosOutcomes researchAntibodies AntinuclearDNA/immunologyImmunosuppressive Agentsmedicine.drugmedicine.medical_specialtyFarmacologiaResearchInstitutes_Networks_Beacons/MICRAConsensusImmunologyAdrenal Cortex Hormones/therapeutic useAMERICAN-COLLEGELupus Erythematosus Systemic/bloodSystemic Lupus ErythematosusGeneral Biochemistry Genetics and Molecular BiologyMaintenance Chemotherapy03 medical and health sciencesAntimalarialsRheumatologySeverity of illnessmedicineDisease Activity; Outcomes research; Systemic Lupus Erythematosus; Immunology and Allergy; Rheumatology; Immunology; Biochemistry Genetics and Molecular Biology (all)HumansDisease Activity030203 arthritis & rheumatologyPharmacologyAntibodies Antinuclear/bloodLupus erythematosusbusiness.industryTask forceConstruct validityRENAL FLARESComplement System ProteinsDNAINITIAL VALIDATIONDisease Activity; Outcomes research; Systemic Lupus Erythematosusmedicine.diseaseLupus eritematósAntimalarials/therapeutic usePhysical therapyImmunosuppressive Agents/therapeutic useComplement System Proteins/metabolismCase studiesOutcomes researchbusinessAnnals of the Rheumatic Diseases
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Mutational analysis ofBRCA1andBRCA2in Mediterranean Spanish women with early-onset breast cancer: Identification of three novel pathogenic mutations

2003

In Spain, the contribution of BRCA mutations to the population incidence of early-onset breast cancer was unknown. We carried out a mutational analysis of the BRCA1 and BRCA2 genes in 124 Spanish women diagnosed with breast cancer before the age 41 and who were not selected for a family history of this disease. The genetic study was performed by PCR-SSCP analysis and DNA sequencing. We identified 6 pathogenic BRCA mutations in 7 unrelated probands (5.6%; 95% CI=2.3% to 11.3%): 1 BRCA1 (c.2080delA) and 5 BRCA2 (p.Y3006X, p.Q1994X, c.9204_9217del14, c.9254_9258del5 and c.295+2T>C). Three out of 6 mutations were novel (BRCA2 p.Y3006X, c.9204_9217del14, and c.295+2T>C), and two further mutation…

GeneticsMutationeducation.field_of_studyendocrine system diseasesPopulationCancerBiologymedicine.disease_causemedicine.diseasefemale genital diseases and pregnancy complicationsGermline mutationBreast cancerDNA Mutational AnalysisGeneticsmedicineMissense mutationFamily historyskin and connective tissue diseaseseducationGenetics (clinical)Human Mutation
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