Search results for "double-blind"

showing 10 items of 662 documents

β-Blockers in Patients With Intermittent Claudication and Arterial Hypertension

2011

The use of β-receptor blockers in peripheral arterial disease is controversial for their impact on vasomotor tone. The β-blocker nebivolol possesses vasodilating, endothelium-dependent, NO-releasing properties that might be beneficial in peripheral arterial disease. The aim of the study was to evaluate the effects and tolerability of nebivolol in comparison with metoprolol in these patients. A total of 128 patients with intermittent claudication and essential hypertension were included and double-blind randomized to receive 5 mg of nebivolol (N=65) or 95 mg of metoprolol (N=63) once daily. End points were changes in ankle-brachial index, initial and absolute claudication distance, endothel…

Malemedicine.medical_specialtyBrachial ArteryAdrenergic beta-AntagonistsArterial Occlusive DiseasesBlood PressureEssential hypertensionNebivololDouble-Blind MethodSurveys and QuestionnairesInternal medicinemedicine.arteryInternal MedicinemedicineHumansAnkle Brachial IndexBenzopyransBrachial arteryAntihypertensive AgentsMetoprololbusiness.industryIntermittent Claudicationmedicine.diseaseNebivololIntermittent claudicationTreatment OutcomeBlood pressureTolerabilityEthanolaminesAnesthesiaHypertensionQuality of LifeCardiologyFemalemedicine.symptomClaudicationbusinessMetoprololmedicine.drugHypertension
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AT1-receptor blockade with irbesartan improves peripheral but not coronary endothelial dysfunction in patients with stable coronary artery disease

2007

Activation of the renin-angiotensin-aldosterone system plays an important role in the pathogenesis of endothelial dysfunction and atherosclerosis. Studies evaluating the effect of AT1-receptor blockers on endothelial dysfunction in patients with coronary artery disease (CAD) revealed mixed results. Studies addressing the effects of AT1-receptor blockers on the coronary and peripheral function in the same study population, are still lacking. We therefore aimed to test the effects of long-term therapy with the AT1-receptor blocker irbesartan (IRB) on both, the coronary and peripheral endothelial function in patients with CAD. Seventy-two patients with CAD were randomly assigned to double-blin…

Malemedicine.medical_specialtyBrachial ArteryEndotheliumTetrazolesCoronary Artery DiseaseCoronary artery diseaseIrbesartanDouble-Blind MethodInternal medicinemedicine.arterymedicineHumanscardiovascular diseasesEndothelial dysfunctionBrachial arteryUltrasonographybusiness.industryVascular diseaseBiphenyl CompoundsIrbesartanMiddle Agedmedicine.diseaseCoronary VesselsAngiotensin IIPeripheralVasodilationmedicine.anatomical_structureEndocrinologyRegional Blood FlowCardiologyFemaleEndothelium VascularCardiology and Cardiovascular MedicinebusinessAngiotensin II Type 1 Receptor Blockersmedicine.drugAtherosclerosis
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Effects of captopril on myocardial protection during cardioplegia

1993

Abstract The study aimed at checking effects exerted by captopril (C) on human myocardial ACE system as well as the role played by tissue ACE inhibition in reducing reperfusion damage. A human experimental model was used during cardioplegia due to aorto-coronary-by-pass (CABG). Fifty-four patients with coronary artery disease affecting 3 vessels having suffered from acute myocardial infarction anterior (AMI-ant), homogeneous as far as ejection fraction (35–55%), number of grafts (3), clamping time, age and sex, were randomised in a double blind experiment, and were given captopril or placebo (P). A total of 4 mg/l Captopril was mixed into the cardioplegic solution with blood according to th…

Malemedicine.medical_specialtyCaptoprilEpinephrineMyocardial Reperfusion InjuryPlaceboCoronary artery diseaseNorepinephrineDouble-Blind MethodInternal medicinemedicineHumansDerivationMyocardial infarctionCoronary Artery BypassCreatine KinaseCoronary sinusEjection fractionbusiness.industryCaptoprilMiddle Agedmedicine.diseaseEpinephrineAnesthesiaHeart Arrest InducedCardiologyFemaleAngiotensin ICardiology and Cardiovascular Medicinebusinessmedicine.drugInternational Journal of Cardiology
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Captopril does not affect plasma endothelin-1 during thrombolysis and reperfusion.

1995

Studies showed that endothelin-1 (ET-1) was increased in the acute myocardial infarction (AMI). Experimental studies reported that captopril was able to reduce ET-1 secretion, and that ET-1 was increased during reperfusion. This study was aimed to verify if captopril was able to reduce plasma ET-1 during thrombolysis in AMI. Seventy-three patients, hospitalized for suspected AMI within 4 h from the onset of symptoms suitable for thrombolysis (1st episode), Killip class 1-2, were randomized (double blind) into two groups: group 1 (37 pts), 8 F/29 M, received captopril, 6.25 mg, orally 15 min before thrombolysis. Group 2: (36 pts) 8 F/28 M, received placebo before thrombolysis. All patients m…

Malemedicine.medical_specialtyCaptoprilTime Factorsmedicine.medical_treatmentMyocardial InfarctionAdministration OralAngiotensin-Converting Enzyme InhibitorsBlood PressureMyocardial ReperfusionPlaceboAnginaPlacebosElectrocardiographyDouble-Blind MethodHeart RateInternal medicineFibrinolysismedicineHumansThrombolytic TherapyMyocardial infarctionAngina UnstableCreatine KinaseKillip classbusiness.industryUnstable anginaEndothelinsCaptoprilThrombolysismedicine.diseaseRecombinant ProteinsSurgeryIsoenzymesTissue Plasminogen ActivatorCardiologyFemaleCardiology and Cardiovascular Medicinebusinessmedicine.drugInternational journal of cardiology
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The combination ace-inhibitors plus canreonate in patients with anterior myocardial infarction: safety and tolerability study.

2001

There is recent evidence that aldosterone (ALDO) exerts pro-fibrotic effects, acting via the mineral-corticoid receptors in cardiovascular tissues and partial aldosterone escape during ACE-inhibition treatment occurs.A double blind randomised study was performed to evaluate the feasibility, and tolerability of the administration of the 25 mg/day of canreonate plus captopril versus captopril alone in patients with anterior AMI unsuitable for thrombolysis and/or not receiving thrombolytic treatment, and unreperfused after thrombolysis. Fifty five patients hospitalised for anterior AMI,with a serum creatinine concentration2.0 mg/dl and a serum K concentration5.0 mmol per liter were randomised …

Malemedicine.medical_specialtyCaptoprilmedicine.medical_treatmentAldosterone escapeUrologyMyocardial InfarctionAngiotensin-Converting Enzyme Inhibitorschemistry.chemical_compoundDouble-Blind MethodmedicineHumanscardiovascular diseasesMyocardial infarctionAgedCreatinineE/A ratiobusiness.industryCaptoprilThrombolysisMiddle Agedmedicine.diseaseSurgerychemistryTolerabilityACE inhibitorFeasibility StudiesDrug Therapy CombinationFemaleCanrenoic AcidCardiology and Cardiovascular Medicinebusinessmedicine.drugInternational journal of cardiology
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Deflazacort vs. prednisone in Duchenne muscular dystrophy: trends of an ongoing study

1995

Several studies have demonstrated the slowing effect of corticosteroids on the decline of muscle strength in Duchenne muscular dystrophy (DMD). Deflazacort (DFC) is supposed to have fewer side effects than prednisone (PRED). An ongoing double blind multicenter study is comparing the effects and side effects of deflazacort (0.9 mg/kg/day) and prednisone (0.75 mg/kg/day) in DMD. This interim report includes data for 67 boys between age 5 years and loss of ambulation. Besides the common clinical and laboratory data for chronic corticoid treatment, motor performance has been tested. Interim results, 3-15 months after starting the medication, show some scattering but no grouping of data for all …

Malemedicine.medical_specialtyDuchenne muscular dystrophyAnti-Inflammatory AgentsMuscular DystrophiesDouble-Blind MethodDevelopmental NeurosciencePregnenedionesPrednisoneInternal medicinemedicineHumansChildCreatine KinaseDose-Response Relationship Drugbiologybusiness.industryBody WeightGeneral MedicineAlkaline Phosphatasemedicine.diseaseClinical trialDeflazacortDose–response relationshipEndocrinologyNeurologyMulticenter studyChild PreschoolAnesthesiaPediatrics Perinatology and Child HealthOsteocalcinbiology.proteinPrednisoneNeurology (clinical)medicine.symptombusinessWeight gainMuscle Contractionmedicine.drugBrain and Development
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Treatment of Duchenne muscular dystrophy with ciclosporin A: a randomised, double-blind, placebo-controlled multicentre trial.

2010

Summary Background Duchenne muscular dystrophy is a rare X-linked progressive disease characterised by loss of ambulation at about age 10 years, with death in early adulthood due to respiratory and cardiac insufficiency. Steroids are effective at slowing the progression of muscle weakness; however, their use is limited by side-effects, prompting the search for alternatives. We assessed the effect of ciclosporin A as monotherapy and in combination with intermittent prednisone for the treatment of ambulant patients with this disorder. Methods Our study was a parallel-group, placebo-controlled, double-blind, multicentre trial at trial sites of the German muscular dystrophy network, MD-NET, ove…

Malemedicine.medical_specialtyDuchenne muscular dystrophyMedizinPlacebolaw.invention03 medical and health sciences0302 clinical medicineRandomized controlled trialDouble-Blind MethodlawPrednisoneInternal medicinemedicineHumansMuscular dystrophyChild030304 developmental biology0303 health sciencesbusiness.industryMuscle weaknessmedicine.diseaseCiclosporin3. Good healthSurgeryClinical trialMuscular Dystrophy DuchenneReview Literature as TopicTreatment OutcomeCyclosporineNeurology (clinical)medicine.symptombusiness030217 neurology & neurosurgerymedicine.drugThe Lancet. Neurology
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Sequential Treatment Escalation with Dapagliflozin and Saxagliptin Improves Beta Cell Function in Type 2 Diabetic Patients on Previous Metformin Trea…

2018

AbstractWe investigated the effect of sequential treatment escalation with dapagliflozin and saxagliptin on beta cell function in patients with T2DM insufficiently controlled on metformin monotherapy during a hyperglycaemic clamp investigation. Twenty-six patients (19 males, age 63.5±7.0 years; duration of diabetes 8.8±4.7 years; HbA1c 63.9±15.8 mmol/mol; mean±SD) were enrolled in the study. During a first treatment period (TP1) all patients received 10 mg dapagliflozin for one month, followed by the addition of 5 mg saxagliptin or placebo for another month (TP2). At baseline and at the end of each treatment period, fasting glucose and insulin levels were analysed, and a hyperglycaemic clam…

Malemedicine.medical_specialtyEndocrinology Diabetes and Metabolismmedicine.medical_treatmentClinical BiochemistryAdamantane030209 endocrinology & metabolism030204 cardiovascular system & hematologySaxagliptinBiochemistryGlucagon03 medical and health scienceschemistry.chemical_compound0302 clinical medicineEndocrinologyInsulin resistanceDouble-Blind MethodGlucosidesInsulin-Secreting CellsInternal medicineDiabetes mellitusmedicineHumansBenzhydryl CompoundsDapagliflozinAgedProinsulinbusiness.industryInsulinBiochemistry (medical)DipeptidesGeneral MedicineMiddle Agedmedicine.diseaseMetforminEndocrinologyDiabetes Mellitus Type 2chemistryFemalebusinessmedicine.drugHormone and Metabolic Research
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Double-blind, crossover study of the clinical efficacy and the hemorheological effects of pentoxifylline in patients with occlusive arterial disease …

1984

The effect of a 3 month daily administration of 800 mg pentoxifylline (Trental 400 bds) or placebo was assessed under double blind crossover design in 18 patients (12 males and 6 females) with peripheral occlusive arterial disease in respect of painfree walking distance and various hemorheological and hemostasiological variables, platelet aggregation, serum cholesterol and triglycerides. In first treatment period walking distance significantly increased with pentoxifylline by 46% from baseline 121 ± 15 m and by 4% with placebo from baseline 134 ± 18 m. Pentoxifylline administration furthermore yielded significant decrease in whole blood and plasma viscosity and significant increase in eryt…

Malemedicine.medical_specialtyErythrocytesPlatelet Aggregationmedicine.medical_treatment030204 cardiovascular system & hematologyPlaceboPentoxifyllineDouble blindPlacebos03 medical and health sciences0302 clinical medicineDouble-Blind MethodMedicineHumansIn patient030212 general & internal medicineClinical efficacyPentoxifyllineTriglyceridesAgedChemotherapyClinical Trials as Topicbusiness.industryOcclusive arterial diseaseIntermittent ClaudicationMiddle AgedBlood ViscosityCrossover studySurgeryAdenosine DiphosphateCholesterolAnesthesiaTheobromineFemaleCollagenCardiology and Cardiovascular MedicinebusinessBlood Flow Velocitymedicine.drugAngiology
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Elevated levels of asymmetric dimethylarginine in chronic heart failure: a pathophysiologic link between oxygen radical load and impaired vasodilator…

2010

Asymmetric dimethylarginine (ADMA) is an endogenous inhibitor of nitric oxide-dependent vasodilation. In 113 patients with chronic heart failure (CHF) and 26 controls, ADMA level was studied in relation to peripheral blood flow and vasodilator capacity. Further, the effects of allopurinol on concentrations of reactive oxygen species (ROS) and ADMA and peripheral vasodilator capacity were tested in a double-blind design. ADMA level was found to be elevated in CHF patients as compared with controls and increased in parallel with New York Heart Association (NYHA) class and exercise capacity (all P < 0.0001). The level of ADMA predicted resting blood flow (P < 0.05) and postischemic vasodilator…

Malemedicine.medical_specialtyHeart diseaseAllopurinolAllopurinolheart failureVasodilationmedicine.disease_causeArgininechemistry.chemical_compoundDouble-Blind Methodendothelial functionInternal medicineMedicineHumansoxidative stressPharmacology (medical)Xanthine oxidaseAgedPharmacologybusiness.industryFree Radical ScavengersMiddle Agedmedicine.diseaseUric AcidVasodilationEndocrinologyCross-Sectional Studieschemistryheart failure; oxidative stress; endothelial functionHeart failureChronic DiseaseUric acidCitrullineFemalebusinessAsymmetric dimethylarginineReactive Oxygen SpeciesOxidative stressmedicine.drug
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