Search results for "dioxins"

showing 10 items of 47 documents

SIMILARITIES IN BIOACCUMULATION PATTERNS OF POLYCHLORINATED DIBENZO-p-DIOXINS AND FURANS AND POLYCHLORINATED DIPHENYL ETHERS IN LABORATORY-EXPOSED OL…

2003

Sediment and chironomid samples were collected from a river in the vicinity of and downstream from a closed chlorophenol production plant, and they were analyzed for polychlorinated dibenzo-p-dioxins and furans (PCDD/F) and polychlorinated diphenyl ethers (PCDE). Bioaccumulation of these compounds from the sediments by the oligochaeta, Lumbriculus variegatus, and semipermeable membrane devices (SPMDs) was studied in a 28-d bioaccumulation test. The sum concentrations of PCDD/Fs and PCDEs in sediments varied spatially, peaking at the site below the production plant, but the congener composition remained fairly constant throughout the river. The sum concentrations of chironomids, oligochaetes…

Geologic SedimentsLumbriculus variegatusPolychlorinated Dibenzodioxins010504 meteorology & atmospheric sciencesHealth Toxicology and MutagenesisLumbriculidae010501 environmental sciences01 natural sciencesChironomidaePolychlorinated diphenyl ethersRiversAnimalsSoil PollutantsEnvironmental ChemistryTissue DistributionOligochaetaWater pollutionBenzofurans0105 earth and related environmental sciencesbiologyChemistryReproducibility of ResultsSedimentMembranes ArtificialEnvironmental ExposureEnvironmental exposureDibenzofurans Polychlorinatedbiology.organism_classificationPolychlorinated BiphenylsCongenerBioaccumulationEnvironmental chemistryEthersEnvironmental Toxicology and Chemistry
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Guanine 6-O-Methylation Pattern within the Dioxin Responsive Element of theCYP1A1 Enhancer Shows Two Critical Guanines for AhR/ARNT Binding

2006

The core-recognition motif for TCDD-liganded AhR/ARNT complex of the dioxin-responsive element (DRE) contains four guanine residues, three on the antisense (5'-T T / A GCGTG-3') and one on the sense (5'-CACGC A / T A-3') strand. It has been reported that, in methylation-protection and methylation-interference assays, the TCDD-liganded AhR/ARNT contacts all four guanine residues. On the other hand, it is known that some anticancer drugs, and various environmental and workplace chemicals, including strongly human carcinogenic nitrosoamines, lead to the highly miscoding 6-O-methylation of guanine. In the present study, we have investigated whether specific methylation of guanine at the 6-O-pos…

GuaninePolychlorinated DibenzodioxinsAryl hydrocarbon receptor nuclear translocatorGuanineBioengineeringLigandsResponse ElementsBiochemistryMicechemistry.chemical_compoundCell Line TumorSense (molecular biology)Cytochrome P-450 CYP1A1AnimalsEnhancerMolecular BiologyCarcinogenBinding SitesAryl Hydrocarbon Receptor Nuclear TranslocatorGeneral ChemistryGeneral MedicineMethylationDNA MethylationMolecular biologyEnhancer Elements GeneticReceptors Aryl HydrocarbonchemistryBiochemistrySense strandMolecular MedicineChemistry & Biodiversity
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Effect of 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) on Hormones of Energy Balance in a TCDD-Sensitive and a TCDD-Resistant Rat Strain

2014

One of the hallmarks of the acute toxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) is a drastically reduced feed intake by an unknown mechanism. To further elucidate this wasting syndrome, we followed the effects of a single large dose (100 μg/kg) of TCDD on the serum levels of several energy balance-influencing hormones, clinical chemistry variables, and hepatic aryl hydrocarbon receptor (AHR) expression in two rat strains that differ widely in their TCDD sensitivities, for up to 10 days. TCDD affected most of the analytes in sensitive Long-Evans rats, while there were few alterations in the resistant Han/Wistar strain. However, analyses of feed-restricted unexposed Long-Evans rats i…

LeptinFOOD-INTAKETCDDFGF21Polychlorinated Dibenzodioxinsmedicine.medical_treatmentAHRwasting syndromeacute toxicity413 Veterinary science8-tetrachlorodibenzo-p-dioxinlcsh:Chemistry2378-tetrachlorodibenzo-<i>p</i>-dioxin; TCDD; wasting syndrome; energy balance; hormones; acute toxicity; strain differences; AHRPPAR-ALPHAInsulinMESSENGER-RNA EXPRESSIONInsulin-Like Growth Factor Ita315Receptorlcsh:QH301-705.5AH RECEPTORSpectroscopyenergiatasebiologyChemistryLeptinGeneral MedicineCENTRAL LEPTIN INFUSIONstrain differencesComputer Science ApplicationsLiverGhrelinAdiponectinARYL-HYDROCARBON RECEPTOR7medicine.medical_specialty3education2GlucagonCatalysisArticleInorganic ChemistrySpecies SpecificityInternal medicinemedicineAnimals2378-tetrachlorodibenzo-p-dioxinRats Long-EvansRNA MessengerPhysical and Theoretical ChemistryRats WistarCARBOXYKINASE PEPCK ACTIVITYMolecular BiologyI IGF-IhormonesGrowth factorOrganic ChemistryBody WeightAryl hydrocarbon receptorGlucagonenergy balancehormonitRatsFibroblast Growth FactorsEndocrinologylcsh:Biology (General)lcsh:QD1-999Receptors Aryl Hydrocarbonbiology.proteinGROWTH-FACTOR 21Energy MetabolismHormoneInternational Journal of Molecular Sciences
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TCDD induces c-jun expression via a novel Ah (dioxin) receptor-mediated p38–MAPK-dependent pathway

2005

The aryl hydrocarbon receptor (AhR) has a fundamental role during postnatal liver development and is essential for mediating dioxin toxicity. However, the genetic programs mediating, both, the toxic and physiological effects downstream of the transcription factor AhR are in major parts unknown. We have identified the proto-oncogene c-jun as a novel target gene of AhR. Induction of c-jun depends on activation of p38-mitogen-activated protein kinase (MAPK) by an AhR-dependent mechanism. None of the kinases that are known to phosphorylate p38-MAPK is activated by AhR. Neither the dephosphorylation rate of p38-MAPK is reduced. Furthermore, increased p38-MAPK phosphorylation in response to dioxi…

MAPK/ERK pathwayCancer ResearchPolychlorinated DibenzodioxinsProto-Oncogene Proteins c-junp38 mitogen-activated protein kinasesBiologyTransfectionProto-Oncogene Masp38 Mitogen-Activated Protein KinasesGenes ReporterCell Line TumorGeneticsHumansRNA NeoplasmRNA Small InterferingProtein kinase AMolecular BiologyTranscription factorDNA PrimersBase SequenceKinasec-junrespiratory systemAryl hydrocarbon receptorrespiratory tract diseasesGene Expression Regulation NeoplasticReceptors Aryl HydrocarbonMitogen-activated protein kinasebiology.proteinCancer researchOncogene
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Dioxin emissions and soft-tissue sarcoma: results of a population-based case-control study.

2004

International audience; BACKGROUND: In 1998, the French Ministry of Environment revealed that of 71 French municipal solid waste incinerators processing more than 6 metric tons of material per hour, dioxin emission from 15 of them was above the 10 ng international toxic equivalency factor/m3 (including Besançon, emitting 16.3 ng international toxic equivalency factor/m3) which is substantially higher than the 0.1 international toxic equivalency factor/m3 prescribed by a European directive of 1994. In 2000, a macrospatial epidemiological study undertaken in the administrative district of Doubs, identified two significant clusters of soft-tissue sarcoma and non Hodgkin lymphoma in the vicinit…

MESH : Case-Control StudiesMESH : MaleMESH: Environmental ExposureMESH : AgedMESH : Child PreschoolMESH : Infant NewbornMESH : SarcomaMESH : DioxinsMESH : ChildMESH: Risk FactorsMESH: ChildMESH : AdolescentMESH: IncinerationMESH : Middle AgedMESH : FemaleMESH : Data Interpretation StatisticalMESH : FranceMESH : IncinerationMESH: AgedMESH: AdolescentMESH: HumansMESH: Middle AgedMESH: DioxinsMESH: Infant NewbornMESH: Child PreschoolMESH : HumansMESH: AdultMESH : Infant[ SDV.SPEE ] Life Sciences [q-bio]/Santé publique et épidémiologieMESH : AdultMESH: InfantMESH: Case-Control StudiesMESH : Risk FactorsMESH: MaleMESH: France[SDV.SPEE] Life Sciences [q-bio]/Santé publique et épidémiologieMESH: Sarcoma[SDV.SPEE]Life Sciences [q-bio]/Santé publique et épidémiologieMESH: Data Interpretation StatisticalMESH: FemaleMESH : Soft Tissue NeoplasmsMESH : Environmental ExposureMESH: Soft Tissue Neoplasms
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Interspecies differences in cancer susceptibility and toxicity.

1999

One of the most complex challenges to the toxicologist represents extrapolation from laboratory animals to humans. In this article, we review interspecies differences in metabolism and toxicity of heterocyclic amines, aflatoxin B1, 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD), and related compounds, endocrine disrupters, polycyclic aromatic hydrocarbons, tamoxifen, and digitoxin. As far as possible, extrapolations to human toxicity and carcinogenicity are performed. Humans may be more susceptible to the carcinogenic effect of heterocyclic amines than monkeys, rats, and mice. Especially, individuals with high CYP1A2 and 3A4 activities and the rapid acetylator phenotype may be expected to have …

MaleAflatoxinAflatoxin B1Cardiotonic AgentsPolychlorinated DibenzodioxinsAntineoplastic Agents HormonalHamsterEndocrine SystemPharmacologyToxicologychemistry.chemical_compoundMiceDigitoxinSpecies SpecificityHeterocyclic CompoundsCricetinaeNeoplasmsBenzo(a)pyreneAnimalsHumansPharmacology (medical)General Pharmacology Toxicology and PharmaceuticsCarcinogenCYP1A2EstrogensGlutathioneAntiestrogenRatsTamoxifenBenzo(a)pyrenechemistryToxicityMicrosomes LiverFemaleDisease SusceptibilityRabbitsDrug metabolism reviews
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Polychlorinated diphenyl ethers, dibenzo-p-dioxins, dibenzofurans and biphenyls in seals and sediment from the Gulf of Finland.

1997

Polychlorinated diphenyl ethers (PCDEs), 2,3,7,8-substituted polychlorinated dibenzo-p-dioxins (PCDDs) and dibenzofurans (PCDFs), and polychlorinated biphenyls (PCBs) were analyzed in seals from the Gulf of Finland and in sediments from the Gulf of Finland and near Gotland. The sediments included one surface core from both sampling sites. The seal material consisted of 14 ringed seals and 6 grey seals that all were found dead and examined for pathology. The main aims were to scrutinize levels and patterns of PCDEs for the first time in seals from the Baltic Sea and to estimate whether chlorinated compounds mentioned have an influence on an exceptional high mortality that occurred among ring…

MaleAgingEnvironmental EngineeringYounger agePolychlorinated DibenzodioxinsSeals EarlessHealth Toxicology and MutagenesisNutritional StatusGas Chromatography-Mass SpectrometryPolychlorinated diphenyl ethersDry weightBlubberEnvironmental ChemistryAnimalsFinlandBenzofuransEcologyChemistryHigh mortalityPublic Health Environmental and Occupational HealthSedimentNutritional statusGeneral MedicineGeneral ChemistryPollutionPolychlorinated BiphenylsBaltic seaAdipose TissueEnvironmental chemistryFemaleWater Pollutants ChemicalChemosphere
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Method development for the simultaneous determination of polybrominated, polychlorinated, mixed polybrominated/chlorinated dibenzo-p-dioxins and dibe…

2013

An analytical methodology was developed for simultaneous determination of five groups of persistent organic pollutants (POPs) including polybrominated, polychlorinated and mixed brominated-chlorinated dibenzo-p-dioxins and dibenzofurans (PBDD/DFs, PCDD/DFs and PXDD/DFs, respectively), polychlorinated biphenyls (PCBs) and polybrominated diphenyl ethers (PBDEs) in fish samples. The presented analytical approach was based on well established and robust method for determination of PCDD/DFs and PCBs, which was augmented with gas chromatography coupled to high resolution mass spectrometry (GC-HRMS) to include PBDEs, as well as poorly investigated PBDD/DFs and PXDD/DFs at toxicologically significa…

MaleEnvironmental EngineeringHealth Toxicology and MutagenesisSalmo salarFractionationDioxinsGas Chromatography-Mass SpectrometryPolybrominated diphenyl ethersHalogenated Diphenyl EthersEnvironmental ChemistryAnimalsHrgc hrmsBenzofuransPollutantChemistryPublic Health Environmental and Occupational HealthGeneral MedicineGeneral ChemistryPollutionMethod developmentLatviaPolychlorinated BiphenylsEnvironmental chemistryChlorinated Dibenzo-p-dioxinsChromatography GelFish <Actinopterygii>FemaleGas chromatographyWater Pollutants ChemicalEnvironmental MonitoringChemosphere
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Transcriptional profiling of rat hypothalamus response to 2,3,7,8-tetrachlorodibenzo-ρ-dioxin

2015

In some mammals, halogenated aromatic hydrocarbon (HAH) exposure causes wasting syndrome, defined as significant weight loss associated with lethal outcomes. The most potent HAH in causing wasting is 2,3,7,8-tetrachlorodibenzo-r-dioxin (TCDD), which exerts its toxic effects through the aryl hydrocarbon receptor (AHR). Since TCDD toxicity is thought to predominantly arise from dysregulation of AHR-transcribed genes, it was hypothesized that wasting syndrome is a result of to TCDD-induced dysregulation of genes involved in regulation of food-intake. As the hypothalamus is the central nervous systems' regulatory center for food-intake and energy balance. Therefore, mRNA abundances in hypothala…

MaleFOOD-INTAKETCDDPolychlorinated DibenzodioxinsTime FactorsTranscription GeneticMicroarrayTISSUE GROWTH-FACTORAHRAH GENE BATTERY413 Veterinary scienceToxicologyToxicogeneticsfeed restrictionTranscriptomeNAD(P)H Dehydrogenase (Quinone)RESISTANT RATheterocyclic compoundsMESSENGER-RNA EXPRESSIONhypothalamusWastingreproductive and urinary physiologyOligonucleotide Array Sequence Analysisbiologyta31413. Good healthPROBE LEVELHypothalamusToxicityENERGY-BALANCEmedicine.symptommicroarrayARYL-HYDROCARBON RECEPTORendocrine systemmedicine.medical_specialtyta3111Species SpecificityInternal medicineCytochrome P-450 CYP1A1medicineAnimalsRats Long-EvansRNA MessengerWasting SyndromeRats WistarWasting SyndromeGene Expression Profilingta1184Lethal doseAryl hydrocarbon receptorstomatognathic diseasesEndocrinologyINDUCED ANOREXIAGene Expression Regulationbiology.proteinToxicology
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Cancer Risk Near a Polluted River in Finland

2004

The River Kymijoki in southern Finland is heavily polluted with polychlorinated dibenzo-p-dioxins and dibenzofurans and may pose a health threat to local residents, especially farmers. In this study we investigated cancer risk in people living near the river (less than 20.0 km) in 1980. We used a geographic information system, which stores registry data, in 500 m times 500 m grid squares, from the Population Register Centre, Statistics Finland, and Finnish Cancer Registry. From 1981 to 2000, cancer incidence in all people (N = 188884) and in farmers (n = 11132) residing in the study area was at the level expected based on national rates. Relative risks for total cancer and 27 cancer subtype…

MaleMini-Monograph: Information SystemsHealth Toxicology and MutagenesisPCDFPCDD010501 environmental sciences01 natural sciences0302 clinical medicinedioxinsNeoplasmsEpidemiologyRegistries030212 general & internal medicineChildFinlandMiddle AgedGISPolychlorinated Biphenyls3. Good healthChild PreschoolepidemiologyFemaleRisk assessmentRecord linkageAdultmedicine.medical_specialtyAdolescentRisk Assessment03 medical and health sciencesRiversmedicinecancerHumansSocioeconomic statusAgedBenzofurans0105 earth and related environmental sciencesbusiness.industryInfant NewbornPublic Health Environmental and Occupational HealthInfantCancerDibenzofurans Polychlorinatedmedicine.diseaseConfidence intervalCancer registryEpidemiologic Studies13. Climate actionRelative riskrecord linkagebusinessWater Pollutants ChemicalDemographyEnvironmental Health Perspectives
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