Search results for "degeneration"

showing 10 items of 601 documents

TOPORS, implicated in retinal degeneration, is a cilia-centrosomal protein.

2011

et al.

Retinal degenerationUbiquitin-Protein LigasesBiologymedicine.disease_causeRetinaCell Line03 medical and health scienceschemistry.chemical_compoundMiceNuclear proteins0302 clinical medicineIntraflagellar transportGeneticsmedicineBasal bodyAnimalsHumansPhotoreceptor CellsCiliaMolecular BiologyZebrafishGenetics (clinical)Cells CulturedZebrafish030304 developmental biologyCentrosome0303 health sciencesRetinaMutationUbiquitinCiliumRetinal DegenerationNuclear ProteinsRetinalTOPORS proteinGeneral MedicineArticlesmedicine.diseasebiology.organism_classification3. Good healthCell biologyNeoplasm ProteinsProtein Transportmedicine.anatomical_structurechemistryNeoplasm proteinssense organs030217 neurology & neurosurgeryHuman molecular genetics
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Vesicle transport and photoreceptor death: fishing for molecular links.

2013

Intracellular vesicle transport defects can induce retinal degeneration and photoreceptor cell death, but the molecular connections between these processes remains poorly understood. Reporting in Developmental Cell, Nishiwaki et al. (2013) suggest that a vesicle fusion cis-SNARE complex component translates vesicular transport defects into photoreceptor cell apoptosis.

Retinal degenerationVesicle fusionLipid bilayer fusionIntracellular vesicleApoptosisCell BiologyBiologymedicine.diseaseMembrane FusionGeneral Biochemistry Genetics and Molecular BiologyPhotoreceptor cellCell biologyVesicular transport proteinSoluble N-Ethylmaleimide-Sensitive Factor Attachment Proteinsmedicine.anatomical_structureProto-Oncogene Proteins c-bcl-2ApoptosismedicineRetinal Cone Photoreceptor CellsAnimalsMolecular BiologyDevelopmental BiologyDevelopmental cell
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Retinal Sensibility to Drugs in Normal Rats and Carriers of Inherited Retinal Degeneration

1972

Secondary retinitis pigmentosa of pseudo-retinitis pigmentosa can be brought about by certain diseases, especially exanthematic or viral ones.

Retinal degenerationcongenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtygenetic structuresmedicine.diagnostic_testbusiness.industryRetinalmedicine.diseaseeye diseaseschemistry.chemical_compoundchemistryOphthalmologyRetinitis pigmentosaotorhinolaryngologic diseasesMedicinesense organsbusinessElectroretinography
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Phosphodiesterase inhibition induces retinal degeneration, oxidative stress and inflammation in cone-enriched cultures of porcine retina.

2013

nherited retinal degenerations affecting both rod and cone photoreceptors constitute one of the causes 74 of incurable blindness in the developed world. Cyclic guanosine monophosphate (cGMP) is crucial in the 75 phototransduction and, mutations in genes related to its metabolism are responsible for different retinal 76 dystrophies. cGMP-degrading phosphodiesterase 6 (PDE6) mutations cause around 4e5% of the retinitis 77 pigmentosa, a rare form of retinal degeneration. The aim of this study was to evaluate whether phar- 78 macological PDE6 inhibition induced retinal degeneration in cone-enriched cultures of porcine retina 79 similar to that found in murine models. PDE6 inhibition was induced…

Retinal degenerationgenetic structuresPurinonesPhosphodiesterase InhibitorsSwineEstrès oxidatiuApoptosisBiologyRetinaCellular and Molecular Neurosciencechemistry.chemical_compoundOrgan Culture TechniquesRetinitis pigmentosamedicineIn Situ Nick-End LabelingAnimalsNeurociènciesCyclic GMPRetinaCalpainCaspase 3Retinal DegenerationPhosphodiesteraseRetinalmedicine.diseaseMolecular biologySensory SystemsOphthalmologyOxidative Stressmedicine.anatomical_structurechemistryBiochemistryRetinal Cone Photoreceptor CellsSwine Miniaturesense organsZaprinastRetinal DystrophiesRetinitis PigmentosaVisual phototransduction
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Retinol and retinyl esters in pigment epithelium of rats with inherited retinal degeneration

1976

A comparative study of the retinol and retinyl ester concentrations was performed in the retinal pigment epithelium of the normal and affected rats. Our findings indicate that in dystrophic rat retinol content increases, whereas the amount of retinyl esters is always lower than normal. An hypothesis can be made on the deficiency of enzymic activities which regulate retinol retinol and retinyl esters levels in the pigment epithelium.

Retinal degenerationgenetic structuresRetinyl estersCellular and Molecular NeurosciencePigmentchemistry.chemical_compoundmedicineAnimalsPigment Epithelium of EyeVitamin AMolecular BiologyPharmacologyRetinal pigment epitheliumChemistryRetinal DegenerationAge FactorsRetinolCell Biologymedicine.diseaseEpitheliumRatsmedicine.anatomical_structureBiochemistryvisual_artvisual_art.visual_art_mediumMolecular MedicineExperientia
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Efficient gene delivery to photoreceptors using AAV2/rh10 and rescue of the Rho–/– mouse

2015

As gene therapies for various forms of retinal degeneration progress toward human clinical trial, it will be essential to have a repertoire of safe and efficient vectors for gene delivery to the target cells. Recombinant adeno-associated virus (AAV) serotype 2/2 has been shown to be well tolerated in the human retina and has provided efficacy in human patients for some inherited retinal degenerations. In this study, the AAV2/8 and AAV2/rh10 serotypes have been compared as a means of gene delivery to mammalian photoreceptor cells using a photoreceptor specific promoter for transgene expression. Both AAV2/8 and AAV2/rh10 provided rescue of the retinal degeneration present in the rhodopsin kno…

Retinal degenerationlcsh:QH426-470TransgeneGenetic enhancementvirusesGene deliveryBiologyBioinformaticsArticlechemistry.chemical_compoundGeneticsmedicinelcsh:QH573-671Molecular BiologyRetinalcsh:CytologyRetinalmedicine.diseaseCell biologylcsh:Geneticsmedicine.anatomical_structurechemistryRhodopsinKnockout mousebiology.proteinMolecular MedicineCorrigendumMolecular Therapy. Methods & Clinical Development
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Combination of Resveratrol with omega-3 fatty acids synergize to counteract VEGF-R pathway in sick retinal pigment epithelium cells mimicking AMD

2015

Purpose The aim of this work is to assess whether RSV (Resveratrol) can act synergistically with omega-3 fatty acids to modulate VEGF (Vascular Endothelial Growth Factor) signaling pathway in order to identify a new and more effective therapy for the treatment of AMD (Age-Related Macular Degeneration). Methods In this study, undifferentiated and differentiated human retinal pigment epithelial cells (ARPE-19) were used. The cells were treated with an omega-3/RSV preparation (Resvega®), or a RSV-free formulation or RSV alone for 24 h. The expression of key proteins in VEGF signaling pathway was evaluated by Western Blotting. Results We observed that the combination omega-3/RSV preparation (Re…

Retinal pigment epitheliumRetinalGeneral MedicineBiologyMacular degenerationResveratrolmedicine.diseaseVascular endothelial growth factorBlotOphthalmologychemistry.chemical_compoundmedicine.anatomical_structurechemistryVEGF Signaling PathwayImmunologymedicineCancer researchSignal transductionActa Ophthalmologica
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Proteostasis Deregulation in Neurodegeneration and Its Link with Stress Granules: Focus on the Scaffold and Ribosomal Protein RACK1.

2022

The role of protein misfolding, deposition, and clearance has been the dominant topic in the last decades of investigation in the field of neurodegeneration. The impairment of protein synthesis, along with RNA metabolism and RNA granules, however, are significantly emerging as novel potential targets for the comprehension of the molecular events leading to neuronal deficits. Indeed, defects in ribosome activity, ribosome stalling, and PQC—all ribosome-related processes required for proteostasis regulation—can contribute to triggering stress conditions and promoting the formation of stress granules (SGs) that could evolve in the formation of pathological granules, usually occurring during ne…

Ribosomal ProteinsRACK1neurodegenerationtranslationGeneral MedicineCytoplasmic GranulesReceptors for Activated C KinaseStress GranulesNeoplasm ProteinsSettore BIO/10 - BiochimicaFrontotemporal DementiaProteostasisRNAHumansCells
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Prevalence of early and late age-related macular degeneration in India: the INDEYE study.

2010

Purpose.: To estimate the prevalence of early and late age-related macular degeneration (AMD) in India. Methods.: Of 7518 people aged 60 years and older identified from randomly sampled villages in North and South India, 5853 (78%) attended an eye examination including fundus photography. Fundus images were graded according to the Wisconsin Age-Related Maculopathy Grading System. Results.: Fundus images were ungradable in 1587 people, mainly because of cataract. People 80 years of age and older were less likely to attend the eye examination and more likely to have ungradable images. For ages 60 to 79 years, the percent prevalence (95% confidence interval [CI]) were late AMD 1.2 (0.8–1.5); a…

Rural Populationmedicine.medical_specialtyUrban Populationgenetic structuresVisual AcuityPrevalenceIndiaDiagnostic Techniques OphthalmologicalDrusenFundus (eye)Macular DegenerationAge DistributionOphthalmologyEpidemiologyPrevalencemedicineHumansSex DistributionAgedAged 80 and overmedicine.diagnostic_testbusiness.industryFundus photographyArticlesMiddle AgedMacular degenerationmedicine.diseaseeye diseasesEye examinationMaculopathysense organsbusiness
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Protective, antioxidant and antiproliferative activity of grapefruit integropectin on sh-sy5y cells

2021

Tested in vitro on SH-SY5Y neuroblastoma cells, grapefruit IntegroPectin is a powerful protective, antioxidant and antiproliferative agent. The strong antioxidant properties of this new citrus pectin, and its ability to preserve mitochondrial membrane potential and morphology, severely impaired in neurodegenerative disorders, make it an attractive therapeutic and preventive agent for the treatment of oxidative stress-associated brain disorders. Similarly, the ability of this pectic polymer rich in RG-I regions, as well as in naringin, linalool, linalool oxide and limonene adsorbed at the outer surface, to inhibit cell proliferation or even kill, at high doses, neoplastic cells may have open…

SH-SY5YAntioxidantCell SurvivalQH301-705.5medicine.medical_treatmentMitochondrionPharmacologymedicine.disease_causeanticancerNeuroprotectionSettore BIO/09 - FisiologiaAntioxidantsArticleCatalysisInorganic ChemistryNeuroblastomachemistry.chemical_compoundX-Ray DiffractionCell Line Tumorhydrodynamic cavitationmedicineHumansoxidative stressPhysical and Theoretical ChemistryBiology (General)neurological diseaseMolecular BiologyNaringinQD1-999SpectroscopyCell ProliferationantitumorMembrane Potential MitochondrialpectinCell growthChemistryOrganic ChemistryneurodegenerationGeneral MedicinephytochemicalsIn vitroComputer Science ApplicationsmitochondriaChemistryNeuroprotective AgentsPectinscell cycleOxidative stressCitrus paradisi
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