Search results for "epilepsy"

showing 10 items of 420 documents

Comparison of effects of Valproate and the newly synthesized Valproil-L-Tryptophan on epileptiform activity in rat brain slices

2008

Valproic acidepilepsy
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Respuesta cardiovascular al estrés en pacientes con epilepsia farmacorresistente: datos preliminares

2021

El estrés es un factor de riesgo para las enfermedades cardiovasculares en la población general. La epilepsia se ha considerado un modelo adecuado de estrés crónico, con mayor incidencia de mortalidad cardiovascular que la población general. Este estudio proporciona datos preliminares sobre la respuesta cardiovascular ante un estresor cognitivo y una evaluación neuropsicológica en pacientes con epilepsia, explorando las relaciones entre la respuesta cardiovascular y el rendimiento cognitivo dependiendo del hemisferio responsable de la epilepsia. Treinta y cinco pacientes con epilepsia farmacorresistente, 17 con el área epileptógena(AE) en el hemisferio izquierdo (LH) y 18 con AE en el hemis…

Variabilidad cardíaca:1 - Filosofía y psicología::159.9 - Psicología [CDU]Heart rateDrug-resistant epilepsyEstrés agudoFrecuencia cardíacaAcute stressRespuesta cardiovascularHeart rate variabilityEpilepsia farmacorresistenteCardiovascular response
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An N-terminal deletion variant of HCN1 in the epileptic WAG/Rij strain modulates HCN current densities.

2015

Rats of the Wistar Albino Glaxo/Rij (WAG/Rij) strain show symptoms resembling human absence epilepsy. Thalamocortical neurons of WAG/Rij rats are characterized by an increased HCN1 expression, a negative shift in Ih activation curve, and an altered responsiveness of Ih to cAMP. We cloned HCN1 channels from rat thalamic cDNA libraries of the WAG/Rij strain and found an N-terminal deletion of 37 amino acids. In addition, WAG-HCN1 has a stretch of six amino acids, directly following the deletion, where the wild-type sequence (GNSVCF) is changed to a polyserine motif. These alterations were found solely in thalamus mRNA but not in genomic DNA. The truncated WAG-HCN1 was detected late postnatal …

WAG/Rij ratThalamusXenopusI hIhlcsh:RC321-571thalamocortical relay neuronsCellular and Molecular Neurosciencelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryMolecular Biologyhealth care economics and organizationsOriginal Researchchemistry.chemical_classificationMessenger RNAbiologycDNA libraryKinaseChemistrybiology.organism_classificationMolecular biologyHCNAmino acidgenomic DNAabsence epilepsyBiochemistryHeterologous expressionhuman activitiesNeuroscienceFrontiers in molecular neuroscience
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Weakness and focal sensory deficits in the postictal state.

2010

Postictal motor deficits may occur in patients following partial and generalized tonic-clonic seizures. Frequency is unclear, epidemiology being hampered by heterogeneous populations and variable methods of detection. Postictal paresis may affect any body part, may be bilateral, and may occur more frequently in seizures involving the sensorimotor cortex. Duration varies depending on the precise mode of testing from a few minutes to 36 hours. Sensory deficits following seizures have been rarely reported but may be missed if not specifically tested for. The lateralizing value of postical paresis is high (>90%), pointing to a seizure origin in the opposite frontal lobe. Postictal paresis often…

Weaknessmedicine.medical_specialtyBrainSensory systemAudiologymedicine.diseaseLesionBehavioral NeuroscienceEpilepsymedicine.anatomical_structureNeurologyFrontal lobeSeizuresAnesthesiamedicineHumansParalysisNeurology (clinical)medicine.symptomPsychologyParesisMotor cortexPostictal stateEpilepsybehavior : EB
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Age-dependent epileptic encephalopathy associated with an unusual co-occurrence of ZEB2 and SCN1A variants.

2020

Mowat-Wilson syndrome is a genetic disorder associated with a variable phenotype including peculiar facial features associated with intellectual disability, epilepsy, language impairment, and multiple congenital anomalies caused by heterozygous mutation of the ZEB2 gene. The ZEB2 protein is a complex transcription factor that encompasses multiple functional domains that interact with the regulatory regions of target genes including those involved in brain development. Recently, it has been documented that ZEB2 regulates the differentiation of interneuron progenitors migrating from the medial ganglionic eminence to cortical layers by repression of the Nkx2-1 homeobox transcription factor. It…

ZEB2genotype-phenotype correlationSettore MED/38 - Pediatria Generale E SpecialisticaSettore M-PSI/08 - Psicologia ClinicaIntellectual DisabilityHumansMowat-Wilson syndromeEEGgenotype-phenotype correlationSCN1AHirschsprung DiseaseEEGChildGenetic Association StudiesZEB2Zinc Finger E-box Binding Homeobox 2EpilepsyEEG; epilepsy; GABAergic interneurons; genotype-phenotype correlation; Mowat-Wilson syndrome; SCN1A; ZEB2FaciesElectroencephalographySettore MED/39 - Neuropsichiatria InfantileGABAergic interneuronsMowat-Wilson syndromeepilepsyNAV1.1 Voltage-Gated Sodium ChannelGABAergic interneuronsMicrocephalySettore MED/26 - NeurologiaFemaleEpileptic disorders : international epilepsy journal with videotape
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Zinc Finger Proteins in Neuro-Related Diseases Progression

2021

Zinc finger proteins (ZNF) are among the most abundant proteins in eukaryotic genomes. It contains several zinc finger domains that can selectively bind to certain DNA or RNA and associate with proteins, therefore, zinc finger proteins can regulate gene expression at the transcriptional and translational levels. In terms of neurological diseases, numerous studies have shown that many zinc finger proteins are associated with neurological diseases. The purpose of this review is to summarize the types and roles of zinc finger proteins in neuropsychiatric disorders. We will describe the structure and classification of zinc finger proteins, then focus on the pathophysiological role of zinc finge…

Zinc fingerGeneral NeuroscienceRNANeurosciences. Biological psychiatry. Neuropsychiatryautism spectrum disorderReviewBiologyneuro-related diseasesGenomeCell biologyschizophreniabody regionschemistry.chemical_compoundzinc finger proteinschemistryIschemic strokeGene expressionischemic strokeepilepsyAlzheimer’s diseaseDNARC321-571NeuroscienceFrontiers in Neuroscience
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Imaging and biochemical biomarkers of psychiatric comorbidities in a rat epilepsy model

2018

Aims: Biomarkers of epilepsy-associated psychiatric comorbidities represent a powerful tool for an early diagnosis and personalized therapy in patients with epilepsy. Here, in order to identify new candidates as biomarkers of epilepsy comorbidities, alterations in brain metabolic activity and serotonergic neurotransmission in a rat post-status epilepticus model were explored and cross-correlated with alterations in various neurobehavioral and biochemical parameters. Methods: Status epilepticus was induced in twelve female Sprague Dawley rats by lithium chloride-pilocarpine treatment. µPET analysis to determine [18F]FDG uptake and [18F]MPPF binding were performed in the chronic phase of epil…

[(18)F]MPPFBehaviorBDNFEpilepsyPETAnimal model[(18)F]FDG
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Hsp60 response in experimental and human temporal lobe epilepsy due to hyppocampal sclerosis

2015

Hsp60 is widely distributed in the brain, and its alteration has been involved in different neurological disorders. Epilepsy is considered one of the most common neurological disorders and typically involves the hippocampal formation. Compelling evidence describes a role of mitochondria, oxidative stress and both innate and adaptive immunity during epileptogenesis in temporal lobe epilepsy due to hyppocampal sclerosis (TLE-HS). Here, we investigate the Hsp60 involvement in experimental and human epilepsy. Firstly, expression and distribution of Hsp60 in epileptic hippocampi of a rat model of temporal lobe epilepsy (TLE), based on the phenomenon of maximal dentate gyrus activation (MDA), usi…

animal structuresTemporal lobe epilepsy; Hsp60; stress response; hippocampus.nervous systemSettore BIO/16 - Anatomia UmanafungiTemporal lobe epilepsy Hsp60 stress response hippocampus.chemical and pharmacologic phenomenanervous system diseases
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Cannabinoid-‐mediated modulation of hippocampal hyperexcitability: focus on the interplay with nitrergic system in different rat models of temporal l…

2015

Temporal lobe epilepsy (TLE) is the most common type of partial complex seizure in adulthood [1]. Within the framework of hyperexcitability, growing interest has risen on the impact of cannabinoids on the control of paroxysmal phenomena [2], despite their reputation as psychotropic substances with addictive properties [3; 4]. In this regard, it was reported that the on-demand production of endocannabinoids from over-activated postsynaptic cells inhibits neurotransmitter release, hence protecting against excitotoxicity in the hippocampus [5; 6]. Nonetheless, the potential anticonvulsant action of cannabinoids has not been fully addressed. Indeed, CB-mediated effects in animal models are attr…

cannabinoids nitric oxide epilepsy electrophysiology behaviour.Settore BIO/09 - Fisiologia
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Cannabis Influences the Putative Cytokines-Related Pathway of Epilepsy among Egyptian Epileptic Patients

2019

The study aims to investigate: (1) the prevalence of cannabis among epileptic patients seen at Mansoura University Hospital, (2) serum levels and gene expression of cytokines in epilepsy patients and the controls. and (3) the possibility that cannabis use affects the cytokine levels in epilepsy patients, triggering its future use in treatment. We recruited 440 epilepsy patients and 200 controls matched for age, gender, and ethnicity. Of the epileptic patients, 37.5% demonstrated lifetime cannabis use with a mean duration of 15 &plusmn

cannabismedicine.medical_specialtypurified cannabidiol (cbd)inflammatory cytokinesmedicine.medical_treatmenttreatment-resistant epilepsyArticlelcsh:RC321-571Proinflammatory cytokine03 medical and health sciencesEpilepsy0302 clinical medicineInternal medicinemedicineanticonvulsant drugslcsh:Neurosciences. Biological psychiatry. Neuropsychiatryseizures030304 developmental biology0303 health sciencesbiologybusiness.industryGeneral NeuroscienceInterleukintetrahydrocannabinol (thc)Cannabis useUniversity hospitalbiology.organism_classificationmedicine.diseaseCytokineepilepsyTumor necrosis factor alphaCannabisbusiness030217 neurology & neurosurgeryBrain Sciences
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