Search results for "corte"

showing 10 items of 2212 documents

Neural oscillations in the infralimbic cortex after electrical stimulation of the amygdala. Relevance to acute stress processing

2017

The stress system coordinates the adaptive reactions of the organism to stressors. Therefore, dysfunctions in this circuit may correlate to anxiety-related disorders, including depression. Comprehending the dynamics of this network may lead to a better understanding of the mechanisms that underlie these diseases. The central nucleus of the amygdala (CeA) activates the hypothalamic–pituitary–adrenal axis and brainstem nodes by triggering endocrine, autonomic and behavioral stress responses. The medial prefrontal cortex plays a significant role in regulating reactions to stressors, and is specifically important for limiting fear responses. Brain oscillations reflect neural systems activity. S…

0301 basic medicineTime FactorsInfralimbic cortexLocal field potentialBiologyAmygdalaRats Sprague-Dawley03 medical and health sciences0302 clinical medicinemedicineAnimalsPrefrontal cortexEvoked PotentialsCerebral CortexNeuronsAfferent PathwaysGeneral NeuroscienceCentral nucleus of the amygdalaAmygdalaElectric StimulationRatsElectrophysiology030104 developmental biologymedicine.anatomical_structureSynaptic plasticityFemaleBrainstemNeuroscience030217 neurology & neurosurgery
researchProduct

Dibutyl Phthalate (DBP)-Induced Apoptosis and Neurotoxicity are Mediated via the Aryl Hydrocarbon Receptor (AhR) but not by Estrogen Receptor Alpha (…

2016

Dibutyl phthalate (di-n-butyl phthalate, DBP) is one of the most commonly used phthalate esters. DBP is widely used as a plasticizer in a variety of household industries and consumer products. Because phthalates are not chemically bound to products, they can easily leak out to enter the environment. DBP can pass through the placental and blood–brain barriers due to its chemical structure, but little is known about its mechanism of action in neuronal cells. This study demonstrated the toxic and apoptotic effects of DBP in mouse neocortical neurons in primary cultures. DBP stimulated caspase-3 and LDH activities as well as ROS formation in a concentration (10 nM–100 µM) and time-dependent (3–…

0301 basic medicineTime Factorsgenetic structuresPPARγPeroxisome proliferator-activated receptorApoptosis010501 environmental sciencesToxicology01 natural sciencesDBPMicechemistry.chemical_compoundERβReceptorCells CulturedERαCerebral CortexNeuronschemistry.chemical_classificationbiologyCaspase 3General NeurosciencePhthalateDibutyl PhthalatePhthalateOriginal ArticleSignal transductioncirculatory and respiratory physiologymedicine.medical_specialtyCell SurvivalDibutyl phthalateNeuroscience(all)03 medical and health sciencesInternal medicinemedicineAnimalsEstrogen Receptor betaRNA Messengercardiovascular diseasesEstrogen receptor beta0105 earth and related environmental sciencesDose-Response Relationship DrugAhREstrogen Receptor alphaNeuronAryl hydrocarbon receptorPPAR gamma030104 developmental biologyEndocrinologyReceptors Aryl Hydrocarbonchemistrybiology.proteinReactive Oxygen SpeciesEstrogen receptor alphaNeurotoxicity Research
researchProduct

Rescue of Hypovitaminosis A Induces Non-Amyloidogenic Amyloid Precursor Protein (APP) Processing.

2015

Retinoic acid, the bioactive metabolite of beta-carotene or vitamin A, plays a pleiotropic, multifunctional role in vertebrate development. Studies in rodents revealed that a diet deficient in vitamin A results in a complex neonatal syndrome (the VAD syndrome), manifested in many organs. In humans, the function of retinoic acid (RA) extends into adulthood, where it has important roles in fertility, vision, and suppression of neoplastic growth. In recent years, it has also been suggested that retinoic acid might potentially act as a therapeutically relevant drug in attenuating or even preventing neurodegenerative diseases such as Alzheimer's disease (AD). Here, we report that VAD leads to an…

0301 basic medicineVitaminmedicine.medical_specialtyADAM10Retinoic acidTretinoin03 medical and health scienceschemistry.chemical_compoundADAM10 ProteinAmyloid beta-Protein PrecursorMiceNeuroblastoma0302 clinical medicineKeratolytic AgentsTretinoinInternal medicineNeuroblastomaGene expressionPresenilin-2medicineAmyloid precursor proteinAnimalsHumansGene Regulatory NetworksRats WistarCells CulturedCerebral CortexNeuronsAmyloid beta-PeptidesbiologyVitamin A Deficiencymedicine.diseaseAcitretinPeptide FragmentsVitamin A deficiencyDisease Models Animal030104 developmental biologyEndocrinologyNeurologychemistryAnimals Newbornbiology.proteinFemaleNeurology (clinical)030217 neurology & neurosurgerymedicine.drugCurrent Alzheimer research
researchProduct

A Multilevel Functional Study of aSNAP25At-Risk Variant for Bipolar Disorder and Schizophrenia

2017

The synaptosomal-associated protein SNAP25 is a key player in synaptic vesicle docking and fusion and has been associated with multiple psychiatric conditions, including schizophrenia, bipolar disorder, and attention-deficit/hyperactivity disorder. We recently identified a promoter variant inSNAP25,rs6039769, that is associated with early-onset bipolar disorder and a higher gene expression level in human prefrontal cortex. In the current study, we showed that this variant was associated both in males and females with schizophrenia in two independent cohorts. We then combinedin vitroandin vivoapproaches in humans to understand the functional impact of the at-risk allele. Thus, we showedin vi…

0301 basic medicine[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiology[SDV.NEU.PC] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Psychology and behaviorbrain imagingAmygdala03 medical and health sciences0302 clinical medicineNeuroimagingSynaptic vesicle dockingmedicinegeneticsBipolar disorderAllelePrefrontal cortexComputingMilieux_MISCELLANEOUSbipolar disorder[SDV.NEU.PC]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Psychology and behavior[SCCO.NEUR]Cognitive science/NeuroscienceGeneral Neuroscience[SCCO.NEUR] Cognitive science/Neuroscience[SDV.NEU.NB] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiology[SDV.NEU.SC]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Cognitive SciencesSNAP25medicine.diseaseschizophrenia030104 developmental biologymedicine.anatomical_structureSNARESNAP25CohortPsychologyNeuroscience[SDV.NEU.SC] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Cognitive Sciences030217 neurology & neurosurgeryThe Journal of Neuroscience
researchProduct

Selective α-synuclein knockdown in monoamine neurons by intranasal oligonucleotide delivery: potential therapy for parkinson’s disease

2018

Progressive neuronal death in brainstem nuclei and widespread accumulation of α-synuclein are neuropathological hallmarks of Parkinson’s disease (PD). Reduction of α-synuclein levels is therefore a potential therapy for PD. However, because α-synuclein is essential for neuronal development and function, α-synuclein elimination would dramatically impact brain function. We previously developed conjugated small interfering RNA (siRNA) sequences that selectively target serotonin (5-HT) or norepinephrine (NE) neurons after intranasal administration. Here, we used this strategy to conjugate inhibitory oligonucleotides, siRNA and antisense oligonucleotide (ASO), with the triple monoamine reuptake …

0301 basic medicineanimal diseasesDopamineOligonucleotidesGene ExpressionPharmacologySynaptic TransmissionPrefrontal cortexMiceDA neurotransmission0302 clinical medicineDrug DiscoveryMonoaminergicNeural PathwaysRNA Small InterferingCells Cultured5-HT neurotransmissionChemistryGene Transfer TechniquesParkinson DiseaseVentral tegmental areaSubstantia Nigramedicine.anatomical_structureCaudate putamenGene Knockdown Techniquesalpha-SynucleinMolecular MedicineRNA InterferenceOriginal ArticleMonoamine reuptake inhibitormedicine.drugSignal TransductionSerotoninSubstantia nigraASO03 medical and health sciencesProsencephalonα-synucleinDopamineIntranasal administrationGeneticsmedicineAnimalsHumansMolecular BiologyAdministration IntranasalPharmacologyPars compactaDopaminergic NeuronsGenetic TherapyCorpus Striatumnervous system diseases030104 developmental biologyMonoamine neurotransmitterGene Expression Regulationnervous systemsiRNAParkinson’s diseaseLocus coeruleus030217 neurology & neurosurgery
researchProduct

A new “sudden fright paradigm” to explore the role of (epi)genetic modulations of the DAT gene in fear-induced avoidance behavior

2020

Alterations in dopamine (DA) reuptake are involved in several psychiatric disorders whose symptoms can be investigated in knock out rats for the DA transporter (DAT-KO). Recent studies evidenced the role of epigenetic DAT modulation in depressive-like behavior. Accordingly, we used heterozygous (HET) rats born from both HET parents (termed MIX-HET), compared to HET rats born from WT-mother and KO-father (MAT-HET), implementing the role of maternal care on DAT modulation. We developed a "sudden fright" paradigm (based on dark-light test) to study reaction to fearful inputs in the DAT-KO, MAT-HET, MIX-HET, and WT groups. Rats could freely explore the whole 3-chambers apparatus; then, they wer…

0301 basic medicineanimal structuresEmotionsStimulus (physiology)Epigenesis GeneticReuptakechoice behavior03 medical and health sciencesBehavioral Neuroscience0302 clinical medicineDopamineDAT-KO ratAvoidance LearningGeneticsmedicineAnimalsFear conditioningEpigeneticsprefrontal cortex.Prefrontal cortexdopamine transporterDopamine transporterDopamine Plasma Membrane Transport ProteinsBehavior AnimalbiologyFearfear conditioningRatsDisease Models Animal030104 developmental biologyNeurologyAttention Deficit Disorder with Hyperactivitybiology.proteinSettore BIO/14 - Farmacologiaconditioned preferenceHistone deacetylaseNeuroscience030217 neurology & neurosurgerymedicine.drug
researchProduct

Development of the whisker-to-barrel cortex system.

2018

This review provides an overview on the development of the rodent whisker-to-barrel cortex system from late embryonic stage to the end of the first postnatal month. During this period the system shows a remarkable transition from a mostly genetic-molecular driven generation of crude connectivity, providing the template for activity-dependent structural and functional maturation and plasticity, to the manifestation of a complex behavioral repertoire including social interactions. Spontaneous and sensory-evoked activity is present in neonatal barrel cortex and control the generation of the cortical architecture. Half a century after its first description by Woolsey and van der Loos the whiske…

0301 basic medicineanimal structuresSensory processingBehavior AnimalGeneral NeuroscienceRepertoiremedicine.medical_treatmentCortical architectureEmbryonic StageSomatosensory CortexBarrel cortexBiology03 medical and health sciences030104 developmental biology0302 clinical medicinemedicine.anatomical_structureTouch PerceptionCortex (anatomy)VibrissaemedicineAnimalsNerve NetNeuroscience030217 neurology & neurosurgeryCurrent opinion in neurobiology
researchProduct

Barrel cortex: What is it good for?

2017

The rodent whisker system, with barrel cortex as its most prominent structure, has evolved into a powerful model system to study sensory processing. However, despite the vast amount of data collected on barrel cortex neural activity patterns, as well as its circuitry and plasticity, the precise behavioral and cognitive operations for which this structure is needed are still elusive. Proposed functions of barrel cortex include detection, discrimination, coordination of whisker movements during exploratory locomotion or active touch, and associative learning. Departing from a definition of what exactly constitutes a function and how the involvement of a brain area in a specific task can be es…

0301 basic medicineanimal structuresSensory processingmedicine.medical_treatmentBarrel (horology)Somatosensory system03 medical and health sciencesNeural activityMice0302 clinical medicineCognitionmedicineAnimalsBehavior AnimalGeneral NeuroscienceCognitionSomatosensory CortexBarrel cortexAssociative learningRats030104 developmental biologyVibrissaePsychologyNeuroscience030217 neurology & neurosurgeryCognitive loadNeuroscience
researchProduct

Characterization and isolation of immature neurons of the adult mouse piriform cortex

2015

Physiological studies indicate that the piriform or primary olfactory cortex of adult mammals exhibits a high degree of synaptic plasticity. Interestingly, a subpopulation of cells in the layer II of the adult piriform cortex expresses neurodevelopmental markers, such as the polysialylated form of neural cell adhesion molecule (PSA-NCAM) or doublecortin (DCX). This study analyzes the nature, origin, and potential function of these poorly understood cells in mice. As previously described in rats, most of the PSA-NCAM expressing cells in layer II could be morphologically classified as tangled cells and only a small proportion of larger cells could be considered semilunar-pyramidal transitiona…

0301 basic medicinebiologyNeurogenesisDoublecortinCell biology03 medical and health sciencesCellular and Molecular NeurosciencePrimary olfactory cortex030104 developmental biology0302 clinical medicinenervous systemDevelopmental NeuroscienceAntigenNeuroblastPiriform cortexSynaptic plasticitybiology.proteinNeural cell adhesion moleculeNeuroscience030217 neurology & neurosurgeryDevelopmental Neurobiology
researchProduct

MECP2 impairs neuronal structure by regulating KIBRA

2016

Using a Drosophila model of MECP2 gain-of-function, we identified memory associated KIBRA as a target of MECP2 in regulating dendritic growth. We found that expression of human MECP2 increased kibra expression in Drosophila, and targeted RNAi knockdown of kibra in identified neurons fully rescued dendritic defects as induced by MECP2 gain-of-function. Validation in mouse confirmed that Kibra is similarly regulated by Mecp2 in a mammalian system. We found that Mecp2 gain-of-function in cultured mouse cortical neurons caused dendritic impairments and increased Kibra levels. Accordingly, Mecp2 loss-of-function in vivo led to decreased Kibra levels in hippocampus, cortex, and cerebellum. Togeth…

0301 basic medicinecongenital hereditary and neonatal diseases and abnormalitiesCerebellumMethyl-CpG-Binding Protein 2Dendritic morphologyHippocampusDisease modelsHippocampusArticlelcsh:RC321-571MECP2Mice03 medical and health sciencesMemoryRNA interferencemental disordersmedicineAnimalsHumanslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryCerebral CortexNeuronsGene knockdownMECP2 duplication syndromebiologybiology.organism_classificationMECP2nervous system diseasesCortex (botany)Disease Models AnimalDrosophila melanogaster030104 developmental biologymedicine.anatomical_structureNeurologyCerebral cortexDrosophilaDrosophila melanogasterNeuroscienceNeurobiology of Disease
researchProduct