Search results for "developmental disorders"

showing 10 items of 87 documents

Urinaryp-cresol is elevated in young French children with autism spectrum disorder: a replication study

2014

The aromatic compound p-cresol (4-methylphenol) has been found elevated in the urines of Italian autistic children up to 8 years of age. The present study aims at replicating these initial findings in an ethnically distinct sample and at extending them by measuring also the three components of urinary p-cresol, namely p-cresylsulfate, p-cresylglucuronate and free p-cresol. Total urinary p-cresol, p-cresylsulfate and p-cresylglucuronate were significantly elevated in 33 French autism spectrum disorder (ASD) cases compared with 33 sex- and age-matched controls (p < 0.05). This increase was limited to ASD children aged ≤8 years (p < 0.01), and not older (p = 0.17). Urinary levels of p-cresol a…

Pervasive developmental disordersMalePathologyHealth Toxicology and MutagenesisClinical BiochemistryBiochemistryClinical biochemistryCresolsorganic contaminants; neurotoxicity; Gut flora; pervasive developmental disorders; p-cresylsulfateUrinary levelsneurotoxicityChildSettore BIO/12P-cresylsulfateSettore MED/39 - Neuropsichiatria InfantileGut flora neurotoxicity organic contaminants p-cresylsulfate pervasive developmental disordersHealthAutism spectrum disorderChild Preschoolp-cresylsulfateBiomarker (medicine)FemaleFrancemedicine.medical_specialtyChild Development DisordersAdolescentUrinary systemGlucuronatesSulfuric Acid EstersOrganic contaminantsGut flora; Neurotoxicity; Organic contaminants; p-cresylsulfate; Pervasive developmental disorders; Adolescent; Case-Control Studies; Child; Child Development Disorders; Pervasive; Child; Preschool; Cresols; Female; France; Glucuronates; Humans; Male; Sulfuric Acid Esters; Biochemistry; Clinical Biochemistry; Health; Toxicology and MutagenesisInternal medicineparasitic diseasesNeurotoxicitymedicineHumansToxicology and MutagenesisPreschoolSettore BIO/10 - BIOCHIMICAPervasiveGut florabusiness.industryCase-control studypervasive developmental disordersmedicine.diseaseChild Development Disorders PervasiveCase-Control StudiesAutismorganic contaminantsGut flora; Neurotoxicity; Organic contaminants; p-cresylsulfate; Pervasive developmental disorders; Adolescent; Case-Control Studies; Child; Child Development Disorders Pervasive; Child Preschool; Cresols; Female; France; Glucuronates; Humans; Male; Sulfuric Acid Esters; Biochemistry; Clinical Biochemistry; Health Toxicology and MutagenesisbusinessBiomarkers
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Genome-wide variant calling in reanalysis of exome sequencing data uncovered a pathogenic TUBB3 variant.

2021

Almost half of all individuals affected by intellectual disability (ID) remain undiagnosed. In the Solve-RD project, exome sequencing (ES) datasets from unresolved individuals with (syndromic) ID (n = 1,472 probands) are systematically reanalyzed, starting from raw sequencing files, followed by genome-wide variant calling and new data interpretation. This strategy led to the identification of a disease-causing de novo missense variant in TUBB3 in a girl with severe developmental delay, secondary microcephaly, brain imaging abnormalities, high hypermetropia, strabismus and short stature. Interestingly, the TUBB3 variant could only be identified through reanalysis of ES data using a genome-wi…

ProbandExome sequencingAdolescentDevelopmental Disabilitieslnfectious Diseases and Global Health Radboud Institute for Molecular Life Sciences [Radboudumc 4]Mutation MissenseComputational biologyBiologyGenomeExonAll institutes and research themes of the Radboud University Medical CenterTubulinIntellectual DisabilitySolve-RDExome SequencingGeneticsCoding regionMissense mutationHumansTUBB3GeneGenetics (clinical)Exome sequencingSequence (medicine)Neurodevelopmental disorders Donders Center for Medical Neuroscience [Radboudumc 7]ERN ITHACABrainMetabolic Disorders Radboud Institute for Molecular Life Sciences [Radboudumc 6]General MedicineGenome-wide variant callingStrabismusFaceMicrocephalyFemaleEuropean journal of medical genetics
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Excess of de novo variants in genes involved in chromatin remodelling in patients with marfanoid habitus and intellectual disability.

2020

PurposeMarfanoid habitus (MH) combined with intellectual disability (ID) (MHID) is a clinically and genetically heterogeneous presentation. The combination of array CGH and targeted sequencing of genes responsible for Marfan or Lujan–Fryns syndrome explain no more than 20% of subjects.MethodsTo further decipher the genetic basis of MHID, we performed exome sequencing on a combination of trio-based (33 subjects) or single probands (31 subjects), of which 61 were sporadic.ResultsWe identified eight genes with de novo variants (DNVs) in at least two unrelated individuals (ARID1B, ATP1A1, DLG4, EHMT1, NFIX, NSD1, NUP205 and ZEB2). Using simulation models, we showed that five genes (DLG4, NFIX, …

ProbandMale[SDV]Life Sciences [q-bio]intellectual deficiencyMESH: NFI Transcription Factorschromatin remodelingMarfan SyndromeCraniofacial AbnormalitiesMESH: ChildIntellectual disabilityMESH: Craniofacial AbnormalitiesMESH: Mental Retardation X-LinkedExomeChildde novo variantsGenetics (clinical)Exome sequencingGeneticsMESH: ExomeMESH: Middle AgedbiologyMESH: Genetic Predisposition to DiseaseMiddle AgedNFIXMESH: Young AdultFemaleAdultMESH: MutationAdolescentChromatin remodelingMESH: Intellectual DisabilityMESH: Marfan SyndromeEHMT1Young AdultMESH: Whole Exome SequencingIntellectual DisabilityExome SequencingGeneticsmedicineHumansGenetic Predisposition to Diseasemarfanoid habitusGeneMESH: Neurodevelopmental DisordersMESH: AdolescentMESH: HumansGenetic heterogeneityMESH: Chromatin Assembly and DisassemblyMESH: Histone-Lysine N-MethyltransferaseMESH: AdultHistone-Lysine N-Methyltransferasemedicine.diseaseChromatin Assembly and DisassemblyMESH: MaleNFI Transcription FactorsNeurodevelopmental DisordersMutationbiology.proteinMental Retardation X-LinkedMESH: FemaleJournal of medical genetics
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Subjective Experience of Episodic Memory and Metacognition: A Neurodevelopmental Approach

2013

Episodic retrieval is characterized by the subjective experience of remembering. This experience enables the co-ordination of memory retrieval processes and can be acted on metacognitively. In successful retrieval, the feeling of remembering may be accompanied by recall of important contextual information. On the other hand, when people fail (or struggle) to retrieve information, other feelings, thoughts and information may come to mind. In this review, we examine the subjective and metacognitive basis of episodic memory function from a neurodevelopmental perspective, looking at recollection paradigms (such as source memory, and the report of recollective experience) and metacognitive parad…

Reconstructive memoryRecallAutobiographical memoryrecollectionneurodevelopmental disordersCognitive NeuroscienceBrain DevelopmentmetamemoryReview Articleepisodic memorylcsh:RC321-571Behavioral NeuroscienceNeuropsychology and Physiological PsychologyRetrospective memoryProspective memoryExplicit memorySemantic memoryPsychologyEpisodic memorylcsh:Neurosciences. Biological psychiatry. NeuropsychiatryCognitive psychologyNeuroscienceFrontiers in Behavioral Neuroscience
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Risk of mortality among children, adolescents, and adults with autism spectrum disorder or attention deficit hyperactivity disorder and their first-d…

2017

BACKGROUND: Autism spectrum disorder (ASD) and attention deficit hyperactivity disorder (ADHD) are childhood onset neurodevelopmental disorders that may persist into adulthood. ASD and ADHD tend to run in families and may have a significant negative impact on the health and longevity of those with the disorder and their relatives. The aim of this study will be to analyze the risk of mortality among children, adolescents, and adults with ASD or ADHD and their first-degree relatives. METHODS/DESIGN: We will conduct a systematic review and meta-analysis of observational studies. Searches of PubMed/MEDLINE, EMBASE, PsycINFO, SCOPUS, and ISI Web of Science will be used to identify epidemiologica…

Research designMaleEpidemiologyAutism Spectrum DisorderMedicine (miscellaneous)lcsh:MedicineTrastorns de l'atencióAdolescents0302 clinical medicineCause of DeathRisk of mortalityProtocol030212 general & internal medicineChildChild developmentNeurodevelopmental disordersAutism spectrum disorders3. Good healthAutism spectrum disorderResearch DesignMeta-analysisFemaleInfantsCohort studyAdultRiskmedicine.medical_specialtyAdolescentAttention deficit hyperactivity disorder03 medical and health sciencesBibliometriamental disordersmedicineMortalitatAttention deficit hyperactivity disorderAdultsHumansFamilyFirst-degree relativesMortalityPsychiatrybusiness.industrylcsh:Rmedicine.diseaseMeta-analysisAttention Deficit Disorder with HyperactivitySystematic reviewObservational studyAutismebusiness030217 neurology & neurosurgerySystematic Reviews as Topic
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Commonalities and distinctions between two neurodevelopmental disorder subtypes associated with SCN2A and SCN8A variants and literature review.

2022

This study was aimed to analyze the commonalities and distinctions of voltage-gated sodium channels, Nav1.2, Nav1.6, in neurodevelopmental disorders. An observational study was performed including two patients with neurodevelopmental disorders. The demographic, electroclinical, genetic, and neuropsychological characteristics were analyzed and compared with each other and then with the subjects carrying the same genetic variants reported in the literature. The clinical features of one of them argued for autism spectrum disorder and developmental delay, the other for intellectual disability, diagnoses confirmed by the neuropsychological assessment. The first patient was a carrier of SCN2A (p.…

SCN8AEpilepsyNAV1.2 Voltage-Gated Sodium ChannelAutism Spectrum Disorderautism spectrum disordersObservational Studies as TopicPhenotypeNAV1.6 Voltage-Gated Sodium ChannelNeurodevelopmental DisordersIntellectual DisabilityHumansEEGwhole-exome sequencingSCN2AMolecular geneticsgenomic medicine
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Noli Me Tangere: Social Touch, Tactile Defensiveness, and Communication in Neurodevelopmental Disorders

2019

Tactile defensiveness is a common feature in neurodevelopmental disorders (NDDs). Since the first studies, tactile defensiveness has been described as the result of an abnormal response to sensory stimulation. Moreover, it has been studied how the tactile system is closely linked to socio-communicative development and how the interoceptive sensory system supports both a discriminating touch and an affective touch. Therefore, several neurophysiological studies have been conducted to investigate the neurobiological basis of the development and functioning of the tactile system for a better understanding of the tactile defensiveness behavior and the social touch of NDDs. Given the lack of rece…

Sensory stimulation therapySettore M-PSI/02 - Psicobiologia E Psicologia FisiologicaInformationSystems_INFORMATIONINTERFACESANDPRESENTATION(e.g.HCI)General Neuroscienceautism spectrum disorders05 social sciencesneurodevelopmental disorders (NDDs)Sensory systemautism spectrum disordertactile defensivene050105 experimental psychologySettore MED/39 - Neuropsichiatria Infantile03 medical and health sciences0302 clinical medicinesocial touchPerspectiveFocusing attention0501 psychology and cognitive sciencesSocial touchtactile defensivenesssensory reactivityPsychology030217 neurology & neurosurgeryCognitive psychology
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Clinical Utility of a Unique Genome-Wide DNA Methylation Signature for KMT2A-Related Syndrome

2022

Wiedemann–Steiner syndrome (WDSTS) is a Mendelian syndromic intellectual disability (ID) condition associated with hypertrichosis cubiti, short stature, and characteristic facies caused by pathogenic variants in the KMT2A gene. Clinical features can be inconclusive in mild and unusual WDSTS presentations with variable ID (mild to severe), facies (typical or not) and other associated malformations (bone, cerebral, renal, cardiac and ophthalmological anomalies). Interpretation and classification of rare KMT2A variants can be challenging. A genome-wide DNA methylation episignature for KMT2A-related syndrome could allow functional classification of variants and provide insights into the pathoph…

Wiedemann–Steiner syndromeQH301-705.5Intellectual disability[SDV.BC]Life Sciences [q-bio]/Cellular BiologyCatalysisInorganic ChemistryKMT2A geneNeurodevelopmental disorderGrowth DisorderAbnormalities Multiple[SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Biology (General)Physical and Theoretical ChemistryEpisignatureQD1-999[SDV.BC] Life Sciences [q-bio]/Cellular BiologyMolecular BiologySpectroscopyDNA methylationOrganic ChemistryNeurodevelopmental disordersCraniofacial AbnormalitieEpigeneticHypertrichosiGeneral MedicineFacieComputer Science Applications<i>KMT2A</i> geneChemistryepigenetics; DNA methylation; episignature; Wiedemann–Steiner syndrome; <i>KMT2A</i> gene; intellectual disability; neurodevelopmental disordersPhenotype[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]EpigeneticsHuman
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Explicit Oral Narrative Intervention for Students with Williams Syndrome

2018

Narrative skills play a crucial role in organizing experience, facilitating social interaction and building academic discourse and literacy. They are at the interface of cognitive, social, and linguistic abilities related to school engagement. Despite their relative strengths in social and grammatical skills, students with Williams syndrome (WS) do not show parallel cognitive and pragmatic performance in narrative generation tasks. The aim of the present study was to assess retelling of a TV cartoon tale and the effect of an individualized explicit instruction of the narrative structure. Participants included eight students with WS who attended different special education levels. Narratives…

Williams syndromelcsh:BF1-990Lexical diversityContext (language use)Special educationoral narrativeat risk of school failure03 medical and health sciences0302 clinical medicinePsychology0501 psychology and cognitive sciencesNarrativeeffective interventionGeneral PsychologyOriginal Researchpragmatic impairmentneurodevelopmental disorders05 social sciencesCHILDESCohesion (linguistics)Language developmentlcsh:Psychologynarrative interventionPsychologylanguage development030217 neurology & neurosurgeryDiscourse marker050104 developmental & child psychologyCognitive psychologyFrontiers in Psychology
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Genetic and phenotypic dissection of 1q43q44 microdeletion syndrome and neurodevelopmental phenotypes associated with mutations in ZBTB18 and HNRNPU

2017

Subtelomeric 1q43q44 microdeletions cause a syndrome associating intellectual disability, microcephaly, seizures and anomalies of the corpus callosum. Despite several previous studies assessing genotype-phenotype correlations, the contribution of genes located in this region to the specific features of this syndrome remains uncertain. Among those, three genes, AKT3, HNRNPU and ZBTB18 are highly expressed in the brain and point mutations in these genes have been recently identified in children with neurodevelopmental phenotypes. In this study, we report the clinical and molecular data from 17 patients with 1q43q44 microdeletions, four with ZBTB18 mutations and seven with HNRNPU mutations, an…

[SDV.GEN]Life Sciences [q-bio]/GeneticsRepressor Proteins/geneticsddc:618Neurodevelopmental Disorders/geneticsHeterogeneous-Nuclear Ribonucleoproteins/geneticsHeterogeneous-Nuclear RibonucleoproteinsChromosomesRepressor ProteinsPhenotypeChromosomes Human Pair 1Neurodevelopmental DisordersMutationGeneticsPair 1HumansGenetics(clinical)Chromosome Deletion[ SDV.GEN ] Life Sciences [q-bio]/GeneticsOriginal InvestigationHuman
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