Search results for "enzyme inhibitors"

showing 10 items of 559 documents

Experimental diabetes induces hyperreactivity of rabbit renal artery to 5-hydroxytryptamine.

2002

Abstract The influence of diabetes on the response of isolated rabbit renal arteries to 5-hydroxytryptamine (5-HT) was examined. 5-HT induced a concentration-related contraction that was higher in arteries from diabetic rabbits than in arteries from control rabbits. Endothelium removal did not significantly modify 5-HT contractions in arteries from control rabbits but enhanced the response to 5-HT in arteries from diabetic rabbits. Incubation with N G -nitro- l -arginine ( l -NA) enhanced contractions to 5-HT in arteries from control and diabetic rabbits. In arteries with endothelium, this l -NA enhancement was lower in diabetic rabbits than in control rabbits. In arteries without endotheli…

Malemedicine.medical_specialtySerotoninContraction (grammar)EndotheliumIndomethacinIn Vitro TechniquesNitroarginineDiabetes Mellitus Experimentalchemistry.chemical_compoundNitroarginineRenal ArteryInternal medicinemedicine.arteryAlloxanmedicineAnimalsRenal arteryEnzyme InhibitorsPharmacologyDose-Response Relationship Drugbusiness.industryProstanoidDrug Synergismmedicine.anatomical_structureEndocrinologychemistryVasoconstrictionCirculatory systemRabbitsmedicine.symptomNitric Oxide SynthasebusinessVasoconstrictionBlood vesselEuropean journal of pharmacology
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Phosphodiesterase inhibitors suppress alpha2-adrenoceptor-mediated 5-hydroxytryptamine release from tracheae of newborn rabbits.

1998

The outflow of 5-hydroxytryptamine (5-HT) from isolated tracheae of newborn rabbits was determined by high pressure liquid chromatography with electrochemical detection. This 5-HT outflow reflects release from neuroendocrine epithelial cells of the airway mucosa, as previously shown. Phenylephrine, via alpha2B-adrenoceptors, caused a transient increase in 5-HT outflow, maximally by about 250%, an effect mediated by liberation of intracellular Ca2+, as previously shown. The non-selective phosphodiesterase inhibitor 2-isobutyl-1-methylxanthine (IBMX) concentration-dependently inhibited phenylephrine-induced 5-HT release (completely at 100 microM, IC50: 1.3 microM). Likewise, benzafentrine (in…

Malemedicine.medical_specialtySerotoninIBMXSiguazodanPhosphodiesterase InhibitorsPhosphodiesterase 3BiologyEpitheliumchemistry.chemical_compoundCyclic nucleotidePhenylephrineInternal medicine1-Methyl-3-isobutylxanthineQuinoxalinesmedicineAnimalsPhosphodiesterase inhibitorEnzyme InhibitorsPhenylephrineRolipramPharmacologyOxadiazolesPhosphodiesteraseTracheaEndocrinologychemistryAnimals NewbornFemaleRabbitsReceptors Adrenergic beta-2Adrenergic alpha-Agonistsmedicine.drugEuropean journal of pharmacology
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The effects of enzalutamide and abiraterone on skeletal related events and bone radiological progression free survival in castration resistant prosta…

2017

Two new drugs, the CYP17 inhibitor abiraterone acetate and the androgen receptor (AR) antagonist enzalutamide, have recently shown to prolong OS prior chemotherapy or in docetaxel treated mCRPC patients, using steroidal therapy or placebo as control group. Updated analyses underlined the role of these new agents on two prostate-specific endpoints as radiographic progression-free survival (rPFS) and time to first skeletal-related event (tSRE). On the basis of these reports, we made an indirect comparison between abiraterone and enzalutamide. We obtained a clinically but not significant difference favouring enzalutamide over abiraterone in terms of rPFS (HR 0.48, 95% CI 0.22–1.02). No signi…

Malemedicine.medical_specialtySettore MED/06 - Oncologia Medicamedicine.medical_treatmentUrologyAbiraterone AcetateBone NeoplasmsAbiraterone; Enzalutamide; mCRPC; rPFS; tSRE; Hematology; Oncology; Geriatrics and GerontologyPlaceboDisease-Free Survivallaw.invention03 medical and health scienceschemistry.chemical_compoundProstate cancer0302 clinical medicineRandomized controlled triallawNitrilesPhenylthiohydantoinEnzalutamideAndrogen Receptor AntagonistsMedicineEnzalutamideCytochrome P-450 Enzyme InhibitorsHumans030212 general & internal medicineProgression-free survivalAbirateronetSRERandomized Controlled Trials as TopicChemotherapybusiness.industryAbiraterone acetateHematologymCRPCmedicine.diseaseProstatic Neoplasms Castration-ResistantTreatment OutcomechemistryDocetaxelrPFSOncology030220 oncology & carcinogenesisBenzamidesDisease ProgressionGeriatrics and Gerontologybusinessmedicine.drugCritical reviews in oncology/hematology
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Characterization of thyroid hormone sulfotransferases

1998

Sulfation is an intriguing pathway of thyroid hormone metabolism since it facilitates the degradation of the hormone by the type I deiodinase (D1). This study reports the preliminary characterization of iodothyronine sulfotransferase activities of rat and human liver cytosol and recombinant rSULT1C1 and hSULT1A1 isoenzymes. All these enzyme preparations catalyzed the sulfation of--in decreasing order of efficiency--3,3'-diiodothyronine (3,3'-T2)3,3',5-triiodothyronine (T3) approximately 3,3',5'-triiodothyronine (rT3)thyroxine (T4). 3,3'-T2 sulfotransferase activity was found to be higher in male than in female rat liver, which has also been shown by others for the expression of rSULT1A1 and…

Malemedicine.medical_specialtySulfotransferaseThyroid HormonesDeiodinaseToxicologyIsozymeSulfationCytosolPhenolsInternal medicinemedicineAnimalsHumansEnzyme InhibitorsRats Wistarchemistry.chemical_classificationbiologyChemistrySulfatesThyroidGeneral MedicineRatsIsoenzymesCytosolKineticsEndocrinologymedicine.anatomical_structureEnzymeBiochemistryLiverbiology.proteinFemaleSulfotransferasesHormone
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Effects of clinical and laboratory variables at admission and of in-hospital treatment with cardiovascular drugs on short term prognosis of ischemic …

2011

Abstract Introduction No information exists, to our knowledge, about the possible role of cardiovascular drug administration in the acute phase of ischemic stroke and possible effects on stroke outcome. The aim of our study was to evaluate the relationship between in-hospital treatment with cardiovascular drugs in patients with acute ischemic stroke and some outcome indicators. Methods and Results 1096 subjects enrolled in the GIFA study, who had a main discharge diagnosis of ischemic stroke represent the final sample. Drugs considered for the analysis were the following: ACE-inhibitors (ACEI), angiotensin II receptor blockers (ARBs), statins, calcium-channel-blockers (CCBs), antiplatelet (…

Malemedicine.medical_specialtyTime FactorsEndocrinology Diabetes and MetabolismHypercholesterolemiaMedicine (miscellaneous)Angiotensin II Receptor BlockersCharlson indexAngiotensin-Converting Enzyme InhibitorsComorbidityBrain IschemiaInternal medicineActivities of Daily LivingmedicineHumansIn patientcardiovascular diseasesStrokeGeriatric AssessmentAgedRetrospective StudiesNutrition and Dieteticsbusiness.industryCardiovascular AgentsHeparinmedicine.diseaseCalcium Channel BlockersPrognosisStrokeHospital treatmentItalyIschemic strokeHypertensionPhysical therapyFunctional statusFemaleCardiology and Cardiovascular MedicinebusinessCognition DisordersPlatelet Aggregation Inhibitorsmedicine.drugNutrition, metabolism, and cardiovascular diseases : NMCD
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Acute relaxant effects of 17-beta-estradiol through non-genomic mechanisms in rabbit carotid artery.

2002

Estrogens could play a cardiovascular protective role not only by means of systemic effects but also by means of direct effects on vascular structure and function. We have studied the acute effects and mechanisms of action of 17-beta-estradiol on vascular tone of rabbit isolated carotid artery. 17-Beta-estradiol (10, 30, and 100 microM) elicited concentration-dependent relaxation of 50 mM KCl-induced active tone in male and female rabbit carotid artery. The stereoisomer 17-alpha-estradiol showed lesser relaxant effects in male rabbits. Endothelium removal did not modify relaxation induced by 17-beta-estradiol. The NO synthase inhibitor L-NAME (100 microM) only reduced significantly relaxati…

Malemedicine.medical_specialtyVascular smooth muscleContraction (grammar)Potassium ChannelsCharybdotoxinEndotheliumMuscle RelaxationClinical BiochemistryNicardipineEstrogen receptorCycloheximideBiochemistrychemistry.chemical_compoundCalcium ChlorideNicardipineEndocrinologyInternal medicinemedicineAnimalsChannel blockerEnzyme InhibitorsMolecular BiologyPharmacologyEstradiolOrganic ChemistryCalcium Channel BlockersEndocrinologymedicine.anatomical_structureCarotid ArteriesNG-Nitroarginine Methyl EsterchemistryPotassiumCalciumFemaleCalcium ChannelsEndothelium VascularRabbitsNitric Oxide Synthasemedicine.drugSteroids
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Increased responsiveness of human pulmonary arteries in patients with positive bronchodilator response.

1996

Abstract 1. The effects of noradrenaline, endothelin-1, acetylcholine and sodium nitroprusside were studied in isolated pulmonary arteries obtained from 14 patients undergoing lobectomy for lung carcinoma. Seven patients had shown increased response to a bronchodilator test prior to operation. In the remaining patients (control) the bronchodilator test was negative. 2. Artery rings from patients with a positive bronchodilator response showed greater contraction to noradrenaline (pD2 = 6.44 +/- 0.1; Emax = 93 +/- 9% of response to 100 mM KCl) and endothelin-1 (pD2 = 8.92 +/- 0.1; Emax = 130 +/- 16%) than the rings from control patients (pD2 = 6.04 +/- 0.08; Emax = 56 +/- 8% for noradrenaline…

Malemedicine.medical_specialtyVascular smooth musclemedicine.drug_classVasodilator AgentsVasodilationIn Vitro TechniquesPulmonary ArteryInternal medicineBronchodilatormedicineHumansVasoconstrictor AgentsEndotheliumEnzyme InhibitorsAgedPharmacologybusiness.industryBronchodilator Agentsmedicine.anatomical_structureNG-Nitroarginine Methyl EsterBronchodilator AgentsDilatorAnesthesiaCardiologySodium nitroprussidemedicine.symptomBronchial HyperreactivityNitric Oxide SynthasebusinessVasoconstrictionArterymedicine.drugResearch ArticleBritish journal of pharmacology
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Role of nitric oxide in pheromone-mediated intraspecific communication in mice

2009

Nitric oxide is known to take part in the control of sexual and agonistic behaviours. This is usually attributed to its role in neural transmission in the hypothalamus and other structures of the limbic system. However, socio-sexual behaviours in rodents are mainly directed by chemical signals detected by the vomeronasal system, and nitric oxide is abundant in key structures along the vomeronasal pathway. Thus, here we check whether pharmacological treatments interfering with nitrergic transmission could affect socio-sexual behaviour by impairing the processing of chemical signals. Treatment with an inhibitor of nitric oxide synthesis (Nomega-Nitro-l-arginine methyl ester hydrochloride, L-N…

Malemedicine.medical_specialtyVomeronasal organExperimental and Cognitive PsychologyStimulationBiologyNitric OxideChoice Behaviorintraspecific communicationNitric oxideMiceBehavioral Neurosciencechemistry.chemical_compoundDiscrimination PsychologicalLimbic systemnitric oxideInternal medicinemedicineAnimalsEnzyme InhibitorsSex AttractantsInstinctAnalysis of VarianceDose-Response Relationship DrugBedding and LinensOlfactory PerceptionStimulation ChemicalAnimal CommunicationNG-Nitroarginine Methyl Estermedicine.anatomical_structureEndocrinologychemistryHypothalamusrodentsSex pheromonePheromoneSex AttractantsFemalePhysiology & Behavior
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Role of PCSK9 in the course of ejection fraction change after ST-segment elevation myocardial infarction: a pilot study

2020

Altres ajuts: Conselleria d'Educació, Investigació, Cultura i Esport GV/2018/116 Proprotein convertase subtilisin/kexin type 9 (PCSK9) has emerged as a therapeutic target for reducing plasma low-density lipoprotein cholesterol. Beyond lipid control, recent findings suggest a deleterious effect of this protein in the pathogenesis of postmyocardial infarction left ventricle remodelling and heart failure-related complications. The aim of this study was to assess the relationship between circulating PCSK9 and 6 month cardiac magnetic resonance imaging-derived left ventricular ejection fraction (LVEF) after a first ST-segment elevation myocardial infarction (STEMI). We prospectively evaluated 40…

Malemedicine.medical_specialtylcsh:Diseases of the circulatory (Cardiovascular) systemLeft ventricular ejection fractionCardiac magnetic resonancemedicine.medical_treatmentMyocardial InfarctionInfarctionAngiotensin-Converting Enzyme InhibitorsPilot Projects030204 cardiovascular system & hematologyVentricular Function LeftPCSK9Angiotensin Receptor Antagonists03 medical and health sciences0302 clinical medicineCardiac magnetic resonance imagingOriginal Research ArticlesInternal medicinemedicineST‐segment elevation myocardial infarctionHumansST segmentOriginal Research Article030212 general & internal medicineMyocardial infarctioncardiovascular diseasesAgedKillip classEjection fractionmedicine.diagnostic_testbusiness.industryPercutaneous coronary interventionStroke VolumeMiddle Agedmedicine.diseaseST-segment elevation myocardial infarctionlcsh:RC666-701Heart failureCardiologyST Elevation Myocardial InfarctionProprotein Convertase 9Cardiology and Cardiovascular Medicinebusiness
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Evidence for a role of inducible nitric oxide synthase in gastric relaxation of mdx mice

2006

Alterations of gastric mechanical activity have been reported in mdx mouse, animal model for Duchenne muscular dystrophy. This study examined if alterations in the vasoactive intestinal polypeptide (VIP) system are present in mdx stomach. Gastric mechanical activity was recorded in vitro as changes of endoluminal pressure and neurally or pharmacologically evoked relaxations were analysed in mdxvs normal stomach. Reverse-transcription polymerase chain reaction was used to detect inducible nitric oxide synthase (iNOS) expression. Relaxations to sodium nitroprusside in mdx stomach showed no difference in comparison with normal preparations. In normal stomach, VIP produced relaxation, which was…

Malemedicine.medical_specialtymdx mousePhysiologyMuscle RelaxationVasoactive intestinal peptideNitric Oxide Synthase Type IIStimulationDUCHENNES MUSCULAR-DYSTROPHYSettore BIO/09 - FisiologiaNitric oxidechemistry.chemical_compoundMiceOrgan Culture TechniquesInternal medicineQuinoxalinesmedicineAnimalsRNA MessengerEnzyme InhibitorsReceptorOxadiazolesbiologyEndocrine and Autonomic SystemsReverse Transcriptase Polymerase Chain ReactionStomachStomachGastroenterologySMOOTH-MUSCLE CELLSMuscle SmoothPEPTIDE RELEASENitric oxide synthaseMuscular Dystrophy DuchenneDisease Models AnimalEndocrinologymedicine.anatomical_structureNG-Nitroarginine Methyl Esterchemistrybiology.proteinMice Inbred mdxReceptors Vasoactive Intestinal PeptideSodium nitroprussideIminesmedicine.drugVasoactive Intestinal Peptide
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