Search results for "disease model"

showing 10 items of 1116 documents

Inhibition of class I histone deacetylase with an apicidin derivative prevents cardiac hypertrophy and failure

2008

AIMS: Recent studies have demonstrated the importance of chromatin remodelling via histone acetylation/deacetylation for the control of cardiac gene expression. Specific histone deacetylases (HDACs) can, in fact, play a positive or negative role in determining cardiac myocyte (CM) size. Here, we report on the effect on hypertrophy development of three inhibitors (HDACi) of class I HDACs. METHODS AND RESULTS: The compounds were first analysed in vitro by scoring hypertrophy, expression of foetal genes, and apoptosis of neonatal rat CMs stimulated with phenylephrine, an alpha1-adrenergic agonist. This initial screening indicated that a truncated derivative of apicidin with class I HDAC specif…

Cardiac function curvemedicine.medical_specialtyHypertrophy Heart failurePhysiologymedicine.drug_classBiologyPeptides CyclicHistone DeacetylasesCell LineMuscle hypertrophychemistry.chemical_compoundPhysiology (medical)Internal medicinemedicineAnimalsHumansMyocytes CardiacEnzyme InhibitorsRats WistarCells CulturedHeart FailurePressure overloadHistone deacetylase inhibitorHypertrophic cardiomyopathyHypertrophymedicine.diseaseRatsHistone Deacetylase InhibitorsDisease Models AnimalEndocrinologychemistryEchocardiographyHeart failureHypertrophy Left VentricularHistone deacetylaseCardiology and Cardiovascular MedicineApicidinCardiovascular Research
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Epicardial delivery of collagen patches with adipose-derived stem cells in rat and minipig models of chronic myocardial infarction.

2013

Although transplantation of adipose-derived stem cells (ADSC) in chronic myocardial infarction (MI) models is associated with functional improvement, its therapeutic value is limited due to poor long-term cell engraftment and survival. Thus, the objective of this study was to examine whether transplantation of collagen patches seeded with ADSC could enhance cell engraftment and improve cardiac function in models of chronic MI. With that purpose, chronically infarcted Sprague-Dawley rats (n = 58) were divided into four groups and transplanted with media, collagen scaffold (CS), rat ADSC, or CS seeded with rat ADSC (CS-rADSC). Cell engraftment, histological changes, and cardiac function were …

Cardiac function curvemedicine.medical_specialtySwinemedicine.medical_treatmentBiophysicsMyocardial InfarctionAdipose tissueBioengineeringRevascularizationBiomaterialsRats Sprague-DawleyVasculogenesisFibrosisInternal medicinemedicineAnimalsMyocardial infarctionTissue Scaffoldsbusiness.industryHeartmedicine.diseaseRatsTransplantationDisease Models Animalsurgical procedures operativeAdipose TissueMechanics of MaterialsChronic DiseaseCeramics and CompositesCardiologySwine MiniatureCollagenStem cellbusinessPericardiumStem Cell TransplantationBiomaterials
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Adipose Stromal Vascular Fraction Improves Cardiac Function in Chronic Myocardial Infarction Through Differentiation and Paracrine Activity

2012

Fresh adipose-derived cells have been shown to be effective in the treatment of acute myocardial infarction (MI), but their role in the chronic setting is unknown. We sought to determine the long-term effect of the adipose derived-stromal vascular fraction (SVF) cell transplantation in a rat model of chronic MI. MI was induced in 82 rats by permanent coronary artery ligation and 5 weeks later rats were allocated to receive an intramyocardial injection of 107 GFP-expressing fresh SVF cells or culture media as control. Heart function and tissue metabolism were determined by echocardiography and 18F-FDG-microPET, respectively, and histological studies were performed for up to 3 months after t…

Cardiac function curvemedicine.medical_specialtymedicine.medical_treatmentHeart VentriclesBiomedical EngineeringMyocardial Infarctionlcsh:MedicineAdipose tissue030204 cardiovascular system & hematologyRevascularizationRats Sprague-Dawley03 medical and health sciences0302 clinical medicineFibrosisInternal medicineParacrine CommunicationmedicineAdipocytesMyocardial RevascularizationAnimalsMyocardial infarctionAngiogenic ProteinsVentricular remodeling030304 developmental biology0303 health sciencesTransplantationTissue Inhibitor of Metalloproteinase-1Ventricular Remodelingbusiness.industrylcsh:RCell DifferentiationTissue Inhibitor of MetalloproteinasesCell BiologyStromal vascular fractionmedicine.diseaseRatsTransplantationDisease Models AnimalPhenotypeEchocardiographyPositron-Emission TomographyChronic DiseaseCardiologyCytokinesFemaleStromal CellsbusinessCell Transplantation
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Hemodynamic Parameters Change Earlier Than Tissue Oxygen Tension in Hemorrhage

2010

BACKGROUND: Untreated hypovolemia results in impaired outcome. This study tests our hypothesis whether general hemodynamic parameters detect acute blood loss earlier than monitoring parameters of regional tissue beds. MATERIALS AND METHODS: Eight pigs (23-25 kg) were anesthetized and mechanically ventilated. A pulmonary artery catheter and an arterial catheter were inserted. Tissue oxygen tension was measured with Clark-type electrodes in the jejunal and colonic wall, in the liver, and subcutaneously. Jejunal microcirculation was assessed by laser Doppler flowmetry (LDF). Intravascular volume was optimized using difference in pulse pressure (dPP) to keep dPP below 13%. Sixty minutes after p…

Cardiac outputMean arterial pressureColonHypovolemiaSus scrofaHemodynamicsBlood PressureHemorrhageBlood volumeHypovolemiaLaser-Doppler FlowmetryIntravascular volume statusmedicineAnimalsCardiac OutputPulmonary wedge pressureBlood Volumebusiness.industryMicrocirculationHemodynamicsBlood flowHeart ArrestOxygenDisease Models AnimalJejunumLiverAnesthesiaAcute DiseaseSurgerymedicine.symptombusinessJournal of Surgical Research
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Mechanisms of Increased Vascular Superoxide Production in an Experimental Model of Idiopathic Dilated Cardiomyopathy

2005

Objective— In the present study, we sought to identify mechanisms underlying increased oxidative stress in vascular tissue in an experimental animal model of chronic congestive heart failure (CHF). Methods and Results— Superoxide and nitric oxide (NO) was measured in vessels from cardiomyopathic hamsters (CHF hamsters) and golden Syrian hamsters. We also determined expression of endothelial nitric oxide synthase (NOSIII), the soluble guanylyl cyclase, the cGMP-dependent kinase, and the NADPH oxidase. To analyze the contribution of the renin-angiotensin system to oxidative stress, CHF hamsters were treated with the angiotensin-converting enzyme inhibitor captopril for 200 days (120 mg · kg …

Cardiomyopathy DilatedMalemedicine.medical_specialtyCaptoprilNitric Oxide Synthase Type IIIReceptors Cytoplasmic and NuclearAngiotensin-Converting Enzyme InhibitorsNitric Oxidemedicine.disease_causeNitric oxideRenin-Angiotensin Systemchemistry.chemical_compoundSoluble Guanylyl CyclaseSuperoxidesCricetinaeInternal medicineIdiopathic dilated cardiomyopathymedicineAnimalsHeart FailureNADPH oxidaseMesocricetusbiologybusiness.industrySuperoxideMyocardiumBody WeightMicrofilament ProteinsNADPH OxidasesCaptoprilOrgan SizePhosphoproteinsDisease Models AnimalOxidative StressEndocrinologychemistryGuanylate CyclaseACE inhibitorbiology.proteinFemaleCardiology and Cardiovascular MedicinebusinessSoluble guanylyl cyclaseCell Adhesion MoleculesOxidative stressmedicine.drugArteriosclerosis, Thrombosis, and Vascular Biology
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May the force be with you: Transfer of healthy mitochondria from stem cells to stroke cells

2018

Stroke is a major cause of death and disability in the United States and around the world with limited therapeutic option. Here, we discuss the critical role of mitochondria in stem cell-mediated rescue of stroke brain by highlighting the concept that deleting the mitochondria from stem cells abolishes the cells’ regenerative potency. The application of innovative approaches entailing generation of mitochondria-voided stem cells as well as pharmacological inhibition of mitochondrial function may elucidate the mechanism underlying transfer of healthy mitochondria to ischemic cells, thereby providing key insights in the pathology and treatment of stroke and other brain disorders plagued with…

Cardiorespiratory Medicine and HaematologyMitochondrionRegenerative medicineRats Sprague-Dawley0302 clinical medicineStem Cell Research - Nonembryonic - Humanenergy metabolismStrokeStem CellsBrainCerebral ischemiaMitochondriaStrokeNeurologycellular bioenergeticStem Cell Research - Nonembryonic - Non-HumanStem cellmedicine.symptomCardiology and Cardiovascular Medicine1.1 Normal biological development and functioningClinical SciencesEnergy metabolismregenerative medicineInflammation03 medical and health sciencesUnderpinning researchmedicineAnimalsHumansNeurology & NeurosurgeryAnimalbusiness.industryMechanism (biology)NeurosciencesStem Cell Researchmedicine.diseaseRatsBrain DisordersTransplantationDisease Models AnimalinflammationDisease ModelsCommentarycellular bioenergeticsSprague-DawleyNeurology (clinical)businessNeuroscience030217 neurology & neurosurgerytransplantationJournal of Cerebral Blood Flow & Metabolism
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External stenting with a new polyester mesh reduces neointimal hyperplasia of vein grafts in a sheep model.

2007

Objective External stents placed around vein grafts have demonstrated effectiveness in reducing neointimal hyperplasia by preventing distension of the thin-walled vein grafts when exposed to arterial pressure. However, the ideal stent material has yet to be defined. The following study investigates the short- and long-term effects of an innovative polyester mesh stent designed with optimized adaptation of circumferential compliance. Methods Following in vitro definition of the ideal macro-porous polyester stent material, a total of 12 sheep underwent implantation of bilateral carotid artery vein graft bypasses. In six sheep, the short-term outcome (four weeks of implantation) was investigat…

Carotid Artery Diseasesmedicine.medical_specialtyCarotid Artery CommonCarotid arteriesmedicine.medical_treatmentPolyesters030232 urology & nephrologyBiomedical EngineeringMedicine (miscellaneous)BioengineeringVein graft030204 cardiovascular system & hematologyDistensionBiomaterials03 medical and health sciencesBlood Vessel Prosthesis Implantation0302 clinical medicineMedicineAnimalsSaphenous VeinNeointimal hyperplasiaPolyester meshHyperplasiaSheepbusiness.industryGraft Occlusion VascularStentGeneral MedicineSurgical Meshequipment and suppliesmedicine.diseaseSurgeryCompliance (physiology)Disease Models Animalsurgical procedures operativeSurgical meshStentsbusinessTunica IntimaDilatation PathologicThe International journal of artificial organs
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Biostable Scaffolds of Polyacrylate Polymers Implanted in the Articular Cartilage Induce Hyaline-Like Cartilage Regeneration in Rabbits

2017

[EN] Purpose: To study the influence of scaffold properties on the organization of ¿in vivo¿ cartilage regeneration. Our hypothesis is that stress transmission to the cells seeded inside the scaffold pores or surrounding it, which is highly dependent on the scaffold properties, determine differentiation of both mesenchymal cells and dedifferentiated autologous chondrocytes. Methods: Four series of porous scaffolds made of different polyacrylate polymers, previously seeded with cultured rabbit chondrocytes or without cells preseeded, were implanted in cartilage defects in rabbits. Subchondral bone was always injured during the surgery in order to allow blood to reach the implantation site an…

Cartilage ArticularHyalinScaffold0206 medical engineeringBiomedical EngineeringMedicine (miscellaneous)Biocompatible MaterialsBioengineering02 engineering and technologyBiomaterialsBiopolymersChondrocytesTissue engineeringIn vivomedicineAnimalsRegenerationTissue engineeringOriginal Research ArticleHyalineScaffoldschemistry.chemical_classificationTissue ScaffoldsGuided Tissue RegenerationRegeneration (biology)CartilageMesenchymal stem cellCell DifferentiationMesenchymal Stem CellsGeneral MedicinePolymerAnatomy021001 nanoscience & nanotechnology020601 biomedical engineeringAnimal modelsDisease Models AnimalCartilagemedicine.anatomical_structureAcrylateschemistryFISICA APLICADAMAQUINAS Y MOTORES TERMICOSRabbits0210 nano-technologyBiomedical engineeringThe International Journal of Artificial Organs
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Genetic abrogation of the fibronectin-α5β1 integrin interaction in articular cartilage aggravates osteoarthritis in mice.

2018

The balance between synthesis and degradation of the cartilage extracellular matrix is severely altered in osteoarthritis, where degradation predominates. One reason for this imbalance is believed to be due to the ligation of the α5β1 integrin, the classic fibronectin (FN) receptor, with soluble FN fragments instead of insoluble FN fibrils, which induces matrix metalloproteinase (MMP) expression. Our objective was to determine whether the lack of α5β1-FN binding influences cartilage morphogenesis in vivo and whether non-ligated α5β1 protects or aggravates the course of osteoarthritis in mice. We engineered mice (Col2a-Cre;Fn1RGE/fl), whose chondrocytes express an α5β1 binding-deficient FN, …

Cartilage ArticularMale0301 basic medicineIntegrinsKnee JointGlycobiologylcsh:MedicineCartilage morphogenesisOsteoarthritisMatrix metalloproteinaseBiochemistryExtracellular matrixMice0302 clinical medicineAnimal CellsMedicine and Health Scienceslcsh:ScienceConnective Tissue CellsStainingMultidisciplinarybiologyChemistryExtracellular MatrixCell biologymedicine.anatomical_structureConnective TissueProteoglycansMatrix Metalloproteinase 3AnatomyCellular Structures and OrganellesCellular TypesResearch ArticleIntegrin alpha5beta1Signal TransductionIntegrinMice TransgenicResearch and Analysis Methods03 medical and health sciencesChondrocytesPhysical Conditioning AnimalMatrix Metalloproteinase 13OsteoarthritisCell AdhesionmedicineAnimalsHumansRegenerationCytoplasmic Staining030203 arthritis & rheumatologyCartilagelcsh:RBiology and Life SciencesProteinsCell Biologymedicine.diseaseFibronectinsFibronectinDisease Models AnimalBiological TissueCartilage030104 developmental biologyProteoglycanSpecimen Preparation and Treatmentbiology.proteinSafranin Staininglcsh:QCollagensArticular CartilagePLoS ONE
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The CO-releasing molecule CORM-3 protects against articular degradation in the K/BxN serum transfer arthritis model.

2010

Contains fulltext : 89015.pdf (Publisher’s version ) (Closed access) Carbon monoxide-releasing molecules can counteract inflammatory responses. The aim of this study was to investigate whether tricarbonylchloro(glycinate)ruthenium (II) (CORM-3) is able to control the effector phase of experimental arthritis. Arthritis was induced in C57Black-6 mice by an intraperitoneal injection of serum from arthritic K/BxN mice. CORM-3 was administered intraperitoneally at 10 mg/kg/day (5 mg/kg twice a day) from days 0 to 10 and animals were sacrificed on day 11. Serum levels of osteocalcin and prostanoids were measured by enzyme-linked immunosorbent assay and radioimmunoassay. Gene expression was determ…

Cartilage ArticularMaleSerummedicine.medical_specialtymedicine.medical_treatmentIntraperitoneal injectionArthritisMice TransgenicHMGB1Auto-immunity transplantation and immunotherapy [N4i 4]RutheniumMicechemistry.chemical_compoundMice Inbred NODInternal medicineOrganometallic CompoundsmedicineAnimalsPharmacologyCarbon MonoxidebiologyChemistryProstaglandin D2 synthaseRadioimmunoassaymedicine.diseaseArthritis ExperimentalMice Inbred C57BLDisease Models AnimalEndocrinologyRANKLbiology.proteinOsteocalcinProstaglandin D2Infection and autoimmunity [NCMLS 1]European Journal of Pharmacology
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