Search results for "digestive"

showing 10 items of 2631 documents

The Unfolded Protein Response Plays a Predominant Homeostatic Role in Response to Mitochondrial Stress in Pancreatic Stellate Cells.

2016

Activated pancreatic stellate cells (PaSC) are key participants in the stroma of pancreatic cancer, secreting extracellular matrix proteins and inflammatory mediators. Tumors are poorly vascularized, creating metabolic stress conditions in cancer and stromal cells that necessitate adaptive homeostatic cellular programs. Activation of autophagy and the endoplasmic reticulum unfolded protein response (UPR) have been described in hepatic stellate cells, but the role of these processes in PaSC responses to metabolic stress is unknown. We reported that the PI3K/mTOR pathway, which AMPK can regulate through multiple inputs, modulates PaSC activation and fibrogenic potential. Here, using primary a…

0301 basic medicinelcsh:MedicineApoptosisMitochondrionAMP-Activated Protein KinasesEndoplasmic ReticulumBiochemistrychemistry.chemical_compoundMiceeIF-2 KinasePhosphatidylinositol 3-Kinases0302 clinical medicineFluorescence MicroscopyCell SignalingTumor Microenvironment2.1 Biological and endogenous factorsSmall interfering RNAsAetiologylcsh:ScienceEnergy-Producing OrganellesCancerMice KnockoutMicroscopyMultidisciplinarySecretory PathwayCell DeathTOR Serine-Threonine KinasesLight MicroscopySignaling CascadesCell biologyMitochondriaNeoplasm ProteinsUp-RegulationNucleic acidsCell Processes030220 oncology & carcinogenesisCellular Structures and OrganellesResearch ArticleSignal TransductionProgrammed cell deathCell PhysiologyGeneral Science & TechnologyAutophagic Cell DeathKnockoutBiologyBioenergeticsResearch and Analysis MethodsStress Signaling Cascade03 medical and health sciencesGeneticsAutophagyAnimalsNon-coding RNAPancreasPI3K/AKT/mTOR pathwaylcsh:RAutophagyAMPKBiology and Life SciencesCell BiologyCell MetabolismGene regulationPancreatic NeoplasmsEnzyme Activation030104 developmental biologychemistryHepatic stellate cellUnfolded protein responseUnfolded Protein ResponseRNAlcsh:QGene expressionInterleukin-4Digestive DiseasesRottlerinTranscription Factor CHOP
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Commensal Bacterium Rothia aeria Degrades and Detoxifies Gluten via a Highly Effective Subtilisin Enzyme

2020

Celiac disease is characterized by a chronic immune-mediated inflammation of the small intestine, triggered by gluten contained in wheat, barley, and rye. Rothia aeria, a gram-positive natural colonizer of the oral cavity and the upper digestive tract is able to degrade and detoxify gluten in vitro. The objective of this study was to assess gluten-degrading activity of live and dead R. aeria bacteria in vitro, and to isolate the R. aeria gluten-degrading enzyme. Methods: After an overnight fast, Balb/c mouse were fed a 1 g pellet of standard chow containing 50% wheat (and 4% gliadin) with or without 1.6 &times

0301 basic medicinelcsh:TX341-641detoxifymedicine.disease_causedigestive systemBacterial cell structure<i>Rothia</i>Microbiology03 medical and health sciences0302 clinical medicinemedicineZymographydegradationchemistry.chemical_classificationepitopeNutrition and DieteticsbiologyRothia aeria<i>Bacillus</i>food and beveragesnutritional and metabolic diseasesbiology.organism_classificationGlutenneutralizecuredigestive system diseasesEnzyme assay030104 developmental biologychemistryglutencommensalbiology.proteinsubtilisin030211 gastroenterology & hepatologyDigestionGliadinlcsh:Nutrition. Foods and food supplyceliac diseaseBacteriaFood ScienceNutrients
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Implications of SARS-COV-2 infection in the diagnosis and management of the pediatric gastrointestinal disease

2021

AbstractGastrointestinal diseases such as celiac disease, functional gastrointestinal disorders (FGIDs), inflammatory bowel disease (IBDs) and acute or chronic diarrhea are quite frequent in the pediatric population. The approach, the diagnosis and management can be changed in the 2019 coronavirus disease (COVID-19) pandemic era. This review has focused on: i) the current understanding of digestive involvement in severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infected children and adolescents and the clinical implications of COVID-19 for pediatric gastroenterologists, ii) the impact of COVID-19 on the clinical approach to patients with pre-existing or onset diseases, including…

0301 basic medicinemedicine.medical_specialtyAdolescentGastrointestinal Diseasesmedicine.medical_treatmentFunctional gastrointestinal disordersACE2ReviewDiseaseChronic liver diseaseInflammatory bowel diseaseDigestive endoscopyPediatricsGastrointestinal symptomsInflammatory bowel disease03 medical and health sciencesLiver diseaseFunctional gastrointestinal disorder0302 clinical medicineACE2; COVID-19; Digestive endoscopy; Functional gastrointestinal disorders; Gastrointestinal symptoms; Inflammatory bowel disease; Liver disease; Pediatrics; Adolescent; COVID-19; Child; Gastrointestinal Diseases; Humans; Infection Control; SARS-CoV-2Gastrointestinal symptomPandemicmedicineHumansRisk factorIntensive care medicineChildInfection Controlbusiness.industrySARS-CoV-2lcsh:RJ1-570COVID-19lcsh:PediatricsImmunosuppressionmedicine.disease030104 developmental biologyGastrointestinal disease030211 gastroenterology & hepatologybusinessLiver disease
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Neutrophil to Lymphocyte Ratio as a Predictor of Poor Prognosis in Metastatic Pancreatic Cancer Patients Treated with Nab-Paclitaxel plus Gemcitabine…

2018

Background. High neutrophil to lymphocyte ratio (NLR) has shown to be a predictor of poor outcomes in various malignancies, including pancreatic cancer. Methods. We assessed 70 consecutive pts with histologically confirmed mPC who received chemotherapy with nab-paclitaxel/gemcitabine at two different European oncologic centers between January 2012 and November 2015. Variables assessed for prognostic correlations included age ≥ 66, sex, Karnofsky PS score, primary tumor site, baseline CA19.9 level ≥ 59xULN, 12-week decrease of the CA19.9 level ≥ 50% from baseline, basal bilirubin level, baseline NLR, biliary stent implantation, and liver metastasis. Survival analyses were generated according…

0301 basic medicinemedicine.medical_specialtyArticle Subjectmedicine.medical_treatmentGastroenterologyMetastasis03 medical and health sciences0302 clinical medicineInternal medicinePancreatic cancermedicineNeutrophil to lymphocyte ratiolcsh:RC799-869ChemotherapyHepatologyProportional hazards modelbusiness.industryGastroenterologymedicine.diseasePrimary tumorGemcitabine030104 developmental biology030220 oncology & carcinogenesisPropensity score matchinglcsh:Diseases of the digestive system. Gastroenterologybusinessmedicine.drugResearch ArticleGastroenterology Research and Practice
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2021

The mechanisms underlying the transport of leptin into the brain are still largely unclear. While the leptin receptor has been implicated in the transport process, recent evidence has suggested an additional role of LRP2 (megalin). To evaluate the function of LRP2 for leptin transport across the blood-brain barrier (BBB), we developed a novel leptin-luciferase fusion protein (pLG), which stimulated leptin signaling and was transported in an in vitro BBB model based on porcine endothelial cells. The LRP inhibitor RAP did not affect leptin transport, arguing against a role of LRP2. In line with this, the selective deletion of LRP2 in brain endothelial cells and epithelial cells of the choroid…

0301 basic medicinemedicine.medical_specialtyBiologyBlood–brain barrierCatalysisInorganic Chemistry03 medical and health sciences0302 clinical medicineInternal medicinemedicinePhysical and Theoretical ChemistryMolecular BiologySpectroscopyLeptin receptorLeptindigestive oral and skin physiologyOrganic ChemistryGeneral MedicineLRP2Fusion proteinIn vitroComputer Science Applications030104 developmental biologymedicine.anatomical_structureEndocrinologyChoroid plexushormones hormone substitutes and hormone antagonists030217 neurology & neurosurgeryFunction (biology)International Journal of Molecular Sciences
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Changes in the Peripheral Endocannabinoid System as a Risk Factor for the Development of Eating Disorders

2017

BACKGROUND AND OBJECTIVE Eating Disorder (ED) is characterized by persistently and severely disturbed eating behaviours. They arise from a combination of long-standing behavioural, emotional, psychological, interpersonal, and social factors and result in insufficient nutrient ingestion and/or adsorption. The three main EDs are: anorexia nervosa, bulimia nervosa, and binge eating disorder. We review the role of peripheral endocannabinoids in eating behaviour. DISCUSSION The neuronal pathways involved in feeding behaviours are closely related to catecholaminergic, serotoninergic and peptidergic systems. Accordingly, feeding is promoted by serotonin, dopamine, and prostaglandin and inhibited b…

0301 basic medicinemedicine.medical_specialtyCannabinoid receptorEndocrinology Diabetes and Metabolismmedicine.medical_treatmentNutritional StatusFeeding and Eating Disorders03 medical and health sciencesIslets of LangerhansReceptor Cannabinoid CB1Binge-eating disorderInternal medicinemedicineImmunology and AllergyAnimalsHumansOpioid peptideMuscle Skeletal030109 nutrition & dieteticsBulimia nervosabusiness.industrydigestive oral and skin physiologyBody WeightBrainFeeding Behaviormedicine.diseaseEndocannabinoid systemEating disordersEndocrinologyAdipose TissueLiverAnorexia nervosa (differential diagnoses)CannabinoidbusinessEnergy MetabolismEndocannabinoidsSignal Transduction
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The consumption of snacks and soft drinks between meals may contribute to the development and to persistence of gastro-esophageal reflux disease

2019

Abstract The hypothesis The habit of snacking and drinking soft beverages between breakfast, lunch and dinner, which is very widespread in the western world, could be a primum movens, thereby contributing to the development and subsequent persistence of gastroesophageal reflux disease (GERD). What does the proposed hypothesis based on? The high prevalence of GERD suggests that it is very probably caused by factors, which are intrinsic and widespread in a western lifestyle. Ingesting snacks or imbibing soft drinks between breakfast, lunch and dinner causes additional gastric acid secretion, acid pocket formation, and additional transient lower esophageal sphincter relaxations (TLESRs) with a…

0301 basic medicinemedicine.medical_specialtyCarbonated BeveragesOverweightGastroenterologyEsophageal Sphincter LowerGastric AcidHiatal hernia03 medical and health sciencesEsophagus0302 clinical medicineRisk FactorsInternal medicinePrevalencemedicineHumansObesityEsophagusLife StyleGastro-esophageal Reflux GERD Lifestyle modifications Transient Lower Esophageal Sphincter Relaxation TLESR Snacking and Soft drinks consumption Hiatal Hernia Overweight ObesitySnackingbusiness.industrydigestive oral and skin physiologyRefluxfood and beveragesFeeding BehaviorGeneral MedicineModels TheoreticalOverweightmedicine.diseaseObesitydigestive system diseasesDietHernia Hiatal030104 developmental biologymedicine.anatomical_structureGastroesophageal RefluxGERDGastric acidSnacksmedicine.symptombusiness030217 neurology & neurosurgery
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Deconvolution of the cellular origin in hepatocellular carcinoma: Hepatocytes take the center stage.

2016

The expression of biliary/progenitor markers by hepatocellular carcinoma (HCC) is often associated with poor prognosis and stem cell-like behaviors of tumor cells. Hepatocellular adenomas (HCA) also often express biliary/progenitor markers and frequently act as precursor lesions for HCC. However, the cell of origin of HCA and HCC that expresses these markers still remains unclear. Therefore, to evaluate if mature hepatocytes give rise to HCA and HCC tumors, and to understand the molecular pathways involved in tumorigenesis, we lineage-labeled hepatocytes by injecting adeno-associated virus (AAV) containing thyroxine-binding globulin (TBG) promoter driven-Cre into RosaYFP mice. Yellow fluore…

0301 basic medicinemedicine.medical_specialtyCarcinoma HepatocellularHepatologyLiver NeoplasmsBiologymedicine.diseaseGastroenterologydigestive system diseasesArticle03 medical and health sciences030104 developmental biologyCellular originInternal medicineHepatocellular carcinomamedicineCancer researchCarcinomaHepatocytesHumansCenter (algebra and category theory)DeconvolutionStage (cooking)Hepatology (Baltimore, Md.)
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Systemic treatment of HCC in special populations

2020

Summary Recent years have seen significant progress in the systemic treatment of hepatocellular carcinoma (HCC), including the advent of immunotherapy. While several large phase III trials have provided the evidence for a multi-line treatment paradigm, they have focused on a highly selected group of patients by excluding potentially confounding comorbidities. As a result, high quality evidence for the systemic treatment of HCC in patients with various comorbidities is missing. This review summarises current knowledge on the use of approved medicines in patients with HIV, autoimmune disease, cardiovascular disease, diabetes, fibrolamellar HCC, mixed HCC-cholangiocarcinoma, decompensated cirr…

0301 basic medicinemedicine.medical_specialtyCarcinoma Hepatocellularmedicine.medical_treatmentComorbidityDisease03 medical and health sciences0302 clinical medicineDiabetes mellitusInternal medicinemedicineHumansMultiple Chronic ConditionsAutoimmune diseaseHepatologybusiness.industryLiver NeoplasmsConfoundingDisease ManagementImmunotherapymedicine.diseasedigestive system diseasesPortal vein thrombosis030104 developmental biologyHepatocellular carcinoma030211 gastroenterology & hepatologyMetabolic syndromebusinessJournal of Hepatology
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Hepatic benefits of HCV cure

2020

Direct-acting antiviral (DAA)-induced HCV clearance conceivably leads to improved outcomes at all stages of liver disease. However, available data suggest that the maximum measurable benefit is obtained by treating patients before they reach the stage of compensated advanced chronic liver disease (cACLD). Ideally, all patients with chronic hepatitis C should be treated before they develop advanced fibrosis or cirrhosis, since even if sustained virologic response (SVR) reduces the risk of hepatic events (e.g. decompensation and hepatocellular carcinoma [HCC]) and improves survival, further progression of liver disease and adverse outcomes, including hepatic deaths, cannot be entirely avoided…

0301 basic medicinemedicine.medical_specialtyCirrhosisHepatocellular carcinomaPortal venous pressureSurvival.Chronic liver diseaseGastroenterology03 medical and health sciencesLiver disease0302 clinical medicineInternal medicinemedicineDecompensationCirrhosiHepatologybusiness.industryHepatitis Cmedicine.diseaseHepatitis Cdigestive system diseases030104 developmental biologyHepatocellular carcinomaPortal hypertension030211 gastroenterology & hepatologybusiness
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