Search results for "drug resistance"

showing 10 items of 953 documents

Synthesis and evaluation of (S)-2-(2-[18F]fluoroethoxy)-4-([3-methyl-1-(2-piperidin-1-yl-phenyl)-butyl-carbamoyl]-methyl)-benzoic acid ([18F]repaglin…

2004

18F-labeled non-sulfonylurea hypoglycemic agent (S)-2-(2-[(18)F]fluoroethoxy)-4-((3-methyl-1-(2-piperidin-1-yl-phenyl)-butylcarbamoyl)-methyl)-benzoic acid ([(18)F]repaglinide), a derivative of the sulfonylurea-receptor (SUR) ligand repaglinide, was synthesized as a potential tracer for the non-invasive investigation of the sulfonylurea 1 receptor status of pancreatic beta-cells by positron emission tomography (PET) in the context of type 1 and type 2 diabetes. [(18)F]Repaglinide could be obtained in an overall radiochemical yield (RCY) of 20% after 135 min with a radiochemical purity higher than 98% applying the secondary labeling precursor 2-[(18)F]fluoroethyltosylate. Specific activity w…

Fluorine RadioisotopesCancer ResearchBiodistributionMetabolic Clearance RateReceptors DrugContext (language use)Sulfonylurea ReceptorsRats Sprague-DawleyIslets of Langerhanschemistry.chemical_compoundPiperidinesmedicineRadioligandAnimalsTissue DistributionRadiology Nuclear Medicine and imagingPotassium Channels Inwardly RectifyingBenzoic acidChemistryBiological activityLigand (biochemistry)RepaglinideRatsDissociation constantBiochemistryOrgan SpecificityRats Inbred LewIsotope LabelingPositron-Emission TomographyFeasibility StudiesMolecular MedicineATP-Binding Cassette TransportersCarbamatesMultidrug Resistance-Associated ProteinsRadiopharmaceuticalsNuclear chemistrymedicine.drugNuclear Medicine and Biology
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Design of antitumor drugs targeting c-kit receptor by a new mixed ligand-structure based method

2020

An important challenge, in the medicinal chemistry field, is the research of novel forceful drugs to overcome tumor-acquired resistance. The c-Kit tyrosine kinase receptor (TKR) represents a suitable target for the carcinogenesis control of gastro-intestinal stromal (GIST), leukemia, and mastocytosis tumors; nevertheless, several hotspot mutations of the protein limit the efficacy of a few clinical administered TKRs inhibitors. In this study, a new in silico protocol based on ligand and structure-based combined method is proposed, with the aim to identify a set of new c-Kit inhibitors able to complex c-Kit mutated proteins. A recent and freely available web-server DRUDIT is used for the lig…

Gastrointestinal Stromal TumorsIn silicoAntineoplastic AgentsComputational biologyDrug resistanceIn silico protocolsmedicine.disease_causeLigandsReceptor tyrosine kinase03 medical and health sciences0302 clinical medicineDRUDIT web-serverc-KitMaterials ChemistrymedicineHumansPhysical and Theoretical ChemistryProtein Kinase InhibitorsSpectroscopy030304 developmental biology0303 health sciencesbiologyChemistryLigandMixed ligandmedicine.diseaseComputer Graphics and Computer-Aided DesignLeukemiaProto-Oncogene Proteins c-kitDocking (molecular)Drug Resistance Neoplasm030220 oncology & carcinogenesisDrug resistanceMutationMolecular dockingbiology.proteinCarcinogenesis
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NAIP-deltaEx10-11: a novel splice variant of the apoptosis inhibitor NAIP differently expressed in drug-sensitive and multidrug-resistant HL60 leukem…

2002

Alterations of neuronal apoptosis inhibitory protein (NAIP), a member of the inhibitory of apoptosis protein (IAP) family of inhibitors of apoptosis, have been previously associated with different neurodegenerative disorders. This study indicated the existence of a novel NAIP splice variant. This isoform, NAIP-deltaEx10-11, was found in tumor cell lines of different origin and in normal adult brain. Analysis of the putative protein predicted that the NAIP variant lacks part of the third BIR domain as well as the COOH-terminal tail of regular NAIP. This might suggest that it is endowed with a reduced antiapoptotic activity. This view is supported by the fact that NAIP-deltaEx10-11 mRNA and p…

Gene isoformCancer ResearchApoptosis InhibitorHL60ApoptosisHL-60 CellsNerve Tissue ProteinsBiologyExonchemistry.chemical_compoundmedicineRNA PrecursorsTumor Cells CulturedHumansProtein IsoformsRNA NeoplasmSequence DeletionGeneticsBrain ChemistryAlternative splicingHematologyExonsmedicine.diseaseDrug Resistance MultipleNeuronal Apoptosis-Inhibitory ProteinNeoplasm ProteinsProtein Structure TertiaryLeukemiaAlternative SplicingOncologychemistryApoptosisDrug Resistance NeoplasmCancer researchNAIPLeukemia research
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ΔNp73β is oncogenic in hepatocellular carcinoma by blocking apoptosis signaling via death receptors and mitochondria

2010

p73 belongs to the p53 family of transcription factors known to regulate cell cycle and apoptosis. The Trp73 gene has two promoters that drive the expression of two major p73 isoform subfamilies: TA and ΔN. In general, TAp73 isoforms show proapoptotic activities, whereas members of the N-terminally truncated (ΔN) p73 subfamily that lack the transactivation domain show antiapoptotic functions. We found that upregulation of ΔNp73 in hepatocellular carcinoma (HCC) correlated with reduced survival. Here, we investigated the molecular mechanisms accounting for the oncogenic role of ΔNp73 in HCC.ΔNp73β can directly interfere with the transcriptional activation function of the TA (containing the t…

Gene isoformCarcinoma HepatocellularMolecular Sequence DataApoptosisBiologyModels BiologicalTransactivationDownregulation and upregulationCell Line TumorHumansProtein IsoformsMolecular BiologyTranscription factorGenes DominantOligonucleotide Array Sequence Analysisbcl-2-Associated X ProteinRegulation of gene expressionBase SequenceSettore BIO/11Gene Expression ProfilingTumor Suppressor ProteinsLiver NeoplasmsNuclear ProteinsTumor Protein p73PromoterReceptors Death DomainCell BiologyCell cyclePrognosisMitochondriaCell biologyDNA-Binding ProteinsEnzyme ActivationGene Expression Regulation NeoplasticDrug Resistance NeoplasmCaspasesCancer researchTumor Suppressor Protein p53Signal transductionPrecancerous ConditionsSignal TransductionDevelopmental BiologyCell Cycle
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Targeting Cancer Chemotherapy Resistance by Precision Medicine-Driven Nanoparticle-Formulated Cisplatin.

2021

Therapy resistance is the major cause of cancer death. As patients respond heterogeneously, precision/personalized medicine needs to be considered, including the application of nanoparticles (NPs). The success of therapeutic NPs requires to first identify clinically relevant resistance mechanisms and to define key players, followed by a rational design of biocompatible NPs capable to target resistance. Consequently, we employed a tiered experimental pipeline fromiin silico/ito analytical andiin vitro/ito overcome cisplatin resistance. First, we generated cisplatin-resistant cancer cells and used next-generation sequencing together with CRISPR/Cas9 knockout technology to identify the ion cha…

General Physics and AstronomyAntineoplastic Agentschemistry.chemical_compoundIn vivoCell Line TumorNeoplasmsmedicineHumansGeneral Materials ScienceDoxorubicinProspective StudiesPrecision MedicineCisplatinbusiness.industryHead and neck cancerGeneral EngineeringMembrane Proteinsmedicine.diseasePaclitaxelchemistryDrug Resistance NeoplasmCancer cellCancer researchNanomedicineNanoparticlesPersonalized medicineCisplatinbusinessmedicine.drugACS nano
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Cyclosporin A resistance of herpes simplex virus-induced "fusion from within" as a phenotypical marker of mutations in the Syn 3 locus of the glycopr…

1994

We here report research in which nine strains of Herpes simplex virus (HSV) with fusing activity were investigated in order to establish precise phenotypical markers of mutations in the carboxy terminus of glycoprotein B (gB). The gene region encoding the carboxy terminus of gB was isolated, then cloned, and finally sequenced. Our investigation showed that seven strains have different mutations in the syn 3 locus. We observed no base difference in the gB gene region encoding the carboxy terminus of gB of two other strains. Strains with a mutation in the carboxy terminus of gB induced fusion from within (FFWI) in the presence of Cyclosporin A (CyA) at a concentration up to 150 µM. There are …

Genetic MarkersGenes ViralLocus (genetics)Biologymedicine.disease_causeVirusCell LineCell FusionViral Envelope ProteinsVirologyCyclosporin aGeneticsmedicineAnimalsHumansSimplexvirusMolecular BiologyGenechemistry.chemical_classificationGeneticsCell fusionDrug Resistance MicrobialGeneral MedicinePhenotypeMolecular biologyHerpes simplex virusPhenotypechemistryMutationCyclosporineGlycoproteinViral Fusion ProteinsVirus genes
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An operon for histidine biosynthesis in Streptomyces coelicolor

1973

On the assumption that a cluster of five his genes (eight cistrons) in S. coelicolor corresponds to an operon, a genetic analysis of a constitutive mutant was carried out. This strain has a multi-site mutation localized at the (conventional) right end of the his cluster and is derepressed for at least two enzymes coded by genes of the cluster. The study of suitable heterozygous clones (heteroclones), showed the mutation to be cis-dominant, suggesting that the operator region is affected. Most likely the strain has a deletion connecting the his operon to an adjacent amm (ammonium requirement) operon as demonstrated by its inability to utilize nitrate as nitrogen source and to complement or r…

Genetics MicrobialHeterozygoteOperator (biology)Genetic LinkageOperonBiologyGenetic analysisOperonGeneticsHistidineAminesMolecular BiologyGeneAllelesCrosses GeneticGenes Dominantchemistry.chemical_classificationGeneticsNitratesStrain (chemistry)Streptomyces coelicolorChromosome MappingDrug Resistance Microbialbiology.organism_classificationStreptomycesQuaternary Ammonium CompoundsButyratesEnzymechemistryMutation (genetic algorithm)Molecular and General Genetics MGG
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Interactions between the yeast mitochondrial and nuclear genomes: isogenic suppressive and hypersuppressive petites differ in their resistance to the…

1990

In a previous paper we have shown that the alkaloid lycorine inhibits growth of rho+, mit- and rho-, strains of Saccharomyces cerevisiae, whereas strains devoid of mitochondrial DNA (rho degrees) are resistant to more than 200 micrograms/ml of the alkaloid. In this report we show that hypersuppressive petites are almost as resistant as rho degrees mutants, whereas isogenic rho- petites, which have retained longer segments of the genome, are sensitive to the drug.

GeneticsMitochondrial DNAMutationbiologyAlkaloidSaccharomyces cerevisiaeMutantDrug Resistance MicrobialSaccharomyces cerevisiaeGeneral Medicinebiology.organism_classificationLycorinemedicine.disease_causeDNA MitochondrialMolecular biologyGenomeYeastPhenanthridineschemistry.chemical_compoundchemistryAmaryllidaceae AlkaloidsGeneticsmedicineDNA FungalCurrent Genetics
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Construction of a Trp commercial baker?s yeast strain by using food-safe-grade dominant drug resistance cassettes

2003

We have designed a food-safe-grade module for gene disruptions in commercial baker's yeast strains, which contains the G418 resistance cassette, KanMX4, flanked by direct repeats from the MEL1 gene of Saccharomyces cerevisiae. This module was used to obtain a Trp(-) auxotrophic mutant of the polyploid HY strain by successive targeting to the TRP1 locus and later in vivo excision of the kan(r) marker. Southern blot analysis indicated that HY contains five copies of the TRP1 gene. However, after four disruption rounds, a strain named HYtrpM(4), unable to grow in the absence of tryptophan, was selected. Southern and Northern analysis of HYtrpM(4) cells showed that a remaining functional wild-t…

GeneticsSaccharomyces cerevisiae ProteinsIsomerase activitybiologyGenes FungalSaccharomyces cerevisiaeDrug Resistance MicrobialSaccharomyces cerevisiaeGeneral Medicinebiology.organism_classificationApplied Microbiology and BiotechnologyMicrobiologyMolecular biologyYeastFungal ProteinsTransformation GeneticPlasmidFood TechnologyDirect repeatGene conversionGeneAldose-Ketose IsomerasesBiotechnologyPlasmidsSouthern blotFEMS Yeast Research
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Predominance of the fimH30 Subclone Among Multidrug-Resistant Escherichia coli Strains Belonging to Sequence Type 131 in Italy

2013

GeneticsSettore MED/07 - Microbiologia E Microbiologia ClinicaST131 E. coli fimH30 sub-clone in ItalyBiologymedicine.disease_causeAnti-Bacterial AgentsMicrobiologyMultiple drug resistanceInfectious DiseasesType (biology)Drug Resistance Multiple BacterialCorrespondenceEscherichia colimedicineHumansImmunology and AllergyEscherichia coliEscherichia coli InfectionsFluoroquinolonesSequence (medicine)Journal of Infectious Diseases
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