Search results for "expression"

showing 10 items of 5168 documents

Power training and postmenopausal hormone therapy affect transcriptional control of specific co-regulated gene clusters in skeletal muscle

2010

At the moment, there is no clear molecular explanation for the steeper decline in muscle performance after menopause or the mechanisms of counteractive treatments. The goal of this genome-wide study was to identify the genes and gene clusters through which power training (PT) comprising jumping activities or estrogen containing hormone replacement therapy (HRT) may affect skeletal muscle properties after menopause. We used musculus vastus lateralis samples from early stage postmenopausal (50–57 years old) women participating in a yearlong randomized double-blind placebo-controlled trial with PT and HRT interventions. Using microarray platform with over 24,000 probes, we identified 665 diffe…

AgingCandidate geneTranscription GeneticvaihdevuodetmenopaussiBioinformaticsEstrogen deprivation0302 clinical medicineGene expressionestrogenTranscriptional regulation0303 health sciencesEstrogen Replacement TherapyGeneral MedicineMiddle AgedestrogeeniPostmenopausemedicine.anatomical_structureFemalevoimaharjoitteluMenopausemedicine.symptomTranscriptome-wide studymedicine.medical_specialtyPlyometric trainingmedicine.drug_classBiologyArticletranskriptomin laajuuinen tutkimus03 medical and health sciencesplyometrinen harjoitteluInternal medicinemedicineHumansSkeletal muscle characteristicsKEGGMuscle SkeletalExerciseGene030304 developmental biologyhormonikorvaushoitoSkeletal muscleMuscle weaknessdeprivaatioPower trainingAgeingEndocrinologyluurankolihaksetHormone replacement therapyEstrogenGeriatrics and Gerontology030217 neurology & neurosurgeryAGE
researchProduct

Living autobiographically: Concepts of aging and artistic expression in painting and modern dance.

2016

This article discusses the ways in which artists have incorporated or failed to incorporate the aging process of their bodies into their art. Using Russian ballet dancer Mikhail Baryshnikov and the French painter Claude Monet as cases in point, we explore situations in which physical changes brought about by aging compromises artists' ability to engage with their artistic medium. Connecting Monet's oeuvre and Baryshnikov's dance performances to life writing accounts, we draw on John Paul Eakin's concept of "living autobiographically": In this vein, life writing research does not only have to take into account concepts of identity as they emerge from life writing narratives, but it also need…

AgingDanceAnthropologyMedicine in the ArtsIdentity (social science)03 medical and health sciences030502 gerontologyHumansNarrativeSociologyDancingPaintingHealth Policy06 humanities and the artsGeneral Medicine060202 literary studiesModern danceLife writingIssues ethics and legal aspectsAutobiographies as TopicExpression (architecture)Aesthetics0602 languages and literaturePaintingsBallet dancer0305 other medical scienceArtJournal of aging studies
researchProduct

Immunological and immunogenetic markers in sporadic Alzheimer’s disease

2006

Background: Common polymorphisms of genes controlling inflammation-modulating cytokines and acute-phase proteins which play important roles in the pathogenesis of Alzheimer''s disease (AD) have been shown to be associated with AD. Aims: The immunological and immunogenetic markers potentially useful for the AD risk evaluation and diagnosis are briefly reviewed. Conclusion: The state-of-the-art of immunological and immunogenetic markers of AD indicates that new tools and strategies are necessary to identify gene products useful as diagnostic tools.

AgingDiseaseImmunogeneticsDiagnostic toolsProteomicsPathogenesisApolipoproteins EAlzheimer DiseaseHumansMedicineOligonucleotide Array Sequence AnalysisInflammationAlzheimer’s disease cytokines immunogenetics inflammation proteomicsPolymorphism GeneticGeriatrics gerontologybusiness.industryDNARisk evaluationGene Expression RegulationPositron-Emission TomographyImmunologyCytokinesMicrogliaGeriatrics and GerontologybusinessBiomarkersAcute-Phase ProteinsAging Clinical and Experimental Research
researchProduct

Function of Glia in Aging and the Brain Diseases.

2019

Microglia cells during aging, neurodegeneration and neuroinflammation show different morphological and transcriptional profiles (related to axonal direction and cell adhesion). Furthermore, expressions of the receptors on the surface and actin formation compared to young are also different. This review delves into the role of glia during aging and the development of the diseases. The susceptibility of different regions of the brain to disease are linked to the overstimulation of signals related to the immune system during aging, as well as the damaging impact of these cascades on the functionality of different populations of microglia present in each region of the brain. Furthermore, a decr…

AgingDiseaseReviewBiologyBlood–brain barrier03 medical and health sciences0302 clinical medicineImmune systemmedicineHumansCell adhesionReceptorNeuroinflammationBrain DiseasesMicrogliaglia.NeurodegenerationBrainGeneral Medicinemedicine.diseasemedicine.anatomical_structureGene Expression RegulationBlood-Brain Barrier030211 gastroenterology & hepatologyMicrogliaNeuroscienceNeuroglia
researchProduct

A53T-Alpha-Synuclein Overexpression Impairs Dopamine Signaling and Striatal Synaptic Plasticity in Old Mice

2010

BACKGROUND: Parkinson's disease (PD), the second most frequent neurodegenerative disorder at old age, can be caused by elevated expression or the A53T missense mutation of the presynaptic protein alpha-synuclein (SNCA). PD is characterized pathologically by the preferential vulnerability of the dopaminergic nigrostriatal projection neurons. METHODOLOGY/PRINCIPAL FINDINGS: Here, we used two mouse lines overexpressing human A53T-SNCA and studied striatal dysfunction in the absence of neurodegeneration to understand early disease mechanisms. To characterize the progression, we employed young adult as well as old mice. Analysis of striatal neurotransmitter content demonstrated that dopamine (DA…

AgingDopaminelcsh:MedicineMicechemistry.chemical_compoundHomer Scaffolding ProteinsReceptor Cannabinoid CB1lcsh:ScienceLong-term depressionNeurotransmitterChromatography High Pressure LiquidIn Situ Hybridization FluorescenceOligonucleotide Array Sequence AnalysisMice KnockoutNeuronal PlasticityMultidisciplinaryReverse Transcriptase Polymerase Chain ReactionDopaminergicNeurodegenerationGenetics and Genomics/Gene ExpressionElectrophysiologyalpha-SynucleinResearch ArticleRadioimmunoprecipitation Assaymedicine.medical_specialtyNeuronal Calcium-Sensor ProteinsHOMER1Substantia nigraNeurotransmissionBiologyNeurological DisordersInternal medicinemedicineAnimalsHumansddc:610Cyclic Nucleotide Phosphodiesterases Type 7Activating Transcription Factor 2lcsh:RNeuropeptidesmedicine.diseaseMolecular biologyCorpus StriatumMice Mutant StrainsEndocrinologyGenetics and Genomics/Disease ModelschemistrySynaptic plasticitylcsh:QCarrier ProteinsPLoS ONE
researchProduct

Long-lived Temnothorax ant queens switch from investment in immunity to antioxidant production with age

2019

Abstract Senescence is manifested by an increase in molecular damage and a deterioration of biological functions with age. In most organisms, body maintenance is traded-off with reproduction. This negative relationship between longevity and fecundity is also evident on the molecular level. Exempt from this negative trait association, social insect queens are both extremely long-lived and highly fecund. Here, we study changes in gene expression with age and fecundity in ant queens to understand the molecular basis of their long lifespan. We analyse tissue-specific gene expression in young founding queens and old fecund queens of the ant Temnothorax rugatulus. More genes altered their express…

AgingEcologyEvolutionAntsGene Expression Profilinglcsh:RFat Bodylcsh:MedicineBrainArticleAntioxidants570 Life sciencesFertilityAnimalslcsh:QFemalelcsh:ScienceTranscriptomicsTranscriptome570 Biowissenschaften
researchProduct

Influence of gene action across different time scales on behavior.

2002

Genes can affect natural behavioral variation in different ways. Allelic variation causes alternative behavioral phenotypes, whereas changes in gene expression can influence the initiation of behavior at different ages. We show that the age-related transition by honey bees from hive work to foraging is associated with an increase in the expression of the foraging ( for ) gene, which encodes a guanosine 3′,5′-monophosphate (cGMP)–dependent protein kinase (PKG). cGMP treatment elevated PKG activity and caused foraging behavior. Previous research showed that allelic differences in PKG expression result in two Drosophila foraging variants. The same gene can thus exert different types of influe…

AgingForagingGenes InsectHierarchy SocialBiologyGene expressionCyclic GMP-Dependent Protein KinasesAnimalsRNA MessengerAlleleSocial BehaviorGeneCyclic GMPAllelesIn Situ HybridizationMushroom BodiesGeneticsAppetitive BehaviorMultidisciplinaryBehavior AnimalDose-Response Relationship DrugReverse Transcriptase Polymerase Chain ReactionGene Expression ProfilingfungiBrainHoney beeFeeding BehaviorBeesPhenotypeUp-RegulationGene expression profilingPhenotypeMushroom bodiesDrosophilaScience (New York, N.Y.)
researchProduct

Up-Regulation of leucocytes Genes Implicated in Telomere Dysfunction and Cellular Senescence Correlates with Depression and Anxiety Severity Scores

2012

BACKGROUND: Major depressive disorder (MDD) is frequently associated with chronic medical illness responsible of increased disability and mortality. Inflammation and oxidative stress are considered to be the major mediators of the allostatic load, and has been shown to correlate with telomere erosion in the leucocytes of MDD patients, leading to the model of accelerated aging. However, the significance of telomere length as an exclusive biomarker of aging has been questioned on both methodological and biological grounds. Furthermore, telomeres significantly shorten only in patients with long lasting MDD. Sensitive and dynamic functional biomarkers of aging would be clinically useful to eval…

AgingGene Expressionlcsh:MedicineAnxietySocial and Behavioral Sciences0302 clinical medicineBiomarkers of agingMolecular Cell BiologyLeukocytesPathologyPsychologylcsh:ScienceCellular SenescenceDepression (differential diagnoses)Psychiatry0303 health sciencesMultidisciplinaryDepressionChromosome BiologyGenomicsMiddle AgedTelomereAllostatic loadUp-RegulationTelomeresMental HealthMedicineMajor depressive disorderAnxietyBiomarker (medicine)Femalemedicine.symptomResearch ArticleAdultSenescenceClinical PathologyPsychological StressBiologybehavioral disciplines and activitiesMolecular Genetics03 medical and health sciencesDiagnostic Medicinemental disordersGeneticsmedicineHumansBiologyCyclin-Dependent Kinase Inhibitor p16030304 developmental biologyDepressive Disorder Majorlcsh:RComputational BiologyHuman GeneticsDNAmedicine.diseaseTelomereOxygenGene Expression RegulationImmunologyStathminlcsh:QBiomarkers030217 neurology & neurosurgeryDNA DamagePLoS ONE
researchProduct

Expression of Toll-Like Receptors in the Developing Brain

2012

Toll-like receptors (TLR) are key players of the innate and adaptive immune response in vertebrates. The original protein Toll in Drosophila melanogaster regulates both host defense and morphogenesis during development. Making use of real-time PCR, in situ hybridization, and immunohistochemistry we systematically examined the expression of TLR1-9 and the intracellular adaptor molecules MyD88 and TRIF during development of the mouse brain. Expression of TLR7 and TLR9 in the brain was strongly regulated during different embryonic, postnatal, and adult stages. In contrast, expression of TLR1-6, TLR8, MyD88, and TRIF mRNA displayed no significant changes in the different phases of brain develop…

AgingGene Expressionlcsh:MedicineMiceMolecular Cell BiologyMorphogenesislcsh:ScienceReceptorImmune ResponseRegulation of gene expressionMultidisciplinaryNeocortexToll-Like ReceptorsBrainGene Expression Regulation DevelopmentalAcquired immune systemInnate ImmunityCell biologyInfectious Diseasesmedicine.anatomical_structureMedicineResearch ArticleImmunologyCentral nervous systemMorphogenesisIn situ hybridizationBiologyMolecular GeneticsImmune ActivationDevelopmental NeuroscienceGeneticsmedicineAnimalsHumansRNA MessengerBiologyImmunity to Infectionslcsh:RImmunityComputational BiologyImmune DefenseAxonsHEK293 CellsTRIFImmune SystemCellular NeuroscienceImmunologyClinical Immunologylcsh:QTranscriptomeDevelopmental BiologyNeurosciencePLoS ONE
researchProduct

Anti-Aging Effects of GDF11 on Skin

2020

International audience; Human skin is composed of three layers: the epidermis, the dermis, and the hypodermis. The epidermis has four major cell layers made up of keratinocytes in varying stages of progressive differentiation. Skin aging is a multi-factorial process that affects every phase of its biology and function. The expression profiles of inflammation-related genes analyzed in resident immune cells demonstrated that these cells have a strong ability to regenerate adult skin stem cells and to produce endogenous substances such as growth differentiation factor 11 (GDF11). GDF11 appears to be the key to progenitor proliferation and/or differentiation. The preservation of youthful phenot…

AgingHuman skinReviewSkin Aginglcsh:Chemistry0302 clinical medicineSkin Physiological Phenomenalcsh:QH301-705.5SpectroscopySkin0303 health sciencesintegumentary systemGeneral Medicine3. Good healthComputer Science ApplicationsCell biologyGrowth Differentiation Factorsmedicine.anatomical_structureBone Morphogenetic ProteinsIntercellular Signaling Peptides and ProteinsDisease SusceptibilityStem cellSignal TransductionBiologyCatalysisInorganic Chemistry03 medical and health sciencesImmune systemDermisgrowth factorsmedicineAnimalsHumans[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyPhysical and Theoretical Chemistryskin agingMolecular Biology030304 developmental biologyWound HealingdiseaseEpidermis (botany)Regeneration (biology)Organic Chemistrylcsh:Biology (General)lcsh:QD1-999Gene Expression RegulationregenerationGDF11[SDV.MHEP.DERM]Life Sciences [q-bio]/Human health and pathology/Dermatology030217 neurology & neurosurgeryInternational Journal of Molecular Sciences
researchProduct