Search results for "endothelin-1"

showing 10 items of 40 documents

Endothelin-1 modulates the expression of adhesion molecules on fibroblast-like synovial cells (FLS)

1996

Endothelin-1 is known to possess various biological properties. In the present study we have investigated the effects of Endothelin-1 (Et-1) on the expression of adhesion molecules ICAM-1, VCAM-1 and CD-44 by fibroblast-like synoviocytes. Cultured fibroblast-like synoviocytes were treated with Et-1 in the absence or presence of C1306, a specific endothelin-A-receptor antagonist. Cell surface expression of ICAM-1, VCAM-1 and CD-44 was determined by immunofluorescence studies, Cyto-ELISA and FACS-analysis. ICAM-1, VCAM-1 and CD-44 were constitutively expressed on cultured FLS. After incubation with Et-1 the expression of ICAM-1, VCAM-1 and CD-44 increased. The level of expression of adhesion …

Endothelin Receptor Antagonistsmedicine.medical_specialtymedicine.medical_treatmentImmunologyVascular Cell Adhesion Molecule-1BiologyProinflammatory cytokineRheumatologyDownregulation and upregulationInternal medicinemedicineHumansImmunology and AllergyCells CulturedEndothelin-1Tumor Necrosis Factor-alphaCell adhesion moleculeSynovial MembraneGeneral MedicineAdhesionFibroblastsIntercellular Adhesion Molecule-1Receptor Endothelin AIn vitroCell biologyHyaluronan ReceptorsCytokinemedicine.anatomical_structureEndocrinologyCell cultureSynovial membraneCell Adhesion MoleculesInterleukin-1
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Contribution of endothelin receptors and cyclooxygenase-derivatives to the altered response of the rabbit renal artery to endothelin-1 in diabetes

2006

Abstract The influence of diabetes on regulatory mechanisms and specific receptors implicated in the response of isolated rabbit renal artery to endothelin-1 was examined. Endothelin-1 induced a concentration-dependent contraction that was less potent in arteries from diabetic rabbits than in arteries from control rabbits. Endothelium removal or NG-nitro- l -arginine (L-NOARG) enhanced contractions to endothelin-1 either in control and diabetic arteries. Indomethacin inhibited endothelin-1-induced response in control arteries, but enhanced it in diabetic arteries. In contrast to that observed in rubbed and in L-NOARG treated arteries, in the presence of indomethacin the contractile action o…

Malemedicine.medical_specialtyEndotheliumIndomethacinVasodilationNitroargininePeptides CyclicDiabetes Mellitus ExperimentalRenal ArteryInternal medicinemedicine.arteryAlloxanmedicineAnimalsCyclooxygenase InhibitorsRenal arteryReceptorAntihypertensive AgentsPharmacologyDose-Response Relationship DrugEndothelin-1biologyChemistryReceptor Endothelin AEndothelin 1Endocrinologymedicine.anatomical_structureProstaglandin-Endoperoxide SynthasesVasoconstrictioncardiovascular systembiology.proteinEndothelium VascularRabbitsCyclooxygenaseNitric Oxide Synthasemedicine.symptomEndothelin receptorVasoconstrictionEuropean Journal of Pharmacology
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Central obesity and hypertensionThe role of plasma endothelin

1996

Hypertension and central obesity are two conditions closely linked, but the mechanisms responsible for obesity-associated hypertension are still unclear. In the last few years, several studies addressed the role of endothelin-1 (ET-1) in the development and maintenance of hypertension. This study was designed to evaluate plasma ET-1 in normotensive and hypertensive central obese subjects compared with a lean healthy group. Our final goal was to analyze the relationship between plasma ET-1, blood pressure, and left ventricular structure and function in central obese subjects (both normotensives and hypertensives). ET-levels have been assessed by the radioimmunoassay method in 20 lean normote…

AdultMalemedicine.hormoneMean arterial pressuremedicine.medical_specialtyAmbulatory blood pressureMegalencephalic leukoencephalopathy with subcortical cystsRadioimmunoassayBody Mass IndexEndothelinsVentricular Dysfunction LeftHeart RateInternal medicineInternal MedicinemedicineHumansRadionuclide AngiographyEndothelin-1business.industryStroke VolumeStroke volumeBlood Pressure Monitoring Ambulatorymedicine.diseaseBody HeightObesity MorbidMean blood pressureBlood pressureEndocrinologyEchocardiographyHypertensionBody ConstitutionRegression AnalysisFemalebusinessBody mass indexAmerican Journal of Hypertension
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Mast cells promote homeostasis by limiting endothelin-1-induced toxicity

2004

Endothelin-1 (ET-1) is a 21-amino-acid peptide, derived from vascular endothelial cells, with potent vasoconstrictor activity. ET-1 has been implicated in diverse physiological or pathological processes, including the vascular changes associated with sepsis. However, the factors that regulate ET-1-associated toxicity during bacterial infections, or in other settings, are not fully understood. Both the pathology associated with certain allergic and autoimmune disorders, and optimal host defence against bacterial and parasitic infections are mediated by mast cells. In vitro, mast cells can produce ET-1 (ref. 11), undergo ET-1-dependent and endothelin-A receptor (ET(A))-dependent activation, a…

DiarrheaProteasesDrug-Related Side Effects and Adverse ReactionsCell SurvivalPeritonitisBiologyPeptides CyclicCell DegranulationBody TemperatureMiceChymasesIn vivomedicineAnimalsHomeostasisMast CellsReceptorEgtazic AcidMice KnockoutMultidisciplinaryEndothelin-1Stem CellsBody WeightSerine EndopeptidasesEndogenous mediatorMast cellEndothelin 1In vitroCell biologyMice Inbred C57BLSurvival RateProto-Oncogene Proteins c-kitmedicine.anatomical_structureMutationImmunologyFemaleOligopeptidesInjections IntraperitonealHomeostasisNature
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Anatomical differences in responsiveness to vasoconstrictors in the mesenteric veins from normal and portal hypertensive rats

1996

The present study evaluates the effects of pre-hepatic portal hypertension, induced in rats by partial portal vein ligation, on the responsiveness of rostral (proximal) and caudal (distal) rings from the mesenteric vein. The anatomical origin of the sample influenced the response to vasoconstrictors in sham-operated animals, and this pattern of reactivity was specifically modified in portal-ligated rats. In veins from sham-operated rats, contraction induced by a submaximal concentration of KCl (60 mM) was greater in proximal than in distal rings. Vasopressin and 5-hydroxytryptamine contracted mainly distal rings, methoxamine showed a greater effect on proximal rings, and endothelin-1 and an…

Malemedicine.medical_specialtyVasopressinContraction (grammar)VasopressinsPropranololIliac VeinIn Vitro TechniquesMuscle Smooth VascularMesenteric VeinMethoxaminePotassium ChlorideNitric oxideRats Sprague-Dawleychemistry.chemical_compoundMesenteric VeinsInternal medicineHypertension PortalmedicineAnimalsVasoconstrictor AgentsPharmacologyEndothelin-1business.industryGeneral Medicinemedicine.diseaseRatsAtropineEndocrinologychemistrycardiovascular systemPortal hypertensionbusinessmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Cigarette smoke exposure up-regulates endothelin receptor B in human pulmonary artery endothelial cells: molecular and functional consequences

2010

BACKGROUND AND PURPOSEPulmonary arteries from smokers and chronic obstructive pulmonary disease patients show abnormal endothelium-dependent vascular reactivity. We studied the effect of cigarette smoke extract (CSE) on endothelin receptor B (ETB) expression in human pulmonary artery endothelial cells (HPAECs) and its role in endothelial dysfunction.EXPERIMENTAL APPROACHETB receptor expression was measured by real time RT-PCR, Western blot and immunofluorescence. Cell contraction, intracellular Ca2+, F/G-actin, RhoA activity, myosin light chain phosphorylation, ET, NO, thromboxane (Tx)A2 and reactive oxygen species (ROS) were measured by traction microscopy, fluorescence microscopy, phalloi…

MalePulmonary ArteryNitric OxideTransfectionMuscle Smooth Vascularendothelial dysfunctionendothelin receptor Bpulmonary artery endothelial cellsSmokeTobaccoHumansRNA Small InterferingCells CulturedAgedSulfonamidesrho-Associated KinasesEndothelin-1bosentancigarette smokeEndothelial CellsResearch PapersReceptor Endothelin BUp-RegulationThromboxane B2FemaleEndothelium VascularReactive Oxygen Species
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Glyceraldehyde-3-phosphate dehydrogenase regulates endothelin-1 expression by a novel, redox-sensitive mechanism involving mRNA stability

2008

17 pages.-- PMID: 18809573 [PubMed].-- Printed version published on Dec 2008.

Untranslated regionUmbilical VeinsRNA StabilityRNA StabilityGlyceraldehyde-3'-phosphate dehydrogenase (GAPDH)Plasma protein bindingstomatognathic systemHumansmRNA stabilityS-Glutathionylation3' Untranslated RegionsMolecular BiologyGlyceraldehyde 3-phosphate dehydrogenaseRegulation of gene expressionMessenger RNAEndothelin-1biologyThree prime untranslated regionGlyceraldehyde-3-Phosphate DehydrogenasesArticlesCell BiologyGlutathioneOxidative StressGene Expression RegulationBiochemistryEndothelin-1 (ET-1)biology.proteinOxidation-ReductionProtein Binding
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Plasma endothelin-1 levels in patients with angina pectoris and normal coronary angiograms.

1998

Some patients with typical angina and electrocardiographic evidence of ischemia have normal coronary angiograms. These patients have a reduced coronary flow reserve and abnormal endothelium-dependent vasodilator responses; this syndrome is known as microvascular angina. Among endothelium-derived peptides, endothelin-1 (ET-1) is a potent vasoconstrictor and an important modulator of microvascular function.Plasma ET-1 was measured in 13 patients with typical angina, instrumental evidence of ischemia, and normal arteriograms and in 20 normal control subjects.Mean concentration of ET-1 was 2.89+/-1.24 pmol/L in patients with angina and normal angiograms and 1.99+/-0.81 pmol/L in normal control …

AdultMalemedicine.medical_specialtyIschemiaRadioimmunoassayVasodilationBlood PressureCoronary AngiographyAngina PectorisAnginaElectrocardiographyInternal medicinemedicineHumanscardiovascular diseasesEndothelial dysfunctionRetrospective Studiesmedicine.diagnostic_testEndothelin-1business.industryMicroangiopathyCoronary flow reserveStroke VolumeMiddle Agedmedicine.diseaseEndothelin 1Vasoconstrictioncardiovascular systemCardiologyExercise TestFemaleCardiology and Cardiovascular MedicinebusinessElectrocardiographyAmerican heart journal
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Changes of neurohumoral parameters and endothelin-1 in response to exercise in patients with mild to moderate congestive heart failure

1998

Plasma endothelin levels are increased in patients with moderate and severe CHF. Conflicting data exist about the endothelin-1 (ET) level in patients with mild to moderate CHF and the effect of maximal exercise on plasma ET levels.We determined the plasma levels of ET and various neurohumoral parameters in 93 patients with CHF in functional class II and III of the NYHA classification at rest and after maximal bicycle exercise. Baseline ET level was increased compared to an age-matched healthy volunteer group (6.95+/-0.31 vs 3.29+/-0.17 pg/ml, mean+/-S.E.M., P0.05), without significant differences between NYHA class II and III patients. Maximal exercise did not increase the ET level. In cont…

Malemedicine.medical_specialtyHeart diseaseneurohumoral activationheart failurePeptide hormonePLASMA ENDOTHELINInternal medicinemedicineHumanscardiovascular diseasesExercise physiologyExerciseAgedNeurotransmitter AgentsEndothelin-1BLOOD-FLOWbusiness.industryBlood flowMiddle Agedmedicine.diseaseEndothelin 1Pathophysiologyexercise capacityEndocrinologyHeart failureExercise TestCardiologycardiovascular systemFemaleCardiology and Cardiovascular MedicineEndothelin receptorbusinessendothelin
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Lactoferricin B-derived peptides with inhibitory effects on ECE-dependent vasoconstriction

2010

Endothelin-converting enzyme (ECE), a key peptidase in the endothelin (ET) system, cleaves inactive big ET-1 to produce active ET-1, which binds to ET(A) receptors to exert its vasoconstrictor and pressor effects. ECE inhibition could be beneficial in the treatment of hypertension. In this study, a set of eight lactoferricin B (LfcinB)-derived peptides, previously characterized in our laboratory as angiotensin-converting enzyme (ACE) inhibitory peptides, was examined for their inhibitory effects on ECE. In vitro inhibitory effects on ECE activity were assessed using both the synthetic fluorogenic peptide substrate V (FPS V) and the natural substrate big ET-1. To study vasoactive effects, an…

MalePhysiologyPeptideEndothelin-Converting EnzymesPharmacologyInhibitory postsynaptic potentialBiochemistryCellular and Molecular NeuroscienceEndocrinologymedicineAnimalsAspartic Acid EndopeptidasesHumansReceptorchemistry.chemical_classificationEndothelin-1ChemistryMetalloendopeptidasesReceptor Endothelin AIn vitroIntracellular signal transductionLactoferrinBiochemistryVasoconstrictionHypertensionRabbitsmedicine.symptomPeptidesEndothelin receptorVasoconstrictionEx vivoSignal TransductionPeptides
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