Search results for "digestive"

showing 10 items of 2631 documents

A comparative economic analysis of torrefied pellet production based on state-of-the-art pellets

2017

Abstract Torrefied pellets have fuel properties superior to those of conventional wood pellets and potentially allow greater rates of co-firing and thus larger reductions in net CO 2 emissions. Despite the growing amount of scientific output on torrefaction, the economic feasibility of torrefied pellet production is still a topic of considerable uncertainty. This is an obstacle for decision makers looking to implement sustainable energy policies. This paper compares the economics of torrefied pellets to conventional wood pellets. Working backwards from demonstrated pellet properties, this work attempts to answer the following question: Based on state-of-the-art torrefied pellets, what would…

Capital investment020209 energyPellets02 engineering and technology010501 environmental sciencestorrefied pellets01 natural sciencesPellet0202 electrical engineering electronic engineering information engineeringEconomic analysisProduction (economics)biocoalWaste Management and Disposalta215ta2180105 earth and related environmental sciencesRenewable Energy Sustainability and the Environmentwood pelletsdigestive oral and skin physiologyForestryeconomicsPulp and paper industryTorrefactionpelletsSustainable energytorrefactionEnvironmental scienceHeat of combustionAgronomy and Crop ScienceBiomass and Bioenergy
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Degenerative and regenerative processes involved in midgut pseudotumor formation in the stick insect (Carausius morosus)

2009

Spontaneous and experimentally induced pseudotumor formation in Carausius morosus impairs the midgut tissue homeostasis. Spontaneous pseudotumor formation begins by the break down of a single or a small group of columnar cells (CCs) and is followed by the degeneration of neighboring CCs. There are not only marked similarities but also decisive differences between normal dying CCs in healthy specimens and the degeneration of CCs leading to pseudotumors: in both cases, the apical cell parts with the nucleus are extruded into the midgut lumen, but only during of pseudotumor formation an "amorphous substance" originates from the basal parts of the CCs. Hemocytes are attracted to this substance …

Carausius morosusPathologymedicine.medical_specialtyHemocytesInsectaCellular differentiationColumnar CellGranuloma Plasma CellDigestive System Physiological PhenomenaStomatogastric nervous systemmedicineAnimalsHomeostasisRegenerationProgenitor cellTissue homeostasisCell ProliferationbiologyStem CellsfungiCell DifferentiationMidgutbiology.organism_classificationstomatognathic diseasesAnimal Science and ZoologyStem cellDigestive SystemDevelopmental BiologyJournal of Morphology
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Inhibition of the hydrolytic and transpeptidase activities of rat kidney gamma-glutamyl transpeptidase by specific monoclonal antibodies

1999

Monoclonal antibodies (mAb) against the native form of rat kidney gamma-glutamyl transpeptidase (GGT) were isolated by screening hybridomas with rat kidney brush-border membrane vesicles. They were directed against protein rather than sugar epitopes in that each recognized all GGT isoforms. All of them inhibited partially the enzyme activity of GGT. They were specific in that they inhibited the rat enzyme, but not the mouse or human enzyme. Kinetic analyses were carried out with free GGT and GGT-mAb complexes with d-gamma-glutamyl-p-nitroanilide in the presence or absence of maleate, or in the presence or absence of alanine, cysteine, cystine or glycylglycine as gamma-glutamyl acceptors. mA…

CarbohydratesCystineKidneydigestive systemBiochemistryGlutaminase activityEpitopeMicechemistry.chemical_compoundGlutaminaseAnimalsHumansRats WistarAcivicinchemistry.chemical_classificationMice Inbred BALB CbiologyChemistryGlutaminaseHydrolysisAntibodies Monoclonalgamma-GlutamyltransferaseMolecular biologydigestive system diseasesEnzyme assayRatsIsoenzymesEnzymeBiochemistrybiology.proteinFemaleCysteineEuropean Journal of Biochemistry
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Cyclooxygenases in hepatocellular carcinoma

2006

Many epidemiological studies demonstrate that treatment with non-steroidal anti-inflammatory drugs (NSAIDs) reduce the incidence and mortality of certain malignancies, especially gastrointestinal cancer. The cyclooxygenase (COX) enzymes are well-known targets of NSAIDs. However, conventional NSAIDs non-selectively inhibit both the constitutive form COX-1, and the inducible form COX-2. Recent evidence indicates that COX-2 is an important molecular target for anticancer therapies. Its expression is undetectable in most normal tissues, and is highly induced by pro-inflammatory cytokines, mitogens, tumor promoters and growth factors. It is now well-established that COX-2 is chronically overexpr…

Carcinoma HepatocellularAngiogenesisBiologymedicine.disease_causeModels BiologicalGene Expression Regulation EnzymologicIn vivomedicineHumansNeoplasm InvasivenessGastrointestinal cancerEnzyme InhibitorsCell growthAnti-Inflammatory Agents Non-SteroidalLiver NeoplasmsGastroenterologyGeneral MedicineHCCSmedicine.diseasedigestive system diseasesGene Expression Regulation NeoplasticEditorialModels ChemicalCyclooxygenase 2Hepatocellular carcinomaImmunologyCyclooxygenase 1Cancer researchCarcinogenesisLiver cancer
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Cytotoxicity of oleanolic and ursolic acid derivatives toward hepatocellular carcinoma and evaluation of NF-κB involvement.

2019

Oleanolic and ursolic acids are two ubiquitous isomeric triterpene phytochemicals known for their anticancer activity. A set of derivatives of the two compounds with a modified oxidation state and lipophylicity at C-3 and C-28 positions, were prepared and tested as anticancer agents versus the lines HepG2, Hep3B and HA22T/VGH of hepatocarcinoma, a strongly aggressive tumor that is not responsive toward the standard therapies. New derivatives containing a three carbons side chain on the C-3 position were synthetized in both stereoisomeric forms by the Barbier-Grignard procedure and three of them were found to be active toward all of the three targets. The implication of the transcriptional n…

Carcinoma HepatocellularApoptosis01 natural sciencesBiochemistrychemistry.chemical_compoundUrsolic acid Oleanolic acid HepG2 Hep3B HA22T/VGH Antitumor activity NF−κBUrsolic acidTriterpeneOleaDrug DiscoverymedicineTumor Cells CulturedHumansSettore BIO/15 - Biologia FarmaceuticaOleanolic AcidCytotoxicityMolecular BiologyCell Proliferationchemistry.chemical_classification010405 organic chemistryPlant ExtractsOrganic ChemistryLiver NeoplasmsNF-kappa BNF-κBSettore CHIM/06 - Chimica Organicamedicine.diseaseAntineoplastic Agents Phytogenicdigestive system diseasesTriterpenes0104 chemical sciences010404 medicinal & biomolecular chemistrychemistryMechanism of actionHepatocellular carcinomaMalusSettore BIO/14 - FarmacologiaCancer researchmedicine.symptomBioorganic chemistry
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Analysis of Possible Mechanisms Accounting for Raf-1 Kinase Inhibitor Protein Downregulation in Hepatocellular Carcinoma

2012

Abstract Raf-1 kinase inhibitor protein (RKIP) is a tumor and metastasis suppressor that promotes drug-induced apoptosis in cancer cells. It is frequently downregulated, both at the mRNA and protein level, in hepatocellular carcinoma (HCC), but the mechanisms leading to this reduction are obscure. We sequenced the whole RKIP gene in three human HCC cell lines (HA22T/VGH, HepG2, and Hep3B), and in five clinical HCC samples, but could not find any gene variant that might account for their low RKIP levels. We also examined whether gene methylation may be responsible for the altered RKIP expression. No methylation of the RKIP gene was found in the tumor samples, while among the cell lines only …

Carcinoma HepatocellularLeupeptinsAntineoplastic AgentsPhosphatidylethanolamine Binding ProteinRKIP (Raf-1 kinase inhibitor protein) hepatocellular carcinomaBiologyBiochemistryDownregulation and upregulationRNA interferenceCell Line TumorGeneticsHumansMetastasis suppressorPromoter Regions GeneticMolecular BiologyRegulation of gene expressionKinaseLiver NeoplasmsHep G2 CellsMethylationDNA Methylationdigestive system diseasesGene Expression Regulation NeoplasticMicroRNAsMutationCancer cellDNA methylationAzacitidineSettore BIO/14 - FarmacologiaCancer researchMolecular MedicineRNA InterferenceBiotechnologyOMICS: A Journal of Integrative Biology
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SGLT2 Inhibitors as the Most Promising Influencers on the Outcome of Non-Alcoholic Fatty Liver Disease

2022

Non-alcoholic fatty liver disease (NAFLD), the most frequent liver disease in the Western world, is a common hepatic manifestation of metabolic syndrome (MetS). A specific cure has not yet been identified, and its treatment is currently based on risk factor therapy. Given that the initial accumulation of triglycerides in the liver parenchyma, in the presence of inflammatory processes, mitochondrial dysfunction, lipotoxicity, glucotoxicity, and oxidative stress, can evolve into non-alcoholic steatohepatitis (NASH). The main goal is to identify the factors contributing to this evolution because, once established, untreated NASH can progress through fibrosis to cirrhosis and, ultimately, be co…

Carcinoma HepatocellularSettore MED/09 - Medicina Internatype 2 diabetes mellituSGLT2metabolic syndromeCatalysisInorganic ChemistryNon-alcoholic Fatty Liver DiseaseNAFLDAnimalsHumansHypoglycemic AgentsPhysical and Theoretical ChemistrySodium-Glucose Transporter 2 InhibitorsMolecular BiologySpectroscopyDipeptidyl-Peptidase IV InhibitorsHypoglycemic AgentLiver NeoplasmsOrganic Chemistrynutritional and metabolic diseasesGeneral Medicinedigestive system diseasesComputer Science ApplicationsGlucoseLiverDiabetes Mellitus Type 2HumanInternational Journal of Molecular Sciences
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Expression of WISPs and of their novel alternative variants in human hepatocellular carcinoma cells

2005

WISPs (Wnt-induced secreted proteins) are members of the CCN (CTGF/Cyr61/Nov) family involved in fibrotic disorders and tumorigenesis. They have a typical structure composed of four conserved cysteine-rich modular domains, but variants of CCN members lacking one or more modules, generated by alternative splicing or gene mutations, have been described in various pathological conditions. WISP genes were first described as downstream targets of the Wnt signaling pathway, which is frequently altered in human hepatocellular carcinoma (HCC). In the present study, WISP mRNA expression was analyzed by RT-PCR in four human HCC cell lines (HepG2, HuH-6, HuH-7, HA22T/VGH). Our results show for the fir…

Carcinoma HepatocellularWISPHepatocellular carcinomaApoptosisGene mutationBiologymedicine.disease_causeGeneral Biochemistry Genetics and Molecular BiologyCCN Intercellular Signaling ProteinsWntalternative splicingHistory and Philosophy of ScienceCell Line TumorProto-Oncogene ProteinsCCN Intercellular Signaling ProteinsmedicineHumansRNA MessengerGeneDNA PrimersOncogene ProteinsGeneticsCCNModels GeneticReverse Transcriptase Polymerase Chain ReactionGeneral NeuroscienceLiver NeoplasmsAlternative splicingIntracellular Signaling Peptides and ProteinsWnt signaling pathwaydigestive system diseasesNeoplasm ProteinsInsulin-Like Growth Factor Binding ProteinsRepressor ProteinsCTGFCYR61Cancer researchIntercellular Signaling Peptides and ProteinsRNACarcinogenesisWISPWntTranscription Factors
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Loss of response of carnitine palmitoyltransferase I to okadaic acid in transformed hepatic cells

1998

The specific activity of carnitine palmitoyltransferase I (CPT-I) was similar in mitochondria isolated from rat Fao and human HepG2 hepatoma cells and from rat hepatocytes, but almost twofold higher in permeabilized hepatoma cells than in permeabilized hepatocytes. Short-term exposure to okadaic acid induced a ca. 80% stimulation of CPT-I in hepatocytes, whereas no significant response of the enzyme from hepatoma cells was evident. Thus, the high CPT-I activity displayed by hepatoma cells may be reached by hepatocytes upon challenge to okadaic acid. Reconstitution experiments with purified mitochondrial and cytoskeletal fractions showed that the cytoskeleton of hepatocytes produced a more r…

Carcinoma Hepatocellularendocrine system diseasesMitochondria LiverMitochondrionBiologyBiochemistrychemistry.chemical_compoundLiver Neoplasms ExperimentalOkadaic AcidTumor Cells CulturedmedicineAnimalsHumansheterocyclic compoundsCarnitine O-palmitoyltransferaseCytoskeletonneoplasmsCell Line TransformedPharmacologyCarnitine O-PalmitoyltransferaseLiver NeoplasmsOkadaic aciddigestive system diseasesMitochondriaRatsCell biologyKineticsmedicine.anatomical_structureBiochemistrychemistryCell cultureHepatocyteHepatic stellate cellCarnitine palmitoyltransferase IBiochemical Pharmacology
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Nonalcoholic Fatty Liver Disease, Cardiovascular Risk, and Carotid Inflammation.

2015

Nonalcoholic fatty liver disease (NAFLD) is defined by excessive triglycerides (TGs) accumulation in the liver (>5% of hepatocytes histologically) in the absence of alcohol excess. The NAFLD ranges from simple steatosis to steatohepatitis and cirrhosis. The NAFLD and nonalcoholic steatohepatitis (NASH) are now the number one cause of liver disease in Western countries. The prevalence of NAFLD is increasing but is underreported, and the epidemiology and demographic characteristics vary worldwide. The prevalence is increasing because of the rising occurrence of obesity and type 2 diabetes (T2DM); in fact, NAFLD is considered as the hepatic manifestation of metabolic syndrome (MetS). Nonalcoho…

Carotid Artery Diseasesmedicine.medical_specialtyVery low-density lipoproteinLipoproteins//purl.org/becyt/ford/3.3 [https]Risk FactorsInternal medicineNonalcoholic fatty liver diseasemedicinePrevalenceHumansInflammationAdiponectinbusiness.industryFatty liverNon Alcoholic Fatty Liver Diseasenutritional and metabolic diseasesmedicine.diseasedigestive system diseasesFatty LiverOxidative StressEndocrinologyCardiovascular DiseasesDisease Progression//purl.org/becyt/ford/3 [https]Hepatic lipaseMetabolic syndromeSteatohepatitisInsulin ResistanceCardiology and Cardiovascular MedicinebusinessBiomarkersLipoproteinAngiology
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