Search results for "drug resistance"

showing 10 items of 953 documents

Hypoxia and anemia: effects on tumor biology and treatment resistance

2004

In locally advanced solid tumors, oxygen (O2) delivery is frequently reduced or even abolished. This is due to abnormalities of the tumor microvasculature, adverse diffusion geometries, and tumor-associated and/or therapy-induced anemia. Up to 50-60% of locally advanced solid tumors may exhibit hypoxic and/or anoxic tissue areas that are heterogeneously distributed within the tumor mass. In approximately 30% of pretreatment patients, a decreased O2 transport capacity of the blood as a result of tumor-associated anemia can greatly contribute to the development of tumor hypoxia. While normal tissues can compensate for this O2 deficiency status by a rise in blood flow rate, locally advanced tu…

Genome instabilityAnemiaClinical BiochemistryDrug resistanceBiologyRadiation ToleranceNeoplasmsmedicineHumansHypoxiaRegulation of gene expressionTumor hypoxiaBiochemistry (medical)NF-kappa BNuclear ProteinsAnemiaHematologyHypoxia (medical)Hypoxia-Inducible Factor 1 alpha SubunitPrognosismedicine.diseaseNeoplasm ProteinsDNA-Binding ProteinsGene Expression Regulation NeoplasticOxygenHypoxia-inducible factorsDrug Resistance NeoplasmTumor progressionImmunologyDisease ProgressionCancer researchHypoxia-Inducible Factor 1medicine.symptomCell DivisionTranscription FactorsTransfusion Clinique et Biologique
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Analysis of the Thymidylate Synthase Gene Structure in Colorectal Cancer Patients and Its Possible Relation with the 5-Fluorouracil Drug Response

2009

Thymidylate synthase (TS) catalyzes methylation of dUMP to dTMP and it is the target for the 5-Fluorouracil (5-FU) activity. Barbour et al. showed that variant structural forms of TS in tumour cell lines confer resistance to fluoropyrimidines. We planned to perform the whole TS gene structure by means of sequencing techniques in human colorectal cancer (CRC) samples to try to identify the presence of any possible TS variant form that could be responsible of fluoropyrimidines drug resistance and of the worse prognosis. We performed the TS-DNA gene sequence in 68 CRC from patients of A, B, and C Dukes' stages and different histological grade, but we did not find any mutation in the TS-DNA str…

Genome instabilityArticle Subjectlcsh:QH426-470Colorectal cancerDrug resistancemedicine.disease_causeBioinformaticsBiochemistryThymidylate synthaselcsh:Biochemistrymedicinelcsh:QD415-436Molecular BiologyGeneMutationbiologybusiness.industryMethylationmedicine.diseaselcsh:GeneticsFluorouracilbiology.proteinCancer researchbusinessResearch Articlemedicine.drugJournal of Nucleic Acids
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Checkpoint adaptation in recombination-deficient cells drives aneuploidy and resistance to genotoxic agents.

2020

Abstract Human cancers frequently harbour mutations in DNA repair genes, rendering the use of DNA damaging agents as an effective therapeutic intervention. As therapy-resistant cells often arise, it is important to better understand the molecular pathways that drive resistance in order to facilitate the eventual targeting of such processes. We employ recombination-defective diploid yeast as a model to demonstrate that, in response to genotoxic challenges, nearly all cells eventually undergo checkpoint adaptation, resulting in the generation of aneuploid cells with whole chromosome losses that have acquired resistance to the initial genotoxic challenge. We demonstrate that adaptation inhibit…

Genome instabilitySaccharomyces cerevisiae ProteinsDNA RepairDNA repairAneuploidySaccharomyces cerevisiaeBiologyBiochemistryGenomic Instabilitychemistry.chemical_compoundGene Knockout TechniquesDrug Resistance FungalmedicineCytotoxicityMolecular BiologyRecombination GeneticSirolimusCell BiologyCell Cycle Checkpointsmedicine.diseaseAneuploidyPhenotypeDiploidyCell biologyRad52 DNA Repair and Recombination ProteinchemistryAdaptationPloidyDNADNA repair
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Genotype and Allele Frequencies of Drug-Metabolizing Enzymes and Drug Transporter Genes Affecting Immunosuppressants in the Spanish White Population

2013

Interpatient variability in drug response can be widely explained by genetically determined differences in metabolizing enzymes, drug transporters, and drug targets, leading to different pharmacokinetic and/or pharmacodynamic behaviors of drugs. Genetic variations affect or do not affect drug responses depending on their influence on protein activity and the relevance of such proteins in the pathway of the drug. Also, the frequency of such genetic variations differs among populations, so the clinical relevance of a specific variation is not the same in all of them. In this study, a panel of 33 single nucleotide polymorphisms in 14 different genes (ABCB1, ABCC2, ABCG2, CYP2B6, CYP2C19, CYP2C…

GenotypeCYP2B6Nod2 Signaling Adaptor ProteinOrganic Anion TransportersSingle-nucleotide polymorphismCYP2C19PharmacologyPolymorphism Single NucleotideWhite PeopleCytochrome P-450 Enzyme SystemGene FrequencyGenetic variationGenotypeHumansPharmacology (medical)ATP Binding Cassette Transporter Subfamily B Member 1GlucuronosyltransferaseAllele frequencyCYP2C9Methylenetetrahydrofolate Reductase (NADPH2)PharmacologyGeneticsbiologyMethyltransferasesMultidrug Resistance-Associated Protein 2Tissue DonorsTransplant RecipientsSpainInactivation MetabolicUDP-Glucuronosyltransferase 1A9biology.proteinSLCO1B1Immunosuppressive AgentsTherapeutic Drug Monitoring
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Recombination in Hepatitis C Virus

2011

Hepatitis C virus (HCV) is a Flavivirus with a positive-sense, single-stranded RNA genome of about 9,600 nucleotides. It is a major cause of liver disease, infecting almost 200 million people all over the world. Similarly to most RNA viruses, HCV displays very high levels of genetic diversity which have been used to differentiate six major genotypes and about 80 subtypes. Although the different genotypes and subtypes share basic biological and pathogenic features they differ in clinical outcomes, response to treatment and epidemiology. The first HCV recombinant strain, in which different genome segments derived from parentals of different genotypes, was described in St. Petersburg (Russia) …

GenotypeHepacivirusHepatitis C viruslcsh:QR1-502Genome ViralHepacivirusReviewmedicine.disease_causeGenomelcsh:MicrobiologyVirussuperinfectionEvolution MolecularVirologyDrug Resistance ViralGenotypeGenetic variationmedicineHumansphylogenetic treePhylogenyRecombination GeneticbreakpointGeneticsbiologycongruenceGenetic Variationhomoplasyvirus diseasesHepatitis Cmedicine.diseasebiology.organism_classificationHepatitis CVirologyFlavivirusInfectious DiseasesMutationRNA ViralViruses
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No association between Helicobacter pylori genotypes and antibiotic resistance phenotypes within families.

2002

Background. Triple therapy combining a proton pump inhibitor with two antibiotics, e.g. clarythromycin (CLR), metronidazole (MTZ) or amoxicillin (AMX), represents the standard in Helicobacter pylori eradication regimens. Resistance to antimicrobial agents, particularly MTZ (up to 56% in Western countries) and CLR (up to 15% in southern Europe), is frequently observed and may be associated with treatment failure [1]. Recently, several studies indicated that individual H. pylori colonies from a single anatomic site may not always yield identical genotypes, or the identical patterns of susceptibility to antibiotics [2–5]. Representative for every single patient we analyzed 27 H. pylori antrum …

Genotypemedicine.drug_classAntibioticsDrug resistanceMicrobial Sensitivity TestsPolymerase Chain ReactionMicrobiologyHelicobacter InfectionsAntibiotic resistanceDrug Resistance BacterialmedicineHelicobacterbiologyHelicobacter pyloriGastroenterologyGeneral MedicineAmoxicillinHelicobacter pyloribiology.organism_classificationAntimicrobialAnti-Bacterial AgentsElectrophoresis Gel Pulsed-FieldMetronidazoleInfectious DiseasesPhenotypePolymorphism Restriction Fragment Lengthmedicine.drugHelicobacter
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Occurrence of antibiotics and risk of antibiotic resistance evolution in selected Kenyan wastewaters, surface waters and sediments

2019

Active pharmaceutical ingredients, especially antibiotics, are micropollutants whose continuous flow into hydrological cycles has the potential to mediate antibiotic resistance in the environment and cause toxicity to sensitive organisms. Here, we investigated the levels of selected antibiotics in four wastewater treatment plants and the receiving water bodies. The measured environmental concentrations were compared with the proposed compound-specific predicted no-effect concentration for resistance selection values. The concentration of doxycycline, amoxicillin, sulfamethoxazole, trimethoprim, ciprofloxacin and norfloxacin within the influents, effluents, surface waters and river sediments…

Geologic SedimentsEnvironmental Engineering010504 meteorology & atmospheric sciencesjätevesiWastewater010501 environmental sciences01 natural sciencesantibioticsAntibiotic resistancemedicineEnvironmental Chemistryantimicrobial resistanceantibiotic resistance evolutionwastewaterWaste Management and DisposalEffluentNorfloxacin0105 earth and related environmental sciencesvesien saastuminenChemistrySulfamethoxazoleantibiootitrisk assessmentDrug Resistance MicrobiallääkeaineetKenyaPollutionTrimethoprim6. Clean waterAnti-Bacterial Agents3. Good healthCiprofloxacinpintavesiWastewaterjätevesikuormitus13. Climate actionpitoisuusEnvironmental chemistrySurface waterWater Pollutants ChemicalantibioottiresistenssiEnvironmental Monitoringmedicine.drugScience of The Total Environment
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Rapid adaptation of signaling networks in the fungal pathogen Magnaporthe oryzae

2019

Abstract Background One fundamental question in biology is how the evolution of eukaryotic signaling networks has taken place. “Loss of function” (lof) mutants from components of the high osmolarity glycerol (HOG) signaling pathway in the filamentous fungus Magnaporthe oryzae are viable, but impaired in osmoregulation. Results After long-term cultivation upon high osmolarity, stable individuals with reestablished osmoregulation capacity arise independently from each of the mutants with inactivated HOG pathway. This phenomenon is extremely reproducible and occurs only in osmosensitive mutants related to the HOG pathway – not in other osmosensitive Magnaporthe mutants. The major compatible so…

GlycerolMagnaportheved/biology.organism_classification_rank.speciesMutantGenomeSalt StressTranscriptome0302 clinical medicineOsmoregulationLoss of Function MutationGene Expression Regulation FungalGene Regulatory NetworksSuppressorReestablishment of osmoregulation0303 health sciencesbiologyMagnaporthe oryzaeRewiringAdaptation PhysiologicalRapid adaptationCell biologyMagnaportheOsmoregulationEpigeneticsGenome FungalBiotechnologySignal TransductionResearch Articlelcsh:QH426-470lcsh:BiotechnologyDioxolesFungal Proteins03 medical and health sciencesDrug Resistance Fungallcsh:TP248.13-248.65GeneticsPyrrolesModel organismGene030304 developmental biologyPlant DiseasesOsmotic concentrationved/biologyGene Expression ProfilingEvolution of signaling networksHOG pathwayOryzabiology.organism_classificationlcsh:Genetics030217 neurology & neurosurgery
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Cytotoxicity and chemosensitizing activity of amphiphilic poly(glycerol)-poly(alkylene oxide) block copolymers.

2014

All polymeric chemosensitizers proposed thus far have a linear poly(ethylene glycol) (PEG) hydrophilic block. To testify whether precisely this chemical structure and architecture of the hydrophilic block is a prerequisite for chemosensitization, we tested a series of novel block copolymers containing a hyperbranched polyglycerol segment as a hydrophilic block (PPO-NG copolymers) on multi-drug-resistant (MDR) tumor cells in culture. PPO-NG copolymers inhibited MDR of three cell lines, indicating that the linear PEG can be substituted for a hyperbranched polyglycerol block without loss of the polymers' chemosensitizing activity. The extent of MDR reversal increased with the polymers affinity…

GlycerolPolymers and PlasticsCell SurvivalPolymersBioengineeringAntineoplastic AgentsMicellePolyethylene GlycolsBiomaterialschemistry.chemical_compoundInhibitory Concentration 50Polymer chemistryAmphiphilePEG ratioMaterials ChemistryCopolymerHumansATP Binding Cassette Transporter Subfamily B Member 1CytotoxicityMicelleschemistry.chemical_classificationDrug SynergismPolymerPoloxamerDrug Resistance MultiplechemistryDoxorubicinDrug Resistance NeoplasmMCF-7 CellsDrug Screening Assays AntitumorK562 CellsEthylene glycolHydrophobic and Hydrophilic InteractionsBiomacromolecules
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Physiology and genetics of antibiotic production and resistance.

1993

Actinomycetes have the genetic capability to synthesize many different biologically active secondary metabolites and of these compounds, antibiotics predominate in therapeutic and commercial importance. Intensive research often centres on the use of molecular techniques to investigate the physiology and genetics of antibiotic biosynthesis with a view to improving production. The isolation of clones of Streptomyces hygroscopicus, the producer of geldanamycin, which synthesizes geldanamycin in S. lividans, is reported. Molecular approaches using genes for elongation factors (tuf) were used in attempts to increase the fermentation yield of kirromycin, whilst probes for aphD and sph, genes for …

Glycerolmedicine.drug_classPyridonesLactams MacrocyclicAntibioticsPhysiologyBiologySecondary metaboliteIn Vitro TechniquesMicrobiologychemistry.chemical_compoundActinomycetalesSpiramycinmedicineMolecular BiologyGeneGeneticsProdigiosinSpiramycinDrug Resistance MicrobialGeneral MedicineGeldanamycinbiology.organism_classificationStreptomycesAnti-Bacterial AgentschemistryBiochemistryStreptomycinTobramycinStreptomyces hygroscopicusBacteriamedicine.drugResearch in microbiology
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