Search results for "cyclooxygenase"

showing 10 items of 235 documents

Prostaglandin E2 regulates inducible nitric oxide synthase in the murine macrophage cell line J774.

1995

We have evaluated the role of prostaglandin E2 (PGE2) in the synthesis of nitric oxide (NO) by the activation of the inducible form of nitric oxide synthase (NOS) in the murine macrophage cell line, J774, stimulated with different doses of lipopolysaccharide (LPS). The stimulation of the J774 line with suboptimal doses of LPS (0.1 microgram/mL) caused a production of endogenous PGE2 that was capable of stimulating NOS activity inducing an increase in the NO synthesis, as attested by the fact that cyclooxygenase enzyme inhibitor, indomethacin, significantly reduced NO secretion. On the contrary, a higher dose of LPS (1 microgram/mL) produced high levels of PGE2 that reduced the levels of NOS…

Lipopolysaccharidesmedicine.medical_specialtyLipopolysaccharideIndomethacinEndogenyNitric OxideBiochemistryDinoprostoneNitric oxideCell Linechemistry.chemical_compoundMiceEndocrinologyInternal medicinemedicineAnimalsProstaglandin E2biologyDose-Response Relationship DrugTumor Necrosis Factor-alphaMacrophagesMolecular biologyNitric oxide synthaseEnzyme ActivationEndocrinologychemistryEnzyme inhibitorbiology.proteinlipids (amino acids peptides and proteins)Tumor necrosis factor alphaCyclooxygenaseNitric Oxide Synthasemedicine.drugProstaglandins
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Anandamide inhibits IL-12p40 production by acting on the promoter repressor element GA-12: Possible involvement of the COX-2 metabolite prostamide E 2

2007

The eCB [endoCB (cannabinoid)] system is being considered as a novel therapeutic target for immune disorders. Cytokines of the IL-12 (interleukin-12) family have essential functions in cell-mediated immunity. In the present study, we have addressed the mechanisms of action of the eCB AEA (anandamide) on the regulation of IL-12p40 in activated microglia/macrophages. We demonstrated that AEA can inhibit the expression of p35, p19 and p40 subunits, which form the biologically-active cytokines IL-12 and IL-23 in microglia stimulated with LPS (lipopolysaccharide)/IFNγ (interferon γ). Additionally, we have provided evidence that AEA reduces the transcriptional activity of the IL-12p40 gene in LPS…

Lipopolysaccharidesmedicine.medical_specialtyPolyunsaturated Alkamidesmedicine.medical_treatmentMolecular Sequence DataRepressorArachidonic AcidsBiologyInterleukin-23BiochemistryDinoprostoneInterferon-gammaMicechemistry.chemical_compoundInternal medicinemedicineAnimalsEthanolamidePromoter Regions GeneticReceptors CannabinoidMolecular BiologyCells CulturedRegulation of gene expressionMice Inbred BALB CInterleukin-12 Subunit p40Cell BiologyAnandamideEndocannabinoid systemCell biologyProtein SubunitsEndocrinologyGene Expression RegulationchemistryCyclooxygenase 2lipids (amino acids peptides and proteins)MicrogliaCannabinoidSignal transductionEndocannabinoidsSignal TransductionProstaglandin E
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Hepatocellular Carcinoma: A Difficult Cancer to Treat

2021

Hepatocellular carcinoma (HCC) is a very peculiar cancer because it presents several molecular alterations linked to the activation of survival and antiapoptotic signal pathways that are protein in form and not easily targetable by even the newest targeted therapies. In addition, it is almost always a consequence of liver cirrhosis, a serious disease condition in which several drugs are often not tolerated. This is why the study of HCC was such a challenge for Professor Natale D'Alessandro, to whom this work is dedicated, during the latter years of his career. The aim of this review is to summarize studies on different molecules involved in the development, progression, and chemoresistance …

Liver CirrhosisCancer ResearchCarcinoma HepatocellularCirrhosisDruggabilityDrug resistanceNuclear factor κbDiseaseYY1medicineHumansInterleukin 6IL-6drug resistancebiologybusiness.industryLiver NeoplasmsCancerhepatocellular carcinomamedicine.diseasedigestive system diseasescyclooxygenasesHepatocellular carcinomaNF-?Bbiology.proteinCancer researchEpatocarcinoma ciclossigenasi NF-kB IL-6 farmacoresistenza YY1businessSignal TransductionCritical Reviews™ in Oncogenesis
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Potentiation of the antitumor effects of both selective cyclooxygenase-1 and cyclooxygenase-2 inhibitors in human hepatic cancer cells by inhibition …

2007

The molecular mechanisms behind the anti-neoplastic effects of non-steroidal anti-inflammatory drugs (NSAIDs) are not completely understood and cannot be explained by the inhibition of the cyclooxygenase (COX) enzymes COX-1 and COX-2 alone. We previously reported that both the selective COX-1 inhibitor SC-560 and the selective COX-2 inhibitor CAY10404 exhibit anti-tumor effects in human hepatoma cells. NSAID inhibitors have many COX-independent actions and, among others, the mitogen-activated protein kinase (MAPK) pathways are targets for NSAIDs. Here, we examined the role of MEK/ERK1/2 signaling in the anti-neoplastic effects of both selective COX-1 and COX-2 inhibitors in two human hepato…

MAPK/ERK pathwayCancer ResearchCarcinoma HepatocellularTime FactorsBlotting WesternApoptosisPharmacologyCOX-1 COX-2 NSAIDs MEK1/2 ERK1/2NitrilesButadienesTumor Cells CulturedHumansCyclooxygenase InhibitorsSulfonesEnzyme InhibitorsPhosphorylationProtein kinase ACell ProliferationPharmacologychemistry.chemical_classificationMitogen-Activated Protein Kinase 1Mitogen-Activated Protein Kinase KinasesMitogen-Activated Protein Kinase 3biologyDose-Response Relationship DrugLiver NeoplasmsCytochromes cLong-term potentiationDrug SynergismIsoxazolesFlow CytometryEnzymeOncologychemistryCyclooxygenase 2CaspasesCancer cellbiology.proteinCyclooxygenase 1Molecular MedicineMEK-ERK PathwayPyrazolesDrug Therapy CombinationCyclooxygenaseHepatoma cellCancer biologytherapy
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TLR4 elimination prevents synaptic and myelin alterations and long-term cognitive dysfunctions in adolescent mice with intermittent ethanol treatment.

2015

The adolescent brain undergoes important dynamic and plastic cell changes, including overproduction of axons and synapses, followed by rapid pruning along with ongoing axon myelination. These developmental changes make the adolescent brain particularly vulnerable to neurotoxic and behavioral effects of alcohol. Although the mechanisms of these effects are largely unknown, we demonstrated that ethanol by activating innate immune receptors toll-like receptor 4 (TLR4), induces neuroinflammation and brain damage in adult mice. The present study aims to evaluate whether intermittent ethanol treatment in adolescence promotes TLR4-dependent pro-inflammatory processes, leading to myelin and synapti…

MAPK/ERK pathwaySynaptic dysfunctionImmunologyNitric Oxide Synthase Type IIBrain damageHMGB1Behavioral NeuroscienceMyelinMiceCognitionmedicineAnimalsTLR4AxonHMGB1 ProteinReceptorNeuroinflammationMyelin SheathMice KnockoutMitogen-Activated Protein Kinase KinasesbiologyBinge ethanol treatmentEthanolEndocrine and Autonomic SystemsNF-kappa BCentral Nervous System DepressantsMyelin alterationsAdolescenceToll-Like Receptor 4medicine.anatomical_structureCyclooxygenase 2SynapsesTLR4biology.proteinmedicine.symptomPsychologyCognition DisordersNeuroscienceCognitive behaviorAlcohol-Related DisordersMyelin ProteinsSignal TransductionBrain, behavior, and immunity
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Novel combination of celecoxib and proteasome inhibitor MG132 provides synergistic antiproliferative and proapoptotic effects in human liver tumor ce…

2010

Molecular targeted therapy has shown promise as a treatment for advanced hepatocellular carcinoma (HCC). Celecoxib (Celebrex®) exhibits antitumor effects in human HCC cells, and its mechanism of action is mediated either by its ability to inhibit cyclooxygenase 2 (COX-2) or by a number of various other COX-2 independent effects. Proteasome inhibitors (PIs) can exert cell growth inhibitory and apoptotic effects in different tumor cell types, including HCC cells. The present study examined the interaction between celecoxib and the PI MG132 in two human liver tumor cell lines HepG2 and HA22T/VGH. Our data showed that each inhibitor reduced proliferation and induced apoptosis in a dose-dependen…

MG132TRB3Programmed cell deathLeupeptinsBlotting WesternApoptosisUPRPharmacologyCysteine Proteinase Inhibitorschemistry.chemical_compoundMG132medicineHumansViability assayHCCMolecular BiologyCell ProliferationSettore MED/12 - GastroenterologiaGene knockdownSulfonamidesbiologyCyclooxygenase 2 InhibitorsCell growthReverse Transcriptase Polymerase Chain ReactionDrug SynergismCell BiologyHep G2 CellsCOX-2ER stress responseFlow CytometryapoptosiproteasomechemistryApoptosisCelecoxibSettore BIO/14 - Farmacologiabiology.proteinProteasome inhibitorPyrazolesCyclooxygenaseDevelopmental Biologymedicine.drug
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Convergent synthesis and preliminary biological evaluations of the stilbenolignan (±)-aiphanol and various congeners

2003

Treatment of an equimolar mixture of stilbene 7 and cinnamyl alcohol 8 with silver carbonate in acetone–benzene afforded a ca. 2 : 1 : 2 : 1 mixture of the stilbenolignan (±)-aiphanol (1) and congeners 2–4 each of which show significant anti-angiogenic and COX-2 inhibitory properties.

Magnetic Resonance SpectroscopyConvergent synthesisAngiogenesis InhibitorsBiochemistryInhibitory Concentration 50chemistry.chemical_compoundStilbenesAnimalsOrganic chemistryBioassayCyclooxygenase InhibitorsPhysical and Theoretical ChemistryBenzeneAortaSilver carbonateCyclooxygenase 2 InhibitorsDose-Response Relationship DrugCinnamyl alcoholChemistryOrganic ChemistryMembrane ProteinsStereoisomerismRatsIsoenzymesEnzyme inhibitionCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesCyclooxygenase 1Org. Biomol. Chem.
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Impact of obesity and aging on crestal alveolar bone height in mice

2018

Obesity and aging are associated with periodontitis, which represents a chronic inflammatory disease of the tooth-supporting tissues, i.e. the periodontium. However, if both risk factors also have a negative impact on crestal alveolar bone in a clinically healthy periodontium, has yet to be elucidated and was analyzed in this in-vivo study. Eight C57BL/6 mice were fed a normal diet during the entire study. Half of these mice were sacrificed at week 19 (group 1: younger lean mice), whereas the other half of the animals were sacrificed at week 25 (group 2: older lean mice). In addition, four mice were fed a high-fat diet until their sacrifice at week 19 (group 3: younger obese mice). Mandible…

Male0301 basic medicineAgingmedicine.medical_specialtyNormal dietAlveolar Bone LossGingivaGene ExpressionMandibleDiet High-FatMice03 medical and health sciences0302 clinical medicineInternal medicineAlveolar ProcessMaxillamedicineAnimalsObesityNicotinamide PhosphoribosyltransferaseDental alveolusPeriodontitisAdiponectinbusiness.industryX-Ray Microtomography030206 dentistryGeneral MedicinePeriodontiummedicine.diseaseMolarMice Inbred C57BLCementoenamel junction030104 developmental biologyEndocrinologyCyclooxygenase 2MaxillaCytokinesCrestInflammation MediatorsAnatomybusinessDevelopmental BiologyAnnals of Anatomy - Anatomischer Anzeiger
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Influence of Dimerization of Apocynin on Its Effects in Experimental Colitis

2017

Apocynin has been widely used as an inhibitor of the nicotinamide adenine dinucleotide phosphate oxidase (NADPH-oxidase) system and shows promise as an anti-inflammatory drug. Diapocynin, the dimeric product generated by the oxidation of apocynin in the presence of myeloperoxidase (MPO), is supposed to be its active form. In this study, diapocynin has been chemically synthesized and its activity on several inflammatory mediators in LPS-stimulated RAW 264.7 macrophages and its anti-inflammatory effect on ulcerative colitis induced by dextran sodium sulfate (DSS) in mice analyzed. We found that diapocynin showed higher inhibitory activity than apocynin. The dimer reduced ROS production, TNF-α…

Male0301 basic medicineDimerInterleukin-1betaPharmacologyInhibitory postsynaptic potentialMice03 medical and health scienceschemistry.chemical_compoundIn vivoAnimalsHumansMice Inbred BALB COxidase testMolecular StructurebiologyInterleukin-6Tumor Necrosis Factor-alphaMacrophagesBiphenyl CompoundsNF-kappa BAcetophenonesGeneral ChemistryColitis030104 developmental biologychemistryDiapocyninBiochemistryCyclooxygenase 2MyeloperoxidaseApocyninbiology.proteinGeneral Agricultural and Biological SciencesDimerizationNicotinamide adenine dinucleotide phosphateJournal of Agricultural and Food Chemistry
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Impact of Donor Human Milk in the Preterm Very Low Birth Weight Gut Transcriptome Profile by Use of Exfoliated Intestinal Cells

2019

[Background] Own mother’s milk (OMM) is the optimal nutrition for preterm infants. However, pasteurized donor human milk (DHM) is a valid alternative. We explored the differences of the transcriptome in exfoliated epithelial intestinal cells (EEIC) of preterm infants receiving full feed with OMM or DHM.

Male0301 basic medicineDonor milkGene Expressionintestinal cellsmedicine.disease_causeTranscriptome0302 clinical medicinemother’s milkGene expressionInfant Very Low Birth Weightoxidative stressgeneticsProspective StudiesIntestinal Mucosa2. Zero hungerPrincipal Component AnalysisNutrition and DieteticsCaseinsIntestinal cells3. Good healthdonor milkGestationFemalemedicine.symptomPrematuritylcsh:Nutrition. Foods and food supplyInfant PrematureGestational Agelcsh:TX341-641InflammationBiologyArticleAndrology03 medical and health sciences030225 pediatricsMother’s milkGeneticsmedicineHumansGeneInflammationMilk HumanprematurityInfant NewbornNADPH OxidasesEpithelial CellsNeutrophil cytosolic factor 1Low birth weight030104 developmental biologyMilk BanksOxidative stressinflammationCyclooxygenase 1LactalbuminTranscriptomeOxidative stressFood ScienceNutrients
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