Search results for "endoperoxide"

showing 10 items of 65 documents

An anti-inflammatory ditriazine inhibiting leukocyte functions and expression of inducible nitric oxide synthase and cyclo-oxygenase-2.

2000

A ditriazine derivative (4,10-dichloropyrido[5,6:4,5]thieno[3,2-d':3, 2-d]-1,2,3-ditriazine (DTD)) inhibited neutrophil functions, including degranulation, superoxide generation, and leukotriene B(4) production, without any effect on 5-lipoxygenase activity. This compound reduced nitric oxide (NO) and prostaglandin E(2) production in mouse peritoneal macrophages stimulated with lipopolysaccharide, whereas no influence on the activity of inducible NO synthase, cyclo-oxygenase-2 or cyclo-oxygenase-1 was observed. DTD significantly reduced mouse paw oedema induced by carrageenan and also markedly reduced NO and prostaglandin E(2) levels in exudates from 24-h zymosan-stimulated mouse air pouch.…

LipopolysaccharideLeukotriene B4Neutrophilsmedicine.medical_treatmentAnti-Inflammatory AgentsNitric Oxide Synthase Type IICarrageenanNeutrophil Activationchemistry.chemical_compoundMiceLeukocytesEdemaProstaglandin E2biologyPancreatic ElastaseSuperoxideTriazinesHindlimbNitric oxide synthaseIsoenzymesBiochemistryFemalemedicine.drugProstaglandin EBlood PlateletsLeukotriene B4DinoprostonePhospholipases ANitric oxideMicrosomesmedicineAnimalsHumansNitritesPharmacologyInflammationCell-Free SystemDose-Response Relationship DrugZymosanMembrane ProteinsMolecular biologyThromboxane B2chemistryEicosanoidCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesLuminescent Measurementsbiology.proteinMacrophages PeritonealNitric Oxide SynthaseEuropean journal of pharmacology
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Novel anti-inflammatory chalcone derivatives inhibit the induction of nitric oxide synthase and cyclooxygenase-2 in mouse peritoneal macrophages

1999

AbstractIn a previous work, we tested a series of chalcone derivatives as possible anti-inflammatory compounds. We now investigate the effects of three of those compounds, CH1, CH8 and CH12, on nitric oxide and prostanoid generation in mouse peritoneal macrophages stimulated with lipopolysaccharide and in the mouse air pouch injected with zymosan, where they showed a dose-dependent inhibition with inhibitory concentration 50% values in the μM range. This effect was not the consequence of a direct inhibitory action on enzyme activities. Our results demonstrated that chalcone derivatives inhibited de novo inducible nitric oxide synthase and cyclooxygenase-2 synthesis, being a novel therapeuti…

LipopolysaccharidesChalconeLipopolysaccharidemedicine.drug_classBiophysicsNitric Oxide Synthase Type IILipopolysaccharidePharmacologyBiochemistryDinoprostoneAnti-inflammatoryNitric oxideMicechemistry.chemical_compoundChalconeStructural BiologyGeneticsmedicineAnimalsCyclooxygenase-2Mouse air pouchInducible nitric oxide synthaseMolecular BiologyNitritesDose-Response Relationship DrugbiologyAnti-Inflammatory Agents Non-SteroidalZymosanZymosanProstanoidMouse peritoneal macrophageCell BiologyIsoenzymesNitric oxide synthasechemistryBiochemistryCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesEnzyme InductionMacrophages Peritonealbiology.proteinFemaleCyclooxygenaseNitric Oxide SynthaseFEBS Letters
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Solid-phase synthesis and inhibitory effects of some pyrido[1,2-c]pyrimidine derivatives on leukocyte formations and experimental inflammation.

2001

A number of pyrido[1,2-c]pyrimidines bearing a nitrogen, oxygen, or sulfur functionality at C-1 were synthesized on solid-phase using the iminophosphorane methodology and tested for their effects on leukocyte functions in vitro and antiinflammatory activity. Compound 5c was found to be a strong scavenger of superoxide anion and an inhibitor of chemiluminescence induced by 12-O-tetradecanoylphorbol 13-acetate in human neutrophils. These pyrido[1,2-c]pyrimidines inhibited the generation of PGE(2) by COX-2 in RAW 264.7 macrophages stimulated with lipopolysaccharide. Compounds 7, 5f, 6, and 8 inhibited enzyme activity, whereas the remaining compounds also acted on the induction phase. In additi…

LipopolysaccharidesLipopolysaccharideNeutrophilsChemical synthesisDinoprostoneNeutrophil Activationchemistry.chemical_compoundMiceStructure-Activity RelationshipDrug DiscoveryAnimalsEdemaHumansCells CulturedbiologyPancreatic ElastaseSuperoxideMacrophagesAnti-Inflammatory Agents Non-SteroidalMembrane ProteinsBiological activityFree Radical ScavengersIn vitroEnzyme assayCarrageenanIsoenzymesPyrimidineschemistryEicosanoidBiochemistryCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesLuminescent Measurementsbiology.proteinMolecular MedicineTetradecanoylphorbol AcetateJournal of medicinal chemistry
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6-Dimethylamino 1H-pyrazolo[3,4-d]pyrimidine derivatives as new inhibitors of inflammatory mediators in intact cells.

2003

The synthesis of 6-dimethylamino 1H-pyrazolo[3,4-d]pyrimidines substituted at positions 1 and 4, and their effects on murine macrophage and human neutrophil functions are described. Several compounds and especially 4b-6b are potent inhibitors of PGE2 generation in murine macrophages. This action is related to a direct effect on COX-2 activity without affecting the enzyme expression. Some of these compounds also inhibited COX-1 and COX-2 in human monocytes and 4b showed selectivity for COX-2 inhibition. © 2003 Elsevier Science Ltd. All rights reserved.

LipopolysaccharidesMagnetic Resonance SpectroscopyPyrimidineClinical BiochemistryBlotting WesternPharmaceutical ScienceBiochemistryLeukotriene B4Pyrazolopyrimidinechemistry.chemical_compoundMiceStructure-Activity RelationshipDrug DiscoverymedicineLeukocytesMacrophageAnimalsHumansCyclooxygenase InhibitorsMolecular Biologychemistry.chemical_classificationbiologyCyclooxygenase 2 InhibitorsPancreatic ElastaseMonocyteOrganic ChemistryMembrane ProteinsBiological activityIn vitroIsoenzymesEnzymemedicine.anatomical_structurePyrimidineschemistryBiochemistryEnzyme inhibitorCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesLuminescent Measurementsbiology.proteinCyclooxygenase 1Macrophages PeritonealMolecular MedicinePyrazolesInflammation MediatorsBioorganicmedicinal chemistry
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ttCH, a selective inhibitor of inducible nitric oxide synthase expression with antiarthritic properties

2003

In a previous work, we investigated the effects of a series of dimethoxy- and trimethoxychalcone derivatives, with various patterns of fluorination, on nitric oxide production in lipopolysaccharide-stimulated murine RAW 264.7 cells. The present study was designed to determine if 2,4,6-trimethoxy-2'-trifluoromethylchalcone (ttCH) could modulate the production of nitric oxide (NO) and/or prostaglandins in vitro and in vivo. On the mouse macrophage cell line RAW 264.7, ttCH inhibited dose-dependently NO and prostaglandin E(2) production, with IC(50) in the micromolar range. This compound had no direct inhibitory effect on inducible nitric oxide synthase (iNOS) and cyclooxygenase-2 activities. …

LipopolysaccharidesMalemedicine.medical_treatmentBlotting WesternNitric Oxide Synthase Type IIPharmacologyCarrageenanNitric OxideDinoprostoneCell LineNitric oxideMicechemistry.chemical_compoundIn vivomedicineAnimalsEdemaEnzyme InhibitorsProstaglandin E2InflammationPharmacologybiologyChemistryMacrophagesAnti-Inflammatory Agents Non-SteroidalBiological activityArthritis ExperimentalHindlimbRatsCarrageenanIsoenzymesRadiographyNitric oxide synthaseMechanism of actionBiochemistryCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesRats Inbred Lewbiology.proteinFemaleNitric Oxide Synthasemedicine.symptomProstaglandin Emedicine.drugEuropean Journal of Pharmacology
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Convergent synthesis and preliminary biological evaluations of the stilbenolignan (±)-aiphanol and various congeners

2003

Treatment of an equimolar mixture of stilbene 7 and cinnamyl alcohol 8 with silver carbonate in acetone–benzene afforded a ca. 2 : 1 : 2 : 1 mixture of the stilbenolignan (±)-aiphanol (1) and congeners 2–4 each of which show significant anti-angiogenic and COX-2 inhibitory properties.

Magnetic Resonance SpectroscopyConvergent synthesisAngiogenesis InhibitorsBiochemistryInhibitory Concentration 50chemistry.chemical_compoundStilbenesAnimalsOrganic chemistryBioassayCyclooxygenase InhibitorsPhysical and Theoretical ChemistryBenzeneAortaSilver carbonateCyclooxygenase 2 InhibitorsDose-Response Relationship DrugCinnamyl alcoholChemistryOrganic ChemistryMembrane ProteinsStereoisomerismRatsIsoenzymesEnzyme inhibitionCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesCyclooxygenase 1Org. Biomol. Chem.
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Correlation between expression of cyclooxygenase-2 and the presence of inflammatory cells in human primary hepatocellular carcinoma: Possible role in…

2005

im: To investigate the association of cyclooxygenase-2 (COX-2) expression with angiogenesis and the number and type of inflammatory cells (macrophages/Kupffer cells; mast cells) within primary hepatocellular carcinoma (HCC) tissues and adjacent non-tumorous (NT) tissues. Methods: Immunohistochemistry for COX-2, CD34, CD68 and mast cell tryptase (MCT) was performed on 14 well-characterized series of liver-cirrhosis-associated HCC patients. COX-2 expression and the number of inflammatory cells in tumor lesions and surrounding liver tissues of each specimen were compared. Moreover, COX-2, CD34 staining and the number of inflammatory cells in areas with different histological degrees within eac…

MaleLiver CancerPathologymedicine.medical_specialtyCarcinoma HepatocellularEndotheliumMacrophageAngiogenesisKupffer CellsNeovascularizationCarcinomamedicineHumansMast CellsHCCAgedInflammationbiologyNeovascularization Pathologicbusiness.industryMacrophagesLiver NeoplasmsGastroenterologyMembrane ProteinsGeneral MedicineCOX-2Middle Agedmedicine.diseasedigestive system diseasesAngiogenesimedicine.anatomical_structureMembrane proteinCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesHepatocellular carcinomabiology.proteinTumor promotionFemaleCyclooxygenaseEndothelium Vascularmedicine.symptombusiness
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Modulatory effect of bolinaquinone, a marine sesquiterpenoid, on acute and chronic inflammatory processes

2002

The marine metabolite bolinaquinone is a novel inhibitor of secretory phospholipase A(2) (sPLA(2)), with a potency on the human synovial enzyme (group II) higher than that of manoalide. This activity on the sPLA(2) was confirmed in vivo in the 8-h zymosan rat air pouch on the secretory enzyme accumulation in the pouch exudate. Additionally, bolinaquinone decreased potently the synthesis and release of leukotriene B(4) (LTB(4)) in calcimycin (A23187)-stimulated human neutrophils as a consequence of the inhibition of 5-lipoxygenase activity, as well as PGE(2) and NO production on zymosan-stimulated mouse peritoneal macrophages. This compound exerted anti-inflammatory effects by topical and or…

MaleNeutrophilsGene ExpressionNitric Oxide Synthase Type IIMarine BiologyPharmacologyBone resorptionMicechemistry.chemical_compoundManoalideIn vivoEdemamedicineAnimalsEdemaHumansRats WistarPharmacologybiologyZymosanMembrane ProteinsArthritis ExperimentalPoriferaRatsCarrageenanIsoenzymesRadiographyNitric oxide synthaseDisease Models AnimalchemistryCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesImmunologyMacrophages Peritonealbiology.proteinMolecular MedicineTumor necrosis factor alphaNitric Oxide Synthasemedicine.symptomSesquiterpenes
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A novel cyclo-oxygenase-2 inhibitor modulates catabolic and antiinflammatory mediators in osteoarthritis.

2004

ITB (6-(p-bromophenyl)amino-7-(p-chlorophenyl)indazolo[2',3':1,5]-1,2,4-triazolo[4,3-a]-1,3,5-benzotriazepine) is a novel inhibitor of cyclo-oxygenase-2 (COX-2) with antiinflammatory activity in animal models. In the present study, we investigated the effect of this compound on the production of catabolic or antiinflammatory mediators in osteoarthritis (OA) cartilage. In OA cartilage explants, ITB inhibited the production of prostaglandin E(2) (PGE(2)), tumour necrosis factor-alpha (TNF-alpha) and matrix metalloproteinase-13 (MMP-13) in a concentration-dependent manner, whereas nitrite was partially reduced. On the contrary, ITB increased the production of interleukin (IL)-10 and the expres…

MaleOxygenaseIndazolesmedicine.medical_treatmentAnti-Inflammatory AgentsOsteoarthritisPharmacologyBiochemistryOsteoarthritismedicineHumansCyclooxygenase InhibitorsProstaglandin E2AgedPharmacologyCyclooxygenase 2 InhibitorsChemistryCatabolismCartilageAnti-Inflammatory Agents Non-SteroidalInterleukinMembrane ProteinsAzepinesTriazolesmedicine.diseaseIsoenzymesInterleukin 10Cytokinemedicine.anatomical_structureCartilageBiochemistryCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesFemalemedicine.drugBiochemical pharmacology
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A polymorphism in the cyclooxygenase 2 gene as an inherited protective factor against myocardial infarction and stroke

2004

CONTEXT: Myocardial infarction (MI) and ischemic stroke are thought to be caused by matrix digestion by metalloproteinases (MMPs) leading to rupture of atherosclerotic plaques. Production of macrophage MMP-2 and MMP-9 is induced by cyclooxygenase 2 (COX-2) and prostaglandin E(2) synthesis. Although COX-2 expression may be genetically determined, the relation between COX-2 polymorphisms and the risk of MI and stroke is unclear. OBJECTIVE: To investigate the relationship between the -765G-->C polymorphism of the COX-2 gene and clinically evident plaque rupture. DESIGN, SETTING, AND PARTICIPANTS: Prospective, matched case-control study conducted between March 2002 and October 2003 among 864 pa…

MalePathologySettore MED/09 - Medicina InternaArteriosclerosisCarotid StenosiMyocardial InfarctionInfarctionProstacyclinGastroenterologyCohort StudiesCerebrovascular AccidentrteriosclerosiRisk FactorsGenotypeMedicineCarotid StenosisProspective StudiesMyocardial infarctionMembrane ProteinStrokebiologyGeneral MedicineMiddle AgedIsoenzymesStrokePhenotypeMatrix Metalloproteinase 9Matrix Metalloproteinase 2FemaleHumanmedicine.drugmedicine.medical_specialtyGenotypeArteriosclerosiInternal medicineDiabetes mellitusHumansPolymorphism Geneticbusiness.industryC-reactive proteinProstaglandin-Endoperoxide SynthaseMembrane Proteinsmedicine.diseaseEpoprostenolIsoenzymeProspective StudieAtheromaCyclooxygenase 2Prostaglandin-Endoperoxide Synthasesbiology.proteinCohort Studiebusiness
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